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临床机制
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  clinical mechanism
     The clinical mechanism and prognosis of delayed onset of traumatic extradural hematoma
     外伤性迟发硬膜外血肿的临床机制及预后探讨
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     Objective:To investigate the clinical mechanism of the cervical spinal cord injury without radiographic abnormality(CSCIWRA).
     目的:探讨颈椎在无骨折脱位情况下出现颈髓损伤的临床机制
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     AIM:To explore the clinical mechanism and curative effect of fluoxetine for adjuvant treatment of migraine, and to study its influence on serum lipid.
     目的:探讨氟西汀辅助治疗偏头痛临床机制和疗效及其对血脂的影响。
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     Objective Study the clinical mechanism of female heroin reliers’ amenorrhea and infertility.
     目的 探讨女性海洛因依赖者闭经、不孕的临床机制
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  curative mechanism
     Objective To treat patients with acute c erebral infarction (ACI) with acupuncture therapy at an early stage, and to stud y the curative mechanism.
     目的:探讨针刺药物联合治疗早期脑梗死的临床机制
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  “临床机制”译为未确定词的双语例句
     Mechanism of Ligustrazine in Treating Cerebral Infarction
     川芎嗪治疗脑梗死的临床机制研究
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     The study on the mechanism of acute myocardial infarction with atrial fibrillation in the different time
     急性心肌梗死后不同时期心房颤动发生的临床机制
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     Objective:To evaluate the effect of Sibelium on vertigo patients after cerebral infarction(CI).
     目的 :探讨西比灵辅助治疗脑梗死后眩晕的临床机制和治疗效果。
短句来源
     AIM: To evaluate the effect of flunarizine on early epilepsy patients after cerebral infarction(CI).
     目的 :探讨氟桂利嗪辅助治疗脑梗死后继发的早期癫痫的临床机制和治疗效果。
短句来源
     Objective:To evaluate the effect of Dengzhanxixin on vertigo patients after cerebral infarction(CI).
     目的 :探讨灯盏细辛辅助治疗脑梗死后眩晕的临床机制和治疗效果。
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     P and mechanism of increased INa.
     P 的机制
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     CLINICAL STUDY ON THE MECHANISM OF LOWERING INTRAOCULARPRESSURE AFTER TRABECULECTOMY
     小梁切除术降压机制临床探讨
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     The Mechanism of Remodelling in Atrial Fibrillation and Its Clinical Significance
     心房重构的机制临床意义
短句来源
     The clinical scores showed that there were significant differences statistically.
     临床资料
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     On Mechanisms
     机制
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  clinical mechanism
Undoubtedly a precise understanding of its clinical mechanism of action, which is currently being studied by a number of groups, will lead to a better understanding of the pathogenesis of migraine.
      
Purpose: In order to explore the clinical mechanism of Neiguan (PC 6) in the treatment of acute seizure of migraine and the relationship between Neiguan (PC 6) and cerebral blood flow.
      
Models for injurious compression of cartilage may identify predictors of injury that suggest a clinical mechanism.
      
  curative mechanism
The present data show that its curative mechanism lies in the action of promoting the recovery of colony forming unit-spleen (CFU-S) and reversing immunologically-induced plasma colony forming unit granulocyte/macrophage (CFU-GM) inhibitory activity.
      


Objective:To investigate the clinical mechanism of the cervical spinal cord injury without radiographic abnormality(CSCIWRA).Method:Twenty four patients with CSCIWRA were included and the radiology informations were analysed in the study.Result:27% patients had spinal canal narrowing,83% patients present with one or multiple protrusive cervical intervertebral discs.Conclusion:The CSCIWRA mainly result from the flexion compression-extension force, the force has the tendency to injury discs and the spinal...

Objective:To investigate the clinical mechanism of the cervical spinal cord injury without radiographic abnormality(CSCIWRA).Method:Twenty four patients with CSCIWRA were included and the radiology informations were analysed in the study.Result:27% patients had spinal canal narrowing,83% patients present with one or multiple protrusive cervical intervertebral discs.Conclusion:The CSCIWRA mainly result from the flexion compression-extension force, the force has the tendency to injury discs and the spinal cord was injuried by the damaged discs.The mechanism of the spinal cord injury also associated with degenerative discs and spinal canal narrowing.

目的:探讨颈椎在无骨折脱位情况下出现颈髓损伤的临床机制。方法:观察24例无骨折脱位型颈髓损伤,重点分析其影像学检查特点。结果:27%病例合并椎管狭窄,83%的病例存在椎间盘突出,不同程度压迫脊髓。结论:无骨折脱位型颈脊髓损伤机制中,存在外伤致颈椎间盘损伤,突出的间盘向后压迫损伤颈髓。颈椎间盘损伤受力机制为屈曲→压缩→过伸。颈椎管狭窄、椎间盘退变等是此类脊髓损伤的病理解剖基础

Objective:To evaluate the effect of Sibelium on vertigo patients after cerebral infarction(CI).Methods: Eighty_two vertigo patients with CI were randomly divided into the Sibelium treating group(43 cases)and the control group(39 cases).The control group was given anticoagulant,plasminogen, and drugs to reduce blood pressure.On the basis of routime treatment, Sibelium(5mg,qN,for one month)was given in the treating group.Results:The treating group showed a statistically effective rate of 97 6%(P<0 01).Conclusion:Sibelium...

Objective:To evaluate the effect of Sibelium on vertigo patients after cerebral infarction(CI).Methods: Eighty_two vertigo patients with CI were randomly divided into the Sibelium treating group(43 cases)and the control group(39 cases).The control group was given anticoagulant,plasminogen, and drugs to reduce blood pressure.On the basis of routime treatment, Sibelium(5mg,qN,for one month)was given in the treating group.Results:The treating group showed a statistically effective rate of 97 6%(P<0 01).Conclusion:Sibelium is effective in the treatment of vertigo patients after cerebral infarction.

目的 :探讨西比灵辅助治疗脑梗死后眩晕的临床机制和治疗效果。方法 :脑梗死后眩晕 82例随机分为治疗组 (43例 )和对照组 (39例 )。对照组按病情分别予抗凝、溶栓、降压、降血糖及营养脑神经等常规治疗 ;在此基础上治疗组加服西比灵胶囊。治疗 1个月后进行疗效评定。结果 :治疗组治疗后纤维蛋白原、血小板粘附率降低 ,凝血酶原时间延长 ,眩晕总有效率 97 6 % ,均优于对照组。结论 :西比灵辅助治疗脑梗死后眩晕安全有效。

AIM: To evaluate the effect of flunarizine on early epilepsy patients after cerebral infarction(CI). METHODS: Ninety two early epilepsy patients wiht CI were randomly divided into two groups, the flunarizine treating group( n =47) and the control group( n =45). Roution treatment was given to all patients. The control group was given carbamazepine phenytoin sodium valproate or diazepam differently, and on the basis of this, flunarizine 5 mg,qn,for three months was given to the treating group. RESULTS:...

AIM: To evaluate the effect of flunarizine on early epilepsy patients after cerebral infarction(CI). METHODS: Ninety two early epilepsy patients wiht CI were randomly divided into two groups, the flunarizine treating group( n =47) and the control group( n =45). Roution treatment was given to all patients. The control group was given carbamazepine phenytoin sodium valproate or diazepam differently, and on the basis of this, flunarizine 5 mg,qn,for three months was given to the treating group. RESULTS: After three months therapy, the treating group showed better than the control group in the antiepileptic effect by Ridit analysis( P <0.01). After treatment the fibrinogen, platelet adhesiveness rate and prothrombin time in treating group was(2.8± s 0.7)g·L -1 ,(52±3)%,(16.7±2.0)s, respectively, compared with control group ((5.6±0.7)g·L -1 ,(56±3)%,(14.9±2.2)s). There was significant difference( P <0.01). CONCLUSION: Flunarizine is effective in adjuvant treatment of early epilepsy patients after cerebral infarction.

目的 :探讨氟桂利嗪辅助治疗脑梗死后继发的早期癫痫的临床机制和治疗效果。方法 :脑梗死后继发的早期癫痫的病人 92例 ,随机分为治疗组4 7例 ,对照组 4 5例 ,对照组按不同的发作类型分别服用苯妥英、丙戊酸纳、卡马西平及地西泮等药 ;治疗组则在此基础上加服用氟桂利嗪胶囊 ,每日 5mg ,qn ,疗程 3mo。治疗后进行疗效评定。治疗前后对血液流变学、血脂、肾功能等生化指标进行对比性分析。结果 :治疗后 ,治疗组抗癫痫疗效优于对照组 ,经Ridit分析 ,差异有非常显著意义 (P <0 .0 1)。治疗组治疗后凝血因子I、血小板粘附率 ,凝血酶原时间 ,分别为 (2 .8±s 0 .7) g·L- 1,(5 2± 3) % ,(16.7± 2 .0 )s与对照组比较 ,(3.6± 0 .7) g·L- 1,(5 6± 3) % ,(14.9± 2 .2 )s ,差异有显著意义或非常显著意义 (P <0 .0 5或P <0 .0 1)。结论 :氟桂利嗪辅助治疗脑梗死后继发的早期癫痫有效

 
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