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重症病毒性肝炎
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  severe viral hepatitis
     Expression of Collagen and PDGF-1 of Liver Tissue in the Patients with Severe Viral Hepatitis
     重症病毒性肝炎肝组织胶原蛋白和血小板衍生生长因子-1的表达
短句来源
     Analysis of the Causes of Death in 281 Cases of Severe Viral Hepatitis
     281例重症病毒性肝炎死亡原因分析
短句来源
     Efficacy of glucagoninsulin therapy in severe viral hepatitis.
     胰高糖素——胰岛素治疗重症病毒性肝炎疗效观察
短句来源
     A Discussion on the Causes of Death in Severe Viral Hepatitis A Clinical and Pathologic Analysis of 223 Fatal Cases
     重症病毒性肝炎的死亡原因探讨(223例死亡病例的临床和病理分析)
短句来源
     The Changes of Serum Levels of TNF-α and IL-6 and Its Significance in Patients with Severe Viral Hepatitis
     重症病毒性肝炎患者血清TNF-α和IL-6水平变化及其意义
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  fulminant viral hepatitis
     CLINICAL ANALYSIS OF LARGE DOSE NATURAL α-INTERFERON IN THE TREATMENT OF FULMINANT VIRAL HEPATITIS
     大剂量天然α─干扰素治疗重症病毒性肝炎临床分析
短句来源
     INFECTIOUS COMPLICATING FULMINANT VIRAL HEPATITIS
     重症病毒性肝炎的感染并发症
短句来源
     AN ULTRASTRUCTURAL ATUDY OF THE LIVER WITH FULMINANT VIRAL HEPATITIS
     重症病毒性肝炎超微结构研究
短句来源
     SERIOUS COMPLICATIONS OF FULMINANT VIRAL HEPATITIS WITH HEPATIC FAILURE
     重症病毒性肝炎肝功能衰竭时的严重并发症
短句来源
     An ultrastruotuial observation for the study of the liver tissue in 41cases with fulminant viral hepatitis was made with the technique of SEM &TEM.
     应用扫描电镜技术和透射电镜技术,对41例重症病毒性肝炎患者肝组织进行超微结构观察。
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  “重症病毒性肝炎”译为未确定词的双语例句
     Analysis on Complications of Artificial Liver Support System in the Treatment of Fatal Hepatitis
     人工肝支持系统治疗重症病毒性肝炎的并发症的分析
短句来源
     Nosocomial Infection of Aspergillus Fumigatus Complication Chronic Serious Viral Hepatitis
     慢性重症病毒性肝炎并发医院内曲霉菌感染
短句来源
     During 1976-1991, 15 cases of chronic seriousviral hepatitis were found to be complicated withnosocomial infection of Aspergillus fumigatus.
     1976~1991年我院收治慢性重症病毒性肝炎并发曲霉菌感染15例,感染于住院后3~112天,<30天9例。
短句来源
     Methods\ Use KM-8800 plaxma exchanger made in Japan to conduct 102 time of treatment on 43 fatal viral hepatitis cases through different ways(PE,PP and DHP),and then compared with 44 cases in control group.
     方法 应用日产KM —8800 血液净化仪对43 例重症病毒性肝炎选用不同的方法( 血浆交换、血液灌流、血浆吸附等) 进行102 次治疗,与对照组44 例比较。
短句来源
     ETIOLOGICAL ANALYSIS OF FULMINANT HEPATITIS IN RENAL ALLOGRAFT RECIPIENTS
     肾移植术后重症病毒性肝炎的病因学分析
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  severe viral hepatitis
These ductal epithelial changes are similar to those that occur in livers of some patients with severe viral hepatitis.
      
All the biopsies were typical of acute hepatitis such as that seen in severe viral hepatitis.
      
  fulminant viral hepatitis
Attempted treatment of fulminant viral hepatitis with human fibroblast interferon
      
We concluded that the pre-existing HBsAg carrier state was a major risk factor for development of fulminant viral hepatitis.
      
Antibodies to hepatitis C virus in non-A non-B fulminant viral hepatitis
      
Persistence of HCV replication in non-A, non-B fulminant viral hepatitis
      
Interferon and cyclosporin A in the treatment of fulminant viral hepatitis
      
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Postmortem examinations of 32 cases of fulminant viral hepatitis showed that,22(68.7%)were complicated by simple or mixed infections of various causes.The following were the sites commonly involved,lung(14),peritoneum(8),blood stream(2),urinary tract(2),gall bladder(1)and soft tissue(1).Whilethese presenting manifestations were non-specific and frequently not recognizeduntil death ensued.The causative agents of these infections were,in order offrequency,E.coli,Hemolyitic streptococccus,Staphylococccus aureus,ProteusAlkaligenes,Candida...

Postmortem examinations of 32 cases of fulminant viral hepatitis showed that,22(68.7%)were complicated by simple or mixed infections of various causes.The following were the sites commonly involved,lung(14),peritoneum(8),blood stream(2),urinary tract(2),gall bladder(1)and soft tissue(1).Whilethese presenting manifestations were non-specific and frequently not recognizeduntil death ensued.The causative agents of these infections were,in order offrequency,E.coli,Hemolyitic streptococccus,Staphylococccus aureus,ProteusAlkaligenes,Candida albicans,and Aspergillus.

本文分析了经病理解剖证实的32例重症病毒性肝炎的感染并发症,其发生率为68.7%,以肺部感染最常见,其次为原发性腹膜炎、真菌感染、败血症及泌尿系感染等。这些感染并发症的临床表现常不典型,易被忽略。文中就各种感染并发症的发生原因及诊断问题进行了讨论。

Postmortum examinations of 50 cases of fulminant viral hepatitis with hepatic failure revealed serious complications involving kidney, lung, brain, heart and pancreas. Among 31 cases with acute renal failure being considered to be functional in nature generally,however, 22 cases were revealed cholenephropathy histologi-cally.Pulmonary edema had been considered as a rare complication, but 19 cases (38%) were encountered in this series. Eight developed cerebral edema, including one cerebral hernia. Five had myocarditis....

Postmortum examinations of 50 cases of fulminant viral hepatitis with hepatic failure revealed serious complications involving kidney, lung, brain, heart and pancreas. Among 31 cases with acute renal failure being considered to be functional in nature generally,however, 22 cases were revealed cholenephropathy histologi-cally.Pulmonary edema had been considered as a rare complication, but 19 cases (38%) were encountered in this series. Eight developed cerebral edema, including one cerebral hernia. Five had myocarditis. Five with pathological evidence of pancreatitis. One manifested as acute necrotic pancreatitis. The clinical feature of these complications in some patients were atypical and could easily be neglected. Early diagnosis and management of these complications are briefly discussed.

50例经病理解剖证实的重症病毒性肝炎回顾性分析表明,在重症病毒性肝炎肝衰竭时,肾、肺、脑、心及胰腺均可受累,计有31例并发肝肾综合征、19例并发肺水肿、8例并发脑水肿、5例有心肌炎、5例发现胰腺病变者中1例表现为急性坏死性胰腺炎而猝死。本文就有关并发症的早期诊断和处理问题进行了讨论。

Sixty-eight cases of severe viral hepatitis were divided into 3 groups. G-roup Ⅰ, 30 cases, treated with conventional (basic) therapy, of which 9 cases (9/30) are living, Group Ⅱ, 28 cases, treated with an additional remedy of branched-chain amino acid (BCAA), of which 13 cases (13/28) are surviving; Group Ⅲ, 10 cases, given with combined BCAA and glucagon-insulin therapy, of which 7 cases (7/10) are surviving. Pre and posttreatment liver function tests, blood sugar determinations, and amino acid spectrum analysis...

Sixty-eight cases of severe viral hepatitis were divided into 3 groups. G-roup Ⅰ, 30 cases, treated with conventional (basic) therapy, of which 9 cases (9/30) are living, Group Ⅱ, 28 cases, treated with an additional remedy of branched-chain amino acid (BCAA), of which 13 cases (13/28) are surviving; Group Ⅲ, 10 cases, given with combined BCAA and glucagon-insulin therapy, of which 7 cases (7/10) are surviving. Pre and posttreatment liver function tests, blood sugar determinations, and amino acid spectrum analysis were performed in all 10 cases in Group Ⅲ. Rapid fading of jaundice, decrease of ALT, increase of BCAA/AAA ratio, and clinical alleviation are noticed. The survivor-patients recover gradually after the discontinuation of the BCAA and glucagon-insulin therapy. The authors consider that combination of BCAA and glucagon-insulin togather with general supportive treatment is superior to the conventional one in the management of severe viral hepatitis.

报告68例重症病毒性肝炎以一般支持疗法治疗30例,存活9例,加用支链氨基酸治疗28例,存活13例,而并用支链氨基酸及胰高糖素—胰岛素治疗10例,7例存活。支链氨基酸与胰高糖素—胰岛素并用可明显提高重症病毒性肝炎的存活率,并就其机制进行了讨论。

 
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