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重症病毒性肝炎
相关语句
  severe viral hepatitis
    Analysis of the Causes of Death in 281 Cases of Severe Viral Hepatitis
    281例重症病毒性肝炎死亡原因分析
短句来源
    The Changes of Serum Levels of TNF-α and IL-6 and Its Significance in Patients with Severe Viral Hepatitis
    重症病毒性肝炎患者血清TNF-α和IL-6水平变化及其意义
短句来源
    Alprostadil in the Treatment of Azotemia Resulted from Severe Viral Hepatitis or Cirrhosis of the Liver
    前列地尔治疗重症病毒性肝炎肝硬化所致氮质血症
短句来源
    Expression of Collagen and PDGF-1 of Liver Tissue in the Patients with Severe Viral Hepatitis
    重症病毒性肝炎肝组织胶原蛋白和血小板衍生生长因子-1的表达
短句来源
    Fuzzy Pattern Recognition in Prediction of Severe Viral Hepatitis Progression
    模糊模式识别在重症病毒性肝炎病情转归中的应用
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  “重症病毒性肝炎”译为未确定词的双语例句
    ETIOLOGICAL ANALYSIS OF FULMINANT HEPATITIS IN RENAL ALLOGRAFT RECIPIENTS
    肾移植术后重症病毒性肝炎的病因学分析
短句来源
    Analysis on Complications of Artificial Liver Support System in the Treatment of Fatal Hepatitis
    人工肝支持系统治疗重症病毒性肝炎的并发症的分析
短句来源
    Clinical Characteristics of Spontaneous Bacterial Peritonitis(SBP) in Patients with Severe Type of Viral Hepatitis
    重症病毒性肝炎并发自发性细菌性腹膜炎临床特点分析
短句来源
    Analysis of Death Causes and Complications of Viral Hepatitis Gravis
    重症病毒性肝炎的并发症和死因分析
短句来源
    The Determination of the Blood Concentrations of Cardionatrin, Reninactivity, Angiotensin, Aldosterone in Patients with Viral Hepatic Failure
    重症病毒性肝炎心钠素、肾素—血管紧张素及醛固酮的测定
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  severe viral hepatitis
These ductal epithelial changes are similar to those that occur in livers of some patients with severe viral hepatitis.
      
All the biopsies were typical of acute hepatitis such as that seen in severe viral hepatitis.
      


There were 7 cases of renal allograft recipients developing sub-acute fulminant hepatitis.In order to investigate the etiological agents, we examined serological markers of hepatitides A, B, C,and D. It was found that all patients were HBsAgpositive, 2 of 7 cases were superinfection of HCV,one each was HAV and HDV. It is suggested thaton the basis of HBV infection superinfection of HCV, HDV, HAV, and other hepatitis viruses were very important causes of the fulminant hepatitis in the renal allograft recipients....

There were 7 cases of renal allograft recipients developing sub-acute fulminant hepatitis.In order to investigate the etiological agents, we examined serological markers of hepatitides A, B, C,and D. It was found that all patients were HBsAgpositive, 2 of 7 cases were superinfection of HCV,one each was HAV and HDV. It is suggested thaton the basis of HBV infection superinfection of HCV, HDV, HAV, and other hepatitis viruses were very important causes of the fulminant hepatitis in the renal allograft recipients. In the otherhand, according to clinical feature and other studyresults, hepatotoxicity and nephrotoxicity of cyclosporine A was another main development factorof fulminant hepatitis. Finally, this paper also recommends some preventive methods and suggestions.

对7例患者肾移植术后并发重症病毒性肝炎的病回进行了分析。发现所有患者均为HBSAg阳性的HBV感染者,2例重叠HCV感染,HAV和HDV重叠感染各1例。表明HBV感染及在此基础上其它肝炎病毒合并感染是发生重症肝炎的主要原因;治疗过程中环孢素A的应用和它所引起的肝肾毒性是移植后重症肝炎发生和影响患者预后的一个重要因素。提出应慎重考虑HBV感染者的肾移植适应症,有HCV、HDV重叠感染者不宜施行肾移植术。对HBsAg阳性肾移植患者应加强术后肝功能的监测,及早应用保肝药物。

In this paper the serum levels of TNF-a and IL-6 in 30 patients with severe viral hepatitis (SH) were measured by ELISA. The results showed that the serum levels of both TNF-a and IL-6 in patients with SH were significantly higher than those in patients with acute hepatitis, chronic hepatitis and liver cirrhosis (F<0. 05 and <0. 01), and that the elevated levels of both TNF-a and IL-6 were shown positively correlated with serum bilirubin and prothrombin time (P< 0. 001) . It is suggested that TNF-a and IL-6...

In this paper the serum levels of TNF-a and IL-6 in 30 patients with severe viral hepatitis (SH) were measured by ELISA. The results showed that the serum levels of both TNF-a and IL-6 in patients with SH were significantly higher than those in patients with acute hepatitis, chronic hepatitis and liver cirrhosis (F<0. 05 and <0. 01), and that the elevated levels of both TNF-a and IL-6 were shown positively correlated with serum bilirubin and prothrombin time (P< 0. 001) . It is suggested that TNF-a and IL-6 are closely associated with the degree of hepatocyte necrosis and play an important role on the pathogenesis of SH. It was also found that the serum levels of TNF-a and IL-6 were markedly higher in patients with B type SH superinfected with HAV. HCV. HDV or HEV than those in patients with B type SH alone (P<0. 01 and<0. 005),and in patients with SH complicated by severe bacterial infections than those in patients without infections (P<0. 05 and <0. 02) . It is thus revealed that the significant elevation of both cytokines in SH is closely correlated with the superinfection of hepatitis virus and endotoxemia.

应用双抗体夹心法检测 30例重症病毒性肝炎(SH)患者血清TNF-α和IL-6水平。结果显示,SH患者血清TNF-α和IL-6水平均显著高于急性肝炎、乙型慢性肝炎和乙型肝炎肝硬化患者(P<0.05和P<0.01),其升高水平与血清胆红素和凝血酶原时间呈显著相关(P<0.001),提示TNF-α和IL-6水平与肝细胞坏死程度密切相关,在SH的发病中有重要作用。重叠甲、丙、丁、戊型肝炎病毒感染的乙型SH患者血清TNF-α和IL-6水平显著高于单纯乙型SH患者(P<0.01和P<0.005),合并严重细菌感染的SH患者血清TNF-α和IL-6水平也显著高于无感染的SH患者(P<0.05和P<0.02),表明SH患者血清TNF-α和IL-6的显著升高与肝炎病毒的重叠感染和内毒素血症有密切关系。

The study use large dose natural α-interferon in treatment of 31 patients of fubonant viral hepatitis andcompared with controI group. All of the patients were not received Artificial Liver Support system treatment. The result revealed that the survival rate of treatment group (64. 5 % ) obviously superior to control group (28. 6% ) (P< 0. 005 ).Though some of the patients manifest fever,leukopenia and thrombocytopenia,the mest of them can still receive the treatment after appropriated management'

本文用大剂量天然α-干扰素治疗31例重症病毒性肝炎患者,并以既往和同期未用α-干扰素的49例患者作为对照,所有患者均未接受人工肝治疗。治疗结果表明:应用天然α-干扰素的31例患者存活今为64.5%(20/31)较对照组存活率28.6%(14/49)明显提高(P<0.005)。治疗组中虽有部分患者出现发烧、白细胞及血小板减少,经适当处理多数患者仍可接受治疗。

 
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