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   再生神经 在 内分泌腺及全身性疾病 分类中 的翻译结果: 查询用时:0.511秒
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再生神经
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  regenerating nerve
    Effects of Ethanol on Synthesis and Secretion of Apolipoproteins by Regenerating Nerve
    乙醇对再生神经载脂蛋白的合成与分泌的影响
短句来源
    The amounts of apolipoprotein(apoE) and A 1 were examined in regenerating nerve.
    本文检测了再生神经释放载脂蛋白E和A1的量。
短句来源
    Cultured minced segments of regenerating nerve taken from rats fed an alcohol containing liquid diet released only 50% of apoE but nearly 200% of apoA 1 when compared with a control diet.
    把从含有乙醇的液体饲料喂养的大白鼠取得的再生神经,切成较小的节段培养后,测得释放入培养基中的载脂蛋白E(apoE)的量,仅仅只有对照组的50%,而载脂蛋白A1(apoA1)的量几乎是对照组的200%。
短句来源
    Thus,chronic ethanol ingestion affects apoE synthesis of regenerating nerve by changing its mRNA.
    这样,慢性乙醇摄入影响再生神经apoE的合成是通过改变它的mRNA的水平所致。
短句来源
    The amount of apoA 1 released by regenerating nerve was not significantly lager than that present in the nerve tissue prior to incubation.
    由再生神经释放入培养基中的apoA1与培养前所含的量没有明显的差异。
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  regenerating nerve
Fibroblast growth factor b diminishes the apoptosis of Schwann cells in regenerating nerve fibers.
      
The first is compression of stumps and regenerating nerve by a thick myofibroblast layer that surrounds these tissues and blocks synthesis of a nerve of large diameter (pressure cuff theory).
      
We therefore suggest that SP may modulate the availability of NGF in the microenvironment of the regenerating nerve fiber endings.
      
This phenomenon usually occurs after nerve injury, and is the result of misdirection of the regenerating nerve fibers.
      
At the suture line, fibroblasts encircled the regenerating axons and the Schwann cells to form new compartments resulting in a large number of minifascicles of regenerating nerve.
      
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The amounts of apolipoprotein(apoE) and A 1 were examined in regenerating nerve.Cultured minced segments of regenerating nerve taken from rats fed an alcohol containing liquid diet released only 50% of apoE but nearly 200% of apoA 1 when compared with a control diet.The extent of decrease in medium apoE mRNA in the nerve.Thus,chronic ethanol ingestion affects apoE synthesis of regenerating nerve by changing its mRNA.On the other hand,apoA 1 mRNA couldn't detected from both groups.The amount of apoA 1 released...

The amounts of apolipoprotein(apoE) and A 1 were examined in regenerating nerve.Cultured minced segments of regenerating nerve taken from rats fed an alcohol containing liquid diet released only 50% of apoE but nearly 200% of apoA 1 when compared with a control diet.The extent of decrease in medium apoE mRNA in the nerve.Thus,chronic ethanol ingestion affects apoE synthesis of regenerating nerve by changing its mRNA.On the other hand,apoA 1 mRNA couldn't detected from both groups.The amount of apoA 1 released by regenerating nerve was not significantly lager than that present in the nerve tissue prior to incubation.Therefore,apoA 1 released by the injured nerve may originate from the bloodstream.The increase in apoA 1 content in the injured nerve of alcoholic fed rats is due to an enhanced permeability of the nerve blood barrier.The burst of apo in the injured nerve is likely to play a role in nerve regeneration.The perturbation of apo contents in regenerating nerve by chronic ethanol consumption may relate to the pathogenesis of alcoholic neuropathy.

本文检测了再生神经释放载脂蛋白E和A1的量。把从含有乙醇的液体饲料喂养的大白鼠取得的再生神经,切成较小的节段培养后,测得释放入培养基中的载脂蛋白E(apoE)的量,仅仅只有对照组的50%,而载脂蛋白A1(apoA1)的量几乎是对照组的200%。释放入培养基中apoE减少的程度与神经中mRNA减少的程度一致。这样,慢性乙醇摄入影响再生神经apoE的合成是通过改变它的mRNA的水平所致。另一方面,apoA1的mRNA在实验组和对照组的神经中均不能检测出。由再生神经释放入培养基中的apoA1与培养前所含的量没有明显的差异。因此,由损伤神经释放的apoA1很可能是从血流而来的。而乙醇喂养的老鼠损伤神经中apoA1含量的增加,是由于增强了神经———血液屏障的通透性。载脂蛋白在损伤神经中的堆积很可能在神经再生过程中扮演一个重要的角色。慢性乙醇摄入的老鼠的再生神经中载脂蛋白含量的紊乱可能与乙醇中毒性神经疾病的发病机理有关

 
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