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急性肾毒性
相关语句
  acute renal toxicity
    Intervention effect of ginseng and prednisone on aristolochia manshuriensis induced acute renal toxicity in rats
    人参和泼尼松对关木通致大鼠急性肾毒性的干预作用
短句来源
    Objective: To investigate the intervention effect of ginseng and predinsone on aristolochia manshuriensis induced acute renal toxicity in rats.
    目的:探讨中药人参和泼尼松对关木通所致急性肾毒性的干预作用。
短句来源
    Animal models of acute renal toxicity were established by 40 g·kg~(-1)·d~(-1) of aristolochia manshuriensis decoction via gastric tube.
    kg-1.d-1制备急性肾毒性模型。
短句来源
    Conclusion: Aristolochia manshuriensis(induced) acute renal toxicity in rats could be relieved by ginseng or prednisone alone or combined.
    结论:单独用人参或泼尼松与关木通混合灌胃,或同时用人参、泼尼松和关木通混合灌胃,均能减轻关木通对大鼠的急性肾毒性
短句来源
  “急性肾毒性”译为未确定词的双语例句
    CALMODULIN ANTAGONIST ATTENUATES ACUTE RENAL INJURY INDUCED BY GENTAMYCIN
    钙调素拮抗剂对急性肾毒性肾功能衰竭保护作用的研究
短句来源
    Objective: Acute renal failure (ARF) is one of common severe renal diseases in clinical practice It may be caused by acute renal ischemia or receiving aniinoglycoside antibiotics and traditionally the pathological manifestation is regarded as acute renal tubular necrosis (ATN)Recent studies reviewed necrosis, apoptosis and regeneration of renal tubular epithelial cells occur and the balance between cell apoptosis and cell proliferation determines the outcome of ARF.
    目的:急性肾衰竭是临床常见的危重肾脏病之一,急性缺血或因使用氨基糖苷类抗生素产生急性肾毒性反应是其常见的病因,传统认为其病理特征为急性肾小管坏死,晚近研究显示,实际上在此过程中肾小管上皮细胞可发生坏死、凋亡和再生,增殖和凋亡的动态平衡决定了急性肾衰的转归。
短句来源
    OBJECTIVE To define the involvement of calmodulin and the effect of calmodulin antagonist treatment on acute nephrotoxic injury in rats.
    目的:观察急性肾毒性肾衰时钙调素的变化以及钙调素拮抗剂对肾衰的保护作用。
短句来源
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  acute renal toxicity
ICI D1694 has been shown to be a more active anticancer agent than CB 3717 in model systems, and it is devoid of the acute renal toxicity associated with the administration of the latter drug to mice.
      
Conclusions: By the protraction of continuous infusion of HD-IFO over 6 to 8?days, ifosfamide-induced acute renal toxicity is avoided, while G-CSF support allows the delivery of the planned dose intensity in most of the patients.
      
Accurate monitoring of blood cyclosporin C2 levels is vital to prevent over immunosuppression and acute renal toxicity in patients who receive organ transplant.
      
Acute Renal Toxicity of Doxorubicin (Adriamycin)-Loaded Cyanoacrylate Nanoparticles
      
Acute renal toxicity is usually dose related, and is exacerbated by hypovolemia.
      
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  acute nephrotoxicity
One serious side effect of Cyclosporine A therapy is its acute nephrotoxicity characterized by a marked decrease in the glomerular filtration rate.
      
By way of lowered ultrafiltration coefficient this effect of Cyclosporine A may be partly responsible for its acute nephrotoxicity.
      
Urinary gamma-glutamyl transferase as an indicator of acute nephrotoxicity in rats
      
It is concluded that the measurement of urinary GGT can be used as an indicator of acute nephrotoxicity.
      
The role of glutathione in the acute nephrotoxicity of sodium dichromate
      
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OBJECTIVE To define the involvement of calmodulin and the effect of calmodulin antagonist treatment on acute nephrotoxic injury in rats. METHODOLOGY Rat model of acute renal injury was established by repeated gentamycin injections. Tissue levels of calmodulin were determined by the measurement of phosphodiesterase activity. Renal histology was examined and the severity of tubular injury was scored. Serum creatinine levels, urine protein, β 2 microglobulin and lysozyme excretion were monitored after acute...

OBJECTIVE To define the involvement of calmodulin and the effect of calmodulin antagonist treatment on acute nephrotoxic injury in rats. METHODOLOGY Rat model of acute renal injury was established by repeated gentamycin injections. Tissue levels of calmodulin were determined by the measurement of phosphodiesterase activity. Renal histology was examined and the severity of tubular injury was scored. Serum creatinine levels, urine protein, β 2 microglobulin and lysozyme excretion were monitored after acute renal injury. RESULTS There was no difference in tissue levels of calmodulin before and after acute nephrotoxic injury. However, calmodulin antagonist, chloropromazine treatment significantly decreased renal injury as evidenced by the less severe tubular injury, lower serum creatinine levels and less amount of urine β 2 microglobulin and lysozyme excretion in chloropromazine treated rats than in untreated rats. CONCLUSION There may be a beneficial effect of calmodulin antagonist treatment on acute nephrotoxic injury.

目的:观察急性肾毒性肾衰时钙调素的变化以及钙调素拮抗剂对肾衰的保护作用。方法:用不同剂量钙调素拮抗剂氯丙嗪对庆大霉素肾毒性肾衰动物进行治疗研究。结果:①庆大霉素肾毒性肾衰大鼠肾组织中钙调素活性无改变,氯丙嗪对钙调素有明显抑制作用。②钙调素拮抗剂对庆大霉素肾毒性肾衰有明显保护作用。并能促进损伤肾小管上皮细胞之再生修复。结论:氯丙嗪等钙调素拮抗剂对急性肾衰有明显保护作用,为治疗急性肾衰提供了新的思路及手段

Objective: To investigate the intervention effect of ginseng and predinsone on aristolochia manshuriensis induced acute renal toxicity in rats.Methods: Fiftyfour cleaning grade female(SpragueDawley)(SD) rats were randomly divided into five groups.Animal models of acute renal toxicity were established by 40 g·kg~(-1)·d~(-1) of aristolochia manshuriensis decoction via gastric tube.Prednisone((30 mg·kg~(-1)·d~(-1))) and dried raw ginseng decoction(40 g·kg~(-1)·d~(-1)) were given via gastric tube for(5 days).On...

Objective: To investigate the intervention effect of ginseng and predinsone on aristolochia manshuriensis induced acute renal toxicity in rats.Methods: Fiftyfour cleaning grade female(SpragueDawley)(SD) rats were randomly divided into five groups.Animal models of acute renal toxicity were established by 40 g·kg~(-1)·d~(-1) of aristolochia manshuriensis decoction via gastric tube.Prednisone((30 mg·kg~(-1)·d~(-1))) and dried raw ginseng decoction(40 g·kg~(-1)·d~(-1)) were given via gastric tube for(5 days).On the 4 th and 8 th days after administrating the drug, biochemical indexes of the rats′ blood and urine were detected,and immunohistochemical technique was used to estimate the expressions of transforming growth(factorβ1)(TGFβ1) and type Ⅳ collagen in the renal tissue of rats.The pathologic changes of(kidney) were also observed by microscopy.The SPSS 11.5 packages were used to analyze the data.Results: The rats′ weight and activity decreased significantly but the levels of blood urea nitrogen(BUN),creatinine(Cr),urine Nacetylglucosaminidase(NAG),urine β_2microglobulin(β_2MG),transforming growth(factorβ1)(TGFβ1) and collagenⅣ increased significantly in the groups administered with aristolochia(manshuriensis),especially in the aristolochia manshuriensis group.The changes of BUN,Cr,urine NAG,(TGFβ1) and collagen Ⅳ were attenuated after feeding the rats with prednisone and ginseng compared to those of the rats administered with aristolochia manshuriensis alone.Conclusion: Aristolochia manshuriensis(induced) acute renal toxicity in rats could be relieved by ginseng or prednisone alone or combined.

目的:探讨中药人参和泼尼松对关木通所致急性肾毒性的干预作用。方法:54只清洁级雌性SD大鼠随机分组,以关木通煎剂40 g.kg-1.d-1制备急性肾毒性模型。分别给予泼尼松(30 m g.k-g 1.-d 1)和生晒参煎剂(40 g.k-g 1.-d 1)灌胃5 d;第4和8 d测定血、尿生化等指标,免疫组化染色进行肾组织病理学观察;用SPSS11.5软件进行统计学处理。结果:含关木通各组大鼠体重和活动能力下降,而血尿素氮(BUN)和肌酐(C r)、尿N乙酰葡糖胺(NAG)酶、尿2β微球蛋白(2βM G)、转化生长因子β1(TGFβ1)、Ⅳ型胶原均有不同程度增高,其中关木通组差异最大;与单纯关木通组比较,各实验组BUN、C r、尿NAG酶、TGFβ1、Ⅳ型胶原有不同程度的降低。结论:单独用人参或泼尼松与关木通混合灌胃,或同时用人参、泼尼松和关木通混合灌胃,均能减轻关木通对大鼠的急性肾毒性

 
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