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家兔内脏
相关语句
  rabbit ' s viscera
     In order to study the distribution of tanshinone A in a rabbit's viscera we made the rabbit take the tanshinone A solution first,then killed it after 24 hours. The tanshinone A in the main viscera was determined by highly sensitive micelle fluorometry. The contents of tanshinone A in the heart,liver,kindey and lungs were 2.26,4.03,3.03 and 1.69 g/g,respectively.
     为了对丹参酮ⅡA在家兔内脏中的分布进行研究,在给家兔口服丹参酮液24h之后,处死动物,采用胶束增敏荧光法,对主要脏器所含丹参酮ⅡA进行测定,所测心、肝、肾、肺的含量分别为226,403,303,169μg/g.
短句来源
  “家兔内脏”译为未确定词的双语例句
     EXPERIMENTAL STUDY OF VISCERAL INJURIES OF RABBITS RESULTING FROM NOISE EXPOSURE
     强噪声对家兔内脏损伤的实验研究
短句来源
     SEGMENTAL DISTRIBUTION OF VISCERAL SENSORY NEURONS OF MAJOR SPLANCHNIC NERVE IN THE RABBIT
     家兔内脏大神经传入神经元的节段分布
短句来源
     This paper described the characteristcs of visceral injuries of rabbits exposed to a wide-band noise field of 168 dB sound pressure level.
     本文叙述了声压级为168dB的宽频带噪声对家兔内脏器官损伤的特征。
短句来源
     The Investigation for Visceral Toxicity Comparison of Two Kinds of Inorganic Bone Cement in Rabbits
     两种无机骨水泥对家兔内脏毒性的比较研究
短句来源
     EFFECTS OF HUMORAL FACTORS IN VISCERAAURICULOPOINT RESPONSE IN RABBITS—A Cross Circulation Experiment
     体液因素在家兔内脏-耳穴反应中的作用——交叉循环实验
短句来源
更多       
  相似匹配句对
     AN EXPERIMENTAL STUDY OF DETECTING POLLEN FROM THE RABBIT'S ORGANS DIED OF DROWNING
     溺死家兔内脏花粉检验的研究
短句来源
     EXPERIMENTAL STUDY OF VISCERAL INJURIES OF RABBITS RESULTING FROM NOISE EXPOSURE
     强噪声对家兔内脏损伤的实验研究
短句来源
     VISCERAL FUNGAL DISEASE
     内脏霉菌病
短句来源
     STUDIES ON SUPEROVULATION IN RABBITS
     家兔的超数排卵
短句来源
     stiedai infection in rabbits.
     证明NO是在家兔E.
短句来源
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Experiments were conducted on the rabbits under urethane and chloralose, tubocurarine and artificial respiration being given. In 108 out of 115 animals depressor responses were shown when the viscero-afferent nerves were excited by means of intestinal distension, or electrical stimulation of the central end of either the mesenteric or splanchnic nerve. As bradycardia and vasodilation accompanied the depressor response, it was obvious that the mechanism of the depressor response appeared to be due to inhibition...

Experiments were conducted on the rabbits under urethane and chloralose, tubocurarine and artificial respiration being given. In 108 out of 115 animals depressor responses were shown when the viscero-afferent nerves were excited by means of intestinal distension, or electrical stimulation of the central end of either the mesenteric or splanchnic nerve. As bradycardia and vasodilation accompanied the depressor response, it was obvious that the mechanism of the depressor response appeared to be due to inhibition of the cardiovascular centres. Neither transection of the middle brain, nor the vagus nerve at the neck could alter the pattern of the response. It was, therefore, clearly shown that the afferent impulses entered the neural axis through the sympathetic nerve, and that the center of this response was located below the level of the middle brain. After transection of the dorsal column of the spinal cord at C(1-2), the depressor response did not vanish. Mechanical or electrolytic destruction of the caudo-medial portion of the medulla oblongata abolished viscero-depressor response, while the depressor response induced by stimulating the aortic nerve remained unchanged. Destruction of the dorsal portion of the medulla, on the contrary, abolished the aortic-depressor response, while the viscero-depressor response remained still. The results mentioned above showed that: 1) excitation of the viscero-afferent nerves in rabbit caused inhibition of the cardiovascular centers, inducing depressor reaction, 2) the center of this reaction was located within bulbar-pontine region, 3) the caudo-medial portion of the medulla was related with viscero-depressor response, while its dorsal portion with the aortic-depressor response. This work was done under the direction of Prof. H. C. Chang.

(一)在氨基甲酸乙酯和氯醛糖的麻醉下,兴奋家兔內脏傳入神經,引起明显的减压反应。 (二)內脏-减压反应发生时伴有心率的减慢和外周血管的舒張。 (三)內脏-减压反应的中樞部位在延桥脑水平。 (四)破坏延脑后端腹內侧組織使內脏-减压反应消失,而主动脉神經-减压反应可不受影响。破坏延脑后端背內側組織却得相反的結果,即主动脉神經-减压反应消失,而内脏-减压反应仍然存在。 (五)就內脏-减压反应的傳导途徑,与延脑“减压区”的关系作了一些討論。

This paper described the characteristcs of visceral injuries of rabbits exposed to a wide-band noise field of 168 dB sound pressure level. The experimental results demonstrated that the sites of injuries were dependent on the posture of the animals. For animals in free posture or in prone posture, the most vulnerable organ is the lung, which opens to the outer atmosphere, and the next are the hollow organs such as the stomach and the intestines. For animals exposed in supine posture, the heart is usually as...

This paper described the characteristcs of visceral injuries of rabbits exposed to a wide-band noise field of 168 dB sound pressure level. The experimental results demonstrated that the sites of injuries were dependent on the posture of the animals. For animals in free posture or in prone posture, the most vulnerable organ is the lung, which opens to the outer atmosphere, and the next are the hollow organs such as the stomach and the intestines. For animals exposed in supine posture, the heart is usually as seriously injured as the lung, parts of the stomach, the intestine and the liver which are situated near the abdominal wall also show some significant injuries. Our experimental results revealed that the primary pathological cause of these organs is the mechanical effect of the sound waves.

本文叙述了声压级为168dB的宽频带噪声对家兔内脏器官损伤的特征。实验表明,家兔内脏的损伤部位与动物所处的体位有关。自由体位和俯卧位动物,主要损伤部位是向外环境开放的肺,其次是空腔脏器胃和肠管。仰卧位动物除肺脏受损外,心脏的损伤非常严重,靠近腹壁的肠管,胃体部及肝脏也受损明显。这些关系表明,高声强噪声对内脏的致伤主要是由于声波的机械效应。

The present work was carried out to investigate the role of septal stimulationand the intraseptal opiate receptor in analgesia for visceral pain in conscious rabbits.A bipolar electrode of Teflon-coated silver wires was implanted to stimulate thesplanchnic nerve. The stimulation consisted of a 500ms train of monophasic, re-tangular-wave pulses (0.5ms, 40Hz) and the current increment was 0.1mA per stepdelivered every five seconds. The minimal electric current required to producedefensive response of forelimbs...

The present work was carried out to investigate the role of septal stimulationand the intraseptal opiate receptor in analgesia for visceral pain in conscious rabbits.A bipolar electrode of Teflon-coated silver wires was implanted to stimulate thesplanchnic nerve. The stimulation consisted of a 500ms train of monophasic, re-tangular-wave pulses (0.5ms, 40Hz) and the current increment was 0.1mA per stepdelivered every five seconds. The minimal electric current required to producedefensive response of forelimbs was taken as visceral pain threshold. When theseptal nuclei was stimulated with weak stimulus intensity, the visceral painthreshold was increased, decreased or unchanged. After bilateral injection ofetorphine (2μg/2μl) into the septal nuclei, the visceral pain threshold increasedstrikingly in 5-40 min (P<0.05 or P<0.01). Naloxone (0.2mg/kg) intravenousinjection or 2μg/2μl microinjection into the PAG could reverse the analgesia frometorphine. Intravenous injection of naloxone (0.4mg/kg) was able to reverseacupuncture analgesia partially, leaving the after-effect not influenced. The an-algesic effect of acupuncture was reversed after bilateral injection of naloxone (2μg/2μl) into the septal nuclei and the after-effect was affected as well. The resultssuggest that the activity of intraseptal opiate receptor bring forth analgesic effect,which may be partially mediated through PAG,and the opiate receptor in the septalnuclei are involved in the electroacupuncture analgesia for visceral pain.

本工作以电刺激内脏大神经测定清醒家兔的内脏痛阈。实验观察到,以弱电流刺激隔核对内脏痛阈有升高、降低和无明显变化三种效应;向家兔双侧隔核注入阿片受体激动剂埃托啡(etorphine)2μg/2μl,注药后 5min,内脏痛阈即升高,维持 40min;静脉注射纳洛酮O.2mg/kg或 2μg/2μl纳洛酮注入中脑导水管周围灰质(PAG)均能阻断隔核内注入埃托啡的镇痛作用;电针刺激能抑制内脏痛,静脉注射纳洛酮 0.4mg/kg 能阻断这一效应,但对电针后效应无明显影响,向隔核内注入纳洛酮 2μg/2μl亦能阻断针效,而且后效应消失。结果提示,镇内脏痛与内阿片肽活动有关,隔核的阿片受体被激活即有明显抑制内脏痛的作用,其作用的传出通路可能通过 PAG;隔核的阿片受体参与电针镇内脏痛过程。

 
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