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新型冠状病毒
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  novel coronavirus
     Study on method for detecting RNA nucleic acid from novel coronavirus and its application
     新型冠状病毒RNA核酸的检测方法研究及应用
短句来源
     SARS-Cov and SARS-like-CoV were detected in these specimens using diagnostic kit for novel coronavirus N protein (ELISA), SARS-CoV Virus RNA detection kit, fluorescence PCR, Genchip, RT-PCR and cell isolation culture methods.
     收集的蝙蝠分别取得咽拭813份、血清524份、肺组织853份及直肠粪便853份标本共计3043份,用新型冠状病毒(N-蛋白)测定试剂盒(酶联免疫法)、SARS-CoVRNA检测试剂盒、荧光PCR及基因芯片检测并通过RT-PCR和细胞分离培养检测SARS-CoV和SARS-like-CoV。
短句来源
     Severe acute respiratory syndrome (SARS) whose the other name is atypical pneumonia in China is a newly emerged disease caused by a novel coronavirus, and it can lead to panic in the society because of its droplet infection and high mortality.
     严重急性呼吸综合征(Severe acute respiratory syndrome,SARS)又称传染性非典型肺炎,是由一种人类从未发现过的新型冠状病毒所导致的、严重危害公共卫生安全的疾病。
短句来源
     Conclusions Novel coronavirus like particles were found in the culture of the 1st SARS patient's throat swab in Shanghai.
     结论 在上海市的第 1例确诊 SARS病例的咽试培养中 ,见到新型冠状病毒样的颗粒
短句来源
     It has been proved that severe acute respiratory syndrome (SARS) is caused by a novel coronavirus.
     已经证明严重急性呼吸综合征 (SARS)是由一种新型冠状病毒所引起。
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  the sars coronavirus
     The SSH library provides a basis for further screen the series genes that express the SARS coronavirus specific IgG antibodies.
     SSH文库为筛选新型冠状病毒特异的抗体基因奠定了分子基础。
短句来源
  “新型冠状病毒”译为未确定词的双语例句
     Etiologic study showed that the etiologic agent for worldwiide prevalence of SARS is a new coronavirus species which later was called SARS-associated Coronavirus (SARS-CoV) by WHO.
     病因学研究表明,SARS的病因是一种新型冠状病毒,世界卫生组织将其命名为SARS相关冠状病毒(SARS-associated coronavirus,SARS-CoV)。
短句来源
     On April 16, 2003, WHO confirmed that a new coronavirus named SARS-CoV was the cause agent of SARS.
     2003年4月16日,WHO正式宣布:SARS的病原体是一种新型冠状病毒,并命名为SARS相关冠状病毒(SARS-CoV)。
短句来源
     The severe acute respiratory syndrome (SARS) is a highly virulent emerging disease. The etiological agent of SARS was identified as a new coronavirus(SARS-CoV).
     严重急性呼吸系统综合征(Severe acute respiratory syndrome,SARS)是一种烈性新发传染病,其病原体为一种新型冠状病毒(SARS-CoV)。
短句来源
     Human coronavirus-NL63 was detected in spec imens from children with acute respiratory infection in Beijing, China
     从北京地区急性呼吸道感染患儿标本中检测到新型冠状病毒NL63基因
短句来源
     A compound disinfecting nanoemulsion, which containing 1 700- 1 900 mg/ L chlorhexdine acetate and 1 000 mg/ L nanometer size zinc oxide, is tested for inactivation of new coronaviruses causing severe acute respiratory syndrome (SARS).
     试验观察了一种含1700~1900mg/L醋酸氯己定、1000mg/L纳米氧化锌的复方消毒乳液,对引起重症急性呼吸综合症(SARS) 的新型冠状病毒的杀灭效果。
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  novel coronavirus
Severe acute respiratory syndrome (SARS) is a highly contagious and predominantly pneumonic illness caused by a novel coronavirus now commonly known as SARS-CoV.
      
A novel coronavirus was identified as the cause for severe acute respiratory syndrome (SARS).
      
Severe acute respiratory syndrome (SARS) is caused by a novel coronavirus (CoV), SARS-CoV.
      
The severe acute respiratory syndrome (SARS) is an emerging infection caused by a novel coronavirus which first appeared in southern China at the end of 2002.
      
A novel coronavirus, isolated from the respiratory secretions of patients, has been implicated in the causation of SARS.
      
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  the sars coronavirus
A rate equation approach to model the denaturation or replication behavior of the SARS coronavirus
      
As a newly emerging virus, little is known about the SARS coronavirus, whose outbreak has brought away several hundred people's lives over the world in the year of 2003 and is seriously imperiling the human health.
      
The complex physical and chemical behaviors of the SARS coronavirus can then be attributed to its activation energy, frequency factor, damage function as well as the surrounding environmental conditions.
      
Intriguingly, the SARS coronavirus, the cause of severe acute respiratory syndrome (SARS), utilizes ACE2 as an essential receptor for cell fusion and in vivo infections.
      
Structural proteomics of the SARS coronavirus: a model response to emerging infectious diseases
      
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The severe acute respiratory syndrome (SARS) has been spreading in nearly 30 countries and regions worldwide recently. In response to its outbreak, many laboratories worldwide collaborated to identify the causative agent of SARS. A new type of coronavirus(named SARS CoV)has been isolated consequently, and the complete genomic sequences of the SARS CoV have been published. It is very important, at this stage, to deeply investigate the functional genome of SARS CoV with modern molecular techniques. As a new...

The severe acute respiratory syndrome (SARS) has been spreading in nearly 30 countries and regions worldwide recently. In response to its outbreak, many laboratories worldwide collaborated to identify the causative agent of SARS. A new type of coronavirus(named SARS CoV)has been isolated consequently, and the complete genomic sequences of the SARS CoV have been published. It is very important, at this stage, to deeply investigate the functional genome of SARS CoV with modern molecular techniques. As a new gene silencing method at RNA level, the recently established RNA interference will be a useful approach for the studies of SARS CoV genome.

最近 ,严重急性呼吸综合征 (SARS)在全世界约 30个国家和地区散发或流行 ,众多实验室由此展开了针对 SARS病原体的国际联合攻关。目前 ,已经确认其病原体是一种新型冠状病毒 (命名为 SARS- Co V) ,而且该病毒的全基因组测序已经完成。采用现代分子病毒学实验技术对 SARS病毒的功能基因组进行深入研究 ,有重要的理论和实际意义。作为一种崭新的基因沉默技术 ,RNA干扰无疑将在 SARS病毒基因组研究中发挥重要的作用。

Objective To study the pathological characteristics, pathogenesis and the pathogen of severe acute respiratory syndrome (SARS) by systematic examination of the pathological changes in 30 organs from 6 autopsies of patients dying in different periods of the disease. Methods Pathological changes were studied by light microscopy, electron microscopy, histochemical and immunohistochemical staining. Results ①The basic pathological changes in the patients dying at the early stage of SARS were mainly degenerative...

Objective To study the pathological characteristics, pathogenesis and the pathogen of severe acute respiratory syndrome (SARS) by systematic examination of the pathological changes in 30 organs from 6 autopsies of patients dying in different periods of the disease. Methods Pathological changes were studied by light microscopy, electron microscopy, histochemical and immunohistochemical staining. Results ①The basic pathological changes in the patients dying at the early stage of SARS were mainly degenerative changes, such as cellular degeneration, apoptosis and necrosis in the lung and immune organs (spleen, lymph nodes, and mucous membrane lymph tissue), and circulatory disturbances such as pulmonary edema, hyaloid membrane formataion and hemorrhage. In the patients dying at the middle and later stages, the basic pathological changes were necrosis and excoriation of pulmonary alveolar epithelial cells, interstitial fibrotic proliferation, accompanied by early fibrosis and progressive atrophy of lymphoid tissues. These results confirmed again that the lung and immune organs were the major target organs. ②The process of pulmonary changes could be divided into three periods: the first period was characterized by acute exudative and leakage inflammation (within two weeks of onset of the disease, the main pathological changes were severe diffuse edema and hyaline membrane formation in lung), the second period showed the feature of pulmonary epithelial necrosis and excoriation, accompanied by hyperplastic and organizational inflammation (within third to fourth weeks); the third period was hyperplasia and early fibrosis of pulmonary tissue (within fifth to sixth weeks) . The lung damages were widespread, immediate, progressive, staged and diverse. ③Serious damage and diffuse hemorrhage were observed in the immune organ, such as spleen, lymph nodes. Remarkable atrophy was seen in spleen nodules and lymph folliculi, with early apoptosis and necrosis of lymphocytes. The number of T cells and B cells were extremely decreased; the immune function became very low. The damages to the immune organs were widespread, immediate, and persistent. ④It was confirmed that novel coronavirus(Co v) was the major pathogen, and the chlamydia like particles was also one of the pathogens. At the same time, unknown stick like structures were found in the lung and spleen, and cocci were also found in the lung. Conclusions Examinations of the autopsy specimens from SARS patients showed that the pathogens were diverse, in which Co v was the major pathogen. The pathology involved multiple organs and cells in the human body, producing viremia. The lung and lymphoid tissues were the main target organs; pathological changes of the lung were progressive and could be divided into 3 stages. Pulmonary fibrosis occurred in 4~6 weeks. The atrophy of the immune organs was persistent and progressive. The immune function was low for a long time(The pictures were put into the color pages of this journal.).

目的 系统观察 6例严重急性呼吸综合征(SARS)不同时间死亡患者近 30个脏器的病理变化 ,探讨其病理发生发展过程及病变和病原体特点。方法 采用光镜、透射电镜、组织化学和部分免疫组织化学方法观察各脏器的病理变化。结果 ①重症SARS死亡患者早期基本病变为以肺和免疫器官 (脾、淋巴结、黏膜淋巴组织)为主的全身各脏器发生不同程度的实质细胞变性、凋亡和坏死等变质性改变和肺水肿、透明膜形成及出血等为主的血循环障碍 ;中后期则以肺上皮坏死脱落和间质纤维增生伴早期纤维化及免疫器官进行性萎缩等病变为主 ,再次证实肺和免疫器官为主要靶器官 ;②肺脏病变经历急性渗漏性炎症期 (发病后 2周内 ,主要病变为严重的弥漫性肺水肿和透明膜形成)、肺泡上皮坏死脱落伴增生机化性炎症期 (发病后 3~ 4周 )和纤维增生伴早期纤维化期 (发病后 5~ 6周 ) ,具有广泛性、速发性、进行性和阶段性、多样性等特点 ;③在重症SARS死亡患者中 ,脾和淋巴结等免疫器官发生严重的破坏和广泛出血 ,脾小体和淋巴滤泡极度萎缩 ,淋巴细胞迅速发生凋亡和坏死 ,T、B淋巴细胞数量极度减少 ,免疫功能极度低下 ,其病变同样具有广泛性、速发性和持续性等特点...

目的 系统观察 6例严重急性呼吸综合征(SARS)不同时间死亡患者近 30个脏器的病理变化 ,探讨其病理发生发展过程及病变和病原体特点。方法 采用光镜、透射电镜、组织化学和部分免疫组织化学方法观察各脏器的病理变化。结果 ①重症SARS死亡患者早期基本病变为以肺和免疫器官 (脾、淋巴结、黏膜淋巴组织)为主的全身各脏器发生不同程度的实质细胞变性、凋亡和坏死等变质性改变和肺水肿、透明膜形成及出血等为主的血循环障碍 ;中后期则以肺上皮坏死脱落和间质纤维增生伴早期纤维化及免疫器官进行性萎缩等病变为主 ,再次证实肺和免疫器官为主要靶器官 ;②肺脏病变经历急性渗漏性炎症期 (发病后 2周内 ,主要病变为严重的弥漫性肺水肿和透明膜形成)、肺泡上皮坏死脱落伴增生机化性炎症期 (发病后 3~ 4周 )和纤维增生伴早期纤维化期 (发病后 5~ 6周 ) ,具有广泛性、速发性、进行性和阶段性、多样性等特点 ;③在重症SARS死亡患者中 ,脾和淋巴结等免疫器官发生严重的破坏和广泛出血 ,脾小体和淋巴滤泡极度萎缩 ,淋巴细胞迅速发生凋亡和坏死 ,T、B淋巴细胞数量极度减少 ,免疫功能极度低下 ,其病变同样具有广泛性、速发性和持续性等特点 ;④证实主要病原体为新型冠状病毒 ,衣原体样颗粒也是致病病原体之一 ,同时

Objective To study the pathological and ultramicrostructural characteristics of organ tissues in relation to the clinical course of severe acute respiratory syndrome (SARS). Methods Post-mortem tissue samples of organs (lungs, heart, liver, spleen, kidneys, pancreas, stomach ) were obtained by needle biopsy from four SARS patients who died in middle and late stages 3-5 weeks after the onset of the disease. The pathological samples were studied by light and electron microscope, immunohistochemistry, histochemistry...

Objective To study the pathological and ultramicrostructural characteristics of organ tissues in relation to the clinical course of severe acute respiratory syndrome (SARS). Methods Post-mortem tissue samples of organs (lungs, heart, liver, spleen, kidneys, pancreas, stomach ) were obtained by needle biopsy from four SARS patients who died in middle and late stages 3-5 weeks after the onset of the disease. The pathological samples were studied by light and electron microscope, immunohistochemistry, histochemistry and indirect immuno-fluorescent antibody test. Results The main pathological features were early interstitial pulmonary fibrosis or organizing pneumonia. Fibroblasts were increased in the interalveoli septa and young connective tissue was found to fill the alveoli. Diffuse alveolar damage (DAD) with alveolar pneumocytes proliferation and an increase in macrophages were found. Desquamative alveolitis also existed at the same time. Squamous metaplasia and syncytial giant cells with multinuclei could be seen. CD3 + and CD20 + lymphocytes were markedly decreased and CD68 + macrophages and S-100 + dendritic cells increased in spleen. Proliferation of bone marrow cells became restrained . Hepatocytes were vacuolated with fatty degeneration. Electron microscopy showed the presence of coronavirus-like particles 80-60nm in diameter enveloped in the cytoplasm of the type Ⅱ pneumocytes, endothelial cells and lymphocytes. Conclusions A novel coronavirus is the cause of the newly recognized severe acute respiratory syndrom (SARS). The main target organs are lung and immune system. Different pulmonary pathological features were found in patients dying from the disease in different stages. All of specimens showed positive reaction of SARS-fluorescence antibody.

目的 探讨严重急性呼吸综合征 (SARS)病理学改变和超微结构特点及其与临床病程的关系。方法 应用光镜、组织化学、透射电镜、免疫组化和间接免疫荧光法对病程为 3~ 5周的 4例中晚期SARS死亡病例多器官穿刺组织 (肺脏、心脏、肝脏、脾脏、肾脏、胰腺、胃)进行研究。结果 SARS死亡病例肺部病理改变以肺泡间质纤维增生和肺泡早期纤维化等机化性肺炎表现为主要特征 ,同时有弥漫性肺泡损伤和脱屑性肺炎改变。脾脏内CD3和CD2 0阳性淋巴细胞减少 ,CD6 8阳性的组织细胞和S 10 0阳性的树突状细胞增多。骨髓粒细胞系统增生减退。在肺泡Ⅱ型上皮细胞、血管内皮细胞和脾脏淋巴细胞胞质内可见多种类型的冠状病毒样颗粒 ,大小均为 6 0~2 2 0nm ,有包膜 ,表面似有纤突样结构 ,以具有同心圆外膜、低电子密度核心的A型病毒颗粒和具有高电子密度核心的C型病毒颗粒为多见。间接免疫荧光法血清检测 4例病人SARS病毒IgGAb均呈明确阳性反应。结论 SARS是由新型冠状病毒引起的急性呼吸道传染病 ,主要靶器官是肺脏和免疫器官。SARS不同病程死亡病人的肺部病理学改变有不同的特征。

 
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