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微血管灌注
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  microvascular perfusion
    After administering all shed blood, though the mean aortic pressures of control and ECP groups were recovered nearly to the basic level, the increase of microvascular perfusion in ECP groups was higher significantly than that in control groups.
    在回输失血复苏后,对照组和反搏组主动脉平均压力恢复接近失血前水平,但反搏组微血管灌注的回升却明显高于对照组。
短句来源
  microvascular perfusion
    After administering all shed blood, though the mean aortic pressures of control and ECP groups were recovered nearly to the basic level, the increase of microvascular perfusion in ECP groups was higher significantly than that in control groups.
    在回输失血复苏后,对照组和反搏组主动脉平均压力恢复接近失血前水平,但反搏组微血管灌注的回升却明显高于对照组。
短句来源
  “微血管灌注”译为未确定词的双语例句
    Objective: To establish the model of swan-like memory compressive connector(SMC)treating experimental humeral fracture in rabbits, and apply following techniquesas: biomechanics, microangiography, radioactive microsphere fluometry, bioelectricpotential, bone mineral density, scan electroscope, reverse transcriptase polyerase chainreaction(RT-PCR), immunohistofluorescence dying, and elucidate following questionsabout humeral fractures fixed by SMC: (1)Radiographic and the biomechanicalcharacteristic in fracture healing of humerus treated by SMC, stress shielding ratio(SSR)of SMC.
    目的:建立天鹅记忆接骨器(SMC)治疗肱骨干骨折的动物模型,并应用生物力学、微血管灌注和放射性微球、骨生物电测试、骨矿含量检测以及扫描电镜观察、逆转录聚合酶链反应(RT-PCR)、免疫荧光组织化学等方法,阐明在 SMC 作用下:(1)骨的力学和几何特性演变及 SMC 的应力遮挡率;
短句来源
    The circulation was stopped by tourniquet in one rabbit hindlimb. After 2 or 5 hr of ischemia,the hindlimb was reperfused. During the first 1 hr of reperfusion,dynamic changes in surface microcirculation of extensor digitorum longus tendon were observed by using intravital microscopy.
    应用活体显微镜技术,观察了兔后肢常温止血带缺血2h(n=8)及5(n=8)再灌注后最初1h期间足背肌腱表面微循环动态变化,尤其是白细胞内皮粘附及微血管灌注状况的变化,旨在探讨缺血再灌注损伤的发生机制,从而指导临床治疗。
短句来源
    Dynamic vascular changes were investigated by microangiography,microscopy and computerized image analysis system at regular intervals after operation.
    应用微血管灌注、组织切片、计算机图像分析等方法,于术后定时地观察皮瓣愈合过程中血管形态的动态变化。
短句来源
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  microvascular perfusion
Within irreversibly injured (infarcted) regions microvascular perfusion can vary from nearly normal to nearly zero, even in the presence of an open infarct-related artery ('no-reflow').
      
Historically, non-invasive assessment of heterogeneous microvascular perfusion within myocardial infarcts has been problematic.
      
The new frontiers of CMR in heart failure hold the promise of unique insights quantifying myocardial iron, nonischemic fibrosis, microvascular perfusion, plaque characterization, and CMR-targeted intervention.
      
Both theno-reflow phenomenon and the events initiated by reflow - termed herein as thereflow-paradox - contribute to the failure of the nutritive microvascular perfusion and loss of tissue viability following ischemia and reperfusion.
      
We studied in vivo the effect of cyclosporine A (CsA) on both pancreatic islet vascularization and microvascular perfusion using intravital fluorescence microscopy and the dorsal skinfold chamber model in Syrian golden hamsters.
      
更多          
  microvascular perfusion
Within irreversibly injured (infarcted) regions microvascular perfusion can vary from nearly normal to nearly zero, even in the presence of an open infarct-related artery ('no-reflow').
      
Historically, non-invasive assessment of heterogeneous microvascular perfusion within myocardial infarcts has been problematic.
      
The new frontiers of CMR in heart failure hold the promise of unique insights quantifying myocardial iron, nonischemic fibrosis, microvascular perfusion, plaque characterization, and CMR-targeted intervention.
      
Both theno-reflow phenomenon and the events initiated by reflow - termed herein as thereflow-paradox - contribute to the failure of the nutritive microvascular perfusion and loss of tissue viability following ischemia and reperfusion.
      
We studied in vivo the effect of cyclosporine A (CsA) on both pancreatic islet vascularization and microvascular perfusion using intravital fluorescence microscopy and the dorsal skinfold chamber model in Syrian golden hamsters.
      
更多          


The circulation was stopped by tourniquet in one rabbit hindlimb.After 2 or 5 hr of ischemia,the hindlimb was reperfused. During the first 1 hr of reperfusion,dynamic changes in surface microcirculation of extensor digitorum longus tendon were observed by using intravital microscopy. Particular attention was paid to leukocyte-endothelium adhesion and microvascular perfusion condition. It was found that ①the number of leukocytes adherent to the venular endothelium of ischemic tissue significantly increased following...

The circulation was stopped by tourniquet in one rabbit hindlimb.After 2 or 5 hr of ischemia,the hindlimb was reperfused. During the first 1 hr of reperfusion,dynamic changes in surface microcirculation of extensor digitorum longus tendon were observed by using intravital microscopy. Particular attention was paid to leukocyte-endothelium adhesion and microvascular perfusion condition. It was found that ①the number of leukocytes adherent to the venular endothelium of ischemic tissue significantly increased following reperfusion and the degree of increases appeared to be directly related to the duration of ischemic period ②reperfusion after 5 hr of ischemia could result in the formation of "no reflow phenomenon" in the ischemic tissues ,including"primary. no reflow" and "secondary capillary perfusion failure".These results suggest that① leuko cyte-endothelium adhesion may contribute to the development of ischemia reperfusion inajury; ②in the limbs undergone 5 hr of ischemia,not all injuries that occur after ischemireperfusion can be attributed to ischemia reperfusion injury.The injury seen some in areas may be produced by ischemia alone ,and in others ischemia-reperfusion-secondary ischemia injury may be responsible.

应用活体显微镜技术,观察了兔后肢常温止血带缺血2h(n=8)及5(n=8)再灌注后最初1h期间足背肌腱表面微循环动态变化,尤其是白细胞内皮粘附及微血管灌注状况的变化,旨在探讨缺血再灌注损伤的发生机制,从而指导临床治疗。结果表明:①肢体缺血再灌注后缺血组织微静脉内皮上粘附的白细胞数显著增加,而且缺血时间越长,增加越显著。②肢体缺血5h再灌注后,缺血组织的微循环并不能均匀恢复,部分区域发生"无复流现象",包括原发性无复流和继发性毛细血管灌注衰竭两种形式。提示:①白细胞内皮粘附参与了缺血再灌注损伤的病理生理过程。②肢体缺血5h再灌注后发生的局部组织损伤并非皆属缺血再灌注损伤,部分区域可能系单纯缺血性损伤,部分区域则可能属缺血再灌注一继发性缺血损伤。

The circulation was stopped by tourniquet in one rabbit hindlimb.After 2 or 5 hr of ischemia,the hindlimb was reperfused.During the first 1 hr of reperfusion,dy- namic changes in surface microcirculation of extensor digitorum longus tendon were ob- served by using intravital microscopy.Particular attention was paid to leukocyte-en- dothelium adhesion and microvascular perfusion condition.It was found that①the num- ber of leukocytes adherent to the venular endothelium of ischemic tissue significantly in- creased...

The circulation was stopped by tourniquet in one rabbit hindlimb.After 2 or 5 hr of ischemia,the hindlimb was reperfused.During the first 1 hr of reperfusion,dy- namic changes in surface microcirculation of extensor digitorum longus tendon were ob- served by using intravital microscopy.Particular attention was paid to leukocyte-en- dothelium adhesion and microvascular perfusion condition.It was found that①the num- ber of leukocytes adherent to the venular endothelium of ischemic tissue significantly in- creased following reperfusion and the degree of increases appeared to be directly related to the duration of ischemic period;②reperfusion after 5 hr of ischemia could result in the formation of“no reflow phenomenon”in the ischemic tissues,including“primary no re- flow”and“secondary capillary perfusion failure”.These results suggest that①leuko cyte-endothelium adhesion may contribute to the development of ischemia reperfusion inajury;②in the limbs undergone 5 hr of ischemia,not all injuries that occur after ische- mi-reperfusion can be attributed to ischemia reperfusion injury.The injury seen some in areas may be produced by ischemia alone,and in others ischemia-reperfusion-secondary ischemia injury may be responsible.

应用活体显微镜技术,观察了兔后肢常温止血带缺血2h(n=8)及5h(n=8)再灌注后最初1h 期间足背肌腱表面微循环动态变化,尤其是白细胞内皮粘附及微血管灌注状况的变化,旨在探讨缺血再灌注损伤的发生机制,从而指导临床治疗。结果表明:①肢体缺血再灌注后缺血组织微静脉内皮上粘附的白细胞数显著增加,而且缺血时间越长,增加越显著。②肢体缺血5h 再灌注后,缺血组织的微循环并不能均匀恢复,部分区域发生“无复流现象”,包括原发性无复流和继发性毛细血管灌注衰竭两种形式。提示:①白细胞内皮粘附参与了缺血再灌注损伤的病理生理过程。②肢体缺血5h 再灌注后发生的局部组织损伤并非皆属缺血再灌注损伤,部分区域可能系单纯缺血性损伤,部分区域则可能属缺血再灌注—继发性缺血损伤。

Objective: Dynamic vascular changes during fasciocutaneous flap revascularization and its clinical significance were studied postoperatively.Methods: Fasciocutaneous flap or ordinary flap was desigened at the lateral side of either hindlimb of the pig.Dynamic vascular changes were investigated by microangiography,microscopy and computerized image analysis system at regular intervals after operation.Results:The blood vessels of fasciocutaneous flap grew faster than those of ordinary flap during revascularization.Marked...

Objective: Dynamic vascular changes during fasciocutaneous flap revascularization and its clinical significance were studied postoperatively.Methods: Fasciocutaneous flap or ordinary flap was desigened at the lateral side of either hindlimb of the pig.Dynamic vascular changes were investigated by microangiography,microscopy and computerized image analysis system at regular intervals after operation.Results:The blood vessels of fasciocutaneous flap grew faster than those of ordinary flap during revascularization.Marked revascularization could be noticed at proximal,mid or distal portion of fasciocutaneous flap,but no visible revascularization could be viewed at distal portion of ordinary flap. Conclusions:Vascular changes of fasciocutaneous flap were synchronous with that of its deep fascia.

目的:为探讨筋膜皮瓣移植术后,其愈合过程中血管形态的动态变化及其临床意义。方法:在猪的两后肢外侧,分别设计筋膜皮瓣或普通皮瓣。应用微血管灌注、组织切片、计算机图像分析等方法,于术后定时地观察皮瓣愈合过程中血管形态的动态变化。结果:①在再血管化过程中,筋膜皮瓣血管增生的速度及幅度,均优于普通皮瓣;②筋膜皮瓣近、中、远各部再血管化明显同步,普通皮瓣远端则未见明显再血管化。结论:筋膜皮瓣血供与其深筋膜血供变化同步

 
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