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碘过多
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  iodine excess
     Iodine excess had no significant effects on level of T4,T3 in the serum and the antioxidizing ability of thyroid.
     碘过多对大鼠血清T4、T3水平和甲状腺的抗氧化能力无明显影响。
短句来源
     A study on membrane fluidity of thyroid gland in rats with iodine deficiency or iodine excess
     碘缺乏和碘过多对大鼠甲状腺细胞膜脂流动性的影响
短句来源
     Results Iodine excess can cause goiter, but the extent of goiter was significantly lower than that of iodine deficiency.
     结果 碘过多可导致大鼠甲状腺肿大,但肿大程度明显低于碘缺乏。
短句来源
  “碘过多”译为未确定词的双语例句
     A quantitative morphological study on thyroid gland in rats of iodine deficiency and excess
     碘缺乏与碘过多大鼠甲状腺定量形态学研究
短句来源
     Conclusion For the children living in the IE environment of iodine 300-1500μg/L in water,their brain dvelopment was normal,and IE couldn't be a risk factor of mental retardation.
     结论在水碘300~1500μg/L之间的碘过多环境中生活的学生智力发展正常,高碘不是智力低下的危险因素。
短句来源
     Conclusion Excessive in-take of dietary iodine may be related with AITD.
     结论在AITD患者中存在饮食碘过多的问题,此可能在AITD的发病中起作用。
短句来源
     Conclusions The increase of thyrotoxicosis and hypothyroidism because of excess iodine added to salt.
     结论 甲状腺毒症和甲状腺功能减退症的增加与盐中的碘过多有关。
短句来源
     EFFECT OF IODINE DEFICIENCY AND EXCESS ON GOITER FORMATION AND THYROID FUNCTION IN RATS
     碘缺乏与碘过多对大鼠甲肿形成及功能的影响
短句来源
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  相似匹配句对
     The high iodine intake could suppress the synthesis and release on thyroid hormone.
     摄入过多可以抑制甲状腺激素的合成和释放。
短句来源
     Conclusions The increase of thyrotoxicosis and hypothyroidism because of excess iodine added to salt.
     结论 甲状腺毒症和甲状腺功能减退症的增加与盐中的过多有关。
短句来源
     The spectrophotometric determination of microamount of atomic iodine
     (I)的分光光度法测定
短句来源
     Iodide Goiter
     高甲状腺肿
短句来源
     6.Protection is excessive;
     6.保护过多
短句来源
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  iodine excess
The addition of selenium alleviated the toxic effects of iodine excess on the activities of D1 and GSHPx.
      
Applying this method for the analysis of Antarctic meteorites, an "iodine excess" in some of these samples compared to the normal iodine concentration in non-Antarctic meteorites could be confirmed.
      
Urinary iodine excretion was between 12-22 (n=3) and 40-42.5 (n=5) μg/dL tn 10 eutopic neonates (2 not assessed).Conclusion: Parental consanguinity and iodine excess could be the causative factors for the high incidence of CH.
      
Effects of chronic iodine excess in a cohort of long-term American workers in West Africa.
      
Transient cases might be caused by transplacental passage of TSH receptor blocking antibodies, or iodine excess.
      


The experiment was designed to study the effect of iodine deficiency and excess on goiter formation and thyroid function in rats. Methods: The rats were kept on a normal iodine diet (NI 6.9μgI/d), a low iodine diet(LI 1 .35μgI/d) and a high iodine diet(HI 555μgI/d) for 24 weeks respectively. After 12 and 24 weeks, the rats were killed and the thyroid pituitary were weighed, iodine content in thyroid tissue and urine and the throid hormones in serum and thyroid tissue were measured. Results: The results showed...

The experiment was designed to study the effect of iodine deficiency and excess on goiter formation and thyroid function in rats. Methods: The rats were kept on a normal iodine diet (NI 6.9μgI/d), a low iodine diet(LI 1 .35μgI/d) and a high iodine diet(HI 555μgI/d) for 24 weeks respectively. After 12 and 24 weeks, the rats were killed and the thyroid pituitary were weighed, iodine content in thyroid tissue and urine and the throid hormones in serum and thyroid tissue were measured. Results: The results showed that in LI rats thyroid and pituitary weight were higher, the serum and tissue hormones and iodine in urine and tissue were lower than NI. In HI rats the thyroid and pituitary weight, serum T4 and tissue iodine were shown no difference, serum T3 and tissue T4 declined markedly at 24 week. The urine iodine was significantly higher than NI. Conclusion: Based on the results we concluded that iodine deficiency caused overt goiter and hypothyroidism. Iodine excess did not induce goiter in Wistar rats during our experiment, while did have an inhibitory effect on thyroid function.

目的;研究碘缺乏与碘过多对大鼠甲状腺肿形成及其功能的影响。方法:实验分为三组,适碘组(对照组)、低碘组、高碘组,观察甲状腺及垂体重量,甲状腺组织及尿液的碘含量,血清及甲状腺组织激素水平。结果:低碘组甲状腺及垂体重量明显高于对照组,血清T_4及组织T_3、T_4、组织及尿碘含量均明显低于对照组,血清T_3在12周时代偿性增高,24周时明显下降。高碘组甲状腺及垂体重量,血清T_4及组织碘含量无明显改变,尿碘明显高于对照组,血清T_3及组织T_4于24周时明显低于对照组。结论:碘缺乏可引起大鼠甲肿和功能低下,而高碘尚未能引起甲肿,但抑制了甲状腺激素的合成和释放。

Objective The experiment was designed to dtudy the affect of iodine deficiency and excess on thyroid morphology.Methods The rats were divided into three groups randomly,normal iodine group (NI,as a control),low iodine group (LI) and high iodine group (HI).Morphological observations and stereological analysis were performed by using routine histology method and MIAS 20000 image analysis system.A certain number of thyroid follicle,follicular cavities and epithelial cell were measured for the stereology parameters...

Objective The experiment was designed to dtudy the affect of iodine deficiency and excess on thyroid morphology.Methods The rats were divided into three groups randomly,normal iodine group (NI,as a control),low iodine group (LI) and high iodine group (HI).Morphological observations and stereological analysis were performed by using routine histology method and MIAS 20000 image analysis system.A certain number of thyroid follicle,follicular cavities and epithelial cell were measured for the stereology parameters including mean volume (V),mean surface (S),specific suface (S/V),numerical density (Nv),and spherical factor(SF).Results The results showed that V,S,SF in LI group were lower and Nv,S/V,volume of epithelial cells higher significantly than that in NI group.However,all parameters of follicle in HI group were no difference compared with NI group,but at 24 w the V and SQ of follicular cavities were lower and the volume of epithelial higher markedly than those in control group.Conclusions Iodine deficiency caused typical small follicular goiter,and iodine excess didn't induce goiter in Wistar rats during our experiment.The follicular cavities became decreased,epithelial cells proliferated with high iodine prolonging. [

目的 研究碘缺乏与碘过多对大鼠甲状腺形态结构的影响。方法 将 Wistar大鼠随机分为 3组 ,适碘组 (NI)、高碘组 (HI)、低碘组 (L I) ,观察喂养 12周、2 4周大鼠甲状腺形态结构的变化 ,应用 MIAS- 2 0 0 0型图像分析系统 ,对甲状腺滤泡及滤泡腔进行形态定量测定 ,获得 5项体视学参数 (平均体积 V、平均表面积 S、比表面积 S/ V、数密度 Nv及球形因子 SF)和 1项截面积的定量参数。结果 低碘组大鼠甲状腺滤泡及滤泡腔的 V、S及 SF均明显小于适碘组 ,而 S/ V和 Nv明显大于适碘组 ,上皮细胞体积明显增大 ;高碘组滤泡在实验过程中无明显改变 ,而滤泡腔 V、S在实验的 2 4周时均明显小于适碘组 ,上皮细胞体积增大。结论 碘缺乏导致大鼠甲状腺小滤泡增生性甲肿改变 ,而碘过多在实验中未形成甲肿 ,并随碘过多时间的延长 ,滤泡腔变小 ,上皮细胞增生。

Objective Study the effects of iodine on the red cell immune system and SOD activity in Wistar rats.Methods C3b sensitized yeast cells and C3b unsensitized yeast cells were used to detect C3b receptors and immune complex on red cells,method of xanthine oxidase was applied to detect SOD activity in red cells.Results It was observed that the rosette rate of red cell C3b receptor,the rosette rate of red cell immune complex and SOD activity of red cells in iodine excess Wistar rats were higher than those of...

Objective Study the effects of iodine on the red cell immune system and SOD activity in Wistar rats.Methods C3b sensitized yeast cells and C3b unsensitized yeast cells were used to detect C3b receptors and immune complex on red cells,method of xanthine oxidase was applied to detect SOD activity in red cells.Results It was observed that the rosette rate of red cell C3b receptor,the rosette rate of red cell immune complex and SOD activity of red cells in iodine excess Wistar rats were higher than those of normal rats.There was no statistical difference for those between iodine deficiency and normal groups.Conclusions These results suggest that iodine excess can lead to red cell immune hyperfunction.This could be one of pathlogical factors for autoimmune reactions in thyroid induced by iodine excess. [

目的 研究在碘缺乏和碘过多的情况下 ,大鼠体内红细胞免疫功能与 SOD活性变化。方法 实验利用低碘饲料和碘化钾去离子水复制了不同碘营养状态的大鼠 ,采用 C3b致敏和未致敏的酵母菌及黄嘌呤氧化酶法分别检测红细胞膜上的 C3b受体、免疫复合物及红细胞内 SOD的活性。结果 碘过多时 ,大鼠红细胞 C3b受体花环率、红细胞免疫复合物花环率及红细胞内 SOD活性均较对照组明显增高 ,碘缺乏时与对照组比较差异无显著意义。结论 碘过多时会引起机体红细胞免疫功能的亢进 ,这可能也是构成高碘引起甲状腺肿自身免疫反应发病机制的因素之一。

 
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