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   神经元缺失 的翻译结果: 查询用时:0.029秒
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神经元缺失
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  neuron loss
     This is proposed to be a replacement changes for the NPY neuron loss in the hilus of dentate gyrus after seizures.
     结论:这可能是对癫痫发作后齿状回门区神经肽Y能神经元缺失的一种代偿性变化。
短句来源
     Results By cell counting, the neuron loss mainly occurred in acute phase, worst in CA3 and CA4, moderate in CA1 and CA2, and no loss in the dentate gyrus.
     细胞计数可见海马结构神经元死亡主要发生于急性期,以海马CA3、CA4区最为显著,海马其他各区较轻,齿状回无明显神经元缺失;
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  neuronal loss
     Results IGF 1 treatment significantly reduced neuronal loss in cerebral coxtex compared with the control group[2 d group(24±6)%vs (35±9)%,F=5.19,P< 0.05, 5 d group(30±7)%vs (51±13)%,F=13.47,P< 0.01].
     结果与对照组2d时(35±9)%和5d时(51±13)%相比,IGF-1治疗组的皮层神经元缺失显著减少犤2d组(24±6)%,F=5.19,P<0.05,5d组(30±7)%,F=13.47,P<0.01)。
短句来源
     Results Frequent recurrent AGSs could cause neuronal loss in CA 1 region of hippocampus and hippocampal mossy fiber sprouting got into the inner molecular layer of dentate gyrus in P77PMC rats, and could decrease the latency of Ⅳ/Ⅴ grade of AGSs, increase the durations of AGSs.
     结果 反复多次听源性惊厥样发作可导致P77PMC大鼠海马CA1区的部分神经元缺失和海马齿状回内分子层出现明显的MF发芽表现 ;
短句来源
     Whereas , there was significant neuronal loss in hippocampal CA, of model group rats compared with contral group rats.
     模型组大鼠海马CA:区与对照组比较有明显的神经元缺失
短句来源
     NISSLE staining showed that neuronal lost significantly in hippocampus CA1 region in model groups. In contrast to ginsenside Rg2 treated groups, ginsenside Rg2 could inhibit neuronal loss.
     尼氏染色结果显示:模型组可以造成显著的海马CA1区神经元的缺失,人参皂苷Rg_2的治疗可以抑制Aβ与IBA引起的大鼠海马区神经元缺失
短句来源
     Based on this model, chronic ad-ministration of 764-3 (8 mg/(kg. d), ip), an extract from Chinese traditiona1 medicine,markedly improved the learning and memory impairment in the injured caudate nucleus rats-Moreover, attenuation of the neuronal loss in caudate nucleus following excitotoxic damagewas observed after 764-3 treatment.
     观察到中药单体764-3(8mg/(kg·d),ip)能够明显改善尾核损伤大鼠的学习记忆缺陷,并减轻兴奋毒性所致的尾核神经元缺失
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  “神经元缺失”译为未确定词的双语例句
     Results Neuron death and up-regulation of caspase-3 and Aβ were seen,beginning at 2d,high-peak at 4d,decling at 7d,which was more statistically significant in cDM group than that in cerebral reperfusion group(P< 0.05 ).
     结果cDM组(慢性糖尿病脑缺血再灌注组)海马区神经元缺失,于2d开始出现,4d达高峰,7d减少,而且cDM组Cas-pase-3和Aβ表达上调,均在2d开始出现,4d达高峰,7d减少,在各时间点分别明显高于缺血再灌注组,差异具有统计学意义(P<0.05)。
短句来源
     In Hippocampus CA1 regions the number of neurons was not reduced in groups Normal,VO and BCCO/R, but decreased in GI/R groups (P<0.01), and the number of neurons also had significant differences among the GI/R 7d, 14d and 30d groups (P<0.01).
     尼氏染色观察VO组和BCCO/R组海马CA1区无神经元缺失,而GI/R组锥体神经元减少,不同再灌注时间锥体神经元存活数差别有统计学意义(P<0.01)。
短句来源
     The neuron damage was evidence in the cerebral cortex and hippocampal CA 1 section of the ischemia group,and 12,24,48 and 72h after operation the damage became serious.
     脑缺血组大脑皮层、海马 CA1区神经元缺失明显 ,12 h、2 4 h、4 8h、72 h神经元凋亡逐渐加重。
短句来源
     2) Pathological changes: Death of hippocampus neurons was mainly found in acute phase and obviouly found in CA3, CA4 area on experimental side, but significant neuron defect was not found in dentate gyrus.
     (2)病理学改变:海马神经元死亡主要出现在急性期,以实验侧CA3、CA4区最为明显,齿状回无明显神经元缺失
短句来源
     Results The hippocampal CA 1 section neurons in normothermic ischemic group were significantly deficit as compared with the false operative group in mild hypothermic ischemic group (P< 0.01).
     结果 与假手术组、低温缺血组相比 ,常温缺血组海马CA1区神经元缺失明显 (P <0 .0 1)。
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  neuron loss
These findings indicated that the pattern of neuron loss in the ventral horn is distinct among these diseases depending on size, location and function of the ventral horn cell population.
      
These disease-specific patterns of neuron loss suggest a difference in the process of neuronal degeneration of ventral horn cells among the disease examined.
      
We studied these two aspects of the disease in a group of 50 patients by prospectively assessing several sensory indices and by studying the selectivity of the spinal motor neuron loss.
      
Motor neuron loss in a patient with spinocerebellar ataxia type 6: chance co-occurrence or causally related
      
This can involve changes in neural metabolism or neural shrinkage and neuron loss.
      
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  neuronal loss
In a series of Drosophila mutants with changes in the brain structure, some characters (reduced life span, behavioral changes, and neuronal loss in various brain regions) resemble symptoms observed in human patients with neurodegenerative diseases.
      
It is suggested that both the grey and white matter may undergo a severe destructive process early in the course of the disease, and the possibility is discussed that the white matter involvement is not a result of neuronal loss.
      
The pathological findings were isolated pallidal degeneration with demyelination and moderate neuronal loss, iron pigment accumulation and spheroid bodies in both pallidal nuclei.
      
Anterior horns of the spinal cord showed central chromatolysis of the motor nerve cells and many spheroids without neuronal loss.
      
Disease-specific patterns of neuronal loss in the spinal ventral horn in amyotrophic lateral sclerosis, multiple system atrophy
      
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Animal model of excitotoxic cognitive deficit induced by kainic acid bilaterally injectioninto caudate nucleus was successfully established in rats. Based on this model, chronic ad-ministration of 764-3 (8 mg/(kg. d), ip), an extract from Chinese traditiona1 medicine,markedly improved the learning and memory impairment in the injured caudate nucleus rats-Moreover, attenuation of the neuronal loss in caudate nucleus following excitotoxic damagewas observed after 764-3 treatment. In addition, results indicated...

Animal model of excitotoxic cognitive deficit induced by kainic acid bilaterally injectioninto caudate nucleus was successfully established in rats. Based on this model, chronic ad-ministration of 764-3 (8 mg/(kg. d), ip), an extract from Chinese traditiona1 medicine,markedly improved the learning and memory impairment in the injured caudate nucleus rats-Moreover, attenuation of the neuronal loss in caudate nucleus following excitotoxic damagewas observed after 764-3 treatment. In addition, results indicated that the mechanism of theeffect of 764-3 may be related to the antagonism against the decline of Na+-K+-ATPase andCa2+-Mg2+-ATPase actlvlty in synaptlc membrane resulted from excitotoxicity.

用海人酸损伤大鼠双侧尾核并经多种行为学测定,成功地建立了兴奋毒性损伤所致认知障碍模型;观察到中药单体764-3(8mg/(kg·d),ip)能够明显改善尾核损伤大鼠的学习记忆缺陷,并减轻兴奋毒性所致的尾核神经元缺失。拮抗兴奋毒性所致神经细胞膜Na+-K+-ATP酶和Ca2+-Mg2+-ATP酶活性下降。可能是764-3的部分作用机理。

Abundant deposition of β_amyolid protein, neurofibrillary tangle and cholinergic meurons selective loss in the brain are the main characteristics in the neuropathology of Alzheimer's p disease (AD). This paper briefly reviewed the neuropathological development in recent data concerning AD and explored the probable affecting approach of herbal medicine for this disease. It is suggested that the principles for developing new herbal preparations should be based on TCM theory, the pharmacological achievements in...

Abundant deposition of β_amyolid protein, neurofibrillary tangle and cholinergic meurons selective loss in the brain are the main characteristics in the neuropathology of Alzheimer's p disease (AD). This paper briefly reviewed the neuropathological development in recent data concerning AD and explored the probable affecting approach of herbal medicine for this disease. It is suggested that the principles for developing new herbal preparations should be based on TCM theory, the pharmacological achievements in herbal medicine and the neuropathological development in AD.

阿茨海默病的神经病理学特征主要是β/A4淀粉样蛋白沉积、神经纤维缠结和选择性胆碱能神经元缺失。本文简要地回顾了近年来以上三个方面的研究进展,探讨了中药对阿茨海默病的可能干预途径,提出开发治疗阿茨海默病中药新药的原则应包括:依据传统中医理论、结合已有的中药药理研究成果及阿茨海默病的现代病理研究进展。

Objective:In order to test whether immunohistochemical characterization of neurofilament proteins in biopsy of sural nerves gives reliables and useful diagnostic information using neurofilament monoclonal antibodies. Methods: We studied 4 biopy specimens of sural nerves from patients with adult spinal amyotrophy. The clinical features and electroneurophysiologic findings in 4 patients with adult spinal amyorophy were done in all these patients, specimens were fixed in 4% paraformaldehyde-0.1M PBS, embeded in...

Objective:In order to test whether immunohistochemical characterization of neurofilament proteins in biopsy of sural nerves gives reliables and useful diagnostic information using neurofilament monoclonal antibodies. Methods: We studied 4 biopy specimens of sural nerves from patients with adult spinal amyotrophy. The clinical features and electroneurophysiologic findings in 4 patients with adult spinal amyorophy were done in all these patients, specimens were fixed in 4% paraformaldehyde-0.1M PBS, embeded in wax and analyzed by HE, Luxol fast blue, Gless-Marsland silver impregnation and immunohistochemical staining of NF monoclonal antibodies (ABC method).Results: In the sural nerve biopsy, the most striking abnormality was the presence of axonal degeneration (axons become fine and breakdown),but the myelin sheath and schwann cells were norman. Conclusion: The results indicated that the pathological findings of adult spinal amyotrophy was not only characterized by lower motor neuron involvement with of anterior horn cells but also with the axonal degeneration and NF decrease in axons of the sural nerves.

目的:为了探讨神经丝蛋白在成年型脊肌萎缩症患者活检取材的腓肠神经中的免疫组化特性是否能够提供可信赖和有用的病理诊断信息,以期发现该病在腓肠神经中相应的病理改变特征。方法:对临床与电生理诊断的4例进行性脊肌萎缩症患者行腓肠神经活检,标本经4%多聚甲醛固定,石蜡包埋,切片经HE,坚牢蓝,银浸染和神经丝蛋白单克隆抗体的ABC法免疫组化染色,镜下观察。结果:常规染色与神经特染的镜下主要特点为腓肠神经的轴索变性而髓鞘和许旺氏细胞相对保持完整,免疫组化结果显示轴索中神经丝蛋白明显减少,残留的神经丝呈细丝状或断裂状。结论:成年型进行性脊肌萎缩症的病理特点不仅只表现为脊髓前角神经元的缺失等下运动神经元受损的表现,而且可以累及周围感觉神经,进行性脊肌萎缩症患者的腓肠神经活检病理检查也有相应的特征性改变,即腓肠神经的轴索变性和神经丝蛋白的明显减少。

 
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