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巨细胞病变
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  giant cell lesions
     Expression of macrophage inflammatory protein-1α, a disintegrin-like and metalloproteinase 8 and 12, and CD68 protein in giant cell lesions of jaw and giant cell tumors of long bone
     巨噬细胞炎症蛋白1-α、解整合素样-金属蛋白酶8、12和CD68蛋白在颌骨巨细胞病变及长骨骨巨细胞瘤中的表达
短句来源
     Methods MIP-1α, ADAM8, ADAM12 and CD68 were detected by immunohistochemistry in 24 paraffin-embedded specimens of central giant cell lesions of jaw and giant cell tumors respectively.
     方法采用免疫组织化学SP法检测24例颌骨中心性巨细胞病变,24例长骨骨巨细胞瘤石蜡组织中的MIP-1α、ADAM8、ADAM12和CD68蛋白的表达。
短句来源
     Methods ADAMS and ADAM12 was detected by immunohistochemistry(SP) in 40 paraffin-embedded specimens of central giant cell lesions of jaw, 10 peripheral giant cell lesions, 9 cherubisms, 6 aneurysmal bone cysts.
     方法 采用免疫组织化学的方法检测40例颌骨中心性巨细胞病变,10例颌骨周围性巨细胞病变,9例巨颌症,6例颌骨动脉瘤性骨囊肿石蜡组织中的ADAM8、ADAM12的基因表达。
短句来源
     ADAM12 was positive for some spindle mononuclear stromal cells in central and peripheral giant cell lesions.
     颌骨中心性和周围性巨细胞病变中部分短梭形的细胞ADAM12阳性。
短句来源
     Expression of a disintegrin-like and metalloproteinase protein 8 and 12 in the giant cell lesions of jaw
     解整合素样-金属蛋白酶8和12在颌骨巨细胞病变组织中表达的研究
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  giant cell lesion
     Resection and reconstruction of giant cell lesion of bone in skull base-temporomandibular joint region
     颅底-颞颌关节区骨巨细胞病变的切除及修复
     Methods RANKL and OPG were detected by immunohistochemistry (SP) in 24 paraffin-embedded and 2 frozen specimens of central giant cell lesion of jaw.
     方法采用免疫组织化学的方法检测26例颌骨中心性巨细胞病变中RANKL和OPG蛋白的表达。
短句来源
  “巨细胞病变”译为未确定词的双语例句
     Results ADAM8 and ADAM12 were positive in the cytomembrane and cytoplasm of all multinucleated giant cells and some round mononuclear cells of the lesions;
     结果 颌骨中心性巨细胞病变、颌骨周围性巨细胞肉芽肿、巨颌症、颌骨动脉瘤性骨囊肿中的几乎所有多核巨细胞和部分圆形单核基质细胞均为ADAM8、ADAM12阳性;
短句来源
     Conclusions Multinucleated giant cells probably originated from the fusion of the round mononuclear cells, and ADAM8 and ADAM12 were involved in this process. In addition, ADAM12 might play a role in the maturation of spindle mononuclear stromal cells.
     结论 颌骨巨细胞病变中的多核破骨样细胞可能由圆形单核基质细胞融合而成,而ADAM8、ADAM12参与了此融合过程,同时,ADAM12可能还参与了病变中梭形单核基质细胞的成熟过程。
短句来源
     Results MIP-1α positive signal was located in blood vessels and bone.
     结果MIP-1α在24例颌骨中心性巨细胞病变和24例长骨骨巨细胞瘤中的血管、类骨质和新骨形成区中强表达,而在多核巨细胞和圆形单核细胞中为阴性;
短句来源
     Results RANKL signals were strongly positive in the vascular epithelial cells.
     结果RANKL在颌骨中心性巨细胞病变中的血管、基质中有强烈表达,某些病变中的部分短梭形单核基质细胞和多核巨细胞的胞膜也为RANKL阳性;
短句来源
     OPG was detected in multinucleated giant cells and a fraction of round mononuclear cells.
     结论激活的血管内皮细胞通过调节RANKL的表达来促进颌骨中心性巨细胞病变中的多核破骨样巨细胞的形成;
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  giant cell lesions
Differential diagnosis with other giant cell lesions is discussed.
      
It is a close radiological mimic of giant cell lesions of the mandible.
      
Giant cell reparative granuloma and other giant cell lesions of the bones of the hands and feet
      
Differentiation between tumor and acute infection is often diffi26,27 Central giant cell lesions occur most frequently in children and young adults.
      
Peripheral and central giant cell lesions are relatively coesnon pathologic processes which occur in the jaws and are of uncertain nature.
      
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  giant cell lesion
Histopathologic diagnosis was giant cell lesion and associated aneurysmal bone cyst.
      


Objective To detect the expression of a disintegrin-like and metalloproteinase (ADAM) 8 and 12 gene in the giant cell lesions of jaw and to study their effects on the histogenesis of cells in these lesions. Methods ADAMS and ADAM12 was detected by immunohistochemistry(SP) in 40 paraffin-embedded specimens of central giant cell lesions of jaw, 10 peripheral giant cell lesions, 9 cherubisms, 6 aneurysmal bone cysts. Results ADAM8 and ADAM12 were positive in the cytomembrane and cytoplasm of all multinucleated...

Objective To detect the expression of a disintegrin-like and metalloproteinase (ADAM) 8 and 12 gene in the giant cell lesions of jaw and to study their effects on the histogenesis of cells in these lesions. Methods ADAMS and ADAM12 was detected by immunohistochemistry(SP) in 40 paraffin-embedded specimens of central giant cell lesions of jaw, 10 peripheral giant cell lesions, 9 cherubisms, 6 aneurysmal bone cysts. Results ADAM8 and ADAM12 were positive in the cytomembrane and cytoplasm of all multinucleated giant cells and some round mononuclear cells of the lesions; ADAM12 was positive for some spindle mononuclear stromal cells in central and peripheral giant cell lesions. Conclusions Multinucleated giant cells probably originated from the fusion of the round mononuclear cells, and ADAM8 and ADAM12 were involved in this process. In addition, ADAM12 might play a role in the maturation of spindle mononuclear stromal cells.

目的 检测解整合素样-金属蛋白酶(ADAM)8和12在颌骨巨细胞病变中的表达,探讨它们在颌骨巨细胞病变细胞发生中的作用。方法 采用免疫组织化学的方法检测40例颌骨中心性巨细胞病变,10例颌骨周围性巨细胞病变,9例巨颌症,6例颌骨动脉瘤性骨囊肿石蜡组织中的ADAM8、ADAM12的基因表达。结果 颌骨中心性巨细胞病变、颌骨周围性巨细胞肉芽肿、巨颌症、颌骨动脉瘤性骨囊肿中的几乎所有多核巨细胞和部分圆形单核基质细胞均为ADAM8、ADAM12阳性;颌骨中心性和周围性巨细胞病变中部分短梭形的细胞ADAM12阳性。结论 颌骨巨细胞病变中的多核破骨样细胞可能由圆形单核基质细胞融合而成,而ADAM8、ADAM12参与了此融合过程,同时,ADAM12可能还参与了病变中梭形单核基质细胞的成熟过程。

Objective To investigate the clinicopathologic features of familial cherubism and its differentiation from other giant cell lesions in jaws and the results of surgical treatments with a long-term follow-up Methods Four cases of familial cherubism were reviewed and their clinical and radiographic features, histopathologic appearance, biochemical markers and surgical treatments analysed Results Clinically , cherubism was characterized by bilateral painless swelling of jaws, mandibular deformity was common...

Objective To investigate the clinicopathologic features of familial cherubism and its differentiation from other giant cell lesions in jaws and the results of surgical treatments with a long-term follow-up Methods Four cases of familial cherubism were reviewed and their clinical and radiographic features, histopathologic appearance, biochemical markers and surgical treatments analysed Results Clinically , cherubism was characterized by bilateral painless swelling of jaws, mandibular deformity was common Radiographs showed multilocular radiolucencies with sclerotic thickening border Histopathologically, numerous randomly distributed multinucleated giant cells and vascular spaces within a fibrous connective tissue stroma with or without eosinophilic collagen perivascular cuffing were shown The lesion regressed without treatment in 1 cases Curettage was performed in 3 cases with good results Conclusions Cherubism can be diagnosed according to its typical clinical and radiographical features with a positive family history It might regress without treatment But surgery intervention is suggested to improve physiological function and to solve the psychologic problem of the patients

目的 分析一个巨颌症家系中的 4例巨颌症患者的临床病理学特征、手术方式及随访结果 ,旨在对巨颌症与颌骨其他巨细胞病变的鉴别诊断和手术时机的选择有所帮助。方法 对一个巨颌症家系中的 4例患者的遗传学、临床、影像、血液学、组织学、生物学特征、治疗方式与随访的资料进行分析。结果 巨颌症在临床上表现为双侧颌骨的无痛性肿大 ,常发生在下颌 ;在影像学上表现为边界清楚的多房性透射影 ;在病理学上 ,骨组织被增生的纤维组织代替 ,纤维结缔组织内血管丰富 ,多核巨细胞沿血管排列 ,伴或不伴嗜伊红物质环绕成袖口状 ;1例未进行治疗的患者 ,病变自然终止 ;其余进行手术治疗的 3例患者术后未见复发。结论 巨颌症可根据其典型的临床、影像学特征 ,结合家族史及病理学特征作出诊断。虽然它的生物学特征表明 ,青春期后病变会逐渐消退 ,但病变期进行手术治疗不仅可以改善患者的生理和心理功能 ,且病变不一定复发

Objective To detect the expression of macrophage inflammatory protein-1α(MIP-1α), a disintegrin-like and metalloproteinase (ADAM) 8 and 12 and CD68 protein in giant cell lesions of jaw and giant cell tumors of long bone, and to study their effects on the histogenesis of giant cells in such lesions. Methods MIP-1α, ADAM8, ADAM12 and CD68 were detected by immunohistochemistry in 24 paraffin-embedded specimens of central giant cell lesions of jaw and giant cell tumors respectively. Results MIP-1α positive signal...

Objective To detect the expression of macrophage inflammatory protein-1α(MIP-1α), a disintegrin-like and metalloproteinase (ADAM) 8 and 12 and CD68 protein in giant cell lesions of jaw and giant cell tumors of long bone, and to study their effects on the histogenesis of giant cells in such lesions. Methods MIP-1α, ADAM8, ADAM12 and CD68 were detected by immunohistochemistry in 24 paraffin-embedded specimens of central giant cell lesions of jaw and giant cell tumors respectively. Results MIP-1α positive signal was located in blood vessels and bone. ADAM8, ADAM12 and CD68 positive signals were located in the cell membrane and cytoplasm of all multinucleated giant cells and some round mononuclear cells in the lesions. In addition, some spindle mononuclear stromal cells were positive for ADAM12 in both lesions. Conclusion Multinucleated giant cells probably originate from CD68-postive round mononuclear cells, which are recruited from monocyte-macrophage system by chemokines, such as MIP-1α, followed by cell fusion mediated by ADAM8 and ADAM12.

目的检测巨噬细胞炎症蛋白1-α(MIP-1α)、解整合素样-金属蛋白酶(ADAM)8、12和CD68在颌骨巨细胞病变和长骨骨巨细胞瘤中的表达,探讨其在这两种巨细胞病变中的多核巨细胞发生中的作用。方法采用免疫组织化学SP法检测24例颌骨中心性巨细胞病变,24例长骨骨巨细胞瘤石蜡组织中的MIP-1α、ADAM8、ADAM12和CD68蛋白的表达。结果MIP-1α在24例颌骨中心性巨细胞病变和24例长骨骨巨细胞瘤中的血管、类骨质和新骨形成区中强表达,而在多核巨细胞和圆形单核细胞中为阴性;ADAM8、ADAM12和CD68在颌骨中心性巨细胞肉芽肿和长骨骨巨细胞瘤中几乎所有多核巨细胞和部分圆形单核基质细胞均为阳性。结论颌骨中心性巨细胞病变和长骨骨巨细胞瘤中的多核巨细胞可能由CD68+的圆形单核基质细胞融合而成,这些圆形单核细胞可能是由骨微环境中的趋化因子,募集外周血中循环的单核细胞到病变局部分化而成的具有破骨细胞前体细胞性质的细胞,继而在某些融合蛋白作用下发生融合,成熟为多核破骨样巨细胞。

 
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