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心肌梗死     
相关语句
  myocardial infarction
    Experimental Study on the Isolation, Culture, Induced Differentiation and Therapeutic Effect of Autologous Transplantation into Myocardial Infarction of Rat Bone Marrow Stromal Stem Cells
    大鼠骨髓基质干细胞的分离培养、诱导分化和自体移植治疗心肌梗死的实验研究
短句来源
    Experimental Study on Establishment of Minimal-invasive Chinese Miniswine Model of Myocardial Infarction during Perioperative Period of Thoracotomy and Cardiac Protection of the Endogenous Heme Oxygenase-1
    心肌梗死小型猪围术期模型的构建及内源性血红素氧合酶-1心脏保护作用的实验研究
短句来源
    Establishment of pEGZ-CT-1 retroviral expressing vector lays a foundation for the further preparation of transgenic cells and therapy of myocardial infarction with transplantation.
    pEGZ-CT-1载体的构建,为转基因细胞的制备和移植治疗心肌梗死奠定了基础。
短句来源
    Effect of streptokinase thrombolytic reperfusion on left ventricular function after acute myocardial infarction
    链激酶溶栓治疗急性心肌梗死对左心室功能的改善作用
短句来源
    An autopsy pathological analysis of 63 cases with acute myocardial infarction with or without thrombolysis
    有否静脉溶栓的急性心肌梗死63例尸解病理分析
短句来源
更多       
  acute myocardial infarction
    Effect of streptokinase thrombolytic reperfusion on left ventricular function after acute myocardial infarction
    链激酶溶栓治疗急性心肌梗死对左心室功能的改善作用
短句来源
    An autopsy pathological analysis of 63 cases with acute myocardial infarction with or without thrombolysis
    有否静脉溶栓的急性心肌梗死63例尸解病理分析
短句来源
    Experimental study of acute myocardial infarction model in dogs
    犬急性心肌梗死模型制备的方法学研究
短句来源
    Establishment of Swine Model of Acute Myocardial Infarction by Coronary Occlusion
    冠状动脉堵闭法建立猪心肌梗死模型
短句来源
    Establishing standard acute myocardial infarction animalmodel
    建立一种标准化急性心肌梗死动物模型
短句来源
更多       
  infarction
    Experimental Study on the Isolation, Culture, Induced Differentiation and Therapeutic Effect of Autologous Transplantation into Myocardial Infarction of Rat Bone Marrow Stromal Stem Cells
    大鼠骨髓基质干细胞的分离培养、诱导分化和自体移植治疗心肌梗死的实验研究
短句来源
    Experimental Study on Establishment of Minimal-invasive Chinese Miniswine Model of Myocardial Infarction during Perioperative Period of Thoracotomy and Cardiac Protection of the Endogenous Heme Oxygenase-1
    心肌梗死小型猪围术期模型的构建及内源性血红素氧合酶-1心脏保护作用的实验研究
短句来源
    Establishment of pEGZ-CT-1 retroviral expressing vector lays a foundation for the further preparation of transgenic cells and therapy of myocardial infarction with transplantation.
    pEGZ-CT-1载体的构建,为转基因细胞的制备和移植治疗心肌梗死奠定了基础。
短句来源
    Effect of streptokinase thrombolytic reperfusion on left ventricular function after acute myocardial infarction
    链激酶溶栓治疗急性心肌梗死对左心室功能的改善作用
短句来源
    An autopsy pathological analysis of 63 cases with acute myocardial infarction with or without thrombolysis
    有否静脉溶栓的急性心肌梗死63例尸解病理分析
短句来源
更多       
  myocardial infarct
    The myocardial infarct size occupied ( 57.7 ± 2.0 ) % of the ischemic myocardium.
    心肌梗死范围占缺血心肌的 (5 7.7± 2 .0 ) %。
短句来源
    The effects of myocardial ischemia reperfusion(MIR) and renal ischemic preconditioning (RIP) on hemodynamics, epicardial electrography, myocardial infarct size, cardiomyocytic apoptosis and gene proteins involving apoptosis (Fas, Bcl 2 and Bax) were observed in aneasthetized rabbit myocardium.
    在麻醉家兔心肌缺血 /再灌注 (myocardialischemia/reperfusion ,MIR)模型上 ,观察MIR和肾脏缺血预处理 (renalischemicpreconditioning ,RIP)对血流动力学、心外膜电图、心肌梗死范围、心肌细胞凋亡和凋亡相关调控基因蛋白(Fas、Bcl 2和Bax)的影响。
短句来源
    Results AST, LDH, CK, myocardial infarct size and atrioventricular node expression of BCL-2 protein in ischemic-reperfusion group were all higher than that of normal control group.
    (2 )假手术组AST、LDH、CK未升高 ,TTC法心肌梗死范围不明显 ,免疫组化检测房室结BCL 2基因蛋白未见表达。
短句来源
    RESULTS: Myocardial infarct size was significantly decreased, CK and LDH were decreased in the serum, NO 2 -/NO 3 - was increased in myocardial tissue in SND group as well as in IPC/SWOP group (there was no difference between the two groups). The expression of p38 MAPK and PKC were upregulated in myocardial tissue in SND and IPC/SWOP groups.
    结果 :延迟缺血预处理组及四逆汤预处理组心肌梗死面积、血清CK、LDH的值明显少于I/R组 ,NO2 -/NO3 -含量显著高于I/R组 ,p38MAPK和PKC发生转位且蛋白表达明显高于I/R组。
短句来源
    The expression of neutrophil CD11b/CD18 was measured by flow cytometry. Myocardial infarct extent was calculated according to the formula: percents of infarct area=(infarct area/risk area)×100%.
    ③分别于基础(缺血和再灌注之前)、缺血1h、再灌注2h各时间点,采用流式细胞术测定中性粒细胞CD11b/CD18表达,按梗死面积%=梗死区面积/危险区面积×100%公式计算心肌梗死范围。
短句来源
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  myocardial infarction
Thus, the IC-MCE method is of great value to coronary artery disease (CAD) patients undergoing PCI, especially for those with acute myocardial infarction (AMI).
      
Nine of the 15 patients exhibited acute myocardial infarction (AMI).
      
The effect of dinitrosyl iron complex (DNIC) with L-cysteine on the hemodynamic indices and the size of myocardial infarction, which was induced by 40-min regional ischemia and subsequent 60-min reperfusion, have been studied in rats in vivo.
      
SkQ1 strongly reduces the damaged area in myocardial infarction or stroke and prevents the death of animals from kidney infarction.
      
Association of the CCR2 Chemokine Receptor Gene Polymorphism with Myocardial Infarction
      
更多          
  acute myocardial infarction
Thus, the IC-MCE method is of great value to coronary artery disease (CAD) patients undergoing PCI, especially for those with acute myocardial infarction (AMI).
      
Nine of the 15 patients exhibited acute myocardial infarction (AMI).
      
Synchronization parameters of 0.1-Hz rhythms isolated from the heart rate and the oscillations of the blood volume in microcirculatory vessels were studied in 12 healthy subjects and 32 patients with acute myocardial infarction.
      
In patients with acute myocardial infarction, synchronization of 0.1-Hz rhythms was considerably poorer.
      
A total of 135 consecutive patients with acute myocardial infarction were enrolled in the study.
      
更多          
  infarction
Thus, the IC-MCE method is of great value to coronary artery disease (CAD) patients undergoing PCI, especially for those with acute myocardial infarction (AMI).
      
Nine of the 15 patients exhibited acute myocardial infarction (AMI).
      
The effect of dinitrosyl iron complex (DNIC) with L-cysteine on the hemodynamic indices and the size of myocardial infarction, which was induced by 40-min regional ischemia and subsequent 60-min reperfusion, have been studied in rats in vivo.
      
The relationship between hydroxyl radical (OH·) generation in the zone of ischemia/reperfusion and the size of infarction formed was investigated in 18-22-week-old anaesthetized male SHRSP and Wistar rats using a myocardial microdialysis technique.
      
SkQ1 strongly reduces the damaged area in myocardial infarction or stroke and prevents the death of animals from kidney infarction.
      
更多          
  myocardial infarct
Different protein expression of myocardium from Chinese mini-swine model of myocardial infarct
      
The Trx 1 mouse hearts displayed significantly improved post-ischemic ventricular recovery and reduced myocardial infarct size and apoptosis compared to the corresponding wild-type mouse hearts.
      
In recent years, early reperfusion of the myocardial infarct has become the mainstay of optimal therapeutic management sinct it limits ventricular injury and infarct expansion and improves patient survival [14].
      
The effect of hyaluronidase (HY) on myocardial infarct size after experimental coronary artery occlusion was studied in two groups of dogs.
      
The effect of Pindolol on myocardial infarct size was studied in 10 open chest dogs.
      
更多          
  其他


An observation on focal myocardial fibrotic lesions was made in hypertensive Wistar rats with one clipped kidney in two. In the sixteen rats 81 lesions were found. That companied with small vessles was 38 (46.9%), fibrotic scars 28(34.6%), and granular tissues 15(18.5%). The residual marks of myocardial necrosis remained in most lesions. The inflammatory infiltrations occured in a few cases.It is believed that the myocardial focal fibrosis in sustained arterial hypertension could be induced by the changes in...

An observation on focal myocardial fibrotic lesions was made in hypertensive Wistar rats with one clipped kidney in two. In the sixteen rats 81 lesions were found. That companied with small vessles was 38 (46.9%), fibrotic scars 28(34.6%), and granular tissues 15(18.5%). The residual marks of myocardial necrosis remained in most lesions. The inflammatory infiltrations occured in a few cases.It is believed that the myocardial focal fibrosis in sustained arterial hypertension could be induced by the changes in small arteries and arterioles, although mainly by myocardial infarction. In addition, the mesenchymal cells, eosinophilic leukocytes played certain roles in the development of those lesions under some conditions.

观察了两肾一夹型高血压Wistar大鼠心肌局灶性纤维化病变的病理形态。在16只高血压大鼠心脏整张切片中,发现纤维化病灶81个,其中与小血管伴行者38个(46.9%),纤维组织为主者28个(34.6%),肉芽组织为主者15个(18.5%)。大部分病灶中遗留着心肌细胞坏死的痕迹,炎细胞浸润见于少数病灶。认为持续性全身性动脉血压升高心肌局灶性纤维化可由小血管病变引发,但多由心肌梗死引起,心肌间质细胞及嗜酸性白细胞和嗜中性白细胞在某些情况下也可能起着一定的作用。

The experimental myocardial damages were induced in Wistar rats by noradrenaline infusion (20ug/kg body wt/minute)for a period of 90 minutes daily for three consecutive days. The animals weresacrificed few minutes (acute phase), 7 days(subacute phase),and 30~90 days (chronic phase)afterinfusion. Specimens of myocardium were examined by histopathological, immunohistochemical and ultrastructural methods. Multiple focal necrosis, deficient of myoglobin in myocytes, myofibrillar fragmentation and dissolution were...

The experimental myocardial damages were induced in Wistar rats by noradrenaline infusion (20ug/kg body wt/minute)for a period of 90 minutes daily for three consecutive days. The animals weresacrificed few minutes (acute phase), 7 days(subacute phase),and 30~90 days (chronic phase)afterinfusion. Specimens of myocardium were examined by histopathological, immunohistochemical and ultrastructural methods. Multiple focal necrosis, deficient of myoglobin in myocytes, myofibrillar fragmentation and dissolution were observed in the acute phase. The mdrphofogical feature simulated themyocardial infarctinn without coronary occlusion. Focal myocardial atrophy, scar f0rmation of myocardium, proliferation of fibroblasts and infiltraiton of the neo-colleagenous fibrils in necrotic myocytes were observed in the subacute and chronic phase. This feacture simulated the early cardiomyopathy.

大鼠尾静脉滴去甲肾上腺素(20μg/kg/min)后,分别于即刻、7天、30~90天杀死取材,用病理组织学、肌红蛋白免疫组化染色及透射电镜观察心肌损伤。结果:早期可见心肌多灶性坏死、肌红蛋白缺染、肌原纤维断裂溶解,似冠状动脉无阻塞性心肌梗死;中后期可见心肌灶性萎缩及疤痕形成、成纤维细胞增生活跃、新生胶元原纤维伸入坏死心肌内,类似早期心肌病图象。

Fourty wistar rats were used for experimental study of myocardial infarction (MI )induced by ligation of the left coronary artery. The results showed that the AF and HBFP stalng as well as Ao fluorescence were sensitive in demonstrating the early MI changes occuced 1 ~ 2 hour after coronary arteryligation. The TTC stamg method was reliable for demonstrating the MI margin. Both HBFP and AOfluorescence were under no influeence of the post-mortem autolysis, but the AF reaction were undeithe influence of the early...

Fourty wistar rats were used for experimental study of myocardial infarction (MI )induced by ligation of the left coronary artery. The results showed that the AF and HBFP stalng as well as Ao fluorescence were sensitive in demonstrating the early MI changes occuced 1 ~ 2 hour after coronary arteryligation. The TTC stamg method was reliable for demonstrating the MI margin. Both HBFP and AOfluorescence were under no influeence of the post-mortem autolysis, but the AF reaction were undeithe influence of the early post-mortem autolysis. Four hours were required for complete developemenof MI. The early stage of MI demonstrated by SEM was conslstant with that bv TEM. Both resuhwere influenced by the post-mortem autolysis.

本文报告40只大鼠结扎左冠状动脉所致实验性心肌梗死早期(缺血1~8h)的病理形态学变化。结果发现苏木素-碱性复红-苦味酸(HBFP)和吖啶橙荧光染色诊断早期心肌缺血较敏感,氯化-2,3,5-三苯基四氮唑(TTc)大体染色法显示缺血境界鲜明可靠。此三种方法不受室温(15~20℃)死后自溶12~48h的影响。酸性复红染色(AF)对早期缺血组织较敏感,但受死后自溶影响,妨碍认定缺血区。扫描电镜(SEM)观察心肌缺血改变基本与透射电镜(TEM)相同,与早期自溶改变难以区别。

 
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