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-异丙肾上腺素
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     the content of Mg2+in myocardium was decreased significantly (P<0.05) .
     2.皮下注射异丙肾上腺素(IS。)
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     2. Isoprenaline group ( Iso, toinduce cell apoptosis);
     2、异丙肾上腺素(Iso);
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     ②the hypoxic method of mice to resist isoprenaline;
     ②对抗异丙肾上腺素法;
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     The reaction between isoproterenol and MnO_4~- was analyzed.
     报道了异丙肾上腺素与MnO4-的反应.
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     Effect of L-Arginine on cardiac damage induced by isoproterenol
     L-精氨酸对异丙肾上腺素致害心肌的影响
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  (-) isoproterenol
(-)-Isoproterenol-stimulated adenylate cyclase activity was augmented only in membranes from T3-treated cells and was reduced by 69% in membranes from (-)-norepinephrine treated cells.
      
As compared to normal controls, the response of cyclic AMP production by leukocytes to stimulation by 1-isoproterenol or PGE1 was significantly decreased.
      
When rats were dosed with 14 μg/kg 3H-isoproterenol, 3H-radioactivity was measurable in the liver until 48 h.
      
Methoxamine produces an approximately parallel shift in the log concentration-effect curves for l-isoproterenol, l-norepinephrine, and l-epinephrine, the chronotropic and inotropic curves being shifted about equally.
      
The evidence obtained is consistent with the hypothesis that the methoxamine-isoproterenol antagonism is a simple competitive one with a pA2 of 5.2.
      
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The present paper describes the effect of extractum of root Pueraria pseudo-hirsuta (Erpph) on the β-adrenergic receptor of various tissues of animals. The results showed that this extract had potent β-adrenergic receptor blocking effects. There is evidence that its blocking action on β_1-receptors is stronger than on β_2-receptors.It was found that at the dosage of 0.5~5 mg/ml or 750 mg/kg Erpph could antagonize the heart excitation induced by isoproterenol (0.17~1.7μg/ml or 10 μg/kg) in vivo and in vitro respectively....

The present paper describes the effect of extractum of root Pueraria pseudo-hirsuta (Erpph) on the β-adrenergic receptor of various tissues of animals. The results showed that this extract had potent β-adrenergic receptor blocking effects. There is evidence that its blocking action on β_1-receptors is stronger than on β_2-receptors.It was found that at the dosage of 0.5~5 mg/ml or 750 mg/kg Erpph could antagonize the heart excitation induced by isoproterenol (0.17~1.7μg/ml or 10 μg/kg) in vivo and in vitro respectively. Erpph was also found to block the depressor reaction of the vessels by injection of isoproterenol (10/μg/kg) and adrenalin (10μg/kg) but it has no effect on the pressor reaction to nor-adrenalin (10 μg/kg). On the other hand, Erpph (750 mg/kg)was shown to decrease the resting heart rate and blood pressure (average 43.7% or average 40±6 mmHg, respectively). These facts form a theoretical bssis for using Erpph in angina pectoris, arrhythmia and hypertension.By comparing the effect of this extractum (750 mg/kg) and practolol (0.1 mg/kg) it was also found that they had almost the same β-adrenergic blocking effect, but the former was stronger to depress the blood pressure and the latter was stronger to depress the heart rateFrom these studies, it is concluded that Erpph is a potent and safe β-blocker.

本文报告了葛根浸膏具有较为广泛而显著的β-受体阻滞效应。且对β_1-受体的作用强于β_2-受体。 相当于0.5~5mg/ml或750mg/kg的葛根能分别对抗异丙肾上腺素0.17~1.7μg/ml或10μg/kg诱发的离体或在体心脏的兴奋作用。此外,相当于750mg/kg的葛根除能阻滞10μg/kg异丙肾上腺素及肾上腺素舒血管(β-受体效应)所致的降压作用外,尚能降低正常心率(平均下降43.7%)及血压(平均下降40±6mmHg)的作用,这些结果为葛根用来缓解心绞痛,治疗快速型心律失常及高血压等提供了根据。 将葛根(750mg/kg)与心得宁(0.1mg/kg)抗异丙肾上腺素(10μg/kg)所致的β-受体效应加以比较,表明两者作用大体相似,减慢心率的作用心得宁似乎稍强于葛根。降压作用葛根略优于心得宁,且葛根无明显的抑制心脏的作用。 根据上述事实,可以认为葛根是一种有效的β-受体阻滞剂。

One case of rheumatic multiple valvular disease and 2 cases of myoeardiopathy, suffering from many attacks of heart failure, underwent homogenous heart transplantation since 1978 in our hospital. Morphine anesthesia was applied in all 3 cases. In induction, morphine, diazepam and pancuronium were used in 2 eases and ketamin and pancuronium in 1 case. All were intubated without difficulty. Anesthssia was maintained by gamma hydrobutyrie acid, and morphine and diazepam were injected intravenously when required....

One case of rheumatic multiple valvular disease and 2 cases of myoeardiopathy, suffering from many attacks of heart failure, underwent homogenous heart transplantation since 1978 in our hospital. Morphine anesthesia was applied in all 3 cases. In induction, morphine, diazepam and pancuronium were used in 2 eases and ketamin and pancuronium in 1 case. All were intubated without difficulty. Anesthssia was maintained by gamma hydrobutyrie acid, and morphine and diazepam were injected intravenously when required. These anesthetic agents did not significantly effect blood pressure or heart rate. The authors are of the opinion that (1) morphine, scopolamine and diazepam used as premedicant offered a smooth induction; (2) scopolamine did not cause tachycardia in denervated donor heart but improved microcirculation; (3) when the heart was operating, intravenous dripping of isoprenaline exerted a positive inotropic effect on the heart; (4) hypotension could sometimes be rectified by calcium chloride, especially when donor heart was operating; (5) the data of hemodynamic monitoring must be appropriately interpreted in accordance with clinical findings. In the first case, although large doses of vasopressor drugs raised CVP to 25 cm, normal blood pressure could only be maintained through correction of hypovolemia by transfusion; (6)supplement of potassium during operation was essential.

本文总结了3例心脏移植麻醉的初步体会,认为吗啡麻醉适用于此类手术。重点讨论了麻醉的选择及血液动力学的监护。移植心脏复跳后常规应用异丙肾上腺素具有显著增强心肌功能的作用,氯化钙对心脏移植后血压下降的治疗有较好的效果,强调勿盲目加大升压药用量。在电解质与酸碱平衡方面,强调钾的补充;若无休克发生,不主张大量应用碱性药物。

Heart rhythm disturbances were precipitated in about 80.9% of normal rabbits 0~10 minutes after the intravenous injection of pyquiton at the dosage of 35~45 mg/kg, and sustained for 1~21 minutes. Prior to cardiac arrhythmia, bradycardia was observed in 84.1% of the rabbits. Among the tested antiarrhythmic agents, phenoxybenzamine (dibenzyline) at the dosage of 3 mg/kg exhibited the most dramatic antiarrhythmic action on pyquiton-induced arrhythmia; chlorpromazine hydrochloride (5mg/kg), isoptinum (0.3~0.5 mg/kg),...

Heart rhythm disturbances were precipitated in about 80.9% of normal rabbits 0~10 minutes after the intravenous injection of pyquiton at the dosage of 35~45 mg/kg, and sustained for 1~21 minutes. Prior to cardiac arrhythmia, bradycardia was observed in 84.1% of the rabbits. Among the tested antiarrhythmic agents, phenoxybenzamine (dibenzyline) at the dosage of 3 mg/kg exhibited the most dramatic antiarrhythmic action on pyquiton-induced arrhythmia; chlorpromazine hydrochloride (5mg/kg), isoptinum (0.3~0.5 mg/kg), promethazine hydrochloridum(3~5 mg/kg) and phenytoine sodium (50 mg/kg) could also abolish arrhythmia; lidocaine hydrochloride, quinidine, isopreanline sulfate, and atropine sulfate etc. showed partial and/ or incomplete antiarrhymic action; while proctolol and shangrolin were ineffective. Preadministration of phenytoine sodium (50 mg/kg), chlorpromazine hydrochloride (5 mg/kg) etc. were demonstrated to be effective in preventing the pyquitoninduced arrhythmia.

家兔静脉注射吡喹酮35~45mg/kg后10分钟内,80.9%(17/21)的兔发生心律失常,持续1~21分钟后恢复窦性心律。心律失常出现前心率较正常时缓慢的占84.1%(58/69)。抗心律失常药中,以酚苄明3mg/kg对抗吡喹酮所致心律失常转为窦性心律的作用最好,其次为氯丙嗪(5mg/kg)、异搏停(0.3~0.5mg/kg)、异丙嗪(3~5mg/kg)及苯妥因钠(50mg/kg),利多卡因、奎尼丁、异丙肾上腺素与阿托品等有部分或作用不完全的抗心律失常的效果。心得宁与常咯啉则无效。先给兔注射苯妥因钠(50mg/kg)或氯丙嗪(5mg/kg)等,有较好的防止吡喹酮所致心律失常的作用。

 
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