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拮抗药物
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  “拮抗药物”译为未确定词的双语例句
     To screen NF- κB antagonistic drugs and research singal transduction pathway related to NF- κB, two vectors, p4κB- d2EGFP containing destabilized enhanced green fluorescent protein(d2EGFP) reporter gene and p4κB- EGFP with EGFP gene, were constructed on the base of 4 copies of NF- κB cis - element κB as enhancer, SV40 as basic promoter and neor gene as selective gene.
     建立核因子 κB (NF κB)反应性不稳定增强型绿色荧光蛋白 (d2EGFP)报告系统 ,作为筛选NF κB拮抗药物及研究其相关信号转导途径的工具 . 分别以EGFP与d2EGFP为报告基因、neor 为筛选基因 ,构建成 4×κB基序为增强子、SV40为基本启动子的报告基因载体 p4κB EGFP和 p4κB d2EGFP .
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     Application of Capillary Zone Electrophoresis in Analysis of Antagonists as p-Aminobenzoic Acid, p-Hydroxybenzoic Acid and Sulfanilamide
     毛细管电泳在拮抗药物对氨基苯甲酸、对羟基苯甲酸和磺胺分析测定中的应用
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     Intravitreous injection of VEGF aptamer stabilized or improved vision in 87.5% of patients with subfoveal CNV 3mo after treatment.
     玻璃体腔注射VEGF的拮抗药物,也能稳定患者的视力3mo。
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     The levels of IL-4 and IL-5 in supernatant by mizolastine was lower than those by cetirizine and loratadine,while the levels of TNF-α had no significant difference by all antihistamines.
     培养上清液中的IL-4含量显著低于氯雷他定和西替利嗪组,IL-5的含量显著低于西替利嗪、氯雷他定组(P<0.05),TNF-α的含量与其他拮抗药物组均无统计学差异(P>0.05)。
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     Objective: To explore the effect of mast cell before or after stimulation of antigen (αIgE HRP).
     目的 :研究用Ⅰ型变态反应的有效拮抗药物 :色甘酸钠作对照 ,研究藿香正气口服液在抗原 (αIgE HRP)再次刺激前、后 ,对肥大细胞 (MC)脱颗粒的作用。
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     Drug Addiction
     药物成瘾性
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     Drug Patches
     药物贴片
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     Advance in search for platelet glucoprotein VI and its antagonist
     血小板糖蛋白Ⅵ及其拮抗药物研究进展
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     Angiotensin Ⅱ inhibitors-related adverse effects and their prevention and treatment
     血管紧张素Ⅱ拮抗药物的不良反应及其防治
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     Antagonistic substances which were strong antagonistic to various plant pathology fungi could be produced by Streptomyces spp.
     拮抗链霉菌(Steiptomycesspp.)
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A simple, rapid and sensitive method determining the binding sites of antagonists on calmodulin was introduced.Studies on the intrinsic fluorescence of calmodulin have shown that Tb3+ can efficiently bind to the Ca2+ -binding sites on calmodulin and then induce conformational changes similar to those of Ca2+. Because each of domains III and IV on calmodulin has one tyrosine, the binding of Tb3+ to calmodulin was followed by obvious increase of Tb3+ fluo-rescence at 490 nm and 545nm, which was caused by exciting...

A simple, rapid and sensitive method determining the binding sites of antagonists on calmodulin was introduced.Studies on the intrinsic fluorescence of calmodulin have shown that Tb3+ can efficiently bind to the Ca2+ -binding sites on calmodulin and then induce conformational changes similar to those of Ca2+. Because each of domains III and IV on calmodulin has one tyrosine, the binding of Tb3+ to calmodulin was followed by obvious increase of Tb3+ fluo-rescence at 490 nm and 545nm, which was caused by exciting the calmodulin tyrosine at 280 nm with energy transfer from tyrosine to Tb3+ . By studing the effect of calmodulin antagonists on this fluorescence, it was found that the binding sites of TEP were located on the C-terminal part of calmodulin which contains the domains III and IV; whereas the binding sites of jujuboside A were located on the N-terminal part which contains the domains I and II.

本文报导以Tb~(3+)作为荧光探针,研究钙调蛋白(CaM)与其拮抗药物分子间相互作用的机制.所用方法简便、快速、灵敏.CaM的内源荧光研究表明,Tb~(3+)类似于Ca~(2+),也能诱导CaM分子构象发生改化,由于CaM分子中Ca~(2+)的第Ⅲ、Ⅳ结合位点上各有一个Tyr线基,如(?)280nm激发,则发生从Tyr向Tb~(3+)的能量转移,从而导致Tb~(3+)在490和545nm处的特征荧光发射大大加强.本文检测了药物分子与Tb~(3+)-CaM结合对该荧光发射的影响.实验表明,TFP与CaM的高亲和位点处于CaM分子C-末端部位,即含第Ⅲ、Ⅳ结构域的半分子上:丙拮抗药物酸枣仁皂甙A则优先结合在含第Ⅰ、Ⅱ的结构域的另一半分子(?).

By using the Y-maze method,the effect of -selenocarrageenan(μ-SeC) on spatial discrimination learning and memory retention in mice were studied.The results were as follows:①-SeC(po 180 or 108 mg/kg, qd×14d) given before testing raised discrimination learning ability in mice;②-SeC(po 180, 108 or 64.8mg/kg,qd×14d) improved the discrimination learning impairment induced by scopolamine(ip 4 mg/kg);③-SeC(po 180 mg/kg, qd×14d) improved the memory retention impairment indnced by scopolamine. It was shown that SeC...

By using the Y-maze method,the effect of -selenocarrageenan(μ-SeC) on spatial discrimination learning and memory retention in mice were studied.The results were as follows:①-SeC(po 180 or 108 mg/kg, qd×14d) given before testing raised discrimination learning ability in mice;②-SeC(po 180, 108 or 64.8mg/kg,qd×14d) improved the discrimination learning impairment induced by scopolamine(ip 4 mg/kg);③-SeC(po 180 mg/kg, qd×14d) improved the memory retention impairment indnced by scopolamine. It was shown that SeC exerted the improved effects on the experimental cognitive impairment in mice.

用Y型迷宫箱测试小鼠空间分辨能力,结果:po -硒化卡拉胶( -selenocarragee-nan, -SeC)180mg/kg及108mg/kg(qd×14d)能增强正常小鼠的学习能力,提高学习成绩;po -SeC180、108及64mg/kg(qd×14)对东莨菪碱(ip4mg/kg)所致小鼠学习障碍均有拮抗作用;po -SeC180mg/kg(qd×14)对东莨菪碱(ip5mg/kg)所致小鼠记忆保持障碍有改善作用。以上表明 -SeC对脑的学习功能有促进作用,并能拮抗药物引起的学习与记忆功能的损害,改善大脑功能.

The dose of Xylazole for immobilization of Sika deer(Cervus nippon)is more than that ofScoline and Miannaining,and immobilization operation is also more complex. The dose of Scoline for immobilization of Sika deer is less, but its effective dose is near the lethal dose.Xylazole and Scoline have stronger side effect.Miannaining is more effective, safe and no side effect for immobilization of Sika deer than other two chemicals.

本文通过用静松灵、司可林、眠乃宁三种保定药物保定梅花鹿对比实验,得出:①静松灵一次性给药剂量较大,生产中操作不方便。②司可林一次给药剂量虽小,但有效剂量与致死剂量十分接近,剂量误差易导致鹿死亡。③静松灵和司可林均无有效拮抗药物,且后遗性影响较大。④眠乃宁注射剂量适中,剂量误差不会导致鹿死亡,无后遗性影响。拮抗药物苏醒灵拮抗效果优良,使眠乃宁的应用更安全。

 
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