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凋亡
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  apoptosis
    Relationship between the activity of potassium channels and the proliferation and apoptosis of cultured rat pulmonary artery smooth muscle cells
    钾通道活性与培养大鼠肺动脉平滑肌细胞增殖和凋亡关系的研究
短句来源
    Experimental Study of The Effect of Promoting Apoptosis of Vascular Smooth Muscle Cell with Human Bcl-2 mRNA-cleaving Ribozyme
    切割人bcl-2mRNA核酶对血管平滑肌细胞促凋亡作用的实验研究
短句来源
    Effects of NHE-1 Hammerhead Ribozyme on Apoptosis and Expression of Apoptosis-associated Genes in Pulmonary Artery Smooth Muscle Cells of Rats in Vitro
    Na~+/H~+交换器-1核酶对缺氧大鼠肺动脉平滑肌细胞凋亡凋亡相关基因表达的影响
短句来源
    Different Regulation of Vascular Smooth Muscle Cell Migration, Apoptosis and Proliferation by β-Adrenergic Receptor Subtype Stimulation and Possible Signal Pathway
    β-肾上腺素能受体不同亚型对于血管平滑肌细胞迁移、凋亡及增殖的影响及其信号转导机制
短句来源
    Mitochondrial Apoptotic Pathway Participates in Apoptosis Induced by Bufalin in Human Leukemia HL-60 Cells
    线粒体凋亡途径参与蟾蜍灵诱导的人类白血病细胞HL-60凋亡
短句来源
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  apoptotic
    Mitochondrial Apoptotic Pathway Participates in Apoptosis Induced by Bufalin in Human Leukemia HL-60 Cells
    线粒体凋亡途径参与蟾蜍灵诱导的人类白血病细胞HL-60凋亡
短句来源
    An Expermental Study of Apoptosis of Myocardial Cells Induced by the Ischemia-reperfusion and the Different Influences of the NO, Ischemia Pretreatment, Heat Shock and the Express of the Apoptotic Relevant Genes
    缺血再灌注诱导心肌细胞凋亡及NO、缺血预处理、热休克对其不同影响与凋亡相关基因表达的实验研究
短句来源
    DETECTION OF DNA STRAND BREAKS IN DRUG-INDUCED APOPTOTIC HL-60 AND U937 CELLS BY IN SITU TERMINAL DEOXYNUCLEOTIDYL TRANSFERASE
    DNA断端标记法分析HL-60和U937白血病细胞株凋亡
短句来源
    Results Overexpression of Mfn2 gene promoted the apoptosis of rVSMCs,remarkably decreased the expression of mitochondrial Bcl-2 protein,decreased the expression of Bax and activated Caspases-9 after the rVSMCs were transfected with Mfn2.Conclusion Overexpression of rMfn2 gene triggers apoptosis of rVSMCs mediated by activation of the mitochondrial apoptotic pathway.
    结果高表达Mfn2基因促进rVSMCs凋亡,使rVSMCs线粒体Bcl-2蛋白表达减少、Bax增多,胞质Caspases-9被激活。 结论Mfn2基因促进rVSMCs的凋亡,其机制与活化线粒体凋亡途径有关。
短句来源
    ③ Number of apoptotic myocardial cells: There was no significant apoptosis found after the reperfusion in the sham-operation group (< 5%), and the apoptotic rate in the ischemic postconditioning group was remarkably lower than that in the ischemic reperfusion group [which were respectively (12.5±2.9)%,(21.3±3.8)%,P < 0.01].
    ③心肌凋亡细胞计数:再灌注结束后假手术组未见明显细胞凋亡(<5%),缺血后处理组心肌细胞凋亡率明显低于缺血再灌注组[分别为(12.5±2.9)%,(21.3±3.8)%,P<0.01]。
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  “凋亡”译为未确定词的双语例句
    The Role of Antiapoptotic Bcl-x_L during Megakaryocyte Differentiation and in Essential Thrombocythemia
    抗凋亡蛋白Bcl-x_L在巨核细胞分化中的作用及其在原发性血小板增多症巨核细胞分化中表达的研究
短句来源
    ANALYSIS OF APOPTOSIS-ANTAGONIZING GENEBCL-2 AT TRANSCRIPTIONAL LEVELS IN ACUTELEUKEMIA CELLS FROM BONE MARROW BlOPSIESAND CELL LINES
    抗凋亡基因bcl-2在急性白血病骨髓与细胞株中的转录水平表达
短句来源
    EXPRESSION OF APOPTOSIS- ANTAGONIZING PROTEIN bcl- 2 AND ITS mRNA IN ACUTE LEUKEMIC CELLS
    急性白血病细胞中抗凋亡蛋白bcl-2及其mRNA的表达
短句来源
    Apoptosis-antagonizing protein Bcl-2 expression,distribution in non-Hodgkin's lymphoma and its clinical significance
    抗凋亡蛋白Bcl-2在非霍奇金淋巴瘤中的表达、分布及其临床意义
短句来源
    The expression of Bcl-2,Bax and Caspase-9 was detected by using Western blot.
    Western blot等检测凋亡相关因子Bcl-2、Bax和Caspase-9的含量。
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  apoptosis
Prodigiosin also could induce apoptosis of pancreatic cancer cells at low concentration and results in the fragmentation pattern of DNA.
      
All these results demonstrate that prodigiosin can obviously inhibit the proliferation of pancreatic cancer cells H8898 by arresting the cell cycle and inducing apoptosis.
      
Detection of the apoptosis of Jurkat cell using an electrorotation chip
      
The apoptosis of cells is one of the fields that attract increasing attention in biology today.
      
Usually, the cells are treated with chemicals when detecting apoptosis.
      
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  apoptotic
The glioma cells showed typical apoptotic signs 90 hours after the transient transfection of GFAP-Baxα.
      
The number of microgranulomas, lipogranulomas and apoptotic bodies increased following severity of steatosis, lobular inflammation and fibrosis.
      
Low concentrations of phospholipase A2 and lipoxygenase inhibitors were shown to stimulate cell division, while higher concentrations inhibited it by blocking G1-S transition and inducing apoptotic cellular death.
      
TUNEL assay was used to detect apoptotic cardiomyocytes.
      
The apoptotic changes in cardiomyocytes proved to prevail in early lesion foci (4-18 h), while cardiomyocytes at later stages were prone to necrosis; cardiomyocytes can exhibit signs of apoptosis and necrosis at the same time.
      
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e apphed more sensitive

采用高敏感多重PAP免疫细胞化学技术,研究抗细胞凋亡基因bcl-2在淋巴结何杰金氏病中的表达与分布。结果表明,21例中有7例(33%)可检出bcl-2蛋白阳性的Reed-Sternberg(R-S)细胞;其中2例(混合细胞型)尚含大量反应性淋巴滤泡,其生发中心也呈阳性反心。相反4例作为阳性对照的炎性淋巴滤泡增生中,bcl-2蛋白主要分布于富含长寿命(long-lived)B细胞的套区(mantlezone),而生发中心则均为阴性。结果提示,bcl-2基因高表达介导的细胞凋亡障碍可能与R-S细胞发生有关。

ntigen distribution and DNA ploidy of megakary-ocytes were investigated in 4 patients with acutemegakaryoblastic leukemia. The percentages of cells as well as HLA-DR ̄+, HLA-DR ̄+ cells in mononuclear cellsvaried, suggesting that the malignant clone developedat different stages of differentiation . Analysis of DNAploidy distribution showed that cells were predominately 2N with a small number of 4N . Cell cycleanalysis showed that very few cells were in S-and G2+M phase and the cell cycle of leukemiccells was...

ntigen distribution and DNA ploidy of megakary-ocytes were investigated in 4 patients with acutemegakaryoblastic leukemia. The percentages of cells as well as HLA-DR ̄+, HLA-DR ̄+ cells in mononuclear cellsvaried, suggesting that the malignant clone developedat different stages of differentiation . Analysis of DNAploidy distribution showed that cells were predominately 2N with a small number of 4N . Cell cycleanalysis showed that very few cells were in S-and G2+M phase and the cell cycle of leukemiccells was identical to that of the normal ones.Electron microscopic observation revealed someleukemic cells with characteristics of apoptosis.

采用流式细胞仪对4例急性巨核细胞白血病的抗原分布和巨核细胞DNA的倍体性进行了分析,发现血小板恃异性抗原分布在各个病例间有一定差异,其中一例高达85%以上,HLA-DR ̄+,和细胞在各个病例间分布同样有一定差异,说明白血病巨核细胞系恶性克隆可发生在造血于细胞向巨核细胞分化的不同阶段,病态巨核细胞DNA倍性分布大部分处2N,少部分处于4N,未见高于4N巨核细胞。提示其DNA倍体化过程受阻。细胞处于S期和G_2+M期的细胞<20%,白血病细胞的细胞周期分布与正常骨髓细胞相同,超微结构显示白血病细胞有凋亡现象,提示白血病巨核细胞堆积可能与其凋亡减少有关。

cl-2 mRNA was detected in bone marrow trephinebiopsies from leukemia patients and leukernic cell linesby in situ hybridization. In 21 of 22 biopsies, most ofthe leukemic cells (myelogenous or lymphocytic )ex-pressed high levels of bcl-2 mRNA. BclZ mRNA wasdetectable not only in lymphoid cell lines (Raji , CEM)but also in myeloid cell lines(K562 , HL-60 , U937)except for Molt-4 (T-cell leukemia). These resultssuggested that bcl-2 gene was broadly activated transcriptionally in various hematopoietic neoplasms...

cl-2 mRNA was detected in bone marrow trephinebiopsies from leukemia patients and leukernic cell linesby in situ hybridization. In 21 of 22 biopsies, most ofthe leukemic cells (myelogenous or lymphocytic )ex-pressed high levels of bcl-2 mRNA. BclZ mRNA wasdetectable not only in lymphoid cell lines (Raji , CEM)but also in myeloid cell lines(K562 , HL-60 , U937)except for Molt-4 (T-cell leukemia). These resultssuggested that bcl-2 gene was broadly activated transcriptionally in various hematopoietic neoplasms , andbcl-2 transcripts might influence the biological hehay-ior of leukemic cells by inhibiting apoptosis.

采用原位杂交方法检测了急性白血病骨髓活检标本及细胞株中bcl-2基因表达水平。结果22例白血病患者骨髓中21例(包括髓系和淋巴系)存在bcl-2mRNA高表达;6种白血病细胞株中5种bcl-2mRNA阳性(Molt-4例外),它们分别为CEM、Raji、HL-60、U937及K562。表明造血系统肿瘤中广泛存在bcl-2基因转录水平激活,其基因产物可能通过抑制细胞凋亡过程而影响急性白血病细胞的生物学特性。

 
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