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凋亡
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  apoptosis
    Effecte of hNaDC3 on the Cellular Energy Metabolism and Apoptosis in the Human Renal Proximal Tubular Epithelial Cells and the Role of AngⅡ Regulation
    hNaDC3对肾小管上皮细胞能量代谢和凋亡的影响以及AngⅡ的调控作用
短句来源
    Effective of Growth Inhibition and Apoptosis Induction by Retinoic Acid Combined with Adriamycin or Interferon on Transitional Cell Carcinoma of Bladder
    维甲酸联合阿霉素、干扰素对膀胱癌生长抑制和凋亡诱导作用研究
短句来源
    Role of ERβ on Proliferation and Apoptosis in Hormone Independent Prostate Cancer Cell Line PC-3 and PC-3M
    ERβ对激素非依赖性前列腺癌细胞株PC-3和PC-3M增殖和凋亡的影响
短句来源
    Effect of Calcium Antagonist Amlodipine on Apoptosis of LLC-PK1 Cell
    钙离子拮抗剂对肾小管上皮细胞LLC-PK_1凋亡的作用
短句来源
    Effect of 1-methylhydantoin and Methylguanidine on HK-2 Cells Apoptosis and Expression of Fibrotic Correlation Factors and Intervention of Medicine
    1-甲脲乙醇酸酐、甲基胍对HK-2细胞的凋亡、纤维化相关因子表达及药物干预的研究
短句来源
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  apoptotic
    ② Flow cytometry techniques showed that the total apoptotic ratio of HCA + AGE group was (15.31±2.14)%, and early apoptotic ratio was (9.16±1.38)%, which were significantly different from those in the HCA group and AGE group (both P < 0.01).
    ②流式细胞仪检测内皮细胞凋亡结果:联合干预组总凋亡率为(15.31±2.14)%,早期凋亡率为(9.16±1.38)%,均显著高于同型半胱氨酸或晚期糖基化终产物单独干预组(P<0.01)。
短句来源
    Apoptotic index = TUNEL positive cells/ total cells×100%.
    细胞凋亡指数=凋亡阳性细胞数/细胞总数×100%。
短句来源
    The apoptotic index was (32.46±3.69) % on the first day after birth, and it peaked on the 7th day after the birth, which was (45.36±4.35)%, while the apoptotic cells decreased gradually time increasing.
    小鼠出生后第1天,细胞凋亡指数为(32.46±3.69)%,于生后第7天达到高峰,细胞凋亡指数为(45.36±4.35)%,以后随着日龄增加凋亡细胞逐渐减少;
短句来源
    5-Aza-CdR at certain concentration(0.625,1.25,2.50,5.00mg/ml)obviously decreased the proliferation ratio and increased the apoptotic index in EJ cells compared with those in control group(P<0.05). The inhibition effect of 5-Aza-CdR was dose-dependant(P<0.05).
    与对照组相比,不同浓度的各组5-Aza-CdR(0.625,1.25,2.50,5.00mg/ml)可以显著降低EJ细胞的增殖比和增高EJ细胞的凋亡指数(P<0.05),且与5-Aza-CdR呈浓度依赖关系(P<0.05);
短句来源
    partial cells presented the morphological changes of apoptosis under the fluorescent microscope,the apoptotic rates of the control group and 4 treatment groups were(9.83±1.53)%,(19.50±1.00)%,(24.83±2.52)%,(30.17±2.08)% and(38.50±2.65)%,respectively;
    荧光显微镜下部分细胞发生凋亡形态学改变,对照组和4个姜黄素不同浓度处理组凋亡率分别为(9.83±1.53)%、(19.50±1.00)%、(24.83±2.52)%、(30.17±2.08)%和(38.50±2.65)%;
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  apoptotic status
    Method We detected the apoptotic status and the expression of apoptotic related gene bcl 2 and bax protein in 48 cases of RCC with TdT mediated dUTP biotin nick end labeling (TUNEL) and immunohistochemistry methods.
    方法采用原位DNA片断末端标记和免疫组织化学方法,检测48例肾癌组织细胞凋亡状态及凋亡相关基因bcl2和bax蛋白的表达。
短句来源
    Methods: Immunohistochemistry and TUNEL (TdTmediated dUTPbiotin nick end labeling) were used on 48 cases of RCC and 30 cases of normal tissue near the tumors mentioned above RCC sections to detect cell proliferative and apoptotic status.
    方法:采用免疫组织化学和原位缺口末端标记方法对48例肾癌和30例癌旁正常肾组织进行细胞增殖和凋亡状态的研究。
短句来源
  “凋亡”译为未确定词的双语例句
    Experimental Study of TNF-Related Apoptosis-Inducing Ligand on Human Bladder Cancer
    肿瘤坏死因子相关诱导凋亡配体对人膀胱癌治疗作用的实验研究
短句来源
    Experimental Study of New Anti-apoptotic Protein hPEBP4 as a Potential Therapeutic Target for Human Prostate Cancer
    新型抗凋亡分子hPEBP4作为人前列腺癌治疗靶点的实验研究
短句来源
    Expression of the Apoptosis-Suppressing Gene BCL-2 Product in Renal Carcinoma
    凋亡抑制基因BCL-2蛋白产物在肾癌中的表达
短句来源
    Expression of the bcl 2 and bax oncoprotein in TCC and its clinical significances
    凋亡相关基因bcl-2和bax蛋白在膀胱癌组织中的表达及临床意义
短句来源
    Conclusion:Over expression of HIF-1α may lead to increase of oxygen free radicals,impaired renal function and subsequent aggravation kidney injury by ischemia-reperfusion,all of which could be alleviated or improved by HIF-1α siRNA pretreatment with the probable mechanism of up-regulation of anti-apoptosis genes.
    结论:HIF-1α的过度表达可能导致氧自由基生成增加,肾功能受损,从而加重缺血再灌注肾脏的损伤,而HIF-1αRNAi可以减轻并改善这一状况,其机制可能与上调凋亡抑制基因有关。
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  apoptosis
Prodigiosin also could induce apoptosis of pancreatic cancer cells at low concentration and results in the fragmentation pattern of DNA.
      
All these results demonstrate that prodigiosin can obviously inhibit the proliferation of pancreatic cancer cells H8898 by arresting the cell cycle and inducing apoptosis.
      
Detection of the apoptosis of Jurkat cell using an electrorotation chip
      
The apoptosis of cells is one of the fields that attract increasing attention in biology today.
      
Usually, the cells are treated with chemicals when detecting apoptosis.
      
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  apoptotic
The glioma cells showed typical apoptotic signs 90 hours after the transient transfection of GFAP-Baxα.
      
The number of microgranulomas, lipogranulomas and apoptotic bodies increased following severity of steatosis, lobular inflammation and fibrosis.
      
Low concentrations of phospholipase A2 and lipoxygenase inhibitors were shown to stimulate cell division, while higher concentrations inhibited it by blocking G1-S transition and inducing apoptotic cellular death.
      
TUNEL assay was used to detect apoptotic cardiomyocytes.
      
The apoptotic changes in cardiomyocytes proved to prevail in early lesion foci (4-18 h), while cardiomyocytes at later stages were prone to necrosis; cardiomyocytes can exhibit signs of apoptosis and necrosis at the same time.
      
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  apoptotic status
The apoptotic status was observed in liver of all rats with TUNEL and PKC protein in liver of OJ was studied by immunohistochemical method.
      
It seems clear that MIBI can be used before treatment to detect drug resistance, assess anti-apoptotic status and predict treatment efficacy.
      
Apoptotic status was determined using Hoechst 33258 and propidium iodide to examine morphological changes.
      
As described for panel A, cells that remained GFP positive at the 42-h time point were stained with annexin-V and assessed for their apoptotic status.
      
Figure 2 Calpain inhibition has no effect on insulin content or apoptotic status.
      
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multiparametric analysis was made to

观察了缺血性肾损伤中肾小管上皮细胞死亡的另一种形式—细胞凋亡(apoptosis)。通过钳夹肾蒂造成小鼠缺血再灌注肾损伤模型。结果发现,缺血5分钟再灌注12~24小时肾小管上皮细胞并不出现坏死,但可发生细胞凋亡现象。随着缺血时间的延长,缺血30分钟再灌注6~24小时或缺血45分钟再灌注6~18小时,肾小管和间质出现凋亡小体。肾脏DNA电泳像呈“梯形结构”,并持续24小时后消失。结果提示,缺血再灌注肾损伤中存在apoptosis机制的参与。

Bcl-2 oncoprotein is known to protect cells from apoptosis.The aim of this study was to investigate whether bcl-2 correlates with the development of benign prostatic hyperplasia(BPH).Expression of bcl-2 was investigated in 20 cases of BPH and in 12 cases of normal prostate tissue using immunohistochemical technique.All basal cells and some luminal cells of peripheral zone in normal prostate were positive.In central and transtional zone,bcl-2 expression was limited to some dispersed basal cells.In BPH,bcl-2 was...

Bcl-2 oncoprotein is known to protect cells from apoptosis.The aim of this study was to investigate whether bcl-2 correlates with the development of benign prostatic hyperplasia(BPH).Expression of bcl-2 was investigated in 20 cases of BPH and in 12 cases of normal prostate tissue using immunohistochemical technique.All basal cells and some luminal cells of peripheral zone in normal prostate were positive.In central and transtional zone,bcl-2 expression was limited to some dispersed basal cells.In BPH,bcl-2 was strongly expressed in basal cells and most of the luminal cells.The rate of positive cells and its staining intensity were much higher in BPH than in normal prostate.There was no bcl-2 experession in the mesenchyme.The results suggest that high expression of bcl-2 might have a role in the development of BPH.

采用抗bcl-2单克隆抗体对20例前列腺增生(BPH)和12例正常前列腺组织的冰冻切片进行免疫组织化学染色。结果表明,正常前列腺的外周区bcl-2蛋白主要位于腺体的基底细胞,腺上皮细胞偶见着色,移行区仅部分基底细胞阳性着色。前列腺增生组织中的上皮基底细胞和大部分腔上皮细胞均呈强阳性,阳性的细胞数和染色强度均高于正常前列腺组织。具有抑制细胞凋亡的bcl-2蛋白在BPH组织中的过表达提示BPH的发生可能与上皮细胞可以不依赖于激素的生长和(或)细胞寿命延长,凋亡的细胞数目减少有关。

Serum free culture with no growth stimulating factors supplemented was used to induce apoptosis in human glomerular endothelial cells(GECs).The results showed that DNA electrophoresis of GECs cultured in serum free medium after 48 hours displayed typical“ladder pattern” of apoptosis which indicated oligonucleosomal DNA fragmentation. Moreover,by means of ELISA and RT PCR,expressions of the protein and mRNA of fibronectin were found to be noticeably higher in serum free group than those in normal culture group.These...

Serum free culture with no growth stimulating factors supplemented was used to induce apoptosis in human glomerular endothelial cells(GECs).The results showed that DNA electrophoresis of GECs cultured in serum free medium after 48 hours displayed typical“ladder pattern” of apoptosis which indicated oligonucleosomal DNA fragmentation. Moreover,by means of ELISA and RT PCR,expressions of the protein and mRNA of fibronectin were found to be noticeably higher in serum free group than those in normal culture group.These results suggest that the disturbance of apoptosis in GECs might involve in the accumulation of ECM during glomerulosclerosis.

用不加生长因子的无血清培养方法诱导人肾小球内皮细胞凋亡。结果显示,培养48h后无血清培养组内皮细胞的DNA电泳呈现凋亡细胞典型的“梯形结构”条带;同时用酶联免疫吸附试验(ELISA)和逆转录-聚合酶链反应(RT-PCR)方法发现,凋亡内皮细胞纤维连接蛋白的蛋白质和基因表达均明显增高。提示肾小球内皮细胞的凋亡紊乱可能与肾小球硬化时细胞外基质的积聚有关,为揭示肾小球肾炎及肾小球硬化的发生和转归提供了理论依据。

 
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