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实验性高血压
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  experimental hypertension
     Expression of Adrenomedullin and Its mRNA in the Experimental Hypertension Rats After Focal Cerebral Ischemia and Reperfusion
     实验性高血压大鼠局灶性脑缺血再灌注后肾上腺髓质素及其mRNA的表达
短句来源
     CONCLUSION: Experimental hypertension have irreversible effect on nociceptive nervous pathway.
     结论 :持续实验性高血压可能对伤害性反射的神经通路具有不可逆的调节作用
短句来源
     Aim:To establish the experimental hypertension rat(EHR) model induced by 5 HT depletion in the brain and to investigate its central mechanism.
     目的 :建立中枢 5 羟色胺 (5 HT)缺损所致的实验性高血压大鼠 (EHR)模型并探讨其中枢机理。
短句来源
     Methods The experimental hypertension rats were prepared by the advanced Kellen's methods. The models of middle cerebral artery occlusion in rats were constructed by the line-block-method,and after ischemia for 2 h they were reperfused.
     方法应用改良Kellen的方法制备实验性高血压大鼠,采用栓线法制成大鼠大脑中动脉闭塞模型,阻断血流2 h后进行再灌注。
短句来源
     Effect of Zishuijianghuoyin on Expression of Lymphocyte AngII Receptor (AT-1)mRNA of Experimental Hypertension Rats
     滋水降火饮对实验性高血压大鼠淋巴细胞AngⅡ受体mRNA表达的影响
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  experimental hypertensive
     Hypotensive effects of 1-(2,6-dimethylphenoxy)-2-(3,4-dimethoxyphenylethylamino) propane hydrochloride on experimental hypertensive rats
     1-(2,6-二甲基苯氧基)-2-(3,4-二甲氧基苯乙胺基)丙烷盐酸盐对实验性高血压大鼠的降压作用
短句来源
     The Influence of Ang-(1-7)on Blood Pressure and Its Mechanisms in Experimental Hypertensive Rats
     Ang-(1-7)对实验性高血压大鼠血压的影响及其机制的初步研究
短句来源
     OBJECTIVE: To determine the effects of 1 (2, 6 dimethylphenoxy) 2 (3, 4 dimethoxyphenylethy lamino) propane hydrochloride (DDPH) on proliferation of vascular smooth muscle cells (VSMC) and the expression of growth factor PDGF B, bFGF and oncogene c sis, c myc in experimental hypertensive rats.
     目的:观察1(2,6二甲基苯氧基)2(3,4二甲氧基苯乙胺基)丙烷盐酸盐(DDPH)对实验性高血压大鼠血管平滑肌细胞(VSMC)增殖的作用及对生长因子PDGFB,bFGF及其相关癌基因csis,cmyc表达的影响。
短句来源
     Effects of DDPH on Proliferation of Vascular Smooth Muscle Cells and Heat Stress Protein,p53 in Experimental Hypertensive Rats
     DDPH对实验性高血压大鼠血管平滑肌细胞增殖及对热应激蛋白70和抑癌基因p53的影响
短句来源
     Observation of Blood Pressure in Experimental Hypertensive Rats
     实验性高血压大鼠的血压观察
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  “实验性高血压”译为未确定词的双语例句
     Different hypertensive animal models were used, such as spontaneously hypertensive rats (SHR), salt-sensitive (SS) hypertensive rats and experimentally hypertensive rats (EHR), including hypertensive rats by left renal artery clipping or induced by deoxycorticosterone acetate-salt administration or by receiving pressor agents, to study the threshold of nociceptive reflex. These results were basically in favor of the conclusion of HAH.
     他们应用不同的高血压动物模型,如自发性高血压大鼠(Spontaneously hypertensive rats,SHR)、盐敏感(Salt-sensitive,SS)高血压大鼠和实验性高血压大鼠(Experimentally hypeaensive rats,EHR),包括左肾动脉钳夹高血压大鼠,醋酸脱氧皮质酮(Deoxycorticosterone acetate,DOCA)盐大鼠和升压药诱致高血压大鼠等,对痛反射阈值进行了研究,结果基本支持“高血压相关性痛觉减退”的结论。
短句来源
     Introduction of Some Models of Hypertension in Experimental Rats
     若干实验性高血压大鼠模型的介绍
短句来源
     (3) When NS was microinjected into PGL with the acupuncture of TPA simultaneously could make the blood pressure of hypertension rabbits decrease, mAP、 mLVSP、 LV+dp/dt max decreased significantly, HR decreased slightly.
     (3) 电针TPA对兔静脉内匀速注射去甲肾上腺素造成的急性实验性高血压具有显著的降压效应,mAP、mLVSP及LV+dp/dt max显著下降,HR有所下降。
短句来源
     Method The experimental rabbit hypertension was induced by norepinephrine(NE) with intravenous drip injection,[Ca 2+ ] i and [Mg 2+ ] i were determined .
     方法静脉滴注去甲肾上腺素(NE)造成家兔实验性高血压,对[Ca2+]i及[Mg2+]i进行测定。
短句来源
     (2)When NS 、 NAL was microinjected respectively into PGL, there's no significant change on hypertension rabbits, mAP、 mLVSP、 LV+dp/dt max and HR were not changed significantly.
     (2) 旁巨细胞外侧核(PGL)内分别微量注射生理盐水(NS)、阿片受体阻断剂纳络酮(NAL),对急性实验性高血压无明显影响,mAP、HR及mLVSP、LV+dp/dt max在微量注射前后均无明显变化。
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  experimental hypertension
Mesenteric microcirculation in rats with experimental hypertension
      
Experimental hypertension, produced by constriction of the abdominal aorta, induced hypertrophy of the left ventricle, with increased perimysium and endomysium of the ECM at the level of the papillary muscles 4 weeks after aortic banding.
      
Based on these considerations, numerous preclinicial studies with vasopeptidase inhibitors have been performed and reveal promising results in experimental hypertension.
      
In particular, the excitation of the rostral ventrolateral medulla by excitatory amino acid neurotransmitters and by stimulation of AT1 angiotensin receptors appears to be increased in experimental hypertension.
      
Effects of perindopril on vascular wall and left ventricular hypertrophy in rats with experimental hypertension
      
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  experimental hypertensive
Cerebral cortical arterioles in focal neocortical areas develop increased permeability to plasma proteins and protein tracers in experimental hypertensive encephalopathy.
      
The significance of overexcitation of the central nervous system in the genesis of experimental hypertensive stroke
      
The role of the initial factor in the development of experimental hypertensive stroke may be played by the functional overexcitation of the CNS; fatal cerebral strokes occur in such cases in 27 percent of the animals.
      
The latter circumstance indicates that a functional model of experimental hypertensive stroke has been created.
      
The data presented here show that the experimental hypertensive insult induces degenerative and apoptotic events comparable to those observed in human glaucoma.
      


Experiments were performed on 13 conscious dogs whose carotid or brachialsystolic blood pressure was measured by indirect methods.Electroacupunctureadministered to dog's“Tsu San-Li”or“Ne-Kuan”had no significant influenceon normal blood pressure.But when experimental hypertension was producedby intravenous infusion of noradrenaline at a constant rate,electroacupunctureresulted in a significant decrease of blood pressure,while the heart rate and respira-tory frequency were not affected.The depressor effect was...

Experiments were performed on 13 conscious dogs whose carotid or brachialsystolic blood pressure was measured by indirect methods.Electroacupunctureadministered to dog's“Tsu San-Li”or“Ne-Kuan”had no significant influenceon normal blood pressure.But when experimental hypertension was producedby intravenous infusion of noradrenaline at a constant rate,electroacupunctureresulted in a significant decrease of blood pressure,while the heart rate and respira-tory frequency were not affected.The depressor effect was not due to decreaseof cardiac output,for the heart rate and PEP/LVET didn't alter significantly.Atro-pine injection couldn't block the depressor effect.But the blood flow of the mesen-teric artery increased during the depressor period.So it is suggested that the de-pressor effect of electroacupuncture was due to inhibition of the sympathetic vasocons-trictor center.Small doses of morphine injected intravenously also produced a transcient de-pressor action on experimental hypertension,but the depressor effect of electro-acupuncture could be prevented by pretreatment with intravenous injection of nalo-xone(0.2 mg/kg),while naloxone itself had no effect on blood pressure and heartrate whether the blood pressure of the animal was normal or raised.These results suggest that the depressor effect of electroacupuncture duringnoradreanline hypertension mainly resulted from vasodilatation due to inhibi-tion of the sympathetic vasoconstrictor tone.This inhibition was related to endoge-nous opiate-like substances which might be released somewhere in the central nervoussystem during electroacupuncture.

本工作在清醒犬上进行,介绍了两种间接测量动脉血压的方法。观察到电针“足三里”穴对正常动物的血压、心率、呼吸并无显著影响。当静脉内匀速注射去甲肾上腺素造成急性实验性高血压时,电针具有显著的降压效应,对心率和呼吸则无明显影响。电针产生降压效应时,内脏血管舒张,血流量增加,而心输出量并不减少,且降压效应不被阿托品阻断。在实验性高血压时静脉注射吗啡可产生与电针相类似的降压效果,静脉注射纳洛酮可阻断电针的降压作用。结果表明:电针对实验性高血压的降压效应主要是交感缩血管中枢紧张性受抑制使外周血管舒张而产生的,此种抑制效应与电针时中枢内产生的内源性鸦片样物质有关。

In 13 conscious dogs,stainless steel cannulae were previously implanted intovarious areas of the brain.The heart rate and blood pressure measured by the indirectmethod were recorded on the polygraph.When normal saline(2μl/2min.)was mi-croinjected into the central gray(CG),dorsal area of the hippocampus(HPC)and sup-ramammillary area(SMA),the experimental hypertension produced by intravenousinfusion of noradrenaline(NA)t a constant rate was not affected,and the de-pressor effect of electroacupuncture at Tsu San-Li...

In 13 conscious dogs,stainless steel cannulae were previously implanted intovarious areas of the brain.The heart rate and blood pressure measured by the indirectmethod were recorded on the polygraph.When normal saline(2μl/2min.)was mi-croinjected into the central gray(CG),dorsal area of the hippocampus(HPC)and sup-ramammillary area(SMA),the experimental hypertension produced by intravenousinfusion of noradrenaline(NA)t a constant rate was not affected,and the de-pressor effect of electroacupuncture at Tsu San-Li still existed.Morphine 4μg/2μl/2 min.)microinjected into these areas caused a depressor effect for about 15minutes on NA-induced hypertension,with no significant changes in the heart rate.But when naloxone(4μg/2 μl/2 min.) was microinjected into these areas,the de-pressor effect of electroacupuncture was blocked.In the medial aspect of parietalcortex,morphine microinjection(4 μg/2 μl /2 min.)didn't produce any depressoreffect,and naloxone microinjection didn't influence the depressor effect of electro-acupuncture on NA-induced hypertension.It is suggested that the inhibitory effect on sympathetic vasomotor neurons byelectroacupuncture was due to the release of endogenous opiate-like substance andthe activation of opiate receptors in CG,HPC and SMA,etc..

在清醒犬的中脑中央灰质、海马背侧、乳头体上区等中枢部位微量注射生理盐水(2微升/2分钟),对血压、心率无明显影响,此时电针“足三里”穴对匀速注射去甲肾上腺素所致的实验性高血压具有显著的降压效应。在这种高血压的基础上,在以上三个部位微量注射吗啡(4微克/2微升/2分钟)可见血压短暂下降后又回升到原高血压水平,心率无显著变化。在这些部位微量注射纳洛酮后(4微克/2微升/2分钟)则以上电针降压效应消失。而在大脑顶叶内侧皮层微量注射吗啡对血压、心率无明显影响;在该处微量注射纳洛酮后,电针对实验性高血压仍有明显的降压作用。结果表明:电针对交感缩血管中枢的抑制与电针时中枢内产生的内(脑)啡肽对中脑中央灰质、海马、乳头体上区等部位鸦片受体的激活有关。

The contents of two kinds of enkephalin (EK) in three cerebral regions were measured by using radioimmunoassay method in five types of experimental hypertensive rats. In the "two-kidney two-clip" group and "two-kidney '8'-ligated-salt" group, methionine-EK (MEK) and leucine-EK (LEK) concentrations in the brain stem, hypothalamus and striatum were significantly higher than in those regions of the normal rats. However, in the "one-kidney one-clip" rats, except for an incressc of MEK in the striatum and LEK in...

The contents of two kinds of enkephalin (EK) in three cerebral regions were measured by using radioimmunoassay method in five types of experimental hypertensive rats. In the "two-kidney two-clip" group and "two-kidney '8'-ligated-salt" group, methionine-EK (MEK) and leucine-EK (LEK) concentrations in the brain stem, hypothalamus and striatum were significantly higher than in those regions of the normal rats. However, in the "one-kidney one-clip" rats, except for an incressc of MEK in the striatum and LEK in the hypothalamus, the contents of EK had no significant chages. In the "two-kidney '8'-ligated" rats, the contents of MEK in the brain stem were somewhat decreased. Nevertheless, in the "DOCA-salt" hypertensive rats, MEK and LEK in the brain stem and the striatum were increased, whereas MEK in the hypothalamus were markedly decreased. These changes in DOCA-salt rats became more significant along with the duration of DOCA-salt administration. The results suggest that central EK is implicated in the course of hypertension characterized by sodium and water retention. Besides, DOCA may have an important effect on the contents of MEK in the hypothalamus.

本文测定了五类实验性高血压大鼠三个脑区中二种脑啡肽的含量。在两肾两夹及两肾“8”结扎—盐高血压大限脑干,下丘和纹状体中甲脑啡肽(MEK)和亮脑啡肽(LEK)含量显著高于正常大鼠。在一肾一夹大鼠中除纹状体MEK及下丘LEK增加外,EK含量无显著变化。在两肾“8”结扎大鼠中仅脑干MEK稍有增加。DOCA—盐高血压大鼠与上述几种大鼠不同,它们的脑干和纹状体中EK含量虽然增加,而下丘MEK却明显下降,DOCA—盐大鼠中的这些变化随DOCA—盐处理时间延长而变得更加显著。结果提示以水纳潴留为特征的高血压动物中涉及中枢EK的含量改变。此外,DOCA对下丘MEK含量可能有重要作用。

 
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