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   前胃癌 的翻译结果: 查询用时:0.033秒
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前胃癌     
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  forestomach carcinoma
     RESULTS: Cyclin D1 expression was significantly higher in forestomach carcinoma (64.7%) than in precancerous lesions (29.4%), χ~2=4.344 7,P=0.0371;
     结果:cyclin D1在小鼠前胃癌组织中的表达(64·7%)明显高于癌前期病变(29·4%),χ2=4·3447,P=0·0371;
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     METHODS: Forestomach carcinoma model of ICR mice was established, and VEGF, cyclin D1 and p27~(kip1) expressions were assayed by SABC immunohistochemical method.
     方法:建立ICR小鼠前胃癌模型,SABC法测定VEGF、cyclin D1和p27kip1表达。
短句来源
     VEGF, cyclin D1 and p27~(kip1) expressions in progressing forestomach carcinoma of mice
     小鼠前胃癌组织形成中VEGF和cyclinD1及p27~(kip1)的表达
短句来源
     OBJECTIVE:To investigate expressions of VEGF, cyclin D1 and p27~(kip1) and mechanisms by retinoic acid intervention in progressing forestomach carcinoma of mice.
     目的:研究VEGF、cyclin D1和p27kip1在小鼠前胃癌组织形成过程中的表达及意义,初步探讨维甲酸对小鼠前胃癌的干预机制。
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     Specific CTL activity for U14was37.8%comparing to22%for control mouse forestomach carcinoma(MFC)cells.
     在小鼠体内瘤苗可激发特异的CTL活性,对U14细胞的杀伤率为37.8%,而对照小鼠前胃癌(MFC)细胞的杀伤率为22.0%。
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  forestomach neoplasia
     The inhibitory effect of CLA on mice forestomach neoplasia induced by B(a)P
     共轭型亚油酸对B(a)P诱导小鼠前胃癌的抑制作用:150001黑龙江省哈尔滨医科大学营养与食品卫生教研室
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     Expression changes of connexin 32 in induced forestomach neoplasia of mice
     小鼠前胃癌诱导中Cx32表达的变化
短句来源
     Methods B(a)P was used to induce the forestomach neoplasia of the mice,and immunohistochemistry technique was used to detect the expression regularity of connexin 32 in the procedure of inducing forestomach neoplasia.
     方法 B(a)P诱导小鼠前胃癌 ,免疫组织化学方法检测Cx32在小鼠前胃癌的发生、发展过程中表达的变化。
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     Establishment of mouse forestomach neoplasia model induced by benzopyrene and chemopreventive effects of conjugated linoleic acid
     苯并(a)芘诱导小鼠前胃癌模型的建立及共轭亚油酸对其预防作用
短句来源
     AIM To establish mouse forestomach neoplasia model and observe the relation of inhibitory effect on forestomach neoplasm and lipid peroxidation of conjugated linoleic acid(CLA) of different constituent.
     目的 用苯并 (a)芘〔B(a)P〕建立小鼠前胃癌模型 ,观察不同构成的共轭亚油酸 (CLA)对前胃癌的抑制作用以及与脂质过氧化的关系。
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  forestomach cancer
     Conclusion CQF liquid can interrupt forestomach cancer and precancerous lesions in mice, and is related to VEGF expression.
     结论 CQF液对小鼠前胃癌及癌前病变具有阻断作用 ,其机理可能与对VEGF表达的调控相关。
短句来源
     VEGF is a factor involved in forestomach cancer and precancerous lesions in mice.
     VEGF参与形成小鼠前胃癌及癌前病变
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     can decrease the development of B(a)P-induced forestomach cancer in mice.
     结果 经口投予和腹腔注射VES均可明显降低B(a)P诱导的小鼠前胃癌的发生。
短句来源
     Methods The model of forestomach cancer and precancerous lesions in mice was established by N-nitrososarcosine ethyl ester and sodium nitrite perfusing stomach;
     方法 应用肌氨酸乙酯盐酸盐和亚硝酸钠灌胃建立小鼠前胃癌及癌前病变模型。
短句来源
     Objective To investigate intervention and mechanism of forestomach cancer and precancerous lesions in mice by Chanpi, Quanchong and Fengfang mixed liquid (CQF liquid).
     目的 研究蟾皮、全虫、蜂房 (CQF液 )对小鼠前胃癌及癌前病变的阻断作用及其相应机理。
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  preventriculus squamous carcinoma
     Establishment of liver metastasis model from preventriculus squamous carcinoma in mice
     实验性小鼠前胃癌肝转移模型的建立
短句来源
     Objective To establish a preventriculus squamous carcinoma liver metastases model for experimental studies in mice.
     目的 :建立实验性小鼠前胃癌肝转移模型。
短句来源
     Conclusion This study successfully established the model of preventriculus squamous carcinoma liver metastases in mice. It's a useful model to select antineoplastics and to study metastatic behaviors.
     结论 :本研究成功建立了实验性小鼠前胃癌肝转移模型 ,为筛选抗肿瘤药物和研究肿瘤转移行为提供了有用的模型
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  forestomach carcinoma
The effect of retinoic acid on cell membrane and metastatic ability of mouse forestomach carcinoma cell
      
We studied the effect of all-trans-retinoic acid (RA) on the expression of several surface lectin receptors and cell membrane fluidity of mouse forestomach carcinoma cell line (MFC)in vitro.
      
On the histopathogenesis of rat forestomach carcinoma caused by aristolochic acid
      
The histopathogenesis of rat forestomach carcinoma induced experimentally with aristolochic acid was investigated.
      
Seven cell lines established from a diethylnitrosamine (DEN)-induced forestomach carcinoma (DEN3) of a BALB/c mouse and its six pulmonary metastatic foci were used to study the biological and functional diversity of tumor cells.
      
  forestomach neoplasia
Inhibitory effects of benzyl isothiocyanate administered shortly before diethylnitrosamine on pulmonary and forestomach neoplasia in A/J mice.
      
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