The result of single-factor logistic regression analysis showed that the risk factors were:father aged at least 40 years old,father or family having hearing disorders,mother's poor nutrition,virus infection and contacting metal or chemical poison during pregnancy,having 2 or more pregnancy history,low birth weight and history of NICU;
There were 232 cases(87.5%) of the CP children were accompanied with mental retardation of different degrees,12 cases(4.5%) of hearing disorders,20 cases(7.5%) of epilepsy and 15 cases(5.7%) of visual dysfunction.
A spectrum and cross-correlation analysis of auditory brainstem responses (ABRs) of normal subjents and patiects with hearing disorders was studied at about 200Hz, 500Hz and 800-1000Hz while few components above 1200Hz in the amplitude-frequency curves of ABRs of normal subjects.
Conclusion:TEOAE and ABR hearing screening technique could improve the sensitivity and accuracy of the hearing screening for newborns, diagnose the newborns with hearing impairment and provide the guidance for early intervention and healing.
【Results】Forty six(36.2%)newborns didn't pass the initial DPOAE test among 127 newborns of hypoxic-ischemic encephalopathy,9(19.6%)among 46 infants didn't pass the second DPOAE test,2 infants failed to pass ABR test,the ratio of hearing disorder in newborns of hypoxic-ischemic encephalopathy was 1.57%(2/127);
Two (3.2%) infants, who respectively suffered from Glucose-6-Phosphate-Dehydrogenase (G-6PD) deficiency, and deficiency of vitamine K resulted in kernicterus and intracranial hemorrhage, failed to pass ABR test and were confirmed having a hearing disorder caused by brain damage.
Results 62(36%)newborns didn't pass the first DPOAE test among 172 high risk newborns,10(16.1%)infants didn't pass the second DPOAE test among 62 infants,5 infants failed to pass ABR test,the ratio of hearing disorder in high risk newborns was 2.91%;
Patients with organic lesions (tumors, cysts) involving the temporal lobe cortex may reveal more severe spatial hearing disorders than temporal epilepsy patients with the same localization of the foci of convulsive activity.
Possible neurophysiological mechanisms underlying the found spatial hearing disorders as well as the possibility of using the results obtained to solve the problems of differential diagnosis are considered.
The relationship between metabolic disturbances in the inner ear and the hearing disorders from salicylate intoxication, known from the clinic, is discussed.
The relationship between metabolic disturbances in the inner ear and the hearing disorders caused by mercury intoxication, known to clinical investigation, is discussed.
In contrast to it, a curtailment of the stimulation interval in patients with certain hearing disorders, the pattern of the evoked response changes in such a manner, that we can speak of an atypical response.
There was no clear-cut dependence of speed and quality of articulation on all of the peripheral factors evaluated (Angle Class, overbite, oral motor ability, hearing disorder, logopedics, Castillo-Morales stimulating plate treatment).
Based upon the anamnestic and symptomatic data obtained from 50 children, the stapedius-reflex-audiometry and ERA findings of which speak for a central hearing disorder, the specificity and clinical importance of these methods is to be discussed.
Whether a malfunction is a technical or physiological problem remains to be defined, particularly in very young children, while a psychogenic hearing disorder after implantation must not be excluded in adults.
Auditory neuropathy (AN) is a hearing disorder characterized by the preservation of outer hair cell function despite the absence of auditory brainstem responses.
Diseases of the inner ear which are common in early childhood (like otitis media) reduce the transfer of TEOAEs and may wrongly indicate a cochlear hearing disorder.