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冷损伤血管
相关语句
  cold injured blood vessels
     Changes of NO synthase activity in the cold injured blood vessels and its biological significance
     冷损伤血管NO合成酶活力的变化及其生物学意义
短句来源
     Objective To explore the new therapy by observing the changes of NO synthase(NOS) activity in the cold injured blood vessels and by studying the effects of SOD and Vit C on the changes of NOS activity.
     目的 观察不同程度冷损伤血管NO 合成酶( NOS) 活力的变化,并研究超氧化物歧化酶(SOD) 和Vit C 对这种变化的影响。
短句来源
     Changes of lipid peroxidation(LPO) in cold injured blood vessels
     冷损伤血管脂质过氧化水平的变化及其意义
短句来源
  “冷损伤血管”译为未确定词的双语例句
     After 4 h of cold exposure,NOS activity in the slight ,moderate and severe injured vessels increased by 15.3%,and decreased by 17.9%,29.6% respectively as compared with control.
     冷损伤后4 小时,轻度冷损伤血管NOS 活力较未冷冻血管增加15 .3 % ,中、重度冷损伤血管NOS 活力则显著下降,分别达17 .9 % 、29 .6 % 。
短句来源
     The changes of the NOS activity was time dependent. Treating with SOD(200 U/ml) or Vit C(50 mg/ml) immediatly after cold exposure made NOS and SOD activity in the vessels significantly increase and LDH in the medium decrease.
     血管冷冻后立即给予SOD(200 U/ ml) 或Vit C(50 mg/ml) ,可使冷损伤血管NOS 活力分别较未经SOD 或Vit C 处理者显著升高,并降低了冷损伤血管培养液中LDH 活力,使血管SOD 活力回升。
短句来源
     Change of NOS activity in hypoxia and cold induced blood vessels damage and its biological significance
     缺氧冷损伤血管一氧化氮合酶活性的变化及其生物学意义
短句来源
  相似匹配句对
     Changes of lipid peroxidation(LPO) in cold injured blood vessels
     损伤血管脂质过氧化水平的变化及其意义
短句来源
     No blood vessel or nerve was injured.
     无血管、神经损伤
短句来源
     Papaverine do damage to endothelial cells.
     罂粟碱损伤血管内皮。
短句来源
     Changes of NO synthase activity in the cold injured blood vessels and its biological significance
     损伤血管NO合成酶活力的变化及其生物学意义
短句来源
     COLD AND HOT WET PACKS IN THE TREATMENT OF ANKLE TRAUMA
     热敷治疗踝关节损伤
短句来源
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Objective To explore the new therapy by observing the changes of NO synthase(NOS) activity in the cold injured blood vessels and by studying the effects of SOD and Vit C on the changes of NOS activity.Methods The aortic artery was isolated from Wistar rats and cultured in PBS medium for 1 h,then exposed to -20 ℃ cold environment.LDH activity was measured using Biochemistry Automatic Analysis Meter,NOS and SOD activities in blood vessels were analysed respectively by Griess assay and adrenalin autooxidation...

Objective To explore the new therapy by observing the changes of NO synthase(NOS) activity in the cold injured blood vessels and by studying the effects of SOD and Vit C on the changes of NOS activity.Methods The aortic artery was isolated from Wistar rats and cultured in PBS medium for 1 h,then exposed to -20 ℃ cold environment.LDH activity was measured using Biochemistry Automatic Analysis Meter,NOS and SOD activities in blood vessels were analysed respectively by Griess assay and adrenalin autooxidation method. Results When the vessels were exposed to cold for 1,2,4 min respectively,LDH activity in the cultured medium increased and SOD activity in the vessels decreased at different level,indicating varying degree of vessel injury:slight ,moderate and severe cold injury.After 4 h of cold exposure,NOS activity in the slight ,moderate and severe injured vessels increased by 15.3%,and decreased by 17.9%,29.6% respectively as compared with control.The changes of the NOS activity was time dependent.Treating with SOD(200 U/ml) or Vit C(50 mg/ml) immediatly after cold exposure made NOS and SOD activity in the vessels significantly increase and LDH in the medium decrease. Conclusion The decrease of NOS activity may lead to the disorder in vascular circulation of cold injury,SOD and Vit C could prevent from or alleviate cold injury by increasing NOS activity.

目的 观察不同程度冷损伤血管NO 合成酶( NOS) 活力的变化,并研究超氧化物歧化酶(SOD) 和Vit C 对这种变化的影响。方法 分离Wistar 大鼠主动脉,并在PBS 培育液中培养1 小时,然后使其暴露于- 20 ℃环境。以全自动生化分析仪测定血管培养液中乳酸脱氢酶(LDH) 活力,以Griess化学法观测血管NOS 活力,以肾上腺素自氧化法测定血管SOD 活力。结果 当血管暴露于- 20 ℃环境1 、2 、4 分钟后,血管培养液中LDH 活力的增强幅度和血管SOD 活力的降低程度均逐步增大,表明血管分别相应受到轻、中、重度冷损伤;冷损伤后4 小时,轻度冷损伤血管NOS 活力较未冷冻血管增加15 .3 % ,中、重度冷损伤血管NOS 活力则显著下降,分别达17 .9 % 、29 .6 % 。血管冷冻后立即给予SOD(200 U/ ml) 或Vit C(50 mg/ml) ,可使冷损伤血管NOS 活力分别较未经SOD 或Vit C 处理者显著升高,并降低了冷损伤血管培养液中LDH 活力,使血管SOD 活力回升。结论 血管NOS 活力降低是导致...

目的 观察不同程度冷损伤血管NO 合成酶( NOS) 活力的变化,并研究超氧化物歧化酶(SOD) 和Vit C 对这种变化的影响。方法 分离Wistar 大鼠主动脉,并在PBS 培育液中培养1 小时,然后使其暴露于- 20 ℃环境。以全自动生化分析仪测定血管培养液中乳酸脱氢酶(LDH) 活力,以Griess化学法观测血管NOS 活力,以肾上腺素自氧化法测定血管SOD 活力。结果 当血管暴露于- 20 ℃环境1 、2 、4 分钟后,血管培养液中LDH 活力的增强幅度和血管SOD 活力的降低程度均逐步增大,表明血管分别相应受到轻、中、重度冷损伤;冷损伤后4 小时,轻度冷损伤血管NOS 活力较未冷冻血管增加15 .3 % ,中、重度冷损伤血管NOS 活力则显著下降,分别达17 .9 % 、29 .6 % 。血管冷冻后立即给予SOD(200 U/ ml) 或Vit C(50 mg/ml) ,可使冷损伤血管NOS 活力分别较未经SOD 或Vit C 处理者显著升高,并降低了冷损伤血管培养液中LDH 活力,使血管SOD 活力回升。结论 血管NOS 活力降低是导致冷损伤组织血液循环障碍发生的重要原因;SOD 及Vit C 可通过调节提高血管NOS 活力,防止或减缓机体冷?

Objectives To explore the therapeutic methods for hypoxia and cold induced blood vessel damage by observing the changes of NO synthase (NOS) activity in hypoxia and development of coldm induced blood vessels damage, and studying the effects of SOD and vitamin C on the changes of NOS activity. Methods The main artery was isolated from Wistar rats and exposed to hypoxia and /or cold environment in vitro. LDH activity was measured using an automatic biochemical analyzer and NOS and SOD activities in blood vessels...

Objectives To explore the therapeutic methods for hypoxia and cold induced blood vessel damage by observing the changes of NO synthase (NOS) activity in hypoxia and development of coldm induced blood vessels damage, and studying the effects of SOD and vitamin C on the changes of NOS activity. Methods The main artery was isolated from Wistar rats and exposed to hypoxia and /or cold environment in vitro. LDH activity was measured using an automatic biochemical analyzer and NOS and SOD activities in blood vessels were analyzed respectively by Griess assay and adrenaline autoxidantion method. Results NOS activity in hypoxia, cold and hypoxia cold damaged vessels decreased by 18 2%, 19 1% and 25 9% respectively after exposure as compared with the control vessels. The changes of NOS activity were time dependent. The NOS activity in damaged vessels was negatively correlated with LDH activity in the culture medium of the respective vessels. The SOD activity in hypoxia and /or cold damaged vessels also decreased significantly. Giving SOD (200 u/ml) or vitamin C (50 mg/ml) immediately after hypoxia and/or cold exposure increased the NOS activity and SOD activity in the damaged vessels significantly, as compared with the non treatment controls. Conclusions The decrease of NOS activity is closely related to the development of damage induced by hypoxia and/or cold injury. SOD and vitamin C could protect the blood vessels exposed to hypoxia and/or cold from damage, possibly by increasing NOS activity.

目的 观察缺氧寒冷损伤血管一氧化氮合酶 (NOS)活性的变化 ,并研究超氧化物歧化酶 (SOD)和维生素C(VC)对这种变化的影响 ,为缺氧寒冷损伤的防治开拓新的思路。方法 分离Wistar大鼠主动脉并使其暴露于缺氧或 /和寒冷环境中 ,以全自动生化分析仪测定血管培养液中乳酸脱氢酶 (LDH)活性 ,以Griess化学法观测血管NOS活性 ,以肾上腺素自氧化法测定血管SOD活性。结果 缺氧和寒冷损伤使血管NOS活性较对照组显著下降 ,分别达 18 2 %和 19 1% ,缺氧寒冷复合损伤更使血管NOS活性下降达 2 5 9% ;NOS的这种变化具有明显的时间依赖性 ,并与相应血管培养液中LDH活性有显著负相关关系 ;缺氧或 /和寒冷损伤血管SOD活性亦显著下降。当血管受到缺氧或 /和冷冻后立即给与SOD(2 0 0u/ml)或VC(5 0mg/ml)可使缺氧或 /和寒冷损伤血管NOS活性升高 ,SOD活性复升 ;对血管培养液中LDH的测定表明 ,SOD和VC对缺氧冷损伤血管具有一定的保护作用。结论 血管NOS活性的降低与缺氧冷损伤发生密切相关 ,可望通过抗氧化...

目的 观察缺氧寒冷损伤血管一氧化氮合酶 (NOS)活性的变化 ,并研究超氧化物歧化酶 (SOD)和维生素C(VC)对这种变化的影响 ,为缺氧寒冷损伤的防治开拓新的思路。方法 分离Wistar大鼠主动脉并使其暴露于缺氧或 /和寒冷环境中 ,以全自动生化分析仪测定血管培养液中乳酸脱氢酶 (LDH)活性 ,以Griess化学法观测血管NOS活性 ,以肾上腺素自氧化法测定血管SOD活性。结果 缺氧和寒冷损伤使血管NOS活性较对照组显著下降 ,分别达 18 2 %和 19 1% ,缺氧寒冷复合损伤更使血管NOS活性下降达 2 5 9% ;NOS的这种变化具有明显的时间依赖性 ,并与相应血管培养液中LDH活性有显著负相关关系 ;缺氧或 /和寒冷损伤血管SOD活性亦显著下降。当血管受到缺氧或 /和冷冻后立即给与SOD(2 0 0u/ml)或VC(5 0mg/ml)可使缺氧或 /和寒冷损伤血管NOS活性升高 ,SOD活性复升 ;对血管培养液中LDH的测定表明 ,SOD和VC对缺氧冷损伤血管具有一定的保护作用。结论 血管NOS活性的降低与缺氧冷损伤发生密切相关 ,可望通过抗氧化剂的使用 ,防止或减缓缺氧寒冷环境中机体损伤的发生

 
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