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胞浆内钙离子
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  cytosolic free calcium
     Low intensity He-Ne laser can decrease the cytosolic free calcium concentration of erythrocytes. But no does relation is showed in this experiment.
     结果显示:低强度He-Ne激光照射后的红细胞与无照射组的红细胞相比,红细胞胞浆内钙离子浓度显著降低,但在本实验中钙离子浓度的降低与激光照射剂量无显著相关。
短句来源
     To evaluate the effects and mechanism of Capto pril on the BP of 2-kidney,1-clip renovascular hypertensive rats (2KlC-RHR) in the two phases (acute and chronic phase,respecticely),we measure platelets cytosolic free calcium concentration([Ca2+]i),platelets aggregation(PAg) and plasma angiotensin Ⅱ(AⅡ)levels.
     应用二肾一夹型肾血管性高血压大鼠(2KlC-RHR)模型,观察在高血压稳定期血小板胞浆内钙离子浓度([FCa2+]i)、血小板聚集率及血浆AⅡ的变化,在此基础上研究血管紧张素转化酶抑制剂一卡托普利不同给药阶段的作用。
短句来源
     The conclusion is that low intensity He-Ne laser may increase human erythrocyt deformability by decreasing the cytosolic free calcium concentration,which is effective to improve the microcirculation of patients with hypertension whose initial cytosolic free calcium concentration is higher than normal.
     由此得出结论:降低红细胞胞浆内钙离子浓度可能是低强度He-Ne激光调节红细胞变形性的重要机制。
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  “胞浆内钙离子”译为未确定词的双语例句
     Objective:To study the effects of protease-activated receptor-2(PAR-2)agonists(SLIGKV and tc-LIGRLO),trypsin and its inhibitors[soybean trypsin inhibitor(SBTI)and α_1-antitrypsin(α_1-AT)]on intracellular calcium concentration([Ca~(2+)]_i)in A549 lung epithelial cell.
     目的:研究蛋白酶激活受体(PAR)2激动剂(SLIGKV和tc LIGRLO)、胰蛋白酶及其抑制剂(SBTI、α1AT)对A549肺上皮细胞胞浆内钙离子浓度([Ca2+]i)的影响。
短句来源
     Results After 10min ischemia-like condition,a rapid increase in △FI was observed(17.52±3.16).
     结果 给予缺血处理 10min后 ,神经元胞浆内钙离子△FI明显升高(17.5 2± 3.16 ) ;
短句来源
     Moreover,it was discovered that the increase in intracellular free calcium concentration and the excessive phosphorylation of CaMK Ⅱ during apoptosis of PC12 cells induced by Glu could be inhibited by serum with BS in 20% concentration.
     20%浓度BS含药血清能够拮抗谷氨酸诱导凋亡的PC12细胞胞浆内钙离子浓度的升高,抑制CaMKⅡ的磷酸化水平增高。
短句来源
     The insulin interact with β cell directly by insulin receptor system, which inhibits the sarco (endo) plasmic reticulum Ca2 + -ATPase (SERCA) , increases cytosolic Ca2+ , and induces insulin secretion.
     胰岛素通过存在于β细胞的胰岛素受体信号转导系统直接作用于β细胞,抑制其内质网上的Ca2+-ATP酶活性,使胞浆内钙离子浓度升高,激活蛋白激酶C,诱导胰岛素分泌,对β细胞分泌起正反馈调节;
短句来源
     The Influence of TNF-αand LPS on Plasmic Inner Concentration of Ca~(2+) of Blood-brain Barri-er Endothelial Cells
     TNF-α和LPS对体外培养血脑屏障内皮细胞胞浆内钙离子浓度的影响
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  相似匹配句对
     Correlation Reseach of Concentration Diversity of Intracytoplasm Calcium Ion and Cell Apoptosis in Neuron
     神经元胞浆离子浓度变化和细胞凋亡相关性的研究
短句来源
     Neuronal apoptosis and cytoplasmic calcium
     神经细胞凋亡与胞浆离子
短句来源
     The platelet cytoplasmic Ca 2+ content was monitored using platelet ionized calcium aggregometer.
     利用血小板离子聚集仪检测胞浆含量。
短句来源
     The effect of estrogen on intracellular calcium concentration in human spermatozoa
     雌激素对人精子胞浆游离离子浓度的影响
短句来源
     Effect of Oral Take L-Arginine on [Ca~(2+) ]_i of Human Platelets
     口服左旋精氨酸对血小板胞浆离子浓度的影响
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  cytosolic free calcium
Role of increased cytosolic free calcium concentration in myocardial ischemic injury
      
Increases in cytosolic free calcium concentration ([Ca2+]I) may play an important role in myocardial ischemic injury.
      
Hypoxia increases cytosolic free calcium in porcine pulmonary arterial endothelial cells
      
Alteration of cytosolic free calcium ([Ca2+1]) in hypoxia was studied with fluorescent probe, Fura-2 in cultured porcine pulmonary arterial endothelial cells.
      
Effects of ATP on cytosolic free calcium ([Ca2+]i) in single porcine pulmonary artery endothelial cell were studied.
      
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To evaluate the effects and mechanism of Capto pril on the BP of 2-kidney,1-clip renovascular hypertensive rats (2KlC-RHR) in the two phases (acute and chronic phase,respecticely),we measure platelets cytosolic free calcium concentration([Ca2+]i),platelets aggregation(PAg) and plasma angiotensin Ⅱ(AⅡ)levels. The results are as follows:(1)captopril reduce the BP significantly while A Ⅱ level decrease in the acute phase,with no changes in [Ca2 +]i and PAg;(2)Captopril has obvious antihypertension in the chronic...

To evaluate the effects and mechanism of Capto pril on the BP of 2-kidney,1-clip renovascular hypertensive rats (2KlC-RHR) in the two phases (acute and chronic phase,respecticely),we measure platelets cytosolic free calcium concentration([Ca2+]i),platelets aggregation(PAg) and plasma angiotensin Ⅱ(AⅡ)levels. The results are as follows:(1)captopril reduce the BP significantly while A Ⅱ level decrease in the acute phase,with no changes in [Ca2 +]i and PAg;(2)Captopril has obvious antihypertension in the chronic phase,with [Ca2+]i and PAg decreasing markedly.In conclusion,the results indicate that there are abnormality of the intracellular calcium and platelets activity during the sustain period of hypertension in the 2KlC-RHR, the antihypertension effect of Captopril during the chronic phase may partly be attributed to its inhibition effect on platelets activity.

应用二肾一夹型肾血管性高血压大鼠(2KlC-RHR)模型,观察在高血压稳定期血小板胞浆内钙离子浓度([FCa2+]i)、血小板聚集率及血浆AⅡ的变化,在此基础上研究血管紧张素转化酶抑制剂一卡托普利不同给药阶段的作用。发现近期实验虽然显著降低血压,血浆AⅡ减少,但血小板内[Ca2+]i,血小板聚集率没有变化;远期实验在显著降低血压的同时血小板内[Ca2+]i、血小板聚集率明显减少(P<0.01)。提示2KlC-RHR在高血压稳定期存在细胞内钙离子代谢异常和血小板活性的改变,血小板活性的降低可能参与卡托普利的远期降压作用。

Objective: To investigate the effect of Poria cocos, a Chinese medicinal herb, on cytosolic free calcium concentration (〔Ca 2+ 〕i) in brain nerve cells of neonatal rats. Methods: Double wave length fluorospectrophotometer and laser confocal scanning microscope were used to measure the 〔Ca 2+ 〕i in neurons. Results: The water extract of poria (31~250mg/L) induced a dose dependent increase in 〔Ca 2+ 〕i, but poria at the range of 500mg/L did not cause 〔Ca 2+ 〕i to increase. It was also shown...

Objective: To investigate the effect of Poria cocos, a Chinese medicinal herb, on cytosolic free calcium concentration (〔Ca 2+ 〕i) in brain nerve cells of neonatal rats. Methods: Double wave length fluorospectrophotometer and laser confocal scanning microscope were used to measure the 〔Ca 2+ 〕i in neurons. Results: The water extract of poria (31~250mg/L) induced a dose dependent increase in 〔Ca 2+ 〕i, but poria at the range of 500mg/L did not cause 〔Ca 2+ 〕i to increase. It was also shown that the water extract of poria significantly inhibited the increment of 〔Ca 2+ 〕i induced by 500μmol/L glutamate. The similar results were observed from the experiments in primarily cultured hippcampal neurons of neonatial rats by using laser confocal scanning microscope. Conclusions: The water extract of Poria cocos has the effect of bidirectional regulation on cytosolic free calcium in brain nerve cells.

目的 :探讨茯苓水提液对神经细胞内钙稳态的影响。方法 :应用双波长荧光分光光度计和激光共聚焦扫描显微镜测定新生大鼠大脑皮层神经细胞悬液和海马神经元原代培养在加入茯苓水提液后细胞内钙离子浓度的变化。结果 :茯苓水提液在 3 1~ 2 5 0mg/L时 ,可诱导细胞内钙离子浓度升高 9 9%~ 3 3 7% ,并且这种作用随给药浓度的增大而增强 ;当浓度大于或等于 5 0 0mg/L时 ,无明显升高胞浆内钙离子浓度的作用。 3 1~ 2 0 0 0mg/L茯苓水提液均有不同程度抑制 5 0 0 μmol/L谷氨酸诱导的升钙作用。 结论 :茯苓水提液对神经细胞内钙离子浓度具有双向调节作用。

Objective To investigate the effects of propofol on intracellular Ca 2+ mobilization and L type calcium current in rat isolated cardiac myocytes.Methods Using Fluorescent Ca 2+ indicator Fluo 3AM and patch clamp techniques(whole cell mode),we measured the influences of propofol,at 50μmol·L -1 or 250μmol·L -1 ,on the changes of intracellular Ca 2+ concentration induced by high concentraton KCl or caffeine,and L type Ca 2+ current...

Objective To investigate the effects of propofol on intracellular Ca 2+ mobilization and L type calcium current in rat isolated cardiac myocytes.Methods Using Fluorescent Ca 2+ indicator Fluo 3AM and patch clamp techniques(whole cell mode),we measured the influences of propofol,at 50μmol·L -1 or 250μmol·L -1 ,on the changes of intracellular Ca 2+ concentration induced by high concentraton KCl or caffeine,and L type Ca 2+ current simutaneously in isolated cardiac myocytes in rats.Results 50μmol·L -1 or 250μmol·L -1 propofol attenuated KCl induced calcium influx significantly in dose dependent manner and decreased L type calcium peak current for 24.8% and 49.6% respectively(P<0.05,n=5).Propofol seemed to have no effect on the Ca 2+ increase released by sarcoplasmic reticulum induced by caffeine.Influx of Ca 2+ went back to the basic level after the removal of propofol.Conclusion The negative inotropic effect of propofol seems to be,at least in part,due to decreasing Ca 2+ influx and inhibiting L type Ca 2+ current.And propofol shows little effect on sarcoplasmic reticulum function.

目的 观察异丙酚对心肌细胞钙离子移动和L-型钙通道电流(Ical)的影响。方法 急性分离的大鼠单个心室肌细胞,采用Fluo-3AM 钙荧光指示剂和膜片钳技术,观察50μm ol·L- 1 或250μm ol·L- 1异丙酚对高浓度KCl或咖啡因所诱发细胞内钙离子浓度的变化,以及对L-型钙通道电流的影响。结果 50μm ol·L- 1或250μm ol·L- 1 异丙酚明显减弱KCl所诱发细胞内钙离子的增加,浓度越高其减弱作用越明显;而对咖啡因所致胞浆内钙离子的增加无明显影响。使钙通道电流峰值分别较用药前下降24.8% 和49.6% (P< 0.05,n= 5)。洗去异丙酚后Ica 可基本恢复至基础水平。结论 异丙酚的负性肌力作用至少部分是由于其影响心肌细胞胞膜功能,减少钙离子跨膜内流以及抑制细胞膜缓慢内流的L-钙通道电流所致,而对肌浆网功能似乎无明显作用。

 
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