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   癌组织 在 口腔科学 分类中 的翻译结果: 查询用时:0.019秒
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癌组织
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  carcinoma of
    The expression of PCNA and AgNOR in human papilloma and squamous cell carcinoma of tongue
    舌乳头状瘤及鳞状细胞癌组织的PCNA及AgNOR表达
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    Expression of nitric oxide synthase in squamous cell carcinoma of tongue and its prognosis
    一氧化氮合酶在舌鳞癌组织中的表达及其与预后的关系
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    EXPRESSION AND SIGNIFICANCE OF CD44v6 PROTEIN IN SQUAMOUS CELL CARCINOMA OF THE TONGUE
    CD44v6蛋白在舌鳞癌组织中的表达及其意义
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    Expression and clinicopathologic significance of Cyclin D1 in squamous cell carcinoma of tongue
    舌鳞癌组织中Cyclin D1的表达与临床病理相关性
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    Cyclin A mRNA and cyclin D1 mRNA overexpression in squamous carcinoma of the tongue
    舌鳞癌组织中cyclinA和cyclin D1的基因转录
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  “癌组织”译为未确定词的双语例句
    Study of DNA Ploidy in Oral Leukoplakia and Squamous Cell Carinomas
    口腔白斑及鳞癌组织中DNA倍体值的研究
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    Patients with HIF-1α over-expression had poor prognosis than patients with HIF-1α lower expression.
    预后差的颊癌组织中,HIF-1α的阳性表达水平明显高于预后好组(P<0.05)。
短句来源
    Conclusion:The low expression of P33~(ING1) may be an important factor related to carcinogenesis and development of OSCC.
    结论:P33ING1蛋白在口腔鳞状细胞癌组织中的表达下降,提示P33ING1蛋白可能在口腔鳞癌的发生发展中有重要意义。
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    Methods:The expression of TNF-α was assessed by immunohistochemical method of SABC in paraffin tissue sections of 41 cases of OSCC and 10 of normal oral epithelia.
    方法:应用免疫组化SABC法检测41例口腔鳞癌组织中TNF-α的表达,并用10例正常口腔黏膜组织作为对照。
短句来源
    ResultsPositive rates of hMLH1 and hMSH2 were lower in OSCC(47.8% and 55.1% respectively)than in normal oral mucosa(77.5% and 80.0% respectively). These differences between OSCC and normal oral mucosa were statistically significant(P<0.05).
    结果口腔鳞状细胞癌组织中hMLH1、hMSH2的阳性表达率(47.8%,55.1%)低于正常口腔粘膜组织(77.5%,80.0%),P<0.05,有显著差异。
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  carcinoma of
All three cases were diagnosed as papillary carcinoma of thyroid origin after microscopic examination.
      
Primary thyroglossal duct carcinoma should conform to the following criteria: localization of the carcinoma to a clearly demonstrable thyroglossal duct cyst or tract; clinically or histologically confirmed absence of carcinoma of the thyroid gland.
      
Regulation of matrix metalloproteinase-9 transcription in squamous cell carcinoma of uterine cervix: the role of human papilloma
      
The NOLA2 and RPS3A genes as highly informative markers of human squamous cell carcinoma of lung
      
20,29-Dihydro-20,29-dichloromethylenebetulinic acid proved to be the most cytotoxic toward human melanoma of the Colo 38 and Bro lines and human ovarian carcinoma of the CaOv line (IC50 10 μM).
      
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Abstract Twelve pleomorphic adenomas and eight myoepitheliomas were studied by an immunohistochemistry with antiactin,myosin, S-100 protein and glial fibrillary acidic protein(GFAP) antibodies. The results indicated that in pleomorphic adenoma, actin, myosin, S-100 protein and GFAP expression were seen in the stellate, triangle cells in the myxoid region and in the basal layer cells of the double-layered duct structure, these tumor cells were regarded as neoplastic myoepithelial cells,the squamous metaplasia...

Abstract Twelve pleomorphic adenomas and eight myoepitheliomas were studied by an immunohistochemistry with antiactin,myosin, S-100 protein and glial fibrillary acidic protein(GFAP) antibodies. The results indicated that in pleomorphic adenoma, actin, myosin, S-100 protein and GFAP expression were seen in the stellate, triangle cells in the myxoid region and in the basal layer cells of the double-layered duct structure, these tumor cells were regarded as neoplastic myoepithelial cells,the squamous metaplasia cells were positive in the tumors; myoepithelioma cells were identified by weak to intense actin, myosin, S-100 protein and GFAP positive reaction. This fact suggested that (1) in pleomorphic adenoma, neoplastic myoepithelial cells could transform into the squamous metaplasia cells, if malignant changes occur, they will become squamous cell carcinoma by squamous metaplasis;(2) neoplastic myoepithelial cells of pleomorphic adenoma were the same immunofeature as myoepithelioma cells, more than 5%~10% of ductal components of pleomorphic adenoma is principle differential from pleomorphic adenoma and myoepithelioma.

采用抗平滑肌的肌动蛋白(actin)、肌球蛋白(myosin);S-100蛋白以及胶质纤维酸性蛋白(GFAP)抗体研究涎腺肌上皮瘤和多形性腺瘤。结果发现,肌上皮瘤细胞对上述4种抗体呈不同程度的阳性反应。在多形性腺瘤中,双层导管结构的外周瘤细胞,成片增生的梭形细胞以及粘液样区域内的星形、三角形瘤细胞对上述4种抗体反应阳性,鳞状化生的瘤细胞也反应阳性,导管内层瘤细胞对上述4对抗体反应阴性。本文研究结果表明:①在多形性腺瘤中,肿瘤性肌上皮细胞可以出现鳞状化生,可以通过鳞状化生形成鳞状细胞癌,成为恶性多形性腺瘤中鳞状细胞癌的组织来源;②肌上皮瘤和多形性腺瘤中肿瘤性肌上皮细胞具有相同的免疫特性和分化趋向,用上述抗体不能鉴别这两种肿瘤。

he polymeras chain reaction(PCR) was used to amplify the DNA sequence of

应用聚合酶链反应(PCR)放大口腔癌组织中H-ras癌基因部分DNA顺序,再以特异突变的寡脱氧核苷酸(ODN)探针杂交方法检测口腔癌中H-ras癌基因第12位密码子的G→T突变。结果在21例口腔癌中有6例存在这种突变(28.5%),而10例正常门腔粘膜组织中无1例发生突变。提示:H-ras癌基因第12位密码子→T突变可能是口腔癌中癌基因激活的方式之一。

Theceluardiferentiationbehaviourofadenoidcysticcarcinoma(ACC)ofsalivaryglandwasinvestigatedinvitrowithimmunohistochemistry.TheresultsshowedthatthecelsofACC-2andACC-3cellinescanexpresssomemarkerproteinsinvitroincludingS-100protein,actin,myosinwhichischaracterizedasafeatureofmyoepithelialcels.Whendiferentiationinducer,dB-cAMP,wasaddedtothemonolayercelsculturedindishes,themarkerproteinexpresionincreasedandtheCEAchangedtopositive.ThecelulardiferetiationbehaviourofACCcelsisrelatedtotheproliferatespeedofcels.Somecelsmanifestedfingerring-likewhenthecultureconditionwaschanged,itmeansthatthesecelshavethesecretoryfunction.IftheACCcelsweresuspendedsinglyincolagengelswhichperformedasanextracelularmatrixtheygrewandformedthetubule-likestructure.TheresultswilhelpustounderstandthehistogenesisofACC,thesourceofmyoep-ithelialcelsinACCandthevarietyofhistologicalstructuresofACC....

Theceluardiferentiationbehaviourofadenoidcysticcarcinoma(ACC)ofsalivaryglandwasinvestigatedinvitrowithimmunohistochemistry.TheresultsshowedthatthecelsofACC-2andACC-3cellinescanexpresssomemarkerproteinsinvitroincludingS-100protein,actin,myosinwhichischaracterizedasafeatureofmyoepithelialcels.Whendiferentiationinducer,dB-cAMP,wasaddedtothemonolayercelsculturedindishes,themarkerproteinexpresionincreasedandtheCEAchangedtopositive.ThecelulardiferetiationbehaviourofACCcelsisrelatedtotheproliferatespeedofcels.Somecelsmanifestedfingerring-likewhenthecultureconditionwaschanged,itmeansthatthesecelshavethesecretoryfunction.IftheACCcelsweresuspendedsinglyincolagengelswhichperformedasanextracelularmatrixtheygrewandformedthetubule-likestructure.TheresultswilhelpustounderstandthehistogenesisofACC,thesourceofmyoep-ithelialcelsinACCandthevarietyofhistologicalstructuresofACC.

为探讨涎腺腺样囊性癌(adenoidcysticcarcinoma,ACC)中肌上皮细胞的来源和此癌的组织发生,对其细胞的一些分化行为进行了实验观察。用ABC免疫组织化学方法,证明了ACC-2和ACC-3细胞系细胞,在体外能不同程度地表达肌上皮细胞特有的S-100蛋白、肌动蛋白(actin)和肌浆球蛋白(myosin)成分;加入分化诱导剂dB-cAMP后,可使上述成分表达增强并使癌胚抗原(carci-noembryonicantigen,CEA)由阴性转阳性。作者还观察到ACC细胞的分化与其增殖速度呈负相关关系;改变细胞生长环境时,部分细胞呈“印戒”样,表明其具有分泌功能;将单个细胞悬浮生长于胶原凝胶为基质的培养基可形成典型的管样结构。作者认为,本研究结果支持ACC的组织发生来自闰管细胞的观点,对认识该肿瘤的发生很有参考价值。

 
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