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水平
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    CLINICAL OBSERVATION OF SVATE ON 34 CASES OF THROMBOSIS AND CONCENTRATION OF TXB_2 6-keto-FGF_(1α)
    江浙蝮蛇抗栓酶对脑血栓形成患者血浆TXB_2、6-KeTo-PGF_(1α)水平的影响
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    The study to experiment rabbit's cerebral ischemia their free radical neuropathology and the mechanism of mannital
    实验性脑缺血组织自由基水平、神经病理与甘露醇作用机制的研究
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    CLINICAL ANALYSIS OF BLOOD LIPID ON CEREBROVASCULAR DISEASE
    脑血管病血脂水平临床分析
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    Results:①Estrogen depressed FADD mRNA[(49.67±0.68)% vs(67.36±0.2)%,P<0.01]and protein expression of ischemic cortex[(37.50±1.68)% vs(67.46±1.41)%,P<0.01];
    结果:①雌激素抑制缺血侧皮质升高的FADD mRNA[(49.67+0.68)%vs(67.36±0.2)%,P<0.01]和蛋白表达水平[(37.50±1.68)%vs(67.46+1.41)%,P<0.01],具有显著性统计学意义。
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    Following a 3 h exposure,the expression of N-methyl-D-aspartate receptor 1(NR1) mRNA and protein were measured at different culture days (7,12,17) using real-time PCR and Western blot assays.
    结果在正常DMEM培养液中,NR1mRNA与蛋白的表达在培养7d时处于较高水平,以后逐渐下降,以培养12d为最低。
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(1) The sciatic and medial nerves are stimulated by rectangular impulses with. a definite strength and duration(so called etectro acupuncture), and a narcotic state of animal is induced within 20-30 minutes, during which the animal shows no response to nociceptive stimuli. (2) The blood pressure rises immediately after, the application of electroacuptuncture, and then retnrns to about control level although the electro-acupuncture lasts. with the withdrawal of etecfro-acu Punoture, the bood pressure is completely...

(1) The sciatic and medial nerves are stimulated by rectangular impulses with. a definite strength and duration(so called etectro acupuncture), and a narcotic state of animal is induced within 20-30 minutes, during which the animal shows no response to nociceptive stimuli. (2) The blood pressure rises immediately after, the application of electroacuptuncture, and then retnrns to about control level although the electro-acupuncture lasts. with the withdrawal of etecfro-acu Punoture, the bood pressure is completely recovered, it is therefore shown that electro-acuPuncture does not exert undesirable action on the cardiovascular system. (3) The pressor reflex caused by occlusion of carotid artery shows an exaggeration during the course of elect ro-acupuncture and restores to control leel as the electro- acupuncture is discontinued. (4) During the course of electroacupuneture, the pressor reflex elicited by stimulation of central end of depressor nerve with various strength of Fa adic curreut reveals typical phasic changes of Wedensky s parabiotic inhibition. (5) On the basis of above observations it is suggested that the physiological mechanism of the electro-acupunetare narcosis is the development of parabiosis in the nerve centers as the consequence of prolonged actionof afferent impules from eletroacupuncture regions.

(一)用一定强度和时间的电针刺激家兔,能引起动物的麻醉状态,使动物对一般损伤性刺激不出现反应;停止电针后10——15分钟,动物对损伤性刺激的反应恢复。 (二)动脉血压在电针刚开始时突然增高,随后恢复至接近正常水平,电针停止后血压完全恢复,由此表明电针本身对心臟——血管系统并无不良影响。 (三)阻塞一侧颈总动脉所致的加压反射,在电针过程中表现加强,电针停止后又恢复正常。 (四)以感应电刺激减压神经向中段所致的减压反射,在电针过程中有特異性变化,表现为减压神经强刺激引起的减压效应较针前为弱,而弱刺激的效应反较针前为强,并出现了均等和反常的间生性效应。 (五)从本实验的结果来看,电针麻醉的机制是在于电针刺激的大量冲动,引起了神经中枢间生态的发展,并且这种发展取决于电针刺激的强度和时间,而在各动物之间有一定的个体差異。

Using different ways to increase the intracranial pressure (ICP) of cats, we have observed the changes in the electroencephalogram (EEG) and electrocardiogram (EKG). The results are as follows:1. When ICP is increased step by step to 30-40 mmHg the frequency of EEG increases and the amplitude is lowered. When ICP is increased to 50-70 mmHg the slow wave predominates. Finally, the cortical electric activities disappear when ICP is increased to the level of the blood pressure (BP). There exists a close correlation...

Using different ways to increase the intracranial pressure (ICP) of cats, we have observed the changes in the electroencephalogram (EEG) and electrocardiogram (EKG). The results are as follows:1. When ICP is increased step by step to 30-40 mmHg the frequency of EEG increases and the amplitude is lowered. When ICP is increased to 50-70 mmHg the slow wave predominates. Finally, the cortical electric activities disappear when ICP is increased to the level of the blood pressure (BP). There exists a close correlation between the changes of EEG and the cerebral perfusion pressure (CPP) which is the difference between the mean arterial pressure and the ICP. CPP is decreased both by increase of ICP and by decrease of BP caused by heart failure.2. When ICP is increased quickly to half level of BP no obvious change appears in EEG, if CPP is not below 70-80 mmHg. If CPP decreases with the drop of BP, there will be obvious changes in EEG which will finally develop into electric silence.3. When ICP is rapidly increased to the level approaching BP, EEG disappears within 16-17 minutes.4. An increase in ICP will lower the heart rhythm and cause changes in BP and EKG. The changes of EKG will take place with normal or abnormal BP. This phenomenon might be related to the excitability of the cardiovascular centre.

在猫上用不同方式升高颅内压,观察脑电图和心电图的变化,结果表明: 1.阶梯性升高颅内压至30—40毫米汞柱时,脑电波频率加快,振幅降低。颅内压升高至50—70毫米汞柱时,以慢波占优势。当颅内压升高接近血压水平时,脑电活动消失。脑电图的变化与脑灌注压有密切关系,除颅内高压可以引起脑灌注压下降外,心脏功能的障碍也可促使血压下降,从而致使脑灌注压降低。 2.急骤升高颅内压至血压的二分之一水平时,如脑灌注压不低于70—80毫米汞柱,脑电图上不出现明显异常。如脑灌注压因血压的下降而迅速降低时,脑电图将出现明显异常,直至脑电波消失。 3.颅内压急骤升高至血压相近水平时,脑电活动在16—17分钟时即发生消失。 4.颅内高压可以引起心率减慢、血压和心电图异常;心电图异常改变可发生在血压异常升高后,也有发生在血压正常时,这可能与心血管中枢的兴奋性改变有关。

89 young male subjects were exposed to altitute in a hypobaric chamber for 30-120 minutes in sedentary position. Circulatory parameters were recorded during the exposure and then comprehensive quantitative evaluation was performed by using compensatory rate Z value and degree of dysfunction S. It was found that the circulatory compensatory dysfunction occurred only at simulated altitude of 5,000 and 6,000 m with rates of occurrence amounting 28.9% and 55.0% respectively. The main features of dysfunction were...

89 young male subjects were exposed to altitute in a hypobaric chamber for 30-120 minutes in sedentary position. Circulatory parameters were recorded during the exposure and then comprehensive quantitative evaluation was performed by using compensatory rate Z value and degree of dysfunction S. It was found that the circulatory compensatory dysfunction occurred only at simulated altitude of 5,000 and 6,000 m with rates of occurrence amounting 28.9% and 55.0% respectively. The main features of dysfunction were marked slowing of heart rate, acute lowering of blood pressure, whereas lowering of compensatory rate Z to below -20, arrhythmias appeared in majority of subjects of dysfunction. As the dysfunction became severe, symptoms such as pallor, cold sweating, nausea and asthenia accompanied. Since all the symptoms disappeared immediately after oxygen inhalation,the dysfunction was physiological in nature. It was believed that the mechanism of circulatory dysfunction was originated from the abnormality of regulation and control of higher nervous system, i. e. the normal domination of sympathetic nerve changed to that of vagus nerve, thus the dysfunction of circulatory system resulted accordingly.

青年健康受试者89名,取安静坐位。在低压舱3、4、5、6、7千米模拟高度上停留30~120分钟,运用代偿率Z值和障碍度S对循环各指标进行综合性定量评定。循环障碍仅发生于5,000与6,000米,发生率分别为28.9%与55.0%。主要表现特征是心率锐减,血压急降,代偿率低达障碍水平Z≤-20以下,大多数障碍者伴有心律紊乱,严重时伴有苍白、恶心、冷汗与无力等。均属生理性障碍。吸入氧气后迅速消除。产生机理设想为高层次脑中枢首先调节失常,进而导致植物神经功能紊乱,由正常的交感神经占优势转为迷走神经张力过度增强,致使循环代偿功能随之障碍。

 
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