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肝纤维化     
相关语句
  liver fibrosis
    Liver fibrosis and cytokines
    肝纤维化与细胞因子
短句来源
    Primary Study of Expression of α_1 ( Ⅰ ) and α_1 (Ⅲ) Procollagen mRNA and Relevant Collagens of Early Rat Liver Fibrosis
    大鼠早期肝纤维化α_1(Ⅰ)、α_1(Ⅲ)前胶原mRNA及相应胶原蛋白表达的初步研究
短句来源
    Study on cells associated with human liver fibrosis
    人肝纤维化相关细胞的研究
短句来源
    DYNAMIC STUDY OF α1(Ⅰ)、α1(Ⅲ)AND α1(Ⅳ)PROCOLLAGEN GENE IN CCI_4-INDUCED EXPERIMENTAL LIVER FIBROSIS
    CCl_4诱导大鼠肝纤维化过程中α1(Ⅰ)、α1(Ⅲ)及α1(Ⅳ)前胶原基因表达的动态观察
短句来源
    HETEROSERUM-INDUCED RAT LIVER FIBROSIS MODEL AND ITS MECHANISM
    异种动物血清诱导的大鼠肝纤维化模型及其机制
短句来源
更多       
  hepatic fibrosis
    Inhibitory Effect of Vitamin A on Experimental Hepatic Fibrosis in Rats
    维生素A阻抑大鼠实验性肝纤维化的病理研究
短句来源
    Expression of Vasoactive Intestinal Peptide Receptor in Rat Liver Lipocytes and Its Relevance in Hepatic Fibrosis
    血管活性肠肽受体在大鼠肝脏贮脂细胞的表达及其在肝纤维化中的意义
短句来源
    Objective To investigate the effect of ultrasound contrast agent and ultrasound irradiation on TIMP-1 siRNA expression in liver of hepatic fibrosis rats.
    目的观测超声造影剂及超声辐照对TIMP-1 siRNA质粒转染肝纤维化大鼠肝组织内TIMP-1基因表达的影响。
短句来源
    Studies on the Mechanism of Anti-Schistosome Hepatic Fibrosis Induced by SCP Combining Reagent
    鲨鱼软骨粉(SCP)合剂诱导的抗血吸虫性肝纤维化效应机制的研究
短句来源
    Experimental study on protective effects of tetrandrine on hepatic fibrosis due to biliary obstruction
    粉防己碱对胆汁性肝纤维化保护作用的实验研究
短句来源
更多       
  fibrosis
    Liver fibrosis and cytokines
    肝纤维化与细胞因子
短句来源
    Inhibitory Effect of Vitamin A on Experimental Hepatic Fibrosis in Rats
    维生素A阻抑大鼠实验性肝纤维化的病理研究
短句来源
    Primary Study of Expression of α_1 ( Ⅰ ) and α_1 (Ⅲ) Procollagen mRNA and Relevant Collagens of Early Rat Liver Fibrosis
    大鼠早期肝纤维化α_1(Ⅰ)、α_1(Ⅲ)前胶原mRNA及相应胶原蛋白表达的初步研究
短句来源
    Study on cells associated with human liver fibrosis
    人肝纤维化相关细胞的研究
短句来源
    Expression of Vasoactive Intestinal Peptide Receptor in Rat Liver Lipocytes and Its Relevance in Hepatic Fibrosis
    血管活性肠肽受体在大鼠肝脏贮脂细胞的表达及其在肝纤维化中的意义
短句来源
更多       
  experimental liver fibrosis
    DYNAMIC STUDY OF α1(Ⅰ)、α1(Ⅲ)AND α1(Ⅳ)PROCOLLAGEN GENE IN CCI_4-INDUCED EXPERIMENTAL LIVER FIBROSIS
    CCl_4诱导大鼠肝纤维化过程中α1(Ⅰ)、α1(Ⅲ)及α1(Ⅳ)前胶原基因表达的动态观察
短句来源
    Synthesis and degradation of type Ⅳ collagen in rat experimental liver fibrosis
    大鼠肝纤维化过程中Ⅳ型胶原合成与降解的变化
短句来源
    Effects of prevention aad treatment of tetramethylpyrazine on experimental liver fibrosis in rats
    四甲基吡嗪对实验性大鼠肝纤维化防治作用的研究
短句来源
    Alterations of interstitial collagenase activity in the process of experimental liver fibrosis
    实验性肝纤维化过程中间质胶原酶活性的动态变化
短句来源
    Expression of hepatic cytochrome P450-2E1 in experimental liver fibrosis.
    肝细胞色素P450 2E1在实验性肝纤维化组织中的表达
短句来源
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  liver fibrosis
Conversely, HAART-related hepatotoxicity may enhance the progression of liver fibrosis.
      
Objective: To study the effect of Xuesaitong soft capsule (XST) on liver fibrosis criteria in patients with post-hepatitis fibrosis.Methods: Sixty-four patients with such fibrosis were randomly divided into the treated group and control group.
      
Their liver fibrosis criteria were examined before and after treatment.Results: The levels of serum procollagen III, hyaluronic acid, collagen IV, laminin in the two groups were significantly lower (P>amp;lt;0.
      
The differences between the two groups were insignificant (P>amp;gt;0.05).Conclusion: XST could recover liver dysfunction and had anti-liver fibrosis function.
      
However, response rates are much lower in patients who are difficult to treat, such as those infected with hepatitis C virus genotype 1, high viral load, or advanced liver fibrosis.
      
更多          
  hepatic fibrosis
The Ile50Val polymorphism of the IL4RA gene was tested for association with chronic viral hepatitis and the character of its progression (the stage of hepatic fibrosis).
      
However, a significant difference in genotype frequency distribution was observed for the patients with different stages of hepatic fibrosis.
      
In hepatic fibrosis, the connective tissue biomatrix of the liver changes from the normal matrix, rich in basement membrane collagens, to a matrix enriched in interstitial fibrillar collagens.
      
Viral hepatitis, hepatic fibrosis and hepatocirrhosis etc.
      
Clinical study of Dahuang Zhechong pill in treating posthepatitis B hepatic fibrosis
      
更多          
  fibrosis
Hepatic steatosis, lobular inflammation, hepatocytic ballooning and fibrosis were presented widespread in NAFLD liver tissues.
      
Mild perisinusoidal fibrosis and periportal fibrosis were often observed in stage 1 cases.
      
According to the statistic analysis, hepatic steatosis was positively correlated with lobular inflammation, hepatocytic ballooning and fibrosis (r = 0.587, 0.488, 0.374, respectively, all P value >amp;lt; 0.01).
      
The number of microgranulomas, lipogranulomas and apoptotic bodies increased following severity of steatosis, lobular inflammation and fibrosis.
      
We suggest that the role of portal inflammation should be emphasized besides hepatic steatosis, lobular inflammation, hepatocyte ballooning and fibrosis in diagnosis and evaluation of NAFLD.
      
更多          
  experimental liver fibrosis
Angiostatin inhibits experimental liver fibrosis in mice
      
Enzymohistochemical studies on the liver tissue of rats with experimental liver fibrosis and of the patients with active chronic
      
Studies on liver cirrhosis-effect of elastase on development and recovery of experimental liver fibrosis
      
Studies on experimental liver fibrosis in rats; suppressive effects on an occurrence of liver fibrosis by antiserum prepared fro
      
Effect of elastoproteinase on development and recovery of experimental liver fibrosis (II)
      
更多          
  其他


The inhibitory effect of vitamin A on experimental hepatic fibrosis in rats induced by ad-ministration of carbon tetrachloride was studied with light,electron microscope,immunohistochemical ??and morphologic quantitative technique.The results of investigation indicated that vitamin A could de-crease the deposit of fibronectin,type Ⅰ and type Ⅲ collagen in liver,inhibit transformation of thefat storing cell to fibroblast and alleviate hepatic fibrosis in rats induced by administration of carbontertrachloride.In...

The inhibitory effect of vitamin A on experimental hepatic fibrosis in rats induced by ad-ministration of carbon tetrachloride was studied with light,electron microscope,immunohistochemical ??and morphologic quantitative technique.The results of investigation indicated that vitamin A could de-crease the deposit of fibronectin,type Ⅰ and type Ⅲ collagen in liver,inhibit transformation of thefat storing cell to fibroblast and alleviate hepatic fibrosis in rats induced by administration of carbontertrachloride.In addition,the mechanism and significance of vitamin A inhibition to the hepatic fibro-sis were analysed.

利用光镜、电镜、免疫组化和形态学定量技术动态研究维生素A对大鼠四氯化碳肝纤维化的抑制作用。结果表明,维生素A可减少四氯化碳中毒大鼠肝内纤维连接蛋白和Ⅰ、Ⅲ型胶原沉积,抑制贮脂细胞向成纤维细胞转化,并可明显地减轻肝纤维化程度。本文还对维生素A抑制肝纤维化的机理及意义作了初步探讨。

To ascertain the role of hepatie fibrosis in the formation of portal hyper-tension in cirrhosis, the content of liver collagen was measured by semiautomatic imageanalysis system. The results showed that hepatic collagen area in cirrhotic rat was muchhigher than the normal rat (32390.16±11879.98 vs. 3206.30±864.26μm~2, P<0 .001).The surface-density of hepatic collagen was 1.58±0.43% in the normal, and 15.97±5.86% in the cirrhotics. In the cirrhotic rat, there was a positive correlation betweenportal pressure...

To ascertain the role of hepatie fibrosis in the formation of portal hyper-tension in cirrhosis, the content of liver collagen was measured by semiautomatic imageanalysis system. The results showed that hepatic collagen area in cirrhotic rat was muchhigher than the normal rat (32390.16±11879.98 vs. 3206.30±864.26μm~2, P<0 .001).The surface-density of hepatic collagen was 1.58±0.43% in the normal, and 15.97±5.86% in the cirrhotics. In the cirrhotic rat, there was a positive correlation betweenportal pressure gradient and the surface density of portal tracts (r=0.44, P<0.05 ), orcollagen of hepatic lobule (r=0.79, P<0.01) or the sum of both (r=0.76, P<0.01).These results concluded that hepatic fibrosis is a main factor producing portal hyperten-sion in the rats with CCl_4-induced cirrhosis.

为证实肝纤维化在肝硬化门脉高压形成中的作用,我们用半自动图象分析仪测定了大鼠CCl_4肝硬化的肝脏胶原含量,同时用检压计测定了同组动物的门脉压力,分析了两者的相关件。结果表明,正常大鼠肝内胶原面积为3206.30±864.62μm~2,肝硬化大鼠为32390.16±11879.98μm~2,差异极显著(P<0.001)。前者与参考面积的比值为1.58±0.43%,后者为15.97±5.86%。正常鼠肝脏胶原面积与门脉压力梯度没有相关性,肝硬化鼠血管周围面积(r=0.44,P<0.05)、肝小叶内胶原面积(r=0.79,P<0.01)及肝内胶原总面积(r=0.76,P<0.01)均与门脉压力梯度呈显著正相关。结果提示,在CCl_4肝硬化大鼠,肝纤维化是导致门脉高压的重要原因之一,支持门脉高压形成的后向血流学说。

By the MTT method we found chondroitin sulfate-A (chondroitin-4-sulfate, CS-A)could promote survival and proliferation of mouse NIH-3T3 fibroblasts and human skin fibroblasts. There was a positive correlation between OD values and CS-A concentrations. The result indicated that fibroblasts could grow well in the condition with high level of CS in fibrotic liver. This CS may accelerate fibroplasia. PAP and ABC stainings showed that CS were present on the surface of both kinds of cells, suggesting that CS may be...

By the MTT method we found chondroitin sulfate-A (chondroitin-4-sulfate, CS-A)could promote survival and proliferation of mouse NIH-3T3 fibroblasts and human skin fibroblasts. There was a positive correlation between OD values and CS-A concentrations. The result indicated that fibroblasts could grow well in the condition with high level of CS in fibrotic liver. This CS may accelerate fibroplasia. PAP and ABC stainings showed that CS were present on the surface of both kinds of cells, suggesting that CS may be an integral component of plasma membrane and may take part in cell-cell and cell-matrix interaction.

用MTT法检测了硫酸软骨素A(4-硫酸软骨素,CS-A)对NIH-3T3鼠成纤维细胞和人皮肤成纤维细胞存活与增殖的影响。发现所侧OD值随CS-A浓度的增高而增大,两者呈正相关(3T3鼠成纤维细胞:r=0.8154,P<0.05;人皮肤成纤维细胞:r=0.915,P<0.01),表明CS-A对这两类成纤维细胞的存活和增殖有促进作用。采用PAP和ABC免疫细胞化学染色,观察到两类细胞的胞膜上有CS分布,提示CS可能参与膜结构的组成,对细胞-细胞与细胞-基质之间的相互作用有重要意义;为进一步探讨硫酸软骨素在肝纤维化中的作用提供了可靠的实验依据。

 
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