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肝纤维化
相关语句
  liver fibrosis
    Effects of Prevention and Treatment of Thymic Factor D on Experimental Liver Fibrosis in Rats
    胸腺因子D对实验性大鼠肝纤维化防治作用的研究
短句来源
    The Clinical Study of Kang Ning Capsule in Reducing Liver Fibrosis
    康宁胶囊减轻肝纤维化的临床研究
短句来源
    Effects of dicaffeoylquinic acid against liver fibrosis and lipid peroxidation in rats:an experimental study
    二咖啡酰奎宁酸抗肝纤维化及脂质过氧化作用的实验研究
短句来源
    Quantitative analyses of the combined effects of nano red elemental selenium and glucurolactone on forming liver fibrosis induced by CCl_4 in rats
    纳米红色元素硒与葡萄糖醛酸内酯联用对大鼠肝纤维化形成过程的干预及其定量分析
短句来源
    Experimental study of the therapeutic effect of interferon-α on liver fibrosis in rats
    干扰素-α抗鼠肝纤维化的作用
短句来源
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  hepatic fibrosis
    SIGNIFICANCE OF DETERMINATION OF SOME BIOCHEMICAL INDICES IN HEPATIC FIBROSIS OF ANIMAL SCHISTOSOMIASIS JAPONICA
    血吸虫病肝纤维化动物某些生化指标的检测及其意义
短句来源
    ULTRASTRUCTURAL STUDIES ON EFFECTS OF COLCHICINE IN TREATING HEPATIC FIBROSIS OF SCHISTOSOMIASIS RABBITS
    秋水仙碱治疗家兔血吸虫病肝纤维化超微结构研究
短句来源
    Effects of Cinnarizine on Hepatic Hyaluronic Acid,Hydroxyproline Contens and N-Acetyl-β-Glucosaminidase Activities in Experimental Rat Hepatic Fibrosis
    脑益嗪对大鼠肝纤维化肝透明质酸、羟脯氨酸及N-乙酰-β-氨基葡萄糖苷酶活性的影响
短句来源
    The Effect of Aplactan on Hepatic Cellular Mitochondrion ATPase and Ca ̄(2+) Uptake in Rats with Hepatic Fibrosis
    脑益嗪对肝纤维化肝细胞线粒体ATP酶及Ca~(2+)的影响
短句来源
    Effects of vitamin A on hepatic fibrosis induced by carbon tetrachloride in rats
    维生素A对实验性大鼠肝纤维化的影响
短句来源
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  hepatic fibrogenesis
    Effect of ACE-I and AT-1 receptor blocker on the progression of CCl_4-inducing rat hepatic fibrogenesis
    培哚普利和氯沙坦抗实验性肝纤维化的研究
短句来源
  “肝纤维化”译为未确定词的双语例句
    An Experimental Research of Antifibrotic Effect of α-Tocopheral and Tetrandrine in Rat Liver
    维生素E和汉防己甲素防治肝纤维化的实验研究
短句来源
    [Methods] A total of 40 healthy Wistar rats were divided into four groups randomly: control group, model group, prevention group and treatment group.
    方法 Wistar大鼠 40 只,随机分成正常对照组、模型组、氯沙坦预防组和治疗组,采用 CCL4 皮下注射构建肝纤维化模型。 行 HE 和 VG 染色,判断肝组织炎症和纤维化的程度。
短句来源
    Effects of vitamin E on carbon tetrachloride-induced hepatic fibrosis-An experimental study
    维生素E抗大鼠四氯化碳所致肝纤维化的实验研究
短句来源
    Primary Study of Gamma-Interferon on Anti-Hepatofibrosis in Rats
    γ-干扰素抗大鼠肝纤维化的初步研究
短句来源
    THE EXPERIMENTAI STUDY OF INTERFERON α_(2b) INTERFERING HEPATIC FIBROTIC FORMATION IN RATS
    干扰素α_(2b)抑制大鼠肝纤维化形成的实验研究
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  liver fibrosis
Conversely, HAART-related hepatotoxicity may enhance the progression of liver fibrosis.
      
Objective: To study the effect of Xuesaitong soft capsule (XST) on liver fibrosis criteria in patients with post-hepatitis fibrosis.Methods: Sixty-four patients with such fibrosis were randomly divided into the treated group and control group.
      
Their liver fibrosis criteria were examined before and after treatment.Results: The levels of serum procollagen III, hyaluronic acid, collagen IV, laminin in the two groups were significantly lower (P>amp;lt;0.
      
The differences between the two groups were insignificant (P>amp;gt;0.05).Conclusion: XST could recover liver dysfunction and had anti-liver fibrosis function.
      
However, response rates are much lower in patients who are difficult to treat, such as those infected with hepatitis C virus genotype 1, high viral load, or advanced liver fibrosis.
      
更多          
  hepatic fibrosis
The Ile50Val polymorphism of the IL4RA gene was tested for association with chronic viral hepatitis and the character of its progression (the stage of hepatic fibrosis).
      
However, a significant difference in genotype frequency distribution was observed for the patients with different stages of hepatic fibrosis.
      
In hepatic fibrosis, the connective tissue biomatrix of the liver changes from the normal matrix, rich in basement membrane collagens, to a matrix enriched in interstitial fibrillar collagens.
      
Viral hepatitis, hepatic fibrosis and hepatocirrhosis etc.
      
Clinical study of Dahuang Zhechong pill in treating posthepatitis B hepatic fibrosis
      
更多          
  hepatic fibrogenesis
Great progress has been made over the past 20 years in elucidating the mechanisms of hepatic fibrogenesis.
      
Great progress has been made over the past 20 years in elucidating the mechanisms of hepatic fibrogenesis.
      
The embryonal origin of hepatic stellate cells (HSCs), the principal cells in hepatic fibrogenesis, is still intriguing.
      
The present results indicate that SPARC is expressed by activated HSCs in chronic hepatitis, suggesting the involvement of SPARC in hepatic fibrogenesis after chronic injuries.
      
mansoni egg antigen-induced granuloma formation and hepatic fibrogenesis, resulting in more severe liver pathology.
      
更多          


In order to study the pathogenesis of hepatic fibrosis, serum monoamine oxidase, mucoprotein, plasma proline and hydroxyproline were determined on 7 rabbits infected with Schistosoma japonicum. After infection serum monoamine oxidase and mucoprotein increased markedly, while plasm hydroxyproline and proline decreased evidently. After pyquiton treatment serum mucoprotein rapidly decreased and returned to normal level; whereas serum monoamine oxidase also diminished significantly, but didn't return to normal....

In order to study the pathogenesis of hepatic fibrosis, serum monoamine oxidase, mucoprotein, plasma proline and hydroxyproline were determined on 7 rabbits infected with Schistosoma japonicum. After infection serum monoamine oxidase and mucoprotein increased markedly, while plasm hydroxyproline and proline decreased evidently. After pyquiton treatment serum mucoprotein rapidly decreased and returned to normal level; whereas serum monoamine oxidase also diminished significantly, but didn't return to normal. It is suggested these biochemical indices might be of value in the early diagnosis of hepatic fibrosis and the evaluation of the effectiveness of therapy in schistosomiasis.

观察了血吸虫病家兔感染前后、形成肝纤维化前后和吡喹酮灌胃给药前后的血清单胺氧化酶、粘蛋白、血浆游离脯氨酸和羟脯氨酸的动态变化。感染后血清单胺氧化酶和粘蛋白明显升高,血浆游离羟脯氨酸和脯氨酸显著降低。治疗后血清粘蛋白迅速显著降低,并回复到正常水平;血清单胺氧化酶也显著降低,但未恢复正常。这些生化指标在肝纤维化的早期诊断和疗效考核方面有一定参考价值。

Rabbits were infected with Schistosoma japonicum cercariae and the collagen content, collagenase activities in the liver and monoamine oxidase(MAO), α2-M in serum were examined post-infection. The results showed that 8 weeks post-infection (pi), the liver collagen content in infected rabbits was 23.14±5.23 (normal control value: 10.56±1.73 mg/g wet weight), and was 32.89±7.04 11 weeks p.i, 27.29±8.86 18 weeks p.i, but 28.34±11.95mg/g wet weight 14 weeks after pyquiton treatment. Collagenase activities were 32.99±7.34...

Rabbits were infected with Schistosoma japonicum cercariae and the collagen content, collagenase activities in the liver and monoamine oxidase(MAO), α2-M in serum were examined post-infection. The results showed that 8 weeks post-infection (pi), the liver collagen content in infected rabbits was 23.14±5.23 (normal control value: 10.56±1.73 mg/g wet weight), and was 32.89±7.04 11 weeks p.i, 27.29±8.86 18 weeks p.i, but 28.34±11.95mg/g wet weight 14 weeks after pyquiton treatment. Collagenase activities were 32.99±7.34 (compared with normal group, P〈0.05), 16.88 ± 5.99, 11.13±3.35 (P〈0.05) and 14.30±5.41u, respectively. Serum MAO increased from the 4th week on but tended to return to normal after pyquiton treatment. A negative relationship was noticed between collagenese activities and collagen content (P〈0.01). It was suggested that the abnormal collagen metabolism was related to immunological reaction caused by granuloma formed by schistosome eggs and the increase in collagenase activities is an important factor in the catabolism of liver collagen.

家兔感染日本血吸虫尾蚴后第8周,其肝内胶原含量增加至正常值的2倍,11周时为正常值的3倍;杀虫治疗后14周仍为正常值的2.5倍左右。血清MAO活性在第4周已显著增高,杀虫治疗后便回复到正常范围。肝组织胶原活性在第8周时,显著高于正常值(P<0.05),11周时显著下降,18周(杀虫治疗后1周)对进一步低下,显著低于正常值(P<0.01)。肝组织胶原酶活性与肝内胶原含量之间呈显著的负相关(r=-0.647,P<0.01).表明血吸虫病肝纤维化形成过程中肝内胶原代谢的异常,似与血吸虫卵引起的肉芽肿免疫反应密切相关。提示对于血吸虫病肝纤维化,除给予病原治疗外,尚须设法提高肝组织胶原酶活性,促进已沉积于肝内胶原的分解。

Eight young female rats freely drank fluoridized water containing 5ppm F~- for 13months,so animal model of chronic fluorosis was established.As a control,anotherfour rats freely drinking running water were used.Light and electron microscopicobservations showed the following lesions in the livers:(1) fatty change,focal nerco-sis,and cholestasis of the hepatocytes;(2)active hepatocyte proliferation;and (3)prominent cell proliferation,and a tendency towards early hepatic fibrosis.Other 30young female rats drinking...

Eight young female rats freely drank fluoridized water containing 5ppm F~- for 13months,so animal model of chronic fluorosis was established.As a control,anotherfour rats freely drinking running water were used.Light and electron microscopicobservations showed the following lesions in the livers:(1) fatty change,focal nerco-sis,and cholestasis of the hepatocytes;(2)active hepatocyte proliferation;and (3)prominent cell proliferation,and a tendency towards early hepatic fibrosis.Other 30young female rats drinking the same fluoridized water were killed at the end of 2nd,4th,and 6th months of the experiment respectively (10 in each group);as comparedwith the corresponding control rats (10 in each group) drinking running water,theexperimental animals showed similar morphologic changes as described above.In theearly dynamic cytochemical studies on injuried hepatocytes,decreased activity ofSDH,G6Pase,G6PDH,Mg~(2+)-ATPase were noticed.The results indicate NaF is a li-mitative hepatotoxicant.Because the induced concentration of F~- is similar to thatin some areas of endemic fluorosis,it must be emphasized the problem of the liverof the population in the endemic areas.

本文以光镜、电镜和酶组织化学方法观察了大鼠低超常量(5ppm F~-)慢性氟中毒时的肝脏病变及其早期动态变化。1.8只非纯系雌性幼龄大鼠自由饮用含氟量为5ppm 的脱离子水13个月后,发现①肝细胞胞浆呈缺氧性超微变化。②肝细胞脂肪变和点灶性坏死。③肝细胞再生活跃。④肝贮脂肪细胞增生。⑤肝间质细胞增生、胶原纤维增生并呈早期肝纤维化趋势;2.30只 SD 雌性幼龄大鼠自由饮用含氟量为5ppm 的脱离子水2、4和6个月后各处死10只,发现了与上述慢性氟中毒大鼠相似的肝脏病变;酶组织化学观察表明,4和6月氟中毒大鼠肝细胞的 Mg~(++)-ATPase、SDH、G-6-P 和 G-6-PDH 的活性都低于对照组。本文的大鼠实验性氟肝病是以近乎国内高氟区人群饮水氟含量的条件诱发的,故应对高氟区人群的肝脏情况予以关注。

 
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