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实验性肺气肿
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  experimental emphysema
     Protection Effect of TCM 814 on the Experimental Emphysema of Mouse
     中药814对地鼠实验性肺气肿的保护作用
短句来源
     A Model of Experimental Emphysema
     应用木瓜蛋白酶制作实验性肺气肿的动物模型
短句来源
     Intervenient Effect of Retinoic Acid and Vitamin A on the Development of Experimental Emphysema in Rats
     维A酸、维生素A对大鼠实验性肺气肿的早期干预作用
短句来源
     Experimental Study of Preventing Emphysema with Elastase Inhibiting Agents Ⅱ. Protective Effect of Radix Salviae Miltiorrhizae on Human Elastic Fibers and Its Preventive Effect on Experimental Emphysema
     利用弹性蛋白酶抑制剂预防肺气肿的实验研究——Ⅱ.丹参对人弹力纤维的保护作用及对实验性肺气肿的预防效应
短句来源
     Study on Pathogenesis of Experimental Emphysema and Pulmonary Hypertension in Hamsters and Observation in Preventive and Therapeutic Effect of Chinese Herbs 814
     实验性肺气肿、肺动脉高压发病机理及中药814防治的进一步研究
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  “实验性肺气肿”译为未确定词的双语例句
     Objective To observe the changes of surfactant A and B (SP A and SP B) in alveolar type II cells of lungs in hamsters with elastase induced emphysema.
     目的 观察金黄地鼠实验性肺气肿肺泡Ⅱ型上皮表面活性物质蛋白 (SP)A和B的变化。
短句来源
     The Expressions of Monocyte Chemoattactant Protein-1 and Metalloproteinase-9 in the Lung Tissue of Emphysema Rat Models
     实验性肺气肿大鼠肺组织中单核细胞趋化蛋白-1和基质金属蛋白酶-9的表达
短句来源
     Experimental Study on the Treatment by Mesenchymal Stem Cells to Emphysema Animal Model
     骨髓间充质干细胞对大鼠实验性肺气肿治疗的研究
短句来源
     Changes of surfactant A and B in alveolar type Ⅱ cells in hamster with elastase induced emphysema
     金黄地鼠实验性肺气肿肺泡Ⅱ型上皮表面活性物质蛋白A和B的变化
短句来源
     Expression of metalloproteinase-9 and its tissue inhibitor-1 in lung of experimenatial pulmonary emphysema in rats
     MMP-9和TIMP-1在实验性肺气肿大鼠肺组织中的表达及其意义
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  相似匹配句对
     Protection Effect of TCM 814 on the Experimental Emphysema of Mouse
     中药814对地鼠实验性肺气肿的保护作用
短句来源
     A Model of Experimental Emphysema
     应用木瓜蛋白酶制作实验性肺气肿的动物模型
短句来源
     the practice is experiment;
     实践的实验性;
短句来源
     Reconstruction of the Collateral Circulation in the Experimental Fraction
     实验性骨折愈合的血运重建
短句来源
     Surgical treatment of emphysema
     肺气肿的外科治疗
短句来源
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  experimental pulmonary emphysema
High frequency jet ventilation in experimental pulmonary emphysema
      
Pressure in the occluded bronchus in experimental pulmonary emphysema
      
  experimental emphysema
Experimental emphysema was induced by multiple intratracheal instillation of Papain in a dose of 2 mg/kg body weight in 4 bastard dogs.
      
Deformation of the transpulmonary pressure plateau in experimental emphysema
      
Deformation of the transpulmonary pressure plateau obtained during interruption of the air flow for 0.5 sec in 8 healthy rabbits and in 9 rabbits with experimental emphysema caused by intravenous injection of lycopodium spores is described.
      
Experimental emphysema can be produced in dogs by impeding expiration by means of a properly constructed ball valve introduced into the trachea.
      
Numerous studies have demonstrated that the instillation of elastolytic enzymes into the lung can induce experimental emphysema.
      


The protective effect of Radix Salviae Miltiorrhizae on the human elastic fibers and preventive effect on experimental emphysema were observed. The results showed that Radix Salviae Miltiorrhizae can prevent human arterial and pulmonary elastic fibers from the destruction of human sputum and porcine pancreatic elastase; morever, it can prevent the occurrence of experimental emphysema caused by elastase. The study suggest that Radix Salviae Miltiorrhizae may become a favorible drug in the prevention of emphysema....

The protective effect of Radix Salviae Miltiorrhizae on the human elastic fibers and preventive effect on experimental emphysema were observed. The results showed that Radix Salviae Miltiorrhizae can prevent human arterial and pulmonary elastic fibers from the destruction of human sputum and porcine pancreatic elastase; morever, it can prevent the occurrence of experimental emphysema caused by elastase. The study suggest that Radix Salviae Miltiorrhizae may become a favorible drug in the prevention of emphysema.

本研究表明,丹参是一种较理想的弹性蛋白酶抑制剂,它可以保护人肺和血管的弹力纤维使其免受弹性蛋白酶的破坏,并可预防弹性酶引起的实验性肺气肿。从而提示,丹参可能成为一种较理想的预防肺气肿的药物。

Objective To observe the changes of surfactant A and B (SP A and SP B) in alveolar type II cells of lungs in hamsters with elastase induced emphysema. Methods After intratracheal instilling of elastase, hamsters were killed at the 30th, 60th and 90th day of the experiment respectively. Slides of lung tissue were examined under light microscope and measured by calculating the mean linear intercepts. Then the slides were stained under immunohistochemistry procedures and measured by using a morphometric analysis...

Objective To observe the changes of surfactant A and B (SP A and SP B) in alveolar type II cells of lungs in hamsters with elastase induced emphysema. Methods After intratracheal instilling of elastase, hamsters were killed at the 30th, 60th and 90th day of the experiment respectively. Slides of lung tissue were examined under light microscope and measured by calculating the mean linear intercepts. Then the slides were stained under immunohistochemistry procedures and measured by using a morphometric analysis system. Meanwhile, slides were also checked by electron microscopy. Results In comparison with the normal group, MLI of the elastase group was significantly increased ( P <0.01). Immunohistochemistry showed that the percentage of both SP A and SP B positive cells was significantly decreased 30 days after the intratracheal instilling of elastase (from 48.0%±3.0% to 9.5%±4.0% and from 28.0%±4.0% to 13.3%±4.1%, respectively; P <0.01), however, a time-dependent recovery 90 days after instilling was seen. Electron microscopy showed a significantly decrease of the number of lamellae. Conclusion The decrease of SP A and SP B in alveolar type II cells seems to play an important role in the development of emphysema.

目的 观察金黄地鼠实验性肺气肿肺泡Ⅱ型上皮表面活性物质蛋白 (SP)A和B的变化。方法 金黄地鼠气管注入弹性蛋白酶后 ,分别于注入弹性蛋白酶的 30、6 0和 90d处死 ,肺组织经光镜检查和图像分析后 ,再行SP A和SP B免疫组化染色和形态定量 ;同时进行透射电镜观察。结果 注入弹性蛋白酶后第 30、6 0和 90d肺平均内衬间隔明显增大 (P <0 .0 1) ;免疫组化显示注入弹性蛋白酶后 30d肺泡Ⅱ型上皮细胞SP A和SP B阳性率分别由 (48.0± 3.0 ) %降至 (9.5± 4.0 ) %、(2 8.0±4.0 ) %降至 (13.3± 4.1) % ,P <0 .0 1,但随时间延至 90d而有所恢复 ;并且与肺气肿程度呈明显负相关。电镜观察发现 ,注入弹性蛋白酶后 ,Ⅱ型上皮细胞内板层小体数减少。结论 实验性金黄地鼠肺气肿肺泡Ⅱ型上皮细胞内SP A和SP B明显减少可能是肺气肿发展的重要环节之一。

Objective To investigate the effects of moderate intensity endurance training on the morphology and function of diaphragm of emphysematous rats. Methods SD rats were randomly divided into two groups first.One group was emphysematous model;emphysema was induced by single endotracheal instillation of porcine pancreatic elastase 800 U/kg;another group served as normal control; the rats received a single endotracheal instillation of equal volume of 0 9% NaCl. Following a 4 week recovery period each...

Objective To investigate the effects of moderate intensity endurance training on the morphology and function of diaphragm of emphysematous rats. Methods SD rats were randomly divided into two groups first.One group was emphysematous model;emphysema was induced by single endotracheal instillation of porcine pancreatic elastase 800 U/kg;another group served as normal control; the rats received a single endotracheal instillation of equal volume of 0 9% NaCl. Following a 4 week recovery period each group of animals was further randomly divided into exercise endurance training and non exercise groups. The four groups were termed as normal control (NC),normal exercise (NE), emphysematous control(EC) and emphysematous exercise(EE).The NE and EE groups were subjected to a 12 week and 55%~75% VO 2max progressively increasing training on a motor driven treadmill. Results Fatigue produced by stimulations at 20 Hz and 100 Hz revealed that the diaphragm of EE group rats showed a signifcantly slower rate by 9%~17% and 6%~11%( P <0 05),and the oxidative potential as assessed with succinate dehydrogenase (SDH) in the diphragmatic fibers of the EE group increased by 17%~ 20%( P <0 05) as compared with the other groups.Both normal (100%) and emphysematous(91 7%) rats completed the training,showing a good compliance with this exercise program. Conclusions The constractability and oxidative capacity of costal diaphragmatic muscle can be enhanced by the regular treadmill running in emphysematous rats.

目的 观察运动锻炼对肺气肿大鼠膈肌收缩与代谢功能的影响。 方法 SD大鼠 ,随机分肺气肿组 ,经气管内一次性滴注猪胰弹性蛋白酶 80 0 U/kg;正常组 ,一次性气管内滴注等体积0 .9%氯化钠溶液 ;4周后再随机分为正常对照组、正常运动组、肺气肿对照组和肺气肿运动组 ;对正常运动组和肺气肿运动组实施同等强度〔最大氧耗量 ( VO2 max) 55%~ 75%〕和时间 ( 1 2周 )的电动平板跑步锻炼。 结果 与其他组比较 ,肺气肿运动组锻炼后膈肌肋部肌纤维在 2 0 Hz和 1 0 0 Hz持续电刺激时 ,张力衰减速度分别减慢 9%~ 1 7%和 6%~ 1 1 % ( P<0 .0 5) ;与氧化代谢有关的琥铂酸脱氢酶 ( SDH)活性增强 1 7%~ 2 0 % ( P<0 .0 5) ;运动锻炼方案具有良好的顺从性 ,正常大鼠 1 0 0 %和肺气肿大鼠 91 .7%按计划完成了锻炼。 结论 渐进行耐力运动锻炼可明显改善实验性肺气肿大鼠膈肌的疲劳耐力和氧化代谢能力。

 
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