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   凋亡阳性率 的翻译结果: 查询用时:0.014秒
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凋亡阳性率
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  apoptosis positivity
     The expression of apoptosis-related genes bcl-2 and bax protein and apoptosis positivity in cervical carcinoma during radiation
     凋亡阳性率及凋亡相关基因bcl-2、bax在宫颈癌放疗前后表达的意义
短句来源
     The Expression of Apoptosis-Related Genes Bcl-2 and Bax Protein and Apoptosis Positivity in Cervical Carcinoma during Irradiation
     凋亡阳性率及凋亡相关蛋白Bcl-2、bax在宫颈癌放疗前后表达的意义(英文)
短句来源
     Results: The apoptosis positivity before and after irradiation was 76.7% and 100% respectively, with the difference being significant (P<0.05);
     结果30例患者凋亡阳性率放疗前后的表达分别为76.7%和100%,放疗后显著增高(P<0.05);
短句来源
     Results:The apoptosis positivity before and after irradiation was 76.7% and 100% respec- tively,with the difference being significant (P<0.05);
     结果 30例患者凋亡阳性率放疗前后的表达分别为76.7%和100%,放疗后显著增高(P<0.05);
短句来源
     Results The apoptosis positivity before and after radiation were 76.7% and 100%, and it was significantly increased after radiation (P<0.05).
     结果  30例患者凋亡阳性率放疗前后的表达分别为 76 .7%和 10 0 % ,放疗后显著增高 (P <0 .0 5 ) ;
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  “凋亡阳性率”译为未确定词的双语例句
     Results After treated with octreotide at a concentration of 1×10~ -5 mol/L , the early apoptosis rates (14.2 ± 2.6) % and the apoptosis indexes (18.8±3.3)% were significantly higher than the control group(1.2 ± 0.1)% (P< 0.05) and (3.6 ± 0.9) % (P<0.01).
     结果1×10-5mol/L奥曲肽使HepG2细胞早期凋亡阳性率和凋亡指数(AI)增加至(14.2±2.6)%和(18.8±3.3)%,与对照组(1.2±0.1)%(P<0.05)和(3.6±0.9)%(P<0.01)比较差异显著;
短句来源
     Before and during the treatment, the positive rates of apoptosis were increased, ranging from 24%to 60%(P=0.01) in the RT group and from 20.8% to 87.5% (P=0.000) in the CRT group, respectively.
     在治疗前和治疗过程中,RT组和CRT组凋亡阳性率均增加,分别由24%上升到60.0%(P=0.01)和20.8%增加到87.5%(P=0.000),差异显著;
短句来源
     TUNEL assay showed that the apoptotic percentage of HL-60 cells reached 37.5% when incubated with 3200 U/ml rhTNF-α for 48 hours.
     TUNEL法检测显示,3200U/mlrhTNF-α作用于HL-60细胞48小时凋亡阳性率可达37.5%。
短句来源
     The positive rate of apoptosis in treated group was 10.60%,which was signlficantly less than that in control group(13.46%)(P<0.01).
     对照组视神经节细胞层凋亡阳性率13.46%,明显高于实验组的10.60%(P<0.01)。
短句来源
     TUNEL assay showed that BMSCs/tk could obviously show bystander effect and induce apoptosis of glioma cells in vivo with an apoptosis positive ratio of 20. 38%±2. 57% , showing a statistically significant difference in comparison with BMSCs group (2. 56%±0. 52% , P = 0. 023 ) and control group (2. 74%±0. 38% , P = 0. 025 ).
     BMSCs/tk不仅在体外可产生明显的旁观者效应,在荷瘤鼠脑内也显示了明显的诱导肿瘤细胞凋亡的旁观者效应,凋亡阳性率为20.38%±2.57%,与BMSCs组(2.56%±0.52%)和对照组(2.74%±0.38%)相比,差异均有统计学意义(P值分别为0.023和0.025)。
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  相似匹配句对
     The positive rate is 10%.
     阳性为10%。
短句来源
     Conclusion The sperm apoptotic percentage in AsAb positive rats was increased.
     结论AsAb阳性大鼠精子凋亡增加;
短句来源
     Use TUNELmethod to detect apoptosis;
     TUNEL标记法检测凋亡细胞阳性
短句来源
     The positive rate of bac terial cluteres was 56% in the series.
     细菌培养阳性56%。
短句来源
     There were significant relation between the decrease of Fas positive rate and increase apoptosis.
     Fas阳性下降与凋亡增加相关。
短句来源
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Eukaryotic cell apoptosis is usually assessed by flow cytometry on reduction of DNA c0ntent or formation of DNAbreaks. We compared the character of the two methods in detecting apoptosis. The apoptosis of mice (n= 5) thymocytes were in-duced by abdominal cavity injection of dexamethasone (25mg/kg), and after 10hr the cells were separated. Propidium iodium(Pl)staining and TdT enzyme mediated deoxynucleotidy nick end labeling (TUNEL)kits were used to measure the DNA content and theDNA strand breaks, respectively....

Eukaryotic cell apoptosis is usually assessed by flow cytometry on reduction of DNA c0ntent or formation of DNAbreaks. We compared the character of the two methods in detecting apoptosis. The apoptosis of mice (n= 5) thymocytes were in-duced by abdominal cavity injection of dexamethasone (25mg/kg), and after 10hr the cells were separated. Propidium iodium(Pl)staining and TdT enzyme mediated deoxynucleotidy nick end labeling (TUNEL)kits were used to measure the DNA content and theDNA strand breaks, respectively. The positive rates of apoptosis detected with the methods of Pl and TUNEL were 29. 3±8. 1 %(x±s) and 40. 2±8. 7%, respectively, P<0. 01 (matched t-test). Analysing with double parameters of Pl and TUNEL, in thepositive part of Pl staining, there were 4. 7±2. 2% of particles which had n0 increased DNA breaks (TUNEL negative). Theseparticles, we think, may be the debris of cells and contribute to the false positive of Pl detection. On the other hand, about 42. 8±10. 6% of TUNEL positive cells had no reduction of DNA content, which resulted in the false negative of Pl measunement. Our re-sults indicated that the method of DNA breaks labeling with TUNEL is superior to the methods of DNA content assay by Pl stain-ing to detect apoptosis.

本文以地塞米松(25mg/kg)腹腔注射10h后诱导的BALB/c鼠(n=5x)胸腺细胞作为凋亡阳性细胞,比较了碘化丙啶(PI,propidiumiodium)染色DNA含量分析和TUNEL(TdT—mediatedX-dUTPnickendlabeling)标记DNA断裂点分析两种方法在检测凋亡中的差异。流式细胞仪分析结果表明,PI检测凋亡的阳性率29.3±8.1%(x±s)明显低于TUNEL检测的阳性率(40.2±8.7%),P<0.01(配对t检验)。进一步用PI和TUNEL双参数分析显示,在PI检测阳性的颗粒中有4.7±2.2%是TUNEL检测阴性的,即没有DNA断裂点的增加,这些颗粒很可能是细胞碎片,它们导致了PI检测的假阳性。另外TUNEL阳性的细胞中有42.8±10.6%处在S期和G2/M期,因而用PI检测为阴性,造成PI方法的漏检,即假阴性。我们的资料表明,PI检测凋亡与TUNEL相比有很高的漏检率和一定的假阳性,因此PI只适用于凋亡初步分析。要进行准确的定量分析应该用TUNEL法。

Viral myocarditis(VMC)may be induced by coxsackie B 3m Virus (CVB 3m ).The CVB 3m was inoculated into abdominal cavity of BALB/C mice aged 5 weeks in this experiment,then the mice were killed in 9 days.The hearts of the mice were examined using TDT/dUTP Nick end Labeling (T/UNEL)of fragmented DNA and electronic microscopy.The Results showed that detectable rate of VMC was 93.33% and positive rate of apoptosis of myocardium was 80.00%.These results indicated that apoptosis may be one of the mechanisms...

Viral myocarditis(VMC)may be induced by coxsackie B 3m Virus (CVB 3m ).The CVB 3m was inoculated into abdominal cavity of BALB/C mice aged 5 weeks in this experiment,then the mice were killed in 9 days.The hearts of the mice were examined using TDT/dUTP Nick end Labeling (T/UNEL)of fragmented DNA and electronic microscopy.The Results showed that detectable rate of VMC was 93.33% and positive rate of apoptosis of myocardium was 80.00%.These results indicated that apoptosis may be one of the mechanisms causing myocyte damage in myocarditis of mice.

实验通过给5周龄BALB/C小鼠腹腔接种CoxsackieB3m病毒(CVB3m),诱发小鼠急性病毒性心肌炎(VMC),感染病毒9天后处死小鼠,利用光镜、电镜及原位末端标记法对心肌组织进行检测,结果显示:VMC检出率为93.33%,心肌细胞凋亡阳性率为80.00%,凋亡细胞多分布于心内膜下、心外膜下心肌组织,血管内皮细胞和坏死病灶周围,提示细胞凋亡可能是小鼠VMC的发病机制之一。

With a view to further investigating teratogenicity, embryotoxicity and teratogenic mechanism of arsenic on developing embryos, the relationship between apoptosis (programmed cell death, PCD) and arsenic induced teratogenesis was studied during head fold stage by using in situ 3'end labeling of DNA and Nile blue sulfate(NBS) vital staining. Sprague Dawley rats were intraperitoneally injected on gestation day 9.5 with doses of 0,1,4 and 8 mg/kg arsenic respectively, and killed at day 12. the results show that...

With a view to further investigating teratogenicity, embryotoxicity and teratogenic mechanism of arsenic on developing embryos, the relationship between apoptosis (programmed cell death, PCD) and arsenic induced teratogenesis was studied during head fold stage by using in situ 3'end labeling of DNA and Nile blue sulfate(NBS) vital staining. Sprague Dawley rats were intraperitoneally injected on gestation day 9.5 with doses of 0,1,4 and 8 mg/kg arsenic respectively, and killed at day 12. the results show that teratogenic incidence and mortality increased to 35.7% and 23.8% respectively, in apparent dose effect relationship. In NBS staining experience, it was found that arsenic induced PCD in many areas, which related with abnormal development of brain, optic system, somite segmentation, limb bud and so on. In situ 3' end labeling of DNA further supported above mentioned results. The positive rate of apoptotic cell death was up to 55.6%(P<005). This research suggested a positive correlation between abnormal PCD and teratogenesis. It may be seen that over apoptosis is one of teratogenic mechanisms of arsenic. Moreover, our results show that necrosis plays some role in teratogenesis as well.

为进一步揭示砷的致畸性、胚胎毒性和作用机制,采用胚胎活体染色和原位DNA末端标记等技术探讨了不同剂量砷是否能诱导孕9.5天龄大鼠的胚胎细胞发生凋亡(程序性细胞死亡)及其凋亡与畸胎形成的关系。结果表明随着砷浓度(0、1、4和8mg/kg)的增高,畸胎率和死胎率分别由2.6%和0上升到35.7%和23.8%,呈明显剂量-反应关系。活体染色发现在胚胎脑、眼、体节、肢芽和腮弓等部位出现大量散在的凋亡细胞,与畸形发生部位相一致。原位DNA末端标记进一步证实砷能诱发器官形成期胚胎细胞凋亡,8mg/kg砷染毒后,凋亡阳性率高达55.6%(P<0.05),表明凋亡与畸胎发生关系密切,这是砷致畸作用的重要机制之一。实验结果还发现胚胎细胞坏死亦与畸胎形成有关。

 
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