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心力衰竭
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  heart failure
    The Relationship between the Changes of Intralymphocyte Cyclic Nucleotide,Ca~(2+) and the Cardiac Function in Patients with Congestive Heart Failure Treated by China-Made Amrinone
    氨利酮治疗充血性心力衰竭时淋巴细胞内环核苷酸和Ca~(2+)含量的变化与心功能的关系
短句来源
    Effect of Nifedipine on Serum Concentration of Digoxin in Heart Failure Patients
    心痛定对心力衰竭患者血清地高辛浓度的影响
短句来源
    A Short-term Effective Observation on 128 Cases of Congestive Heart Failure Patients Given with Captopril and Diuretics for Treatment
    卡托普利合用利尿剂治疗充血性心力衰竭128例短期疗效观察
短句来源
    Comparison Between the Curative Effects of Captopril and Digoxin for Heart Failure
    卡托普利与地高辛治疗心力衰竭的疗效比较
短句来源
    Clinical study on therapy for congestive heart failure with metoprolol
    美托洛尔治疗充血性心力衰竭的临床研究
短句来源
更多       
  congestive heart failure
    The Relationship between the Changes of Intralymphocyte Cyclic Nucleotide,Ca~(2+) and the Cardiac Function in Patients with Congestive Heart Failure Treated by China-Made Amrinone
    氨利酮治疗充血性心力衰竭时淋巴细胞内环核苷酸和Ca~(2+)含量的变化与心功能的关系
短句来源
    A Short-term Effective Observation on 128 Cases of Congestive Heart Failure Patients Given with Captopril and Diuretics for Treatment
    卡托普利合用利尿剂治疗充血性心力衰竭128例短期疗效观察
短句来源
    Clinical study on therapy for congestive heart failure with metoprolol
    美托洛尔治疗充血性心力衰竭的临床研究
短句来源
    Effects of small dose prostaglandin E 1 on severe congestive heart failure and serum endothelin level
    小剂量前列腺素E_1对严重充血性心力衰竭及内皮素的影响
短句来源
    Effects of endothelin receptor A antagonist FR139317 on rats with congestive heart failure
    内皮素受体A拮抗剂FR139317对充血性心力衰竭大鼠的作用(英文)
短句来源
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  cardiac failure
    Some cases suggest that high-dose,long-term propofol infusion for sedation of patients in the intensive care unit can result in metabolic acidosis, hypertriglyceridemia, hepatomegaly, rhabdomyolysis, acute renal failure, hyperkalemia, ventricular arrhythmia, and cardiac failure wich has been referred to as propofol infusion syndrome (PIS).
    一些病例报告提示:大剂量、长时间输注异丙酚后可能引起代谢性酸中毒、高脂血症、肝脏脂肪浸润和横纹肌溶解、甚至高钾血症、急性肾衰、心律失常、难治性的心力衰竭等严重并发症,甚至导致死亡,即“异丙酚输注综合征”(propofol infusion syndrome,PIS)。
短句来源
    WC observed the effect of Perindopril (Per) on left ventricular diastolic function in rat hearts with cardiac failure induced by myocardial infarction (MI).
    观察了培哚普利(Per)对心肌梗塞(MI)后心力衰竭(HF)大鼠左室舒张功能的影响。
短句来源
  “心力衰竭”译为未确定词的双语例句
    The double drug interaction between propafenone (PF) and digoxin (D) was investigated in twelve healthy volunteers.
    心律失常合并心力衰竭应用普罗帕酮(Propafenone,PF)治疗有时需与地高辛(Digoxin,D)合用。
短句来源
    AIM To investigate the influence of β 3 adrenoreceptor (β 3 AR) and the effect of β 3 AR agonist on the failing hearts.
    目的 探讨 β3 肾上腺素受体 (β3 AR)及其激动剂BRL 37344 (BRL)在心力衰竭中的作用。
短句来源
    the level of β 3 AR mRNA is higher than that in control heart; β 3 AR agonist aggravates cardiac dysfunctions.
    β3 ARmRNA水平在衰竭心脏比非衰竭心脏明显增高 ,应用 β3 AR激动剂可明显加重心力衰竭
短句来源
    Surveyingathe Actions of PGE1 on Congestive Cardiac Failure
    前列腺素E_1对心力衰竭的疗效观察
短句来源
    HERAPEUTIC EFFECTS,HEMODYNAMICS AND MECHANISM OF CLONIDINE IN CONGESTIVE
    可乐定治疗心力衰竭的临床疗效和血液动力学效应及其作用机制
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  heart failure
Cardioprotection by Aldosterone Receptor Antagonism in Heart Failure: 1.
      
Thus, the renin-angiotensin-aldosterone system (RAAS) plays a major role in the development of both hypertension and heart failure and is, therefore, a key target for therapeutic interventions.
      
Commonly prescribed medications for control of hypertension and congestive heart failure are inhibitors of the RAAS, including angiotensin converting enzyme inhibitors (ACE-Is) and angiotensin II (A-II) receptor antagonists.
      
These findings support the pivotal role of aldosterone in the pathophysiology of progressive heart failure.
      
Recently, eplerenone was successfully introduced for the treatment of hypertension and heart failure.
      
更多          
  congestive heart failure
Commonly prescribed medications for control of hypertension and congestive heart failure are inhibitors of the RAAS, including angiotensin converting enzyme inhibitors (ACE-Is) and angiotensin II (A-II) receptor antagonists.
      
Complex clinical-laboratory investigation of children with congestive heart failure developed on the basis of dilated cardiomyopathy and hypertrophic cardiomyopathy has been carried out.
      
The majority of the anomalies are of benign prognosis, but others can be associated with cardiac symptoms and syndromes (angina, dyspnea, syncope, congestive heart failure, myocardial infarction and sudden death).
      
Congestive heart failure (CHF) has emerged as a major worldwide epidemic and its main causes seem to be the aging of the population and the survival of patients with post-myocardial infarction.
      
Thyrotoxicosis is not only able to aggravate preexisting cardiac disease but can also by itself lead to atrial fibrillation, cardiomegaly or congestive heart failure.
      
更多          
  cardiac failure
Myoblast transplantation (MT) is a cell-based gene therapy treatment, representing a potential treatment for Duchenne muscular dystrophy (DMD), cardiac failure and muscle trauma.
      
Its primary goal is to maintain circulation in the state of terminal cardiac failure which cannot be stabilized otherwise.
      
In hyperthyreoidism, the total blood volume increases, the peripheral vascular resistence decreases, and cardiac failure may occur despite an increased cardiac output at rest.
      
The mean life expectancy for untreated patients with Marfan syndrome is 32 years with aortic dissection, aortic rupture or cardiac failure due to mitral and aortic valve regurgitation as the predominant cause of death in >amp;gt;90% of the cases.
      
Aortico-left ventricular tunnel (ALVT) is a rare congenital cardiac malformation that presents with cardiac failure and clinical findings of severe aortic regurgitation in early infancy.
      
更多          


The cardiotonic potency and therapeutic range of corchoroside A were compared with the well-known cardiac glycosides in heart-lung preparations of guinea pigs and cats. Heart failure was produced by adding pentobarbital sodium to the blood in the venous reservoir of heart-lung preparations (HLP) at a uniform rate (1mg/minute) in most experiments. In some other experiments the preparations were allowed to deteriorate spontaneously.A Steady fall in the cardiac output and right atrial pressure rise was taken as...

The cardiotonic potency and therapeutic range of corchoroside A were compared with the well-known cardiac glycosides in heart-lung preparations of guinea pigs and cats. Heart failure was produced by adding pentobarbital sodium to the blood in the venous reservoir of heart-lung preparations (HLP) at a uniform rate (1mg/minute) in most experiments. In some other experiments the preparations were allowed to deteriorate spontaneously.A Steady fall in the cardiac output and right atrial pressure rise was taken as an indication of failure. The cardiac Competence test was used to assess the cardiac function by raising the venous reservoir by 5~10 cm for 15 seconds.The reduction in atrial pressure and the increased cardiac output were taken as an indicator of "threapeutic dose". Following the appearance of therapeutic effects the continued infusion of a cardiac glycoside results in cardiac irregularities. These irregularities are accompanied by a progressive reduction in the cardiac output and a concomitant abrupt rise in atrial pressure with widened pulse waves. These phenomena mentioned above were taken as an indicator of "toxic dose". Further continuation of the infusion results in ventricular fibrillation and was taken as an indicator of "lethal dose".The ratios of the threapeutic dose and toxic dose to the lethal dose in three glycosides have been expressed in per cent of the dose respectively. In the HLP of cats corchoroside A usually yielded higher therapeutic effect than ouabain and strophanthin K. In the HLP of guinea pigs its therapeutic dose was similar with ouabain and higher than strophantin K. Corchoraside A not only improved cardiac function and increased cardiac competence but the manifestation of cardiotonic action was earlier.Under our experimental conditions in the HLP of guinea pigs the per centage ratio of therapeutic dose/lethal dose was 6.05 for corchoroside A, 17.45 for ouabain, 14.41 for strophanthin K and in the HLP of cats 10.86, 21.29, and 22.96 respectively. Hence, corchoroside A has a 2~3 fold greater "safety margin" than the other two glycosides studied. It is statistically significant, The utilization of the HLP of guinea pigs as a novel tool for the determination of the therapeutic range of cardiac glycosides is also discussed.

本文报告强心药黄麻甲甙的治疗作用强度、治疗宽度并与已知强心药作比较性研究。在豚鼠、猫心肺制备上,以戊巴比妥钠引起心力衰竭(少数自发性心力衰竭),分别测定了黄麻甲甙、哇巴因、康毗丁的治疗作用及治疗宽度,以输出量或静脉压的改善作为、“治疗量”的指标;以静脉压重又上升,波幅加大,心律不齐作为“中毒量”指标;以发生心室纤维颤动作为“致死量”指标;计算了三种药物的治疗量/致死量、中毒量/致死量的百分率,黄麻甲甙在二种心肺制备上,对心力衰竭有良好的治疗作用,其治疗量在猫身上小于哇巴因;在豚鼠身上则与哇巴因相近,不仅作用较强,而且作用较其他二种已知强心甙发生得更早。 在治疗量/致死量的百分率方面,在豚鼠心肺制备上,黄麻甲甙为6.05%、哇巴因17.45%、康毗丁14.41%。在猫心肺制备上则分别为10.86%、21.29%及22.96%;即黄麻甲甙的治疗宽度为上述二种强心甙的2~3倍,在统计学上有显著性差异。在中毒量/致死量的百分率方面,三种强心甙差异不大。本文对豚鼠心肺制备的实验方法及其作为测定强心甙治疗宽度的工具,作了简短的讨论。

In order to assess the significance of drug level mornitoring, the relationship between digoxin dosage, serum concentration and clinical effects in 91 patients with chronic heart failure were analysed by multiple linear regression method. The main results are as follows: The limit between toxic and therapeutic digoxin serum concentration was about 1.8ng/ml; the optimum treatment area seemed to be in the range of 0.6-1.4ng/ml. The drug level mornitoring is important to the rational drug administration. However,...

In order to assess the significance of drug level mornitoring, the relationship between digoxin dosage, serum concentration and clinical effects in 91 patients with chronic heart failure were analysed by multiple linear regression method. The main results are as follows: The limit between toxic and therapeutic digoxin serum concentration was about 1.8ng/ml; the optimum treatment area seemed to be in the range of 0.6-1.4ng/ml. The drug level mornitoring is important to the rational drug administration. However, because of the marked difference in the reaction to digoxin from person to person, digitalis intoxication can be thus reduced but can not be prevented totally by the drug level mornitoring.

为探讨药物浓度测定的意义,本文以回归方法分析91例慢性充血性心力衰竭病人所用的地高辛剂量、血药浓度与临床疗效的关系。结果表明:地高辛中毒与治疗浓度间的界限大致为1.8ng/ml,最佳治疗范围0.6~1.4ng/ml。测定血药浓度对合理用药有指导意义。但由于对地高辛的反应个体差异较大,通过血药监测能够减少但并不能完全防止洋地黄中毒。

Meta-nifedipine(m-nif) is a calcium antagonist synthesized by Tianjin Institute of Pharmaceutical Industry. The effects of m-nif on cardiac output(CO), cardiac index(CI), stroke index(SI) were monitored in 6 anesthetized close chest dogs by impedence cardiographic method in comparison with nifedipine(nif). The magnitude of increase of CI and SI by m-nif was more than those of nif for 2. 7-and 4-fold, respectively, at the same dose(20 μg/kg ⅳ). The duration of action of m-nif was also longer than that of nif....

Meta-nifedipine(m-nif) is a calcium antagonist synthesized by Tianjin Institute of Pharmaceutical Industry. The effects of m-nif on cardiac output(CO), cardiac index(CI), stroke index(SI) were monitored in 6 anesthetized close chest dogs by impedence cardiographic method in comparison with nifedipine(nif). The magnitude of increase of CI and SI by m-nif was more than those of nif for 2. 7-and 4-fold, respectively, at the same dose(20 μg/kg ⅳ). The duration of action of m-nif was also longer than that of nif. m-nif produced a marked reduction of the peripheral vasocular resistance. In addition, the pulse pressure was increased because the decrease of diastolic blood pressure was much larger than that of the systolic. In concomitance with its effect in improving cardiac performance, m-nif caused a slight slowing of heart rate(HR). However, nif increased HR.Tension-time index(TTI), as cardiac energy and oxygen consumption index, was decreased significantly by m-nif more than by nif in cats. m-Nif raised oxygen content in the coronary sinus blood and reduced oxygen difference between arterialand coronary sinus blood. The rate of oxygen uptake and the rate of CO_2 production were reduced to a greater degree with m-nif. It appears that effects of m-nif in improving the left ventricular pump function, myocardial oxygen consumption, CO_2 production and reducing peripheral vasocular resistance were stronger and lasted longer than those of nif. Thus, m-nif seems to have more beneficial effects in treatment of congestive heart failure.

间硝苯吡啶(M-Nif)为新合成的钙拮抗剂。实验表明,M-Nif可增加麻醉狗的CI、SI,降低麻醉猫的舒张压、TTI较Nif强而持久。Nif增加心率,M-Nif却无影响或略减少。M-Nif增加冠状窦氧含量,缩小动静脉氧差的强度弱于Nif,但降低心肌氧摄取率。二氧化碳产生率则较Nif强。提示M-Nif治疗充血性心力衰竭优于Nif。

 
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