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冷束缚
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  cold restraint
     Methods With common bile duct ligation (CBDL) in Wistar rats under cold restraint stress, GMBF and the content of Endothelin 1, Angiotensin Ⅱ, H 2, α 1 receptor in gastric mucosa were measured. Before stress anti ET 1 serum, Enalapril, Cimentidine and Phetolamini were administrated, and the change of GMBF was studied.
     方法用结扎总胆管的大鼠冷束缚应激模型,测定胃粘膜血流和胃粘膜中内皮素1(ET-1),血管紧张素Ⅱ(Ang-Ⅱ)和胃粘膜组胺H2受体(H2受体),肾上腺素能α1受体(α1受体)的变化,以及应激前使用抗ET-1血清、依那普利、甲氰米胍、酚妥拉明后胃粘膜血流的改变。
短句来源
     Methods:Plasma and gastric mucosal ET 1 levels, gastric mucosal blood flow(GMBF) and ulcer index (UI) were measured prior to stress and 1,3,6,9,12 h poststress in cold restraint stress induced ulcer model of rats.
     方法:采用大鼠冷束缚应激溃疡模型(CRS),动态检测应激前及应激后1,3,6,9,12h各时相血浆、胃粘膜组织ET-1含量、胃粘膜血流量(GMBF)及溃疡指数(UI)的变化。
短句来源
     Methods:Make the cold restraint stress model of rat. At0,1/4,2,8 h after stress,the levels of β-endorphine in plasma and gastric mucosa were assessed with RIA,the gastric juice pH,gastric mucosa ulcer index(UI) were measured.
     方法:制作大鼠冷束缚应激模型,以放免法测定应激0,1/4,2,8 h血浆及胃黏膜中β-内啡肽的含量,同时测定胃液pH、胃黏膜溃疡指数(UI)。
短句来源
     To investigate the role of ET 1 in the pathogenesis of SU, a model was established by cold restraint stress(CRS) inducing rat ulcer, immunohistochemistry(IHC)ABC method and radioimmunoassay(RIA) methods were used to determine ET 1 content and to study the expression and location of ET 1 in stomach of normal and CRS rats,and to evaluate the ulcer index(UI).
     为探讨内皮素 (ET) 1在应激性溃疡 (SU)发病中的意义 ,以大鼠冷束缚应激溃疡 (CRS)为模型 ,采用免疫组化及放免方法 ,观察研究应激前后大鼠胃粘膜组织ET 1表达分布、含量及胃粘膜损伤溃疡指数 (UI)等变化情况。
短句来源
     (2) The DRRD 6g/kg given orally inhibited markedly gastric mucosal dam- age induced by 0.2N NaOH and 0.6N HC1.(3) The DRRD 6g/kg given orally inhibited markedly the formation of ulcer induced by indomethacin and cold restraint stress,too.
     (2)胃饲6g/kg 的干地黄煎剂对0.2mol/L NaOH、0.6mol/L HCl 所致的胃粘膜损伤均有显著的抑制作用; (3)胃饲6g/kg 的干地黄还能抑制消炎痛型和冷束缚应激型胃溃疡的形成。
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  cold-restraint
     Methods:The gastric ulcer models produced by pylorus ligation,0.2N NaOH,0.6N HCI,indomethacin and cold- restraint stress were used.
     方法:采用幽门结扎型、O. 2N mol/L NaOH 型、0.6mol/L HCl 型、消炎痛型和冷束缚应激型胃溃疡模型。
短句来源
     KA8 Inhibite Gastric Acid Secretion Induced by Cold-restraint Stress and Intracisteral TRH in Rat
     KA8肽抑制TRH和冷束缚引起的胃酸分泌增加
短句来源
     Methods The SU model of rats was induced with cold-restraint stress,and gastric mucosal blood flow(GMBF)was measured with the neutral red clearance method.
     方法采用冷束缚法制备大鼠应激性溃疡模型,中性红清除率法测定大鼠胃粘膜血流量。
短句来源
     Methods: After the gastric mucosal injury model induced by cold-restraint in rats was established, the present study was comleted by: ①observing the effect of endogenous NO on gastric mucosal damage, ②measuring the alterations of stressed gastric sulfhydryl compounds and malonaldehyde(MDA), the end product of lipid peroxidation, and ③evaluating the effect of inhibited NO on sulfhydryl compounds and MDA tissue levels of stressed gastric mucosa.
     方法:建立冷束缚大鼠应激性胃粘膜损伤模型,观察:①内源性NO对胃粘膜损伤的影响; ②应激后大鼠胃粘膜内巯基物质和脂质过氧化终产物丙二醛(MDA)含量的改变;
短句来源
     Results Cold-restraint stress can inhibit significantly gastric emptying and motor activity of small intestine in rats,and by contrast EGb can reverse these effects evoked by cold-restraint stress.
     结果冷束缚应激可抑制大鼠胃排空速度(P<0.05)及小肠推进率(P<0.01),EGb干预可明显拮抗冷束缚应激引起的胃排空速度及小肠推进率降低(P<0.05);
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  “冷束缚”译为未确定词的双语例句
     Methods: The contents of CGRP in plasma of rats were assessed with RIA before and 2,4,6,8 h after stress.
     方法:制作大鼠冷束缚应激动物模型,检测应激前及应激后2,4,6,8h大鼠血浆CGRP含量;
短句来源
     The paper reported the effects of intracerebroventricular injection GABA (4 umol) on four different gastric ulcerations induced by ethanol (0. 2ml, i. g. ) , HCl (0. 2N, 0.2ml, i. g. ) , indomethacin (32mg/kg, i. m. )
     报道小白鼠脑室注射GABA(4ml)对无水乙醇(0.lml灌胃)、盐酸(0.2N,0.2ml灌胃)、消炎痛(32mg·kg-1肌肉注射)和冷束缚应激(1℃)四种不同实验性胃溃疡及其粘液屏障的影响。
短句来源
     The results suggest that in the process of formation of SU,ET 1 may be synthesized and secreted by gastric mucosa of CRS rats,and plays an important role in the pathogenesis of SU.
     提示在冷束缚应激诱导大鼠SU形成过程中 ,胃粘膜组织局部可大量增加ET 1的合成、分泌 ,从而参与SU的病理生理过程 ,可能是胃粘膜损伤的重要致病因子。
短句来源
     The anti-ET-1 preconditioning group underwent anti-ET-1 serum precondition 30,60 or 120min before the coldly restraint.
     ( 3 )抗ET 1血清预处理组 ,于成模后行 3 0 ,60 ,12 0min冷束缚前注射抗ET 1血清。
短句来源
     The Relation of TGF-β_1 Expression and Epithelial Cell Apoptosis on Rat Enteral Mucosae in Cold Stress
     冷束缚应激状态下大鼠小肠粘膜上皮TGF-β_1的表达与细胞凋亡关系的研究
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  cold restraint
To study adrenal contribution, we adrenalectomized rats and studied the effect of 3-wk of cold/restraint stress (1.5 h/d) on them compared to intact animals.
      
The effect of cannabinoid drugs (i.p.) on cold/restraint stress-induced gastric ulcers was studied in rats.
      
Gastric histamine content and gastric ulcer formation in cold/restraint stressed rats: Effects of cinnarizine and flunarizine
      
The effects of the calcium channel blockers cinnarizine (20 mg/kg p.o.) and flunarizine (10 mg/kg p.o.) on gastric and small intestine histamine content and ulcer formation were determined in cold/restraint stressed rats (4°C for 3 h).
      
Effects of (R)->lt;alpha>-methylhistamine and its prodrug BP 2.94 on gastric mucosal lesions in cold/restraint stressed rats
      
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  cold-restraint
Effect of cold-restraint stress on the distribution of trace elements in rat tissues
      
The aim of this study was to assess the effects of cold-restraint stress on blood and the other tissue levels of trace elements.
      
In conclusion, cold-restraint stress may affect the metabolic process by changing the distributions of zinc, magnesium, and manganese in tissues.
      
Immobilization and cold stress, applied separately or in combination (cold-restraint), did not influence significantly any of the analysed parameters compared to those of the control group of non-infected mice.
      
However, after application of cold-restraint plus influenza virus infection, the level of cytochrome P-450 and activity of cytochrome c reductase stayed markedly lower than in infected and non-stressed animals.
      
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We have studied, by means of some samples of the stress ulcer in rats when exposed to cold and restraint,the animal' injury on the gastric mucosa brought about by saline or allopurinol after being pre-processed . Thirty six rats are set at random into two groups: saline and allopurinol. As result, it is discovered that the parameters , in the allopurinol group , of injury on the gastric mucesa and of the degree of ulcer and that the damage of the mucosa are all apparently less than those in the saline group.Here...

We have studied, by means of some samples of the stress ulcer in rats when exposed to cold and restraint,the animal' injury on the gastric mucosa brought about by saline or allopurinol after being pre-processed . Thirty six rats are set at random into two groups: saline and allopurinol. As result, it is discovered that the parameters , in the allopurinol group , of injury on the gastric mucesa and of the degree of ulcer and that the damage of the mucosa are all apparently less than those in the saline group.Here it is confirmed that the oxygen free radicals derivedfrom xanthine oxidase hare played a principal role in the pathogenesis of acute stress ulcer ation of the rat.our suggestion is that the enzyme of xanthine oxidase should determine the source of these free radicals.

用大鼠冷束缚应激性溃疡模型,观察用生理盐水或别嘌呤醇预处理后,动物胃粘膜损伤的情况。结果发现,别嘌呤醇处理组胃粘膜损伤指数、溃疡深度指数及粘膜损伤度均显著低于生理盐水组。提示,来源于黄嘌呤氧化酶的氧自由基,对急性应激性溃疡的发生具有重要作用。

The effects of ginsenosides on the endocrine glands of rats exposed to cold restraint stress were studied by radioimmunoassay and electromicroscopy.In experiment group, the ginsenosides was injected intra-abdominally for successional 5 days in dose of 100mg/kg weighther day.All of them were killed after the 3 hour-cold restraiut stress on the 6th day.The results indicate that in stress, the ginsenosides inhibites the function excitement of the pituitary-adrenal gland system, helps to bring about enormous secretion...

The effects of ginsenosides on the endocrine glands of rats exposed to cold restraint stress were studied by radioimmunoassay and electromicroscopy.In experiment group, the ginsenosides was injected intra-abdominally for successional 5 days in dose of 100mg/kg weighther day.All of them were killed after the 3 hour-cold restraiut stress on the 6th day.The results indicate that in stress, the ginsenosides inhibites the function excitement of the pituitary-adrenal gland system, helps to bring about enormous secretion of the growth hormone and strengthens nonspecific resistance.

应用放射免疫法测定血清激素值和透射电镜观察相应内分泌腺细胞超微结构变化,研究人参皂甙对冷束缚应激大鼠内分泌腺结构和功能的影响。实验组经腹腔注射入参皂甙,每天100mm/kg体重,连续5天,第6天冷束缚应激3小时后处死。实验结果表明,人参皂甙对冷束缚应激大鼠,具有抑制垂体-肾上腺轴的功能活动,促进生长激素大量分泌,增强机体的非特异抵抗力。

KA8 is a C-terminal octapeptide (30-37) of oxyntomodulin and glicentin. KA8 could inhibite gastric secrection. But it is not clear if KA8 is able to inhibite gastric secrection induced by cold-restraint and TRH administration in intracerebroventricle. We designed two experiments. All the rats were fasted for 24 h before experiment. The rats with light ether anaesthesia were undertook the operation of pylorus and cardia ligation. After the rats remained consciousness, the rats were divided into two groups. One...

KA8 is a C-terminal octapeptide (30-37) of oxyntomodulin and glicentin. KA8 could inhibite gastric secrection. But it is not clear if KA8 is able to inhibite gastric secrection induced by cold-restraint and TRH administration in intracerebroventricle. We designed two experiments. All the rats were fasted for 24 h before experiment. The rats with light ether anaesthesia were undertook the operation of pylorus and cardia ligation. After the rats remained consciousness, the rats were divided into two groups. One Was injected by iv KA8 (60μg/kg), another was gaven NS as control, then the rats were sxposed 4 C enviroment for 2 hours. In the second experiment, the rats were implanted with tube in cerebro-ventricle before one week. The rats were administrated TRH (60μg/kg) through the tube and divided into two groups. One was injected by iv KA8, another Was received NS as a control. Each rat was placed in separated cage for 4 hours. All rats were decapitated and plasmas were stored in -20℃ for gastrin determination. The resoults shown that KA8 could significantly inhibited gastric secretion not only for rats exposed to cold, but also for TRH icy administration. But KA8 was not able to infulence the plasma concentration of pentogastrin. It suggest that KA8 inhibit gastric secretion but the biologica activity of KA8 is not associated with pentogastrin.

KA8是oxyntomodulin和glicentin的C端八肽(30-37)。KA8可以抑制胃酸分泌。我们设计了两个实验,证明KA8可以抑制冷束缚和TRH刺激引起的胃酸分泌,血中胃泌素水平无明显变化。这说明KA8不是通过抑制胃泌素而起作用的。

 
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