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细胞谷胱甘肽过氧化物酶
相关语句
  cellular glutathione peroxidase
     Effect of Nano-selenium on Cellular Glutathione Peroxidase Activity in Chick Hepatocytes
     纳米硒对肉鸡肝细胞中细胞谷胱甘肽过氧化物酶活性的影响
短句来源
     Cellular glutathione peroxidase(cGPX) is an important antioxidase in animals.
     细胞谷胱甘肽过氧化物酶 (cGPX)是动物体内一种重要的抗氧化酶。
短句来源
  “细胞谷胱甘肽过氧化物酶”译为未确定词的双语例句
     Effect of Macrophage Colony-Stimulating Factor on Glutathione Peroxidase Activity and Gene Expression in RAW 264.7 Cells
     巨噬细胞集落刺激因子对RAW264.7细胞谷胱甘肽过氧化物酶活性及其基因表达的影响
短句来源
     RT-PCR method for the semiquantitative analysis of cGPX mRNA level in pig muscle
     半定量RT-PCR法分析猪肌肉组织细胞谷胱甘肽过氧化物酶mRNA表达水平
短句来源
     Three selenium containing catalytic antibodies mHB4, mHB5 and mHB7, which acted as mimics of cytosolic glutathione peroxidase(cGPX), were prepared by chemically introducing selenium into monoclonal antibodies HB4, HB5 and HB7. HB4, HB5 and HB7 were raised against a GSH derivative GSH S DNP dibenzyl ester.
     通过单克隆抗体制备技术得到三株特异结合半抗原 4 ( GSH-S-DNP二苄酯 )的单克隆抗体 HB4 ,HB5和 HB7.抗体经两步化学诱变得到具有细胞谷胱甘肽过氧化物酶 ( c GPX)活性的含硒抗体酶 m HB4 ,m HB5和 m HB7,活力分别为 1 70 ,1 867,32 U/μmol.
短句来源
     The activities of intracellular glutathione peroxidase (GSH-Px), superoxided dismutase(SOD) and catalase (CAT) were measured.
     酶生化法检测细胞谷胱甘肽过氧化物酶(GSH-Px)、超氧化物歧化酶(SOD)和过氧化氢酶(CAT)活性;
短句来源
     Methods fluorosis rat were made by drinking diet water with sodium fluoride. GSH-Px activity and the level of SOD and MDA,and also cell cycle and apoptosis rate were tested in rat brain.
     方法应用饮水加入氟化钠进行大鼠染毒实验,测定大鼠脑细胞谷胱甘肽过氧化物酶(GSH-Px)活性、超氧化物歧化酶(SOD)活性和脂质过氧化终产物丙二醛(MDA)产生水平以及脑细胞周期变化及细胞凋亡率。
短句来源
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  相似匹配句对
     STUDY ON GLUTATHIONE PEROXIDASE
     谷胱甘肽过氧化物酶的研究
短句来源
     DETERMINATION OF GLUTATHIONE PEROXIDASE IN RICE SEEDLINGS
     水稻谷胱甘肽过氧化物酶的测定法
短句来源
     Cellular glutathione peroxidase(cGPX) is an important antioxidase in animals.
     细胞谷胱甘肽过氧化物酶 (cGPX)是动物体内一种重要的抗氧化酶。
短句来源
     Regulation of Glutathione Peroxidase Activity by Dicysteine Selenium in Leukemia Cell Lines
     硒代二半胱氨酸对白血病细胞谷胱甘肽过氧化物酶的影响
短句来源
     Effect of Nano-selenium on Cellular Glutathione Peroxidase Activity in Chick Hepatocytes
     纳米硒对肉鸡肝细胞细胞谷胱甘肽过氧化物酶活性的影响
短句来源
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  cellular glutathione peroxidase
Activity of cellular glutathione peroxidase was virtually abolished in Se-deficient rat liver, whereas activity of glutathione S-transferase was 43% higher than in Se-adequate liver.
      
Here, we report the effect of Fe overload in mice lacking cellular glutathione peroxidase (GPX1 knockout [KO] mice), the selenoenzyme thought to account for much of the antioxidant action of Se.
      
The activity of cellular glutathione peroxidase (GPx) was decreased at d 30 and 45 of gestation in liver of gilts fed the low-Se diet.
      
Cellular glutathione peroxidase and catalase activities were decreased on exposure to oxalate.
      
Erythrocytes were analysed for MDA (E-MDA), reduced glutathione (GSH) and cellular glutathione peroxidase (cGPx) activity.
      
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Using a luminol-dependent chemiluminescence assay we found tert-butylhydroperoxide to be a strong inhibitor of respiratory burst of the mouse peritoneal macrophage. However, the protection against the inhibition of respiratory burst induced by tert-butylhydrope-roxide was enhanced after the intraperitoneal injection of polysaccharide from Coriolus versicolor. Further investigation exhibited that glutathione peroxidase activity was markly elevated in PSK-treated macrophages, Meanwhile, higher activity of glutathione...

Using a luminol-dependent chemiluminescence assay we found tert-butylhydroperoxide to be a strong inhibitor of respiratory burst of the mouse peritoneal macrophage. However, the protection against the inhibition of respiratory burst induced by tert-butylhydrope-roxide was enhanced after the intraperitoneal injection of polysaccharide from Coriolus versicolor. Further investigation exhibited that glutathione peroxidase activity was markly elevated in PSK-treated macrophages, Meanwhile, higher activity of glutathione peroxidase was maintained in PSK-treated macrophages incubated with tert-butylhydroperoxide. These results suggested that the immunological function of macrophage was related to the activity of glutathione peroxidase. Non-specific immunopolysaccharide might prevent macrophage from damage induced by reactive oxygen species by enhancing antioxidation capacity.

用化学发光法观察到叔丁基氢过氧化物对培养的小鼠腹腔巨噬细胞呼吸爆发有强烈的抑制作用。云芝多糖经腹腔注射后,能增强巨噬细胞呼吸爆发功能对叔丁基氢过氧化物损伤的抵抗力。云芝多糖处理的巨噬细胞谷胱甘肽过氧化物酶基础活力显著提高,在叔丁基氢过氧化物作用下,云芝多糖处理的巨噬细胞仍有较高的谷胱甘肽过氧化物酶活力。说明巨噬细胞的免疫功能与谷胱甘肽过氧化物酶活力有关,非特异性免疫多糖可提高细胞抗氧化能力,减轻活性氧损伤作用。

In order to find out the mechanism that Polysaccharide Krestin (PSK) prevents the progression of atherosclerosis and the lipoperoxidative injury caused by oxidatively modified low density lipoprotein (OLDL) to macrophages, the alterations of GSHPx,SOD activity and NO release in PSK-treated mouse peri toneal macrophages and the effect of LPS on them were investigated. The results showed that with the peritoneal injection of PSK,the mouse peritoneal macrophages got: 1 )elevated SeGSHPx activity; 2)elevated non-SeGSHPx...

In order to find out the mechanism that Polysaccharide Krestin (PSK) prevents the progression of atherosclerosis and the lipoperoxidative injury caused by oxidatively modified low density lipoprotein (OLDL) to macrophages, the alterations of GSHPx,SOD activity and NO release in PSK-treated mouse peri toneal macrophages and the effect of LPS on them were investigated. The results showed that with the peritoneal injection of PSK,the mouse peritoneal macrophages got: 1 )elevated SeGSHPx activity; 2)elevated non-SeGSHPx and SOD activity; and 3) the enzyme activity can be further improved by lipopolysaccharide (LPS); 4)much No can be found to be released by PSK-treated mouse peritoneal macrophages when stimulated with LPS.

为探讨云芝多糖(PSK)预防动脉粥样硬化及防止氧化修饰低密度脂蛋白(O-LDL)对巨噬细胞的过氧化损伤的作用机理,考察了腹腔注射PSK对小鼠腹腔巨噬细胞谷胱甘肽过氧化物酶、超氧化物歧化酶(SOD)活性及一氧化氮(NO)释放的作用,及脂多糖(LPS)对这一作用的影响。结果显示腹腔注射PSK可以使小鼠腹腔巨噬细胞SeGSHPx酶活性、non-SeGSHPx及SOD活性升高;在LPS作用下可进一步升高上述酶活性,使NO释放量有较大增加。

ABSTRACT Aim The effects of macrophage colony stimulatingfactor (0CSF) on the antioxidative capacity and foamcell formation of murine peritoneal macrophages wereobserved.Methods MCSF was in the conditional cultural liquidsof L929 cell line. Superoxide dismutase(SOD) activi-ty was detected by Pyroyallol method, and seleniumdependent glutathione peroxidase (SeGSHPx) activitywas detected by DTNB method, the survival numberof murine macrophages was detected by MTT method.Results MCSF could enhance SeGSHPx and SOD...

ABSTRACT Aim The effects of macrophage colony stimulatingfactor (0CSF) on the antioxidative capacity and foamcell formation of murine peritoneal macrophages wereobserved.Methods MCSF was in the conditional cultural liquidsof L929 cell line. Superoxide dismutase(SOD) activi-ty was detected by Pyroyallol method, and seleniumdependent glutathione peroxidase (SeGSHPx) activitywas detected by DTNB method, the survival numberof murine macrophages was detected by MTT method.Results MCSF could enhance SeGSHPx and SOD ac-tivities , moreover , it could prevent foam cell formationcaused by active oxygen and elevate the survival num-ber of murine macrophages.Conclusion The enhancement of antioxidase activitiesby MCSF may be one of the reasons that MCSF canprevents foam cell formation and retard the progressionof atherosclerosis.KEY WORDS Macrophage colony stimulating factor;Seleniumdependent glutathione peroxidase: Super-oxide dismutase; Foam cell formation; Atheros-clerosis

越来越多的实验结果证实巨噬细胞集落刺激因子在动脉粥样硬化发生过程中的重要作用,为了探讨巨噬细胞集落刺激因子和活性氧与动脉粥样硬化之间的相互关系,观察了巨噬细胞集落刺激因子对培养的小鼠腹腔巨噬细胞抗氧化酶活性的影响。结果发现巨噬细胞集落刺激因子能使小鼠腹腔巨噬细胞谷胱甘肽过氧化物酶活性提高32%,超氧化物歧化酶的活性提高120%,并减轻叔丁基氢过氧化物促巨噬细胞源性泡沫细胞形成,以及提高巨噬细胞的生存数。此结果提示巨噬细胞集落刺激因子提高杭氧化酶活性的作用可能是其虽增加对氧化修饰低密度脂蛋白和胆固醇的摄入却能阻止泡沫细胞形成及动脉粥样硬化发生发展的原因之一。

 
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