The expression of CD19+CD25+ cells in peripheral blood in patients would reflect degree of liver injury and status of humoral immunity, that may be helpful to instruct clinical therapy and judge the prognosis.
Conclusions Ischemia preconditioning relieves the liver injury after reduced-size liver transplantation during early period,and facilitates the expression of Ref-1 protein in hepatic tissue after transplantation, implying the protection mechanism is at least partially relevant to the promotion of Ref-1 protein expression.
Results Pancreatic injury in 5 cases, gastric injury in 4 cases, hepatic injury in 2 cases, colon injury in 5 cases, diaphragmatic injury in 1 case and left renal injury in 1 case were detected.
Objective To study the mechanism of hepatic injury in aged rats with cerebral ischemic reperfusion from the unbalance between thromboxaneA 2(TXA 2)and prostacyclin(PGI 2)and the tumor necrosis factor(TNF)content.
Methods A retrospective analysis was made to the treatment of 46 cases with hepatic injury and hematocoelia reinfusion in 14 cases with hepatic injury.
Objective:To investigate the possible role of IκB kinaseβ(IKKβ),the pathogenesis of hepatic injury induced by resuscitation of hemorrhagic shock and endotoxin,and to evaluate the preventive effect of anisodamine(6542).
(3)Multiple Logistic regression analysis also showed significant interactions of VCM exposure with CYP2E1,CYP2E1 with GSTT1,and GSTT1 with GSTM1 to liver damage.
Results:The glossy ganoderma in a large dose(0.9g/kg,qd×10d,ig)could significantly decrease the levels of serum ALT(P<0.01) and AST (P<0.05), reduce the spleen lesion(P<0.05)and the liver damage in mice;
Serum F protein could be measured by radioimmunoassay. In the present study,serum values from 36 healthy subjects averaged 186± 31ng/ml-1 while those from 58 HBV hepatitis patients averaged 450±120ng/ ml-1, This new marker may be of value in the diagnosis of liver damage.
Methods Retrospective study of 76 hepatic trauma acses due to traffic accident was conducted for demonstrating the clinical manifestations and comparison of the results of prognosis in different operation time.
Methods Retrospective study of 76 cases of hepatic trauma caused by traffic accident was conducted to demonstrate the clinical manifestation and compare the survival rates in groups with different operative time.
Changes in sphingomyelinase activity, tumor necrosis factor α expression, and level of lipid peroxidation products in the course of development of cholestatic liver injury have been studied.
In conclusion, induction of HO-1 provides protection against liver injury during cold ischemia preservation and improves the preservation of liver graft.
The effects of pectin from the eelgrass Zostera marina on toxic liver injury induced by enteral administration of lead acetate are examined in experiments on rats.
The hepatoprotective potential of earthworm extract (EE) (Lampito mauritii, Kinberg) was evaluated against paracetamol-induced liver injury in Wistar albino rat, in comparison with silymarin, the standard hepatoprotective drug.
Whereas, the degree of hepatic injury was attenuated in PGZ prevention group, liver indexes and serum levels of ALT, ALP were significantly decreased (P>amp;lt;0.05) compared with model group I.
After 16 weeks, notable steatosis, and lobular inflammation were observed in model group II rat liver, while the degree of hepatic injury was attenuated in the PGZ treatment group.
Since leukocytes are much more available than hepatocytes or neuronal cells in humans, we assume that CE in peripheral blood leukocytes (neutrophils and monocytes) can be used as markers for indication of pending liver damage by CPZ.
In this study, we examined the effects of its ethanolic root extract on experimental liver damage in order to evaluate its hepatoprotective effects against hepatotoxicity induced in rats by ethanol at a dosage of 5 g/kg body weight for 60 days.
The degree of liver damage was analysed using serum marker enzyme activities, the total protein, albumin, globulin, ceruloplasmin and liver glycogen contents, and the A/G ratio.
The correlation of serum arylesterase (PON1) activity on phenylacetate determined by an integrated method to classical biochemical indexes of liver damage was investigated for the use of PON1 activity to evaluate liver damage.
The management of hepatic trauma should be, if possible, non-operative and is initially determined by hemodynamic stability, absence of coagulopathy and limited need for blood transfusions.
To evaluate the utility of routine follow-up computed tomography (CT) and/or ultrasound (US) scans in children with blunt hepatic trauma initially managed non-operatively.
Therefore, we believe that in asymptomatic children with blunt hepatic trauma who are clinically stable, routine follow-up imaging studies are of very limited value.
肝脏损伤
liver injury
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