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Genomic DNA was extracted from blood samples of healthy controls and from patients with HBV infection.
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The immunoassay was used to check the BALP of the blood serum that was from 42 primary osteosarcoma patients.
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The patients were followed up from five months to 49 months with an average of 24.3 months.
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Detection of abnormal myocardial perfusion is crucial to the prognosis of patients with coronary artery disease (CAD) after they have undergone percutaneous coronary intervention (PCI).
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For those patients with acute or total occlusion, the levels of myocardial perfusion before and after PCI were similar, as determined by IC-MCE and visually analyzed from 61 segments (P >amp;lt; 0.05).
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In all these cases we actually show that Γ=π1(M) has a finite index subgroup which is mapped onto a nonabelian free group.
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We can ensure that in most cases the hypothesis onG-invariance is satisfied.
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Next we settle the remaining cases, with a computer-aided variation on the old method of Donkin.
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We describe a basis forY, show that it is a polynomial algebra and describe its rank, which we compute explicitly in a number of cases.
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Except for the Borel and some special cases a corresponding result is not known for the semi-centre of the enveloping algebra ofp.
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The patients were followed up from five months to 49 months with an average of 24.3 months.
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The patients' medical records were reviewed and the clinical information was extracted.
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All the patients were followed up postoperatively for more than one year.
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The patients with eye pterygia were divided into groups in terms of occupation, sex, age, length of history, grade of congestion, and size of the heads.
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There were significant differences in incidence among the patients of different occupation, grades of congestion, and size of heads.
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Detection of a new mutation (T1140C) in a patient with Hunter syndrome from Guangdong, China
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This study identified mutations of the idurnate-2-sulfatase (IDS) gene in a patient with Hunter syndrome, and established a basis for the diagnosis of the prenatal gene of Hunter syndrome.
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The patient is a hemizygote, and his mother is a heterozygote.
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It is possible that this mutation severely impairs enzymatic activity and is the underlying basis for the pathology seen in this patient with Hunter syndrome.
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A patient suffered from GERD without esophageal symptoms, which was diagnosed and treated as bronchial asthma during his five emergency admissions.
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