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游离钙浓度
相关语句
  free intracellular calcium concentration
    The effect of postburn serum on the free intracellular calcium concentration of the in vitro cultured Kupffer cells
    烫伤血清对大鼠库普弗细胞胞浆游离钙浓度的影响
短句来源
    Objective To study the effects of post burn serum on the free intracellular calcium concentration ([Ca 2+ ] i) in the isolated cardiac ventricular myocytes and L type calcium (I Ca L ) currentsin the cardiac myocyte membrane.
    目的 观察烫伤血清对分离的成年大鼠心室肌细胞游离钙浓度 ([Ca2 + ]i)及胞膜钙通道电流的影响。
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  “游离钙浓度”译为未确定词的双语例句
    Furthermore, we should observe effect indices of muscle relaxant and clinical signs for recovery after operation. Results: Compared with those before AHHD, Hb and Hct dropped by 34.5% and 32.6%, TPP and Alb dropped by 21.9% and 25.9%, concentrations of K~+ and Ca~(2+) dropped by 7.7% and 47.8% respectively after AHHD.
    结果:与AHHD前相比,AHHD后Hb和Hct分别下降34.5%和32.6%,TPP与Alb分别降低21.9%和25.9%,血钾和游离钙浓度分别降低了7.7%和47.8%,其差异均有统计学意义。
短句来源
    Methods The changes of plasmic protein tyrosine kinase (P TK),protein kinase C (PKC),PI-PLC activivies and cytoplasmic calcium concentr ation were detected,the secretion of IL-2,IL-10 and the proliferating functi on of T cells at different postburn periods.
    方法 检测伤后早期 ( 2~ 168h)T细胞胞浆蛋白酪氨酸激酶(PTK)、蛋白激酶C(PKC)、肌醇磷脂特异性磷脂酶C (PI PLC)的活性改变和胞浆游离钙浓度变化 ,观察伤后T淋巴细胞增殖转化功能及白细胞介素 (IL) 2、IL 10分泌活性 ,分析各信号转导分子活性改变在导致伤后T细胞功能受抑中的作用。
短句来源
    ② The leak of CK, LDH, myocardial content of lactate and MDA, and the intramyocyte calcium after retrograde perfusion was much lower in group B than those of group C and D.
    B组心肌酶漏出、乳酸生成量、心肌细胞内游离钙浓度、心肌细胞膜丙二醛 (MDA)含量低于C组和D组 ,C组低于D组。
短句来源
    Effects of postburned serum on free intracellular calcium in isolated cardiac myocytes of adult rat
    烫伤血清对成年大鼠分离的心肌细胞游离钙浓度的影响
短句来源
    The effect of postburn serum on free intracellular calcium concentration and production of nitric oxide in macrophages
    烧伤血清对大鼠腹腔巨噬细胞内游离钙浓度及其产生NO的影响
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  free intracellular calcium concentration
Aequorin measurements indicated that the contractile deficit in the failing myocardium at higher rates of stimulation is associated with decreased free intracellular calcium concentration.
      
TFA significantly reduced the resting and CaCl2-induced increase of free intracellular calcium concentration ([Ca2+]i) in rabbit plateletin vitro.Conclusion: TFA has an antiplatelet effect via the inhibition on the influx of Ca2+.
      
The free intracellular calcium concentration was slightly but significantly diminished in patients compared with healthy volunteers.
      
Specifically, when keratinocytes were exposed to thapsigargin under calcium-depleted culture conditions the subsequent increase in free intracellular calcium concentration was moderate in NF1 keratinocytes compared to controls.
      
Whole-cell patch-clamp recordings of membrane currents and fura-2 measurements of free intracellular calcium concentration ([Ca2+]i) were used to study Mn2+ influx in rat peritoneal mast cells.
      
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Abstract Model of rat brain injury were performed by a weight drop device.Ca(2+)content within cortex neurons were examined with Fura-2/AM fluorescent method.It was found free Ca(2+)concentration in neuronal synaptosomes increases greatly 30 min.after head injury (6 times than control value)and lasted for 72 hours.Meanwhile,brain edema developed.Calmodulin content in injured cortex increases at the same time.It is suggested that neuronal Ca(2+)overload is one of the key factors in the development of traumatic...

Abstract Model of rat brain injury were performed by a weight drop device.Ca(2+)content within cortex neurons were examined with Fura-2/AM fluorescent method.It was found free Ca(2+)concentration in neuronal synaptosomes increases greatly 30 min.after head injury (6 times than control value)and lasted for 72 hours.Meanwhile,brain edema developed.Calmodulin content in injured cortex increases at the same time.It is suggested that neuronal Ca(2+)overload is one of the key factors in the development of traumatic brain edema.The treatment for severe head injury with Ca(2+)channel blocker,Nimodipine,has gotten a excellent result and reduced the morbility which are confirmed by both animal experiment and clinical trial.

采用大鼠落体致伤脑挫裂伤模型、Fura-2荧光探针Ca~(2+)检测法,发现脑皮质神经细胞突触体胞浆游离钙浓度,于伤后30分钟已显著增高为对照值之10倍,持续至伤后72小时。脑水肿亦随之发生,6小时达高峰。脑皮质活性钙调素含量在伤后6~48小时相应增高。论证脑伤后神经细胞钙超载是导致脑水肿的关键因素之一。实验与临床应用钙通道阻断剂尼莫地平治疗重型颅脑伤,取得良好效果,死亡率明显降低。

AbstractThis experimental study in serious scald miceshowed that the cytosolic . free calcium ion concentrationand the capacity of secreting TNF-α of macrophage(MΦ)were obviously increased while the secretion ofIL-1 was significantly decreased,and accompanied withsimultaneous apparent rising of serum TNF-α in earlypostburn period.These indicate that MΦ have beenhighly or even over activated after burn. But the abilityof antigen presentation of MΦ during this period wasdeeply suppressed rendering the obvious...

AbstractThis experimental study in serious scald miceshowed that the cytosolic . free calcium ion concentrationand the capacity of secreting TNF-α of macrophage(MΦ)were obviously increased while the secretion ofIL-1 was significantly decreased,and accompanied withsimultaneous apparent rising of serum TNF-α in earlypostburn period.These indicate that MΦ have beenhighly or even over activated after burn. But the abilityof antigen presentation of MΦ during this period wasdeeply suppressed rendering the obvious declination ofactivity and proliferation response of antigen pulsedlymphocyte. It might be concluded that the defectiveantigen presentation of MΦ played a critical role in ear-ly immune dysfunction of severe scald mice, and thehighly activated or even overactivated MΦ by excessivesecretion of TNF-α were the molecular basis of al-teratios of MΦ function.

作者在实验中观察到小鼠严重烫伤后其巨噬细胞内游离钙浓度明显增高,分泌肿瘤坏死因子-α的能力明显增强,而分泌之白介素-1则逐渐减少,同时伴血清肿瘤坏死因子-α水平显著升高。这表明巨噬细胞在烫伤后已被充分激活甚至过度活化,烫伤后巨噬细胞的抗原提呈能力明显受抑制,这可使致敏淋巴细胞的活化和增殖反应显著减弱。作者认为严重烫伤后小鼠巨噬细胞抗原提呈功能的明显低下,在伤后早期免疫功能紊乱中起着重要作用,而伤后巨噬细胞的高度活化、肿瘤坏死因子-α分泌过多引起的巨噬细胞过度活化,又是巨噬细胞功能改变的分子基础。

The changes of free calcium level(Ca2+).calmodulin(CaM)and CaM-dependent protein kinase(CaM-PK)activity in the activated T-cells after trauma and their relationship with T-cell functions were studied in mice.It was found that Ca2+in activated T-cells was reduced after trauma,which was closely related to the reduction of interleukin 2(IL-2)mRNA and IL-2 receptor alpha mRNA levels,a decrease of IL-2 production,a suppression of IL-2 receptor alpha expression and a reduction of T-cell transformation in T-cells,CaM...

The changes of free calcium level(Ca2+).calmodulin(CaM)and CaM-dependent protein kinase(CaM-PK)activity in the activated T-cells after trauma and their relationship with T-cell functions were studied in mice.It was found that Ca2+in activated T-cells was reduced after trauma,which was closely related to the reduction of interleukin 2(IL-2)mRNA and IL-2 receptor alpha mRNA levels,a decrease of IL-2 production,a suppression of IL-2 receptor alpha expression and a reduction of T-cell transformation in T-cells,CaM and CaM-PK activities in the activated T-cells were significantly decreased after trauma.Calcium cation ionophore A23187 could restore Ca2+level in activated T-cells of traumatized mice to the control level in vitro and it could also inncrease CaM and CaM-PK activities and IL-2 mRNA and IL-2 receptor alpha mRNA levels significantly though they were still under the normal levels.It is suggested that the reduced Ca2+and CaM and CaM-PK activities are the important factors to result in the suppression of T-cell functions after trauma.

研究了创伤小鼠活化T细胞内游离钙浓度([Ca2+]i)、钙调素(CaM)、钙调素依赖的蛋白激酶(CaM-PK)活性的变化及其与T细胞功能之间的关系。结果显示,创伤后活化T细胞内[Ca2+]i降低,且这一变化同创伤小鼠T细胞白介素2(IL-2Rα)mRNA、IL-2受体α(IL-2Rα)mRNA水平降低,IL-2生成减少,IL-2Rα表达受抑,T淋巴细胞转化(TLT)降低密切相关。创伤后活化T细胞内CaM、CaM-PK活性明显降低。钙离子导入剂A_(23187)在体外可提高创伤小鼠活化T细胞内[Ca2+]i至正常对照水平,并明显提高其CaM、CaM-PK活性及IL-2mRNA、IL-2RαmRNA水平,但不能使之恢复正常。提示活化T细胞内[Ca2+]i、CaM、CaM-PK活性降低是导致创伤后T细胞功能受抑的重要原因。

 
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