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肾中毒
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  renal intoxication
     Conclusion Drug induce renal intoxication and allergic nephritis are constantly the factors causing acute renal failure.
     结论 药物性肾中毒,低容量及药物性低血压所致肾低灌注及药物过敏性间质性肾炎是医院获得性急性肾功能衰竭的主要病因。
短句来源
  nephro toxicity
     But the TNF levels in CsA nephrotoxicity group (0.91±0.22 ng/L, 0.4±0.27 ng/L) were not significantly different from those in functionally stable renal graft group.
     而CsA肾中毒组的血、尿TNF(0.91±0.22ng/L、0 40±0.27ng/L)与肾功能稳定组无明显差别。
短句来源
     Additionally UALB (207.5±112. 7mg/L) had been found dominance in acute rejection while Ua1-M(95.0±34.1mg/L) in chronic CSA nephrotoxicity.
     肾移植急性排异以UALB增幅最大(207.5±112.7mg/L),而慢性环泡霉素A(CSA)肾中毒则以Ual-M增高为主(95.0±34.1mg/L)。
短句来源
     The blood concentration of CsA was decreased in group of acute rejection and increased in group of CsA nephrotoxicity.
     CsA血药浓度在急性排斥反应组明显降低 ,而CsA肾中毒组显著升高。
短句来源
     The occurrence of UALB was the most pronounce in acute rejection while Uα1 M being the most in chronic CsA nephrotoxicity.
     肾移植急性排斥以UALB增幅最大(2075±1127mg/L),而慢性CsA肾中毒则以Uα1M增高为主(950±341mg/L)。
短句来源
     Conclusion (1)HSPs may act as one of the earliest indicators of acute nephrotoxicity.
     结论(1)HSPs可作为急性肾中毒损伤的最早标志之一。
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  “肾中毒”译为未确定词的双语例句
     CLINICAL STUDY ON PLASMA 6- Keto-PGF_(1a) AND TxB_2 CHANGES IN CsA NEPHRO-INTOXICATION
     CsA肾中毒时血桨中6-酮-PGF_(1α)和TxB_2的变化
短句来源
     According to Pathogenic mechanism,18 cases of renal fail ure were divided into 13 cases of hepatorenal syndrome,2 cases of acute hyperuricemia nephropathy,2 cases of acute renal intoxicatin,and 1 case of obstractive nephropathy.
     按肾功能衰竭发病机理分为肝肾综合征13例、急性高尿酸血症肾病2例,急性肾中毒2例,梗阻性肾病1例。
短句来源
     Theideal immunosuppressive CsA trough levels were found as follows: The whole blood CsA trough level was 300~450ng/ml within the first month after transplantation, 250~400ng/ml in three months, E00~350ng/mi in six months and 150~250ng/ml since then.
     CsA治疗浓度与发生排斥反应时的浓度及肾中毒浓度均有一定程度的重叠。 结论:认为术后CsA理想的治疗窗浓度应为:术后第1月内为300~450ng/ml,3月内为250~400ng/ml,半年内为200~350ng/ml,以后CsA浓度最好维持在150~250ng/ml。
短句来源
     Methods:Administering CsA 50mg/kg·d to group of rats for 7 days resulted in CsA acute intoxication. Other two groups received Huang Shen Mixture 4ml/kg·d and 8mg/kg·d before CsA.
     方法 :以Cs A5 0 mg/ kg·d灌胃 7d导致急性 Cs A肝肾中毒 ,另二组给药前分别给换肾合剂 4ml/ kg· d和 8ml/k· d。
短句来源
     CONCLUSION:In order to prevent toxic reˉaction in liver and kidney by CsA,blood concentration of CsA in the older-aged renal transplant patients should be monitored.
     结论:应监测老年肾移植患者CsA血药浓度,以防止CsA致肝、肾中毒等不良反应的发生。
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  renal intoxication
The renal alterations are thought to be caused by renal intoxication with damage of capillary walls and circulatory disturbance due to the viral hepatitis and encephalitis.
      
  nephro toxicity
Cyclosporine, used in the early post-transplant period to prevent GVHD, is another agent capable of producing either acute or chronic nephrotox icity.
      
Many risk factors associated with aminoglycoside neph rotoxicity have been identified in humans.
      
Nine patients with cyclosporine nephro toxicity all had normal duplex scans.
      
The mechanism by which FK506 produces neph rotoxicity is unknown.
      
Various roles can be postulated for Ang-1 after acute neph rotoxicity.
      


In recent years, the incidence of gentamicin nephrotoxicity and its induced acute renal failure(ARF) is on the increase. 3 patients with ARF in relation to the gentamicin treatment were studied. Among them, 2 cases of ARF were caused directly by gentamicin nephrotoxicity, one of which was nonoliguric and the other oliguric. Although the third case had traumatic history, the cause of ARF was related to gentamicin nephrotoxicity. In this paper, the causes of ARF in the 3 cases were analysed and such problems as...

In recent years, the incidence of gentamicin nephrotoxicity and its induced acute renal failure(ARF) is on the increase. 3 patients with ARF in relation to the gentamicin treatment were studied. Among them, 2 cases of ARF were caused directly by gentamicin nephrotoxicity, one of which was nonoliguric and the other oliguric. Although the third case had traumatic history, the cause of ARF was related to gentamicin nephrotoxicity. In this paper, the causes of ARF in the 3 cases were analysed and such problems as application of gentamicin and how to apply it clinically were discussed.

报告3例与庆大霉素治疗有关的急性肾功能衰竭,其中2例单纯为庆大霉素肾中毒所致。另1例虽有外伤史,但庆大霉素的肾毒性不容忽视。通过分折上述3例急性肾功能衰竭的发病原因,就临床上如何合理使用庆大霉素等问题进行了讨论。

This study was done to identify the ototoxic liability of gentamicin (GM) to the inner ear and kidney of 99 guinea pigs by electro-physiological and histo pathological methods.Additionally,the pharmacokinetics of GM in the plasma and perilymph were determined.It revealed that the large dosage (80mg/kg/day),admin- istered continually (13-26 days),produced obviously lesions within the inner ear and kidney,and with more than moderate hearing threshold shift.Some correlations existed in the damage between the inner...

This study was done to identify the ototoxic liability of gentamicin (GM) to the inner ear and kidney of 99 guinea pigs by electro-physiological and histo pathological methods.Additionally,the pharmacokinetics of GM in the plasma and perilymph were determined.It revealed that the large dosage (80mg/kg/day),admin- istered continually (13-26 days),produced obviously lesions within the inner ear and kidney,and with more than moderate hearing threshold shift.Some correlations existed in the damage between the inner ear and kidney of same individual,but were not parallel absolutely.By the GM pharmacokinetic analysis,it suggested that the accumulation of drugs in perilymph be a key point of GM ototoxicity by the distur- bance of the blood-labyrinth barrier. We suggested that the valley level of drug concentration of GM in plasma could be a monitoring criterion as important as the peak level and might be a more effective one.

采用电生理学及组织病理学方法对99只豚鼠进行实验研究,以证实庆大霉素对内耳及肾的中毒易损性。同时测定了庆大霉素在血清及外淋巴中的药物代谢动力学诸参数。当大剂量(80mg/kg/day)连续给药(13-26天)时,内耳及肾产生明显病损,并中等度以上听力阈移。同一机体中内耳与肾的损害存在一定的相关性,但非绝对平行。由药代动力学分析,内耳外淋巴中药物蓄积是庆大霉素耳中毒的重要环节,在于破坏了血-迷路屏障。作者建议庆大霉素在血清中的药物谷值浓度和峰值一样,同为临床监测指标,且可能是更为有效的一个监测拒标。

To study the mechanism of CsA-induced nephro-intoxication and find an effective monitory method for early detection, we measured plasma level of 6-keto-PGF1a and TxB2 (a main metabolite of PGI2 and TxA2 ) , using radio-immunoassay (RIA) in 21 kidney transplantation patients for 30 days.All these patients were treated with CsA and prednisone.Nine cases of CsA nephro-intox-ication occurred.One to three days prior to CsA nephro-intoxication, plasma level of 6-keto-PGF1a was decreased markedly (P<0. 05) . These...

To study the mechanism of CsA-induced nephro-intoxication and find an effective monitory method for early detection, we measured plasma level of 6-keto-PGF1a and TxB2 (a main metabolite of PGI2 and TxA2 ) , using radio-immunoassay (RIA) in 21 kidney transplantation patients for 30 days.All these patients were treated with CsA and prednisone.Nine cases of CsA nephro-intox-ication occurred.One to three days prior to CsA nephro-intoxication, plasma level of 6-keto-PGF1a was decreased markedly (P<0. 05) . These results suggest that specific inhibition of PGI2 may be one of the causes inducing CsA nephro-intoxication,and measuring plasma PGI2 level in transplantation Patients may be an effective monitory method for recognition of CsA nephro-intoxication.

本试验检测了21例肾移植病人血浆中6-酮-PGF_1α和TxB_2的浓度。结果表明,当临床出现CsA肾中毒症状前1~3天和当日,血浆中6—酮—PGF_1α水平明显降低。从而分析CsA肾中毒的发生可能是由于PGI_2降低后,使血浆中TxA_2和PGI_2的比例发生变化,TxA_2相应增加,在肾血管中微小血栓形成,使肾小球滤过率降低出现肾中毒的临床症状。我们认为在器官移植后应用CsA的病人中进行PGI_2的监测可以在临床早期发现CsA肾中毒,成为CsA肾中毒的可靠监测指标之一。

 
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