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肾近曲小管细胞
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  isolated rat renal proximal tubules
     Toxicity and cellular mechanisms of mercuric chloride and methylmercuric chloride were studied in isolated rat renal proximal tubules.
     研究了HgCl_2和CH_3HgCl对大鼠游离肾近曲小管细胞的毒性作用及其机理.
短句来源
  renal proximal tubule cells
     AIM:To investigate the influence of bioartificial renal tubule assist device(RAD) constructured by human renal proximal tubule cells (PTCs)on the blood pressure(BP),interleukin(IL) and survival time of acute renal failure(ARF) pigs combined with multiple organ dysfunctions syndrome(MODS)/multiple organ failure(MOF).
     目的观察人肾近曲小管细胞构建的生物人工肾小管辅助装置(bioartificialrenaltubuleassistdevice,RAD)对急性尿毒症并重症腹腔感染致多器官障碍综合征(multipleorgandysfunctionsyndrome,MODS)/多器官衰竭(multipleorganfailure,MOF)猪血压、白细胞介素、生存时间的影响。
短句来源
     Objective To test the role of a bioartificial kidney which consisted of a continuous vein-vein hemofiltration (CVVH) system and renal proximal tubule cells (PTCs)device, also called the renal tubule assist device (RAD),in endotoxin shock.
     目的生物人工肾是由连续静脉-静脉血液滤过循环(CVVH)系统和含有肾近曲小管细胞(PTCs)装置(也称肾小管辅助器, RAD)组成。 本实验重点测试其对内毒素休克猪心血管功能和细胞因子的改变。
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  “肾近曲小管细胞”译为未确定词的双语例句
     Conclusions Human RAD can ameliorate MAP, the level of TNF-α and prolong the survival period of pig models with MODS/MOF and ARF.
     结论应用人肾近曲小管细胞构建的RAD治疗MODS/MOF猪,能显著改善低血压,降低血TNF-α,延长生存时间。
短句来源
     A significant difference on survival time was also observed between the two groups,with(9.07 ± 0.88) h in the cell RAD group vs. (5.10 ± 0.46)h in the sham RAD group, P< 0.01. Conclusion The bioartificial kidney containing PTCs can improve cardiovascular function,reduce cytokine levels,and prolong survival time.
     两组生存时间相比,差异有显著性(P<0.01) ,治疗组(9.07±0.88) h比对照组(5.10±0.46) h长。 结论含有肾近曲小管细胞(PTCs)的生物人工肾影响内毒素休克猪心血管功能,降低促炎因子水平,有效地延长生存期。
短句来源
     Results In control,the shape of renal proximal tubule epithelial cell was regular; in mercury group,the damage was that the range of microvilli was severe mussily,the nuclei manifest the typical apoptosis character.
     结果透射电镜观察,对照组小鼠肾近曲小管细胞形态规则,单纯染汞组表现微绒毛排列紊乱,染色质聚集成块,核破碎释放出凋亡小体,凋亡小体被邻近细胞吞噬等典型细胞凋亡形态。
短句来源
     METHODS:Human PTCs were cultured to fabricate human RAD. ARF pigs with MODS/MOF were divided into 3 groups according to the random number table method:RAD group(n=5),sham RAD group(n=5) and control group(n=5).
     方法原代培养人肾近曲小管细胞并构建RAD,急性肾衰竭并MODS/MOF猪模型按随机数字表法分为3组RAD组(n=5),将RAD与血滤器连接,行连续静-静脉血液滤过(continuousvenovenoushemofiltration,CVVH)治疗24h;
短句来源
     A lot of lysosomes were seen in the cytoplasm of the renal proximal convoluted tubule cells.
     肾近曲小管细胞的胞质内有大量溶酶体。
短句来源
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     A lot of lysosomes were seen in the cytoplasm of the renal proximal convoluted tubule cells.
     管细胞的胞质内有大量溶酶体。
短句来源
     Results In control,the shape of renal proximal tubule epithelial cell is regular.
     结果对照组管细胞形态规则;
短句来源
     THE CULTURE OF PROXIMAL TUBULE CELLS OF RABBIT KIDNEY
     兔管细胞的培养
短句来源
     TOXICITY OF MERCURIC CHLORIDE AND METHYLMERCURIC CHLORIDE IN ISOLATED RAT RENAL PROXIMAL TUBULES
     氯化汞、氯化甲基汞对大鼠管细胞损伤机理的研究
短句来源
     The histological structure was observated under the optical microscope. Results In experiment group,the cytoplasm of proximal convoluted tubule showed hydropic degeneration.
     结果实验组组织中,管细胞的胞质呈水样变性。
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  renal proximal tubule cells
More recent work with rabbit renal proximal tubule cells suggests that the 12,13-metabolites of linoleic acid are more toxic than the 9,10-isomers.
      
We analysed the release of Up4A from cultivated renal proximal tubule cells after stimulation of protein kinase C with oleoyl-2-acetyl-sn-glycerol (OAG).
      
Platinum complex-induced dysfunction of cultured renal proximal tubule cells
      
Short-term peritubular alkalinization from 7.5 to 8.5 hyperpolarized (-8.8 mV) the basolateral membrane potential (V1) in HCO3- free Hepes buffered Necturus renal proximal tubule cells.
      
The transport of the amphiphilic organic anion, P-aminohippurate (PAH), across the luminal (brush-border) and contraluminal (basolateral) membrane of renal proximal tubule cells was studied with membrane vesicles isolated from bovine kidney cortex.
      
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Electron microscopic observations on the ultrastructures of the heart, brain and kidney were made in endotoxic shock model rabbits employing E. coli toxin injected intravenously. The sarcolemma of the cardiac muscle extended outwards forming numerous slender villous projections. Disarrangement and fragmentation of the myofilaments, and even disruption and dissolution of the myofibers were found. Many marginal folds formed from the surfaces of endothelial cells extending into the lumen. Degranulation and distention...

Electron microscopic observations on the ultrastructures of the heart, brain and kidney were made in endotoxic shock model rabbits employing E. coli toxin injected intravenously. The sarcolemma of the cardiac muscle extended outwards forming numerous slender villous projections. Disarrangement and fragmentation of the myofilaments, and even disruption and dissolution of the myofibers were found. Many marginal folds formed from the surfaces of endothelial cells extending into the lumen. Degranulation and distention of the cisternae of rough endoplasmic reticulum are common in neurons, swelling mitochondria, with disordered and disrupted cristae, or mitochondria of irregular sizes and forms could always be seen in both cardiac muscles and neurons. A lot of lysosomes were seen in the cytoplasm of the renal proximal convoluted tubule cells.

静脉注射大肠杆菌内毒素造成家兔内毒素休克模型。电镜观察其主要脏器超微结构的结果:心肌细胞的肌膜向外发出许多细长绒毛样突起,肌丝排列紊乱、断裂,并可出现肌纤维溶解消失等变化。毛细血管内皮形成许多边缘突伸向腔内。神经细胞的粗面内质网常有脱颗粒、囊扩张现象。在心肌与神经细胞内均可见到线粒体肿胀、嵴排列紊乱、结构破坏,及形状、大小差异较大的现象。肾近曲小管细胞的胞质内有大量溶酶体。

Rats presented significant nephrotoxicity after receiving10 mg/kg V_2O_5(ip)once daily for 4 consecutive days.On the secondday,the urine flow and urinary excretion of Na~+,K~+,glucose,enzymes(ALP,LDH,GOT GPT) were found to be increased,but bloodglucose and urine osmolality were found to be decreased.On the 4thday,additional finding was the rise of urinary protein,bloodcreatinine and urea nitrogen,Principal pathological changes appeared in the proximal convoluted tubules with cellular degenerations andnecrosis.The...

Rats presented significant nephrotoxicity after receiving10 mg/kg V_2O_5(ip)once daily for 4 consecutive days.On the secondday,the urine flow and urinary excretion of Na~+,K~+,glucose,enzymes(ALP,LDH,GOT GPT) were found to be increased,but bloodglucose and urine osmolality were found to be decreased.On the 4thday,additional finding was the rise of urinary protein,bloodcreatinine and urea nitrogen,Principal pathological changes appeared in the proximal convoluted tubules with cellular degenerations andnecrosis.The relationship between the nephrotoxicity and lipid per-oxidation was investigated by using zinc acetate (ZnAc)as an protectiveagent:The result revealed that both V_2O_5-induced lipid peroxidationin the kidney and manifestation of the acute nephrotoxicity showed adefinite amelioration after pretreatment with ZnAc.It thus appearedthat V_2O_5-induced lipid peroxidation may be one of the mechanismsin causing renal damage.

给大鼠腹注五氧化二钒(V_2O_5)10mg/kg,每天一次,连续四天,导致明显肾脏损害。染毒两天后,出现尿量、尿钠与钾排泄、尿糖以及尿中ALP、LDH、GOT、GPT酶活性增加;血糖和尿渗透浓度降低。染毒四天后,除上述改变外,还出现尿蛋白、血肌酐和尿素氮增高。主要病理学改变为肾近曲小管细胞变性和坏死。用醋酸锌作为保护剂研究肾损害与脂质过氧化的关系,结果表明,大鼠用锌预处理后,V_2O_5诱导的脂质过氧化和急性肾损同时减轻。提示 V_2O_5诱导的脂质过氧化可能是形成肾损害的机理之一。

Rabbits accepted daily 20mg/kg sodium fluoride in drinking water for 4 and 6 months. The amount of urinary γ-GT excretion increased. The activities of renal AKP, SDH and ACP decreased. The changes in renal tubular cells were swelling degeneration of mitochondria, expansion of endoplasmic reticulum, loss of microvillus. disappearance of basal folds, dropping of bound ribosomes from the face of rough endoplasmic reticulum, depolymerization of polyribosomes, and appearance of auto-phagic lysosomes. When rabbits...

Rabbits accepted daily 20mg/kg sodium fluoride in drinking water for 4 and 6 months. The amount of urinary γ-GT excretion increased. The activities of renal AKP, SDH and ACP decreased. The changes in renal tubular cells were swelling degeneration of mitochondria, expansion of endoplasmic reticulum, loss of microvillus. disappearance of basal folds, dropping of bound ribosomes from the face of rough endoplasmic reticulum, depolymerization of polyribosomes, and appearance of auto-phagic lysosomes. When rabbits accepted daily 30mg/kg sodium tetraborate during the period of fluoride exposure, boron reduced the adverse effects of fluoride on renal function and structure chiefly by decreasing the levels of fluoride in the kidneys.

家兔每日摄入氟化钠20mg/kg4和6个月,尿γ-谷氨酰转肽酶排出量增加,肾碱性磷酸酶、琥珀酸脱氢酶、酸性磷酸酶活性降低。肾近曲小管细胞线粒体肿胀变性,内质网扩张,微绒毛脱失,基底反褶消失,粗面内质网脱粒,核蛋白体解聚,自噬性溶酶体形成。摄氟的同时给予四硼酸钠30mg/kg,则可较明显地减轻氟对肾功能和结构的损伤。主要机理可能是硼与氟在体内形成氟硼酸根随尿排出,减少了氟在肾内的蓄积。

 
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