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ages抑制剂
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  ages inhibitors
     AGEs also may enhance oxidative stress,interact with receptor for AGEs(RAGE) binding on neuron and alter the endoneurial microcirculation,thereby contributes to the development of DN. AGEs inhibitors,soluble RAGE and anti-RAGE IgG have shown great promise in the prevention,delay and reversal of DN.
     单用或联用AGEs抑制剂、抗氧化剂、可溶性RAGE及抗RAGE IgG在防止、缓解、逆转糖尿病神经病变中显示出巨大的潜力。
短句来源
  ages-inhibitors
     Animal model studies and preliminary clinical trials have revealed that AGEs-inhibitors and the cross-links breakers, such as ALT-711, can reduce the severity of pathological changes of advanced glycosylation at several stages, especially the AGEs breaker can reverse stiffening of body tissues, organs and vessels, thus offering new therapeutic approaches for glucose-derived complications of diabetes and ageing.
     动物模型和临床前实验表明AGEs抑制剂以及AGEs交联结构裂解剂 ,如ALT 711,可以从不同的阶段打断AGEs的形成 ,特别是AGEs裂解剂能够逆转或软化由AGEs造成的组织、器官以及血管的硬化 ,因而有希望成为一类新型治疗药物 ,用于治疗糖尿病和衰老过程中由于AGEs引发的各种并发症
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  “ages抑制剂”译为未确定词的双语例句
     The above indexes in the diabetic rats treated with insulin and 2,3 DAP together(DID group) were significantly higher than those in D group(P<0 01) Conclusion:Penile erection was seriously impaired in rats with DM,and the function of penile erection in diabetic rats was improved by the treatment with inhibitors of AGE S
     结果 :DM组大鼠勃起次数明显少于对照组 ,合用胰岛素和 2 ,3-DAP组勃起次数明显多于 DM组 (P <0 .0 1)。 结论 :DM严重损坏大鼠的勃起功能 ,AGES抑制剂治疗能延缓糖尿病性勃起功能障碍的产生
短句来源
  相似匹配句对
     Pyridoxamine-A Natural AGEs/ALEs Inhibitor
     吡哆胺-一种天然的AGEs/ALEs抑制剂
短句来源
     Nitrification Inhibitor
     硝化抑制剂
短句来源
     Research and Development of Aldosterone Inhibitors
     醛固酮抑制剂的研究与开发
短句来源
     The activity of PKC could be enhanced by AGEs. Conclusions AGEs can upregulate VCAM-1 expression and PKC activity of HUVECs, and increase adhesion of HUVECs to PBMCs.
     蛋白激酶C(PKC)抑制剂可明显降低AGEs所致的VCAM-1表达增加及HUVECs-PBMCs黏附率增加。
短句来源
     AGEs have a tendency to polymerize.
     AGEs具有高度交联性。
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Objective:To study the effects of diabetes on the erectile function in rats and whether the function of penile erection in diabetic rats was improved by the treatment with inhibitors of AGE S Methods:Male Sprague Dawley rats were injected intraperitoneally with streptozotozin(STZ) to make experimental models of diabetes mellitus(DM).Diabetic rats were treated with insulin and 2,3 DAP for 8 or 16 weeks,then injected with apomorphine in all of the rats,penile erections were observed in order to evaluate their...

Objective:To study the effects of diabetes on the erectile function in rats and whether the function of penile erection in diabetic rats was improved by the treatment with inhibitors of AGE S Methods:Male Sprague Dawley rats were injected intraperitoneally with streptozotozin(STZ) to make experimental models of diabetes mellitus(DM).Diabetic rats were treated with insulin and 2,3 DAP for 8 or 16 weeks,then injected with apomorphine in all of the rats,penile erections were observed in order to evaluate their erectile function Results:Compaired with the controls(C group),penile erectile times in the DM rats(D group) were decreased significantly(P<0 01).The above indexes in the diabetic rats treated with insulin and 2,3 DAP together(DID group) were significantly higher than those in D group(P<0 01) Conclusion:Penile erection was seriously impaired in rats with DM,and the function of penile erection in diabetic rats was improved by the treatment with inhibitors of AGE S

目的 :探讨糖尿病 (DM)对大鼠勃起功能的影响 ,AGES抑制剂治疗是否能提高 DM大鼠的勃起功能。方法 :应用雄性 SD大鼠腹腔内注射链脲佐菌素 (STZ)制造 DM动物模型 ,用胰岛素和二氨基酚嗪 (2 ,3- DAP)对 DM大鼠进行干预治疗 8周、 16周 ,再通过勃起试验来评价其勃起功能。结果 :DM组大鼠勃起次数明显少于对照组 ,合用胰岛素和 2 ,3-DAP组勃起次数明显多于 DM组 (P <0 .0 1)。结论 :DM严重损坏大鼠的勃起功能 ,AGES抑制剂治疗能延缓糖尿病性勃起功能障碍的产生

Glucose and other reducing sugars react non-enzymatically with proteins, leading to the formation of advanced glycosylation end products (AGEs) and AGE-derived protein cross-linking. The accumulations of AGEs contribute to the pathological events leading to diabetic and aging complications. Animal model studies and preliminary clinical trials have revealed that AGEs-inhibitors and the cross-links breakers, such as ALT-711, can reduce the severity of pathological changes of advanced glycosylation at several stages,...

Glucose and other reducing sugars react non-enzymatically with proteins, leading to the formation of advanced glycosylation end products (AGEs) and AGE-derived protein cross-linking. The accumulations of AGEs contribute to the pathological events leading to diabetic and aging complications. Animal model studies and preliminary clinical trials have revealed that AGEs-inhibitors and the cross-links breakers, such as ALT-711, can reduce the severity of pathological changes of advanced glycosylation at several stages, especially the AGEs breaker can reverse stiffening of body tissues, organs and vessels, thus offering new therapeutic approaches for glucose-derived complications of diabetes and ageing.

葡萄糖和其他还原糖与蛋白的非酶糖基化反应导致晚期糖基化终产物 (advancedglycosylationendproducts,AGEs)以及由AGEs引发的蛋白交联物的形成 ,这是糖尿病和衰老过程中引起并发症的主要原因。动物模型和临床前实验表明AGEs抑制剂以及AGEs交联结构裂解剂 ,如ALT 711,可以从不同的阶段打断AGEs的形成 ,特别是AGEs裂解剂能够逆转或软化由AGEs造成的组织、器官以及血管的硬化 ,因而有希望成为一类新型治疗药物 ,用于治疗糖尿病和衰老过程中由于AGEs引发的各种并发症

Alzheimer's disease (AD) is a neuro-degenerative disorder in central nervous system. Drugs for prevention and treatment of AD have been one of the research focuses in recent years. In this paper, the recent research progress in drugs for treatment of AD and their mechanisms are reviewed, including AChE inhibitor,M1 receptor agonist,antioxidants,nerve growth factor,calcium regulator and so on.

阿尔茨海默病(AD)为中枢神经退化性疾病,是危害人类健康的一大综合征,治疗AD药物是近年来研究的一大热点。综述了近5年来治疗AD药物的研究进展,根据作用机制的不同对临床应用的抗AD和有促智活性的药物进行分类详述,主要分为乙酰胆碱酯酶抑制剂等改善胆碱功能的药物、M1受体激动剂、抗氧化药物、消炎镇痛药物、抑制Aβ蛋白形成的药物、神经生长因子、钙调节剂、晚期糖基化终产物(AGE)抑制剂以及中药复方等几类。

 
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