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免疫诱导
相关语句
  immune-induced
    Effects of Composite Blood-Activating Decoction on Bone Marrow Microenvironment in Mice of Immune-Induced Aplastic Anemia
    复方活血汤对免疫诱导再生障碍性贫血小鼠骨髓微环境的作用研究
短句来源
    Influence of Angelica Sinenisis Injection on the Expression Level of Cyclin D_2 in Immune-Induced Aplastic Anemia Mice
    当归注射液对免疫诱导再生障碍性贫血小鼠骨髓细胞周期蛋白D_2表达的影响
短句来源
    Influence of Angelica Sinenisis Injection on Bone Marrow Histology and Ultrastructure in Immune-Induced Aplastic Anemia Mice
    当归注射液对免疫诱导再生障碍性贫血小鼠骨髓细胞组织形态及超微结构的影响
短句来源
    Objective To investigate the influence of angelica sinenisis injection on bone marrow histology and ultrastructure in immune-induced aplastic anemia(AA) mice.
    目的 探讨当归注射液对免疫诱导再生障碍性贫血 (AA)小鼠骨髓细胞组织形态及超微结构的影响。
短句来源
    Conclusions Angelica sinenisis injection performs a protective role in immune-induced AA mice by increasing the quantity of hematopoietic cells and improving the structure of hematopoietic microenvironment.
    结论 当归注射液能通过促进骨髓造血细胞增生 ,改善骨髓微环境对免疫诱导AA小鼠进行保护作用。
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  “免疫诱导”译为未确定词的双语例句
    The influence of angelica polysaccharide on the expression level of cyclin D_2 in immuneinduced aplastic anemia mice
    当归多糖对免疫诱导再生障碍性贫血小鼠骨髓细胞周期蛋白D_2表达的影响
短句来源
    RESULTS:The liver fibrosis degree and level of mRNA and proteins of collagen I/III in the liver were significantly reduced in the decreasing order in oxymatrine preventive group, oxymatrine therapeutic group, and cochicine therapeutic group.
    结果:试验组肝组织结构明显好转,纤维组织增生程度明显低于肝纤维化模型组; I型、III型胶原含量及I型、III型胶原的mRNA表达均低于模型组免疫诱导型肝纤维化大鼠,其效果为试验组I(苦参素预防组)效果最好,试验组II(苦参素治疗组)次之,秋水仙碱治疗组再次之,三组均优于造模组.
短句来源
    METHODS:Rat immunogenic liver fibrosis model was induced by administration human serum albumin (HSA 200 g/L) intravenously, 80 rats were randomly divided into 5 groups.
    方法:采用免疫诱导型肝纤维化大鼠模型,80只Wister大鼠,分为5组,A预防组造模同时开始喂食双甲五灵冲剂;
短句来源
    Objective: To investigate the influence of angelica polysaccharide on the percentage of cells in G_0/G_1 and expression level of cyclin D_2 of bone marrow mononucleate cells (MNC) in immuneinduced aplastic anemia (AA) mice.
    目的:探讨当归多糖对免疫诱导再生障碍性贫血(AA)小鼠骨髓有核细胞增殖周期及其细胞周期蛋白D2 表达的影响。
短句来源
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  immune-induced
Expression of adherent molecule and cyclin by ligustrazine in bone marrow of mice with immune-induced aplastic anemia
      
Mice with immune-induced aplastic anemia (AA) were given 5 mg ligustrazine intraperitoneally twice a day.
      
The results showed that ligustrazine could improve the expression of adherent molecule and cyclin D2 in the bone marrow of mice with immune-induced aplastic anemia, thereby promoting the growth of hematopoietic cells.
      
The permeability of the blood-testis barrier to lanthanum during the immune-induced aspermatogenesis and following vasectomy in
      
We also sequenced three gram-negative binding proteins (GNBPs) and two immune-induced peptides with strong homology to the GNBPs.
      
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Objective: To study the effect of composite blood activating decoction (CBAD) on expression of adherent molecule CD 49 d and cyclin D 2 in bone marrow hematopoietic cells in experimental immune mediated aplastic anemia (AA) mice. Methods: After the model of AA was established, the animals were fed with 0.2ml of 100% CBAD, twice a day for 12 days. Te CD 49 d and cyclin D 2 expression level of bone marrow hematopoietic cells in model mice were measured by flow cytometor analysis system at the 13th...

Objective: To study the effect of composite blood activating decoction (CBAD) on expression of adherent molecule CD 49 d and cyclin D 2 in bone marrow hematopoietic cells in experimental immune mediated aplastic anemia (AA) mice. Methods: After the model of AA was established, the animals were fed with 0.2ml of 100% CBAD, twice a day for 12 days. Te CD 49 d and cyclin D 2 expression level of bone marrow hematopoietic cells in model mice were measured by flow cytometor analysis system at the 13th day of experiment. Results: CD 49 d expression level in CBAD group was significantly higher than that in the AA group (P<0 05), and was similar to that in the normal group (P>0 05). The expression of cyclin D 2 in CBAD group was significantly higher than that in the AA group (P<0 01), but the cell count of G 0+G 1 phase was significantly lower in the CBAD group, as compared with the AA group, P<0 01. Conclusion: CBAD can increase the expression of adherent molecule CD 49 d and cyclin D 2 in bone marrow hematopoietic cells, and promote the growth of hematopoietic cells.

目的:探讨复方活血汤(简称中药)对免疫诱导的再生障碍性贫血(再障)小鼠骨髓造血细胞粘附分子及细胞周期蛋白表达的作用。方法:建立免疫诱导的再障小鼠模型,喂饲100%复方活血汤每次02ml,每日2次,第13日用流式细胞仪检测小鼠骨髓单个核细胞β1整合素家族(CD49d/CD29,α4β1;又称VeryLateAntigens,VLA家族)及细胞周期蛋白D2的表达水平。结果:中药组CD49d表达明显高于再障组(P<005),且与正常组无明显差异;中药组细胞周期蛋白D2表达明显高于再障组(P<001);而其G0+G1期细胞数显著低于再障组(P<001)。结论:复方活血汤能增强免疫再障小鼠骨髓造血细胞粘附分子CD49d和细胞周期蛋白D2的表达,促进造血细胞的增生。

AIM:To study the effect of Oxymatrine on liver fibrosis in immunogenic liver fibrosis rat model. METHODS:Rat liver fibrosis model was induced by human serum albumin (HSA), 60 Wistar rats were randomly divided into 5 groups, control group without any treatment,liver fibrosis model group,oxymatrine preventive group, oxymatrine therapeutic group,and cochicine therapeutic group.The pathological changes of liver were observed by HE and Von-Gieson staining.The expressions of mRNA and proteins of collagen I/III in...

AIM:To study the effect of Oxymatrine on liver fibrosis in immunogenic liver fibrosis rat model. METHODS:Rat liver fibrosis model was induced by human serum albumin (HSA), 60 Wistar rats were randomly divided into 5 groups, control group without any treatment,liver fibrosis model group,oxymatrine preventive group, oxymatrine therapeutic group,and cochicine therapeutic group.The pathological changes of liver were observed by HE and Von-Gieson staining.The expressions of mRNA and proteins of collagen I/III in liver were determined by in situ hybridization and immunohistochemistry. RESULTS:The liver fibrosis degree and level of mRNA and proteins of collagen I/III in the liver were significantly reduced in the decreasing order in oxymatrine preventive group, oxymatrine therapeutic group, and cochicine therapeutic group. CONCLUSION:Oxymatrine may inhibit hepatic inflammation and hepatic synthesis of collagen I/III,and thus prevent and inhibit hepatic fibrosis.

目的:探讨苦参素对实验性大鼠肝纤维化的预防与治疗作用.方法:采用人血清白蛋白免疫损伤Wistar大鼠造成肝纤维化模型,60只Wister大鼠,分为5组:造模组、秋水仙碱治疗组、试验组I(苦参素预防组)、试验组II(苦参素治疗组),另设正常对照组.动态观察大鼠肝组织病例变化,采用HE染色观察肝组织改变及Von-Gieson胶原纤维特殊染色观察肝纤维化程度,并采用免疫组织化学染色及肝组织原位杂交检测I型、III胶原含量及I型、III型胶原mRNA的表达.结果:试验组肝组织结构明显好转,纤维组织增生程度明显低于肝纤维化模型组;I型、III型胶原含量及I型、III型胶原的mRNA表达均低于模型组免疫诱导型肝纤维化大鼠,其效果为试验组I(苦参素预防组)效果最好,试验组II(苦参素治疗组)次之,秋水仙碱治疗组再次之,三组均优于造模组.结论:苦参素可减轻肝脏炎症活动、抑制肝内胶原合成,对实验性大鼠肝纤维化具有防治作用.

AIM:To explore the effect of Chinese herb Shuangjiawuling in preventing hepatic fibrosis in rat with liver fibrosis. METHODS:Rat immunogenic liver fibrosis model was induced by administration human serum albumin (HSA 200 g/L) intravenously, 80 rats were randomly divided into 5 groups. Shuangjiawuling was taken orally simultaneously with (group A), immediately after (group B), or 3 months after ( group C) administration of HAS intravenously. Colchine tablets were taken orally with the administration of HAS (group...

AIM:To explore the effect of Chinese herb Shuangjiawuling in preventing hepatic fibrosis in rat with liver fibrosis. METHODS:Rat immunogenic liver fibrosis model was induced by administration human serum albumin (HSA 200 g/L) intravenously, 80 rats were randomly divided into 5 groups. Shuangjiawuling was taken orally simultaneously with (group A), immediately after (group B), or 3 months after ( group C) administration of HAS intravenously. Colchine tablets were taken orally with the administration of HAS (group D),normal rats were used as control ( Group E ). The pathological changes of liver tissue were observed by HE and Von-Gieson staining. The hepatic mRNA and protein of TIMP-1 and TIMP-2 were analyzed by in site hybridization and immunohistochemistry. RESULTS:Hepatic fibrosis developed gradually and became obviously on the third month after administration of HAS. And strong expressions of TIMP-1 and TIMP-2 were observed, however,its expression and hepatic fibrosis were reduced dramatically following administration of Shuangjiawuling and colchine tablets.The effect of Shuangjiawuling in preventing fi- brosis was superior to that of colchine tablets (P <0.05), and its effect in group A, B, and C decreased in order.CONCLUSION:Chinese herb Shuangjiawuling can inhibit the development of hepatic fibrosis and the expression of TIMPs, and promoted the effect of the degradation of the extracellular matrix.

目的:研究在活血化淤基础上辅以软坚散节自制的双甲五灵冲剂对大鼠肝纤维化模型抗纤维化的治疗机制.方法:采用免疫诱导型肝纤维化大鼠模型,80只Wister大鼠,分为5组,A预防组造模同时开始喂食双甲五灵冲剂;B治疗组1在造模结束的同时开始喂食双甲五灵冲剂;C治疗组2在造模结束3mo后开始喂食双甲五灵冲剂;D秋水仙碱组在造模结束的同时开始喂食秋水仙碱;另设正常对照组10只.采用HE染色观察肝组织改变及Von-Gieson胶原纤维特殊染色以观察肝纤维化程度,并采用肝组织原位杂交及免疫组织化学染色观察大鼠肝脏组织中TIMP-1和TIMP-2mRNA及蛋白的表达强度.结果:免疫诱导型肝纤维化大鼠,在造模结束后肝组织学改变呈进行性加重,以造膜结束后3mo为著,其TIMP-1和TIMP-2mRNA及蛋白呈强阳性表达,而经双甲五灵冲剂治疗后的大鼠与模型组大鼠相比,肝组织结构明显好转,纤维组织明显减少,TIMP-1和TIMP-2mRNA及蛋白表达明显降低(P<0.05),并且其效果为:预防组效果最好,治疗1组较治疗2组效果好,即造模同时开始喂食双甲五灵冲剂最好,造模结束同时用药好于造模结束3mo后用药,三...

目的:研究在活血化淤基础上辅以软坚散节自制的双甲五灵冲剂对大鼠肝纤维化模型抗纤维化的治疗机制.方法:采用免疫诱导型肝纤维化大鼠模型,80只Wister大鼠,分为5组,A预防组造模同时开始喂食双甲五灵冲剂;B治疗组1在造模结束的同时开始喂食双甲五灵冲剂;C治疗组2在造模结束3mo后开始喂食双甲五灵冲剂;D秋水仙碱组在造模结束的同时开始喂食秋水仙碱;另设正常对照组10只.采用HE染色观察肝组织改变及Von-Gieson胶原纤维特殊染色以观察肝纤维化程度,并采用肝组织原位杂交及免疫组织化学染色观察大鼠肝脏组织中TIMP-1和TIMP-2mRNA及蛋白的表达强度.结果:免疫诱导型肝纤维化大鼠,在造模结束后肝组织学改变呈进行性加重,以造膜结束后3mo为著,其TIMP-1和TIMP-2mRNA及蛋白呈强阳性表达,而经双甲五灵冲剂治疗后的大鼠与模型组大鼠相比,肝组织结构明显好转,纤维组织明显减少,TIMP-1和TIMP-2mRNA及蛋白表达明显降低(P<0.05),并且其效果为:预防组效果最好,治疗1组较治疗2组效果好,即造模同时开始喂食双甲五灵冲剂最好,造模结束同时用药好于造模结束3mo后用药,三组均明显优于秋水仙碱组.结论:中药双甲五灵冲剂治疗肝纤维化的机制可能通过抑制TIMP-1和TIMP-2mRNA及蛋白的表达而降低对MMPs的抑制,从而有利于MMPs对细胞外基质的降解.

 
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