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阳虚模型
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  “阳虚模型”译为未确定词的双语例句
     It could also promote the intestine movement, and the charcoal particles promoting rates were 90. 7%± 5. 6% and 90. 6%±5. 4% in the high and low dose groups, 73. 5 % ±6. 0 % in the YDM model group,respectively (P<0. 01 ).
     还可明显促进阳虚小鼠小肠推进运动,高、低剂量组小鼠小肠碳 末推进率分别为90.7%±5.6%和90.6%±5.4%,阳虚模型组小鼠为73.5%±6.0%(P<0.01)。
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     Ia Antigen Expression of Peritoneal Macrophage in Mice Model of Yang Deficiency and the Effect of Aconitine
     阳虚模型小鼠腹腔巨噬细胞Ia抗原表达及乌头碱的作用
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     Methods:20 male SD rats were used and randomly divided into 2 groups:normal group and shenyang-defect model group,and each group had 10 rats.
     方法:选用240-260克雄性SD大鼠20只,随机分为两组:正常组和肾阳虚模型组,每组10只。
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     The results showed : as compared to that of normal rat, DA and NA in cortisol model rat obviously decreased (p<0. 05, p<0. 01, respectively) , while MAO-B increased (p<0. 05).
     结果表明:可的松阳虚模型大鼠与正常大鼠比较其下丘脑多巴胺(DA)、去甲肾上腺素(NA)含量均降低(p<0.05,0.01); 单胺氧化酶—B(MAO—B)活性升高(p<0.05)。
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     In the RA team,the contents of SP and VIP obviously returned to normal,compared with the team of sufferer(P<0.01,P<0.05),and compared with the normally fed team there is no obvious statistic difference (P>0.05).
     黄芪建中汤能明显改善大鼠脾阳虚诸证,使SP含量降低(与脾阳虚模型组比P<0.01)、VIP升高(P<0.05),二者恢复至正常水平(与空白对照组比较P>0.05)。
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     model.
     模型
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     E model of CRM.
     E模型
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     METHODS The Shenyangxu model was established;
     方法:造肾阳虚大鼠模型;
短句来源
     On the 20 th day from feeding with the fodder,the mice were provided with Tabellae Thiamazoli to create Yang Deficiency models.
     第20d建立小鼠阳虚模型
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The binding of [~3H] quinuclidnyl benzilate(QNB) to membrane preparations of hyperthyroid rat brain(similar to yin-deficiency) and hypothyroid rabbit brain (similar to yang-deifciency) represents a specific interaction with muscarinic cholinergic receptor sites. In the hyperthyroid rat brain, the number of binding sites of the receptor decreased (P<0.05), while the affinity of the receptor was higher (P<0.05). In the hypothyroid rabbit brain, the number of binding sites of the receptor increased (P<0.05). It...

The binding of [~3H] quinuclidnyl benzilate(QNB) to membrane preparations of hyperthyroid rat brain(similar to yin-deficiency) and hypothyroid rabbit brain (similar to yang-deifciency) represents a specific interaction with muscarinic cholinergic receptor sites. In the hyperthyroid rat brain, the number of binding sites of the receptor decreased (P<0.05), while the affinity of the receptor was higher (P<0.05). In the hypothyroid rabbit brain, the number of binding sites of the receptor increased (P<0.05). It is found that tonics could regulate and correct the changes and the results suggested that the changes of muscurinic cholinergic receptor in the brain might be one of the characteristics occurring in case of yin-deficiency and yang-deficiency.

本文报告了利用放射性配基受体结合法分析测定了甲亢类阴虚和甲低类阳虚模型动物大脑中枢胆碱能M一受体的变化,以及养阴益气中药复方和温补肾阳中药复方对其影响,尝试从细胞生物学水平上对虚证的发病机理和中药复方治疗虚证的机制作些探讨。结果表明,甲亢类阴虚模型大鼠脑中枢胆碱能M一受体数量减少(P<0.05),素和力提高(P<0.05),养阴益气中药复方能提高其数量(P<0.05),降低其亲和力(P<0.05);相反,甲低类阳虚模型免大脑中枢胆碱能M一受体的数量增多(P<0.05),亲和力无明显改变,温补肾阳中药复方能降低其数量。提示中枢胆碱能M一受体的变化,可能是中医阴虚(甲亢)阳虚(甲低)病理生理功能改变的原因之一,中药对此具有纠正调整作用。

According to the theory of traditional Chinese medicine, In this experiment the Pi-Yang-Xu (deficiency of Yang of spleen) models of mice were reconstructed by principally feeding cabbage and lard, and then they were divided into 3 groups (group Ⅰ、Ⅱ and Ⅲ) and were administered with Xue-Lian polysaccharide solution, Sijunzi Decaetion and physiological saline solution respectively for 7 days. In addition, some normal mice were used as controls (group Ⅵ) and were administered with physiological saline solution...

According to the theory of traditional Chinese medicine, In this experiment the Pi-Yang-Xu (deficiency of Yang of spleen) models of mice were reconstructed by principally feeding cabbage and lard, and then they were divided into 3 groups (group Ⅰ、Ⅱ and Ⅲ) and were administered with Xue-Lian polysaccharide solution, Sijunzi Decaetion and physiological saline solution respectively for 7 days. In addition, some normal mice were used as controls (group Ⅵ) and were administered with physiological saline solution for same days. On the fifth day of adminisration, 1 ml of 5% suspension of soluble starch was injected into abdominal cavity in each mouse. On the next day of stopping administration, the phagocytic index and phagocytic rate of the macrophage from each mouse to RBC from chicken were measured. The results showed that the phagocytic indexes and phagocytic rates in groups Ⅰ, Ⅱ, Ⅲ and Ⅳ were 0.457±0.06559, 40.0±7.75%; 0.260±0.0498, 21.6±4.81%; 0.220±0.0444, 16.8±4.29% and 0.244±0.0532, 20.1±4.99% respectively, both indexes in group Ⅰ were greater than those in other groups distinctly. Statistically, there were significant differences between group Ⅰ and Ⅱ (p<0.001), group Ⅰ and Ⅲ (p<0.O01), gronp Ⅰ aud Ⅵ (p<0.001). It is suggested that the Xue Lian polysaccharide can distinctly promote the phagocytosis of macrophage from Pi-Yang-Xu models of mice

根据中医饮食失节,过食肥甘,饥饿等均可伤元气、损脾胃的理论,本次试验以莲花白、猪脂为主饲喂小鼠,复制成脾阳虚模型,将其分为Ⅰ、Ⅱ、Ⅲ组,Ⅰ组小鼠每日灌服雪莲多糖液0.5ml(含多糖25mg)/只,Ⅱ、Ⅲ组每日分别灌服四君子汤1ml(含生药1g)/只和生理盐水0.5ml/只,另以正常小鼠作对照(Ⅳ组),每日灌服生理盐水0.5ml/只,均各连服7天。在停药前两天,全部小鼠均以含可溶性淀粉5%为溶液1ml/只作腹腔注射,停药后的翌日,检测各组腹腔巨噬细胞对鸡红细胞的吞噬指数和吞噬百分率。结果Ⅰ组均值分别为0.457±0.0655,40.0±7.75%;Ⅱ组0.260±0.0498,21.6±4.81%;Ⅲ组0.220±0.0444、16.8±4.29%;Ⅳ组0.244±0.0532,20.1±4.99%,Ⅰ组两项指标明显高于其余各组,差异均极显著(p<0.001),表明雪莲多糖对脾阳虚小鼠腹腔巨噬细胞的吞噬功能有明显的增强作用。

The experimental yang deficiency modal mice made by hydrocortione were given Y-G-Y ig · l0g · kg-1 · d-1 × 10d, Cortisone content of peasma was drug group: 3.43±0.61, control group: 2.47±0.30mmol/L P<0.001, cAMP: 144.24±33.35 and 109.00± 31.89mmol/L P<0.25, cGMP: 28.37±10.22 and 45.39±15.33mmol/L, P<0.05.

对氢化考的松化阳虚模型小鼠,右归饮可使其血浆皮质醇及cAMP含量明显增加,cGMP含量明显降低。小鼠及免疫抑制模型小鼠给予右归饮,均可使外周血淋巴细胞NANE阳性率、血清溶血素含量、胸腺指数及腹腔巨噬细胞吞噬率显著增高。右归饮的疗效可能与其调节血浆皮质醇含量及cAMP∶cGMP值,增强机体免疫功能有关。

 
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