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放射性肝纤维化
相关语句
  hepatic radiation fibrosis
     Quantitative research on the process of hepatic radiation fibrosis
     放射性肝纤维化过程的定量研究
短句来源
     The results showed that hepatic radiation fibrosis occurred in 30 Gy irradiatied rats.
     结果表明,30Gy组在照射后1年内逐渐发生了放射性肝纤维化病变。
短句来源
     The significance of quantitative changes in the formation of hepatic radiation fibrosis is discussed.
     讨论了上述成分的定量变化在放射性肝纤维化形成中的意义
短句来源
  “放射性肝纤维化”译为未确定词的双语例句
     Conclusion after irradiation 6 months, liver damage of 10Gy group has been essential recovered, classic irradiation liver fibrosis has been appeared at 30Gy group.
     结论 照射后 6个月 10Gy组肝损伤基本恢复 ,30Gy组出现典型的放射性肝纤维化改变
短句来源
     but liver damnify is aggravated with the radiatd dosage enlarged. After irradiation 6 months,Iiver damage of group A has been recovered,classic irradiation liver fibrosis has appeared in group C.
     随着放射剂量加大肝损伤加重,照射后6个月:A组肝损伤基本恢复,C组出现典型的放射性肝纤维化改变。
短句来源
     Immunohistochemical studies on radiation hepatic fibrosis
     放射性肝纤维化的免疫组化研究
短句来源
     But radiation-induced liver injury was a serious complication to affect radiotherapy and prognosi of radiation disease.
     但放射性肝炎及其纤维化是影响腹部放疗疗效和放射病预后的严重并发症,有时放射性肝炎可导致放射性肝纤维化,常造成肝硬化腹水甚至肝衰竭而致死,而且至今尚无有特效的防治措施。
短句来源
     Fibronectin type Ⅲ and Ⅳ collagens also increased in the hepatocytes. The significance of the changes in strorna in radiation hepatic fibrosis is discussed.
     肝细胞浆内纤维连接蛋白、Ⅲ及Ⅳ型胶原增多.最后,作者对上述肝间质成分变化在放射性肝纤维化中的作用进行了讨论.
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     the toxic action of PZA on liver is greater than that of other antitubercular drugs.
     ?
短句来源
     In 2hr radioactivity in lung was the highest,In 24h radioactivity in liver was the highest.
     24h放射性最高。
短句来源
     CT Diagnosis of Local Radiation-Induced Hepatic Injury
     局限性放射性损伤的CT诊断
短句来源
     Quantitative research on the process of hepatic radiation fibrosis
     放射性纤维化过程的定量研究
短句来源
     Immunohistochemical studies on radiation hepatic fibrosis
     放射性纤维化的免疫组化研究
短句来源
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By means of routine technique and several itntnunohistochemical methods, we studied the pathological changes in rat livers which were locally irradiated with 60Co γ-rays of 0, 10, 30, 50 and 60 Gy. The results showed that the liver lesions induced by single γ irradiation were more serious and occurred earlier than those by fractional radiation. Radiation hepatic fibrosis occurred in 30 Gy group with single irradiation in which Kupffer cells obviously increased in the early phase, whereas fat-storing cells did...

By means of routine technique and several itntnunohistochemical methods, we studied the pathological changes in rat livers which were locally irradiated with 60Co γ-rays of 0, 10, 30, 50 and 60 Gy. The results showed that the liver lesions induced by single γ irradiation were more serious and occurred earlier than those by fractional radiation. Radiation hepatic fibrosis occurred in 30 Gy group with single irradiation in which Kupffer cells obviously increased in the early phase, whereas fat-storing cells did later.The type Ⅳ collagen was strongly positive and fibronectin increased in the earll fibrosis, while it was replaced by collagen in the late phase.Much more laminin and type Ⅳ collagen deposited in sinusoids,which were completely enclosed by base membrane and developed capillary vascularizat ion. Fibronectin type Ⅲ and Ⅳ collagens also increased in the hepatocytes. The significance of the changes in strorna in radiation hepatic fibrosis is discussed.

60Coγ射线照射大鼠肝区后,不同时间分批活杀,进行肝脏病理(常规及多种免疫组化染色)研究。结果表明:单次照射病变明显重于分次照射且发生时间早;照后早期枯否细胞增多为显著,而晚期贮脂细胞增多为明显,胞浆内Ⅳ型胶原呈强阳性;纤维连接蛋白在肝纤维化早期明显增多,晚期则为胶原所取代;层粘连蛋白及Ⅳ型胶原在窦壁沉积进行性增多,可见完整基膜包绕肝窦,使之呈毛细血管化;肝细胞浆内纤维连接蛋白、Ⅲ及Ⅳ型胶原增多.最后,作者对上述肝间质成分变化在放射性肝纤维化中的作用进行了讨论.

By means of optic microscope (HE,PAS,collagenous and reticular fiber staining),electron microscope and image analysis,the liver lesions within one year after local irradiation with 60 Co γ rays were studied quantitatively and dynamically.The results showed that hepatic radiation fibrosis occurred in 30 Gy irradiatied rats.In the course of fibrosis,the granules of glycogen in hepatocytes were decreased progressively;the collagenous fiber progressively increased;reticular fibers significantly increased...

By means of optic microscope (HE,PAS,collagenous and reticular fiber staining),electron microscope and image analysis,the liver lesions within one year after local irradiation with 60 Co γ rays were studied quantitatively and dynamically.The results showed that hepatic radiation fibrosis occurred in 30 Gy irradiatied rats.In the course of fibrosis,the granules of glycogen in hepatocytes were decreased progressively;the collagenous fiber progressively increased;reticular fibers significantly increased in 1 3 months after irradiation and decreased after 6 months due to necrosis of hepatocytes and collapse of hepatic cell cords.The significance of quantitative changes in the formation of hepatic radiation fibrosis is discussed.

经60Coγ线照射大鼠肝区,通过光镜(常规HE染色,肝细胞内糖原染色、胶原纤维及网状纤维染色)、电镜和图像分析仪,定量研究了照后1年肘脏的病理改变。结果表明,30Gy组在照射后1年内逐渐发生了放射性肝纤维化病变。在肝纤维化发生过程中,肝细胞内糖原颗粒含量进行性减少,间质中胶原纤维含量进行性增加,网状纤维于照射后1~3个月呈进行性增加。照射后6个月因肝细胞坏死、肝索塌陷,网状纤维呈减少趋势。讨论了上述成分的定量变化在放射性肝纤维化形成中的意义

Objective\ The promotive role of iron on pathogenesis of hepatic fibrosis and the protective role of taurine and L arginine against hepatic fibrosis were studied.Methods\ The model of rat radiation hepatic fibrosis was used.Experimental rats were divided into 0Gy,30Gy,30Gy+iron,30 Gy+taurine and 30Gy+L arginine groups.Serum iron,liver tissue hydroxyproline(Hyp) and malondialdehyde (MDA) were measured one and three months respectively after irradition of hepatic tissue,production and distribution characteristics...

Objective\ The promotive role of iron on pathogenesis of hepatic fibrosis and the protective role of taurine and L arginine against hepatic fibrosis were studied.Methods\ The model of rat radiation hepatic fibrosis was used.Experimental rats were divided into 0Gy,30Gy,30Gy+iron,30 Gy+taurine and 30Gy+L arginine groups.Serum iron,liver tissue hydroxyproline(Hyp) and malondialdehyde (MDA) were measured one and three months respectively after irradition of hepatic tissue,production and distribution characteristics of hepatic tissne type Ⅰ and Ⅲ collagen were observed with a polarizing microscope.Results\ Administration of iron agent could significantly increase hepatic tissue MDA content and serum iron concentration,one month after irradiation ,hepatic tissue Hyp in 30Gy+iron group began to increase,and collagen in hepatic tissue obviously increased.Taurine and L arginine could reduce serum iron concentration and decrease production of hepatic fissue Hyp.Conclusion\ Exogenous iron agent could promote early development of radiation hepatic fibrosis;taurine and arginine could diminish pathologic alteration of hepatic fibrosis to a certain extent.

应用放射性大鼠肝纤维化模型探讨铁剂在放射性肝纤维化发生中的作用及牛磺酸和精氨酸对肝脏的保护作用。方法将大鼠分为0Gy(正常对照)、30Gy和30Gy+铁剂、30Gy+牛磺酸和30Gy+精氨酸组。于肝区照射后1,3个月分别检测血清铁离子、肝组织羟脯氨酸(Hyp)和丙二醛(MDA)含量以及应用偏振光显微镜观察肝内Ⅰ、Ⅲ胶原纤维的生成及分布特点。结果铁剂能明显增加肝组织中MDA的含量和血清铁离子浓度。照射后1个月,铁剂组肝组织中Hyp开始明显增加,进而肝脏中胶原纤维产生增多。牛磺酸和精氨酸能明显降低血清铁离子浓度,减少肝组织Hyp含量。结论外源性铁剂能促进放射性肝纤维化的早期发生;牛磺酸和精氨酸可分别在一定程度上减轻放射性肝纤维化的病变

 
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