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慢性肝纤维化
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  chronic hepatic fibrosis
     Methods Concentrated solution of Dan-shen ranged from 2 g/kg to 8 g/kg (equal to raw herb) combined with extract of Ginkgo Biloba L ranged from 1.5 g/kg to 6 g/kg(equal to raw herb) were administered to chronic hepatic fibrosis rats .
     方法将丹参浓缩液与银杏叶提取物用量(含生药量)分别在2-8g/kg、1.5-6g/kg范围内配伍给慢性肝纤维化大鼠灌胃。
短句来源
     ②The serum GPT and AKP levels of rats chronic hepatic fibrosis induced by tetrachloromathane were remarkably lower than those in the saline control groups(P < 0.05-0.01), and the levels in the GQK treatment group were closed to those in the blank control group.
     ②肝纤克治疗各组及空白对照组四氯化碳致慢性肝纤维化大鼠血清谷丙转氨酶和碱性磷酸酶水平均明显低于盐水对照组(P<0.05~0.01),肝纤克治疗各组接近空白对照组。
短句来源
     ⑤To evaluate the effect of GQK on rat chronic hepatic fibrosis induced by tetrachloromathane: Ninety Wistar rats were selected and randomly divided into 6 groups: blank control group (normal diet and never to give any drugs);
     ⑤观察肝纤克对四氯化碳致大鼠慢性肝纤维化的影响:取Wistar大鼠90只,随机分为6组:空白对照组(给予正常饮食,不给任何药物);
短句来源
     Experiences of Professor Yao Xixian in Treating Chronic Hepatic Fibrosis Based on Blood Stasis Theory
     姚希贤瘀血论治慢性肝纤维化经验
短句来源
     Methods: Animal model of acute hepatic injury induced by carbon tetrachloride and acetaminophen as well as cell model of acute hepatic injury induced by carbon tetrachloride and hydrogen peroxide in vitro were used to observe the protective effect of ginger oils on liver, model of chronic hepatic fibrosis established by subcutaneous injection of carbon tetrachloride was used to observe anti-fibrosis effect of ginger oils and to search its possible molecular mechanisms.
     方法:通过复制四氯化碳和醋氨酚急性肝损伤动物模型,四氯化碳和过氧化氢体外急性肝细胞损伤模型,观察生姜油的保肝作用; 通过皮下注射四氯化碳,复制慢性肝纤维化模型,观察生姜油的抗肝纤维化作用及其可能的分子机制。
短句来源
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  chronic liver fibrosis
     Effects of resveratrol on chronic liver fibrosis in rats
     白藜芦醇对大鼠慢性肝纤维化的影响
短句来源
     Fuganbao has the theraputic capability to the chronic liver fibrosis caused by the CCl 4 in rats,it can run lower the liver fibrosis obviously.
     对 CCl4诱发大鼠慢性肝纤维化有治疗作用 ,可使肝纤维化明显减轻。
短句来源
     Objective:To assess the effects of resveratrol on chronic liver fibrosis induced by carbon tetrachloride (CCl_4) in rats.
     目的:观察白藜芦醇对四氯化碳(CCl4)致大鼠慢性肝纤维化的影响。
短句来源
  “慢性肝纤维化”译为未确定词的双语例句
     Colorimetry was used to measure the contents of ALT, AST, SOD, MDA, TP and Alb. ELISA was used to determine the contents of serum TGF-β1, HA and LN. In situ hybridization was used to determine the expression of TIMP-1 mRNA in hepatic tissue.
     采用比色法测定ALT、AST、SOD、MDA、TP和Alb含量,ELISA法测定血清TGF-β1、HA和LN含量,原位杂交法测定TIMP-1mRNA的表达情况,光镜检查急性肝损伤和慢性肝纤维化肝组织的形态学改变。
短句来源
     Methods We used CCl4 to induce the model of liver fibrosis in 23 rats, which were randomly divided into the control group and the model group.
     方法应用四氯化碳(CCl4)诱导大鼠慢性肝纤维化模型。 23只Wistar大鼠随机分为正常对照组(N组)和肝纤维化模型组(M组)。
短句来源
     Objective To investigate the effect of heluoshugan capsules on acetylcholine receptors and their possible mechanism in resisting hepatic fibrosis induced by carbon tetrachloride(CCl4).
     目的探讨和络舒肝胶囊对CCl4诱导的肝纤维化模型的肝脏中乙酰胆碱受体的影响及其抗肝纤维化的可能机制。 方法应用四氯化碳(CCl4)诱导大鼠慢性肝纤维化模型。
短句来源
     Objective To validate the protective role of Ruangan capsules in antagonizing chronic fibrosis of rat liver caused by carbon tetrachloride (CCl4).
     目的验证软肝胶囊对四氯化碳(CCl4)所致大鼠慢性肝纤维化模型的肝保护作用及抗纤维化作用。
短句来源
     Histopathology showed the effect of anti liver fibrosis of OKL 2.Conclusion:The protection of OKL 2 against liver fibrosis in rats induced by carbon tetrachloride was significant.
     结论 :OKL - 2对 CCl4 诱导的大鼠慢性肝纤维化具有明显的保护作用
短句来源
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  chronic hepatic fibrosis
Two resections were palliative for cholangiocarcinomas; the remainder included 9 hepatocellular carcinomas, 12 colorectal metastases, 2 adenomas, 3 cancers of the gallbladder, and one case of chronic hepatic fibrosis.
      


Human hepatic stimulator substance(hHSS) was extracted from the liver induced labor fetus from 4-6 months healthy pregnant women according to the method of LaBrecque.The bioactivity of hHSS was detected by determination of the rate of 3H-thymidine.incorporation into the hepa tocyte DNA after injection of hHSS into the abdominal cavity of partial hepatectomized rats.The results showed that the amount of DNA syn thesis stimulated by hHSS was 6 times as much as that of control group.The model of experimental chronic...

Human hepatic stimulator substance(hHSS) was extracted from the liver induced labor fetus from 4-6 months healthy pregnant women according to the method of LaBrecque.The bioactivity of hHSS was detected by determination of the rate of 3H-thymidine.incorporation into the hepa tocyte DNA after injection of hHSS into the abdominal cavity of partial hepatectomized rats.The results showed that the amount of DNA syn thesis stimulated by hHSS was 6 times as much as that of control group.The model of experimental chronic liver injury was established by injec tion of carbon tetrachloride(CCl4)hypodermically and feeding of alcohol.The experimental results showed:(1) hHSS could lessen the elevation of the liver HYP content in the chronic CCl4-alcohol intoxicated rats at the 28th and 42th days during the period of chronic liver injury.(2) Pathohistological finding indicated:hHSS could remarkably reduce the degree of liver fibrosis induced by chronic CCl4-alcohol injury in rats.

本工作从健康孕妇水囊引产4~6个月龄的胎儿取肝,采用LaBrecque方法提取肝刺激因子,称人肝刺激因子(humanHepaticStimulatorSubstance,hHSS)。将hHSS注入肝部分切除之大白鼠体内,用3H-胸腺嘧啶核苷掺入肝DNA法测定其生物活性。结果表明,我们提取的hHSS刺激肝细胞DNA合成较对照组增加6倍。采用皮下注射60%CCl4豆油溶液和饮用低浓度的乙醇制备慢性肝损伤动物模型,以观察kHSS对大鼠实验性慢性肝纤维化的影响。实验结果表明,(1)hHSS可以明显降低CCl4-乙醇所致慢性肝损伤时肝组织羟脯氨酸含量的升高,在实验第28、42天差异具显著性(P<0.05,0.01)。(2)病理组织学观察表明,hHSS可以明显降低CCl4-乙致慢性肝损伤时肝组织纤维化的程度。在实验的第42天差异具显著性(P<0.01)。本工作结果提示,肝再生刺激因子可能是一种有希望应用于临床治疗慢性肝病的生物制剂。

Objective:To observe the result of the Fuganbao(复肝宝) in treatment of experimental liver trauma and liver fibrosis.Methods:Fifty mice were served in the experiment of protection of liver and to lower enzyme,and 70 rats in the experiment of antiliver fibrosis.The mice or rats were randomly divided into five groups in each experiment.None medicine was given to the empty contrast team and the model team,The medical contrast team was given to Dahuang Zhechong pill (大黄虫丸,1 000 mg/kg),and the therapy team with little(375...

Objective:To observe the result of the Fuganbao(复肝宝) in treatment of experimental liver trauma and liver fibrosis.Methods:Fifty mice were served in the experiment of protection of liver and to lower enzyme,and 70 rats in the experiment of antiliver fibrosis.The mice or rats were randomly divided into five groups in each experiment.None medicine was given to the empty contrast team and the model team,The medical contrast team was given to Dahuang Zhechong pill (大黄虫丸,1 000 mg/kg),and the therapy team with little(375 mg/kg) and big dose(2 250 mg/kg) was given to the Fuganbao one time per day.The mice used in the experiment of protection liver and to lower enzyme was given to the medicine by oral for four days,and then 2 5% CCl 4(the dose was 0 01 ml/g) injected into their abdomen(but no use in the empty contrast team).After 20 hours,the mice were killed.The serum enzyme of glutamate pyruvate transminase(GPT) was determined and the pathological changes of the liver were observed.The rats used in the experiment of antiliver fibrosis,40% CCl 4 (the dose of 1 25 ml/kg) was injected into their subcutaneous two times per week,but except of the empty contrast team.After six weeks,the correspondence medicine was given to them for five weeks.Then the rats were killed,and the contents of the procollagen Ⅲ peptide(PⅢP) and collagenous protein in the heptic tissue were determined.The pathological changes were also observed.Results:Contrast the big and little dose Fuganbao team with the model team,the contents of GPT and hepatic collagenous protein were significantly different( P <0 05 or P <0 01),PⅢP and degree of the liver trauma and grade of liver fibrosis were significantly different(all P <0 05).Conclusions:Fuganbao can protect the liver when liver trauma is happened with the toxic reaction of CCl 4 in mice.Fuganbao has the theraputic capability to the chronic liver fibrosis caused by the CCl 4 in rats,it can run lower the liver fibrosis obviously.

目的 :观察中药复肝宝抗实验性肝损伤和肝纤维化的疗效。方法 :保肝降酶实验用 5 0只小鼠 ,抗肝纤维化实验用 70只大鼠 ,各随机分为 5组。正常对照组及损伤组不给任何治疗性药物 ,大黄虫丸组给大黄虫丸 10 0 0 mg/ kg,复肝宝大、小剂量组分别给复肝宝 2 2 5 0和 375 m g/ kg,均每日 1次。保肝降酶实验小鼠口服给药 4日后 ,腹腔注射 2 .5 %四氯化碳 (CCl4)油溶液 0 .0 1ml/ g(除正常对照组 ) ,2 0小时后处死 ,测定血清谷丙转氨酶 (GPT) ,并观察肝病理组织学改变。抗肝纤维化实验大鼠 (除正常对照组 )皮下注射 40 % CCl41.2 5 m l/ kg,每周 2次 ,6周后给相应药物 5周 ,然后处死 ,测定 型前胶原肽 (P P)及肝组织中胶原蛋白含量 ,并观察肝病理组织学改变。结果 :复肝宝大、小剂量组与损伤组比较 ,GPT及肝胶原蛋白含量均差异显著 (P<0 .0 5或 P<0 .0 1) ,P P、肝细胞损伤程度及纤维化分级亦有显著性差异 (P均 <0 .0 5 )。结论 :复肝宝对CCl4诱发小鼠急性肝损伤有保护作用 ;对 CC...

目的 :观察中药复肝宝抗实验性肝损伤和肝纤维化的疗效。方法 :保肝降酶实验用 5 0只小鼠 ,抗肝纤维化实验用 70只大鼠 ,各随机分为 5组。正常对照组及损伤组不给任何治疗性药物 ,大黄虫丸组给大黄虫丸 10 0 0 mg/ kg,复肝宝大、小剂量组分别给复肝宝 2 2 5 0和 375 m g/ kg,均每日 1次。保肝降酶实验小鼠口服给药 4日后 ,腹腔注射 2 .5 %四氯化碳 (CCl4)油溶液 0 .0 1ml/ g(除正常对照组 ) ,2 0小时后处死 ,测定血清谷丙转氨酶 (GPT) ,并观察肝病理组织学改变。抗肝纤维化实验大鼠 (除正常对照组 )皮下注射 40 % CCl41.2 5 m l/ kg,每周 2次 ,6周后给相应药物 5周 ,然后处死 ,测定 型前胶原肽 (P P)及肝组织中胶原蛋白含量 ,并观察肝病理组织学改变。结果 :复肝宝大、小剂量组与损伤组比较 ,GPT及肝胶原蛋白含量均差异显著 (P<0 .0 5或 P<0 .0 1) ,P P、肝细胞损伤程度及纤维化分级亦有显著性差异 (P均 <0 .0 5 )。结论 :复肝宝对CCl4诱发小鼠急性肝损伤有保护作用 ;对 CCl4诱发大鼠慢性肝纤维化有治疗作用 ,可使肝纤维化明显减轻。

Objective:To investigate the effect of Keboli 2 Liquor (OKL 2) on experimental liver fibrosis.Methods:Liver fibrosis in rats was induced by carbon tetrachloride injected subcutaneously for 8 weeks.OKL 2 was given simultaneously.The liver function,liver hydroxyproline and histopathology were determined after that.Results:The liver fibrosis was ameliorated,the liver hydroxyproline was reduced and the liver function was improved significantly.Histopathology showed the effect of anti liver fibrosis of OKL 2.Conclusion:The...

Objective:To investigate the effect of Keboli 2 Liquor (OKL 2) on experimental liver fibrosis.Methods:Liver fibrosis in rats was induced by carbon tetrachloride injected subcutaneously for 8 weeks.OKL 2 was given simultaneously.The liver function,liver hydroxyproline and histopathology were determined after that.Results:The liver fibrosis was ameliorated,the liver hydroxyproline was reduced and the liver function was improved significantly.Histopathology showed the effect of anti liver fibrosis of OKL 2.Conclusion:The protection of OKL 2 against liver fibrosis in rats induced by carbon tetrachloride was significant.

目的 :研究可博利 2号口服液 (OKL- 2 )抗肝纤维化的作用。方法 :四氯化碳 (CCl4 )连续 8周皮下注射复制大鼠慢性肝纤维化模型。造模开始时即同时给予 OKL- 2 ,实验结束后分别测定肝功能、肝纤维化指标 (肝羟脯氨酸含量 ) ,并作肝组织病理学检查以观察肝纤维化的程度。结果 :OKL- 2可显著减轻 CCl4 所致肝纤维化程度 ,能明显降低肝羟脯氨酸含量 ,改善肝功能 ,肝组织病理学检查亦显示具有抗肝纤维化作用。结论 :OKL - 2对 CCl4 诱导的大鼠慢性肝纤维化具有明显的保护作用

 
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