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心房电活动
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  atrial electric activity
     Researches have proved that the area between left atrium and pulmonary veins can provide the basis for reentry and that the electric activity of pulmonary veins during the maintainance of atrial fibrillation,which highly depends on left atrial electric activity,is indicative of reentry.
     研究证实,在肺静脉和左心房之间具有形成折返激动的必备基础,而且心房颤动持续过程中的肺静脉电活动具有提示折返激动的特征,并高度依赖于左心房电活动的存在。
短句来源
  atrial electrical activity
     FREQUENCY SPECTRUM ANALYSIS OF ATRIAL ELECTRICAL ACTIVITY IN RABBITS
     家兔心房电活动的频谱分析
短句来源
     THE CHANGE OF ATRIAL ELECTRICAL ACTIVITY SPECTRUM IN THE EXPERIMENT OF ATRIAL FIBRILLATION ON RABBITS
     家兔实验性心房纤颤时心房电活动的频谱变化
短句来源
  “心房电活动”译为未确定词的双语例句
     The measurements were made of refractory periods using programmed premature stimulation in the right atrium both before and after the administeration(Ach group: 10 mmol/L acylcholine 0.5 mL;carbachol group: 10 mmol/L carbachol 0.5 mL). Premature beats were delivered at 2×,4×,10×,20× threshold at coupling intervals starting at 150 msec down to refractoriness and the ERP was recorded at each level.
     分别在给药前后(乙酰胆碱组:10 mmol/L乙酰胆碱0.5 mL;卡巴胆碱组:10 mmol/L卡巴胆碱0.5 mL),于右心房处行程序期前刺激,来测定有效不应期及诱发房颤,刺激强度分别采用2倍、4倍、10倍及20倍起搏域值,如出现房颤,则记录房颤持续最长时间及房颤时心房电活动的平均周期。
短句来源
     Results Comparing with P wave duration of sinus rhythm,P wave duration is prolonged during RAA pacing and shortened during DCS,DSA,and BiA pacing. Conclusion Atrial activation duration are markedly shortened and the dispersion of atrial activation are reduced during DCS,DSA,and BiA pacing,which are in favor of prevention and treatment of paroxysmal atrial fibrillation.
     结果与窦性P波时限相比 ,RAA起搏明显延长P波时限 (P <0 0 1) ,DCS、DSA及BiA起搏则明显缩短P波时限 (P <0 0 1)结论DCS、DSA及BiA起搏明显缩短心房激动时间 ,减少心房电活动的离散度 ,有利于PAf的防治
短句来源
     ② PVP dissociation from atrial waves;
     ②肺静脉自律性电位与心房电活动无关 ;
短句来源
     Conclusion Atrial activation duration are markedly shortened and the dispersion of atrial activation are reduced during DCS, DSA, and BiA pacing, which is in favor of prevention and treatment of paroxysmal atrial fibrillation.
     结论  DCS、DSA及 Bi A起搏明显缩短心房激动时间 ,减少心房电活动的离散度 ,有利于 PAf的防治。
短句来源
     Before the Ach injection, all induced Afs were short paroxysmal Afs with the duration of 9.7 6sec, the average Af cycle length of 122 9ms;
     未注射乙酞胆碱时,所诱发房颤均为短阵性房颤,持续时间为9.7土6se。 ,心房电活动周期为122士gms,注人乙酞胆碱后持续时间则延长为596 .7士28 1 see(p<0.01);
短句来源
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  相似匹配句对
     FREQUENCY SPECTRUM ANALYSIS OF ATRIAL ELECTRICAL ACTIVITY IN RABBITS
     家兔心房活动的频谱分析
短句来源
     NONINVASIVE MAPPING OF RIGHT ATRIAL ACTIVITY
     右心房活动的无创性标测
短句来源
     Effects of pacing at different sites of atria on atrial activation
     不同部位心房起搏对心房活动的影响
短句来源
     Electrical Remodeling Effect on Pulmonary Veins
     重构对心房及肺静脉入口处活动的影响
短句来源
     Electrophysiologic characteristics of the atrium in rabbits with sinus node dysfunction
     窦房结功能障碍时心房活动的变化
短句来源
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  atrial electrical activity
The duration of atrial electrical activity was measured by selecting a minimum electrographic amplitude of 50μV.
      
We hypothesized that a free-floating single-pass defibrillation lead reliably senses the atrial electrical activity.
      


Reprodticing the model of experiment atrial fibrillation on rabbitswith acetylcholine(ACH ).The results showed that the atrial activity spectrumgramwere significantly unnormal during the atrial fibrillation. The power in lower frequencyincreased.1 -4Hz and 1-20Hz spectrum power were significantly higher than that innormal and the highst fregtiency were decreased.It suggested that the power in lowerfrequency of atrial activity spectrum increased during atrial fibrillation.

用乙酰胆硷(Ach)复制家兔实验性心房纤颤模型,采用Y导联引导心电信号经快速傅里叶转换对心房电活动进行频谱分析。结果显示:心房纤颤时心房电活动频谱明显异常,表现为低频段能量增加,1-4Hz和1-20Hz频段能量比正常状态明显增大,1-4H/5-10Hz比值和1-20Hz/>20Hz的比值都明显变大,而最高频率明显减小。提示心房纤颤发作时心房电频谱中低频成分能量增加。

Objective To evaluate the efficacy of dsotalol on experimental snstained atrial fibrillation (AF) and effects on its ratedependent action in atrium of dogs and to study its electrophysiologic mechanisms for terminating AF by using computerbased 112electrode epicardial mapping system. Methods Using sustained cholinergic AF model,clinically used dose (2 mgkg-1) of dsotalol and additional dose of dsotalol (cumulative load 8 mgkg-1) were given to 12 dogs to assess the ability to terminate AF.Atrial epicardial electrophysiologic...

Objective To evaluate the efficacy of dsotalol on experimental snstained atrial fibrillation (AF) and effects on its ratedependent action in atrium of dogs and to study its electrophysiologic mechanisms for terminating AF by using computerbased 112electrode epicardial mapping system. Methods Using sustained cholinergic AF model,clinically used dose (2 mgkg-1) of dsotalol and additional dose of dsotalol (cumulative load 8 mgkg-1) were given to 12 dogs to assess the ability to terminate AF.Atrial epicardial electrophysiologic activities were mapped and atrial effective refractory period (AERP) were measured at four different basic cycle lengths (BCL) at baseline and after drug administration.ZResults Dsotalol of 2 mgkg-1 terminated AF by 167% and 6 mgkg-1 terminated AF by 917%(P<0001).Time of termination were (20312) min and (8214) min and cencentration for termination were (6712) mg/L and (23416) mg/L respectively for dose 2 mgkg-1 and dose 6 mgkg-1.During no vagal stimulation,dsotalol of 6 mgkg-1 increased AERP by (437)% 3) ms to (2359) ms]and increased wavelength by (54202)% 518) cm to (28612) cm] at BCL 400 ms.But the dose incrlased AERP only by (151)% and increased wavelength only by (15874)% at BCL 200ms.During vagal stimulation,dsotalol of 6 mgkg-1 increased AERP by (17937)% and wavelength by (1861212)% 07) cm to (22104) cm]at BCL 400ms.But same dose increased AERP only by (11019)% and wavelength only by (1124%115)% at BCL 200 ms.Effects of dsotalol on AERP and wavelength showed strong reverse rate dependence.Dsotalol had no effects on conduction velocity of atrium. Conclusions Dsotalol could terminate vagotonic AF but larger dose were needed.Dsotalol terminated AF by increasing AERP and wavelength but with strong reverse rate dependence.Effects of reverse rate dependence on AERP and wavelength can limit drug efficacy against AF.

目的应用心外膜标测技术观察索他洛尔对持续迷走性心房颤动(房颤)的效应并研究其对犬心房电生理特性的频率依赖性影响和终止房颤的作用机制。方法建立迷走神经性持续房颤模型,12只犬分别给予索他洛尔临床使用量(2mg·kg-1)和大剂量(6mg·kg-1,累积量为8mg·kg-1)两个剂量,观察该药对房颤的终止情况,同时在用药前后进行心房电活动实时心外膜标测及心房有效不应期的测量。结果索他洛尔2mg·kg-1负荷量可转复房颤16.7%,而6mg·kg-1可转复91.7%(P<0.001)。转复时间分别为(20.3±1.2)min和(8.2±1.4)min。转复时血药浓度分别为(6.7±1.2)mg/L和(23.4±1.6)mg/L。当S1S1为400ms时索他洛尔6mg·kg-1可使无迷走神经刺激情况下的心房有效不应期从(165±3)ms增至(235±9)ms,增加率为(43±7)%,折返波长从(18.5±1.8)cm增至(28.6±1.2)cm,增加率为(54.2±0.2)%。而S1S1为200ms时,心房有效不应期从(135±5)ms增至(155±5)ms,增加率仅为(15±1)%,心房折返波长从(15...

目的应用心外膜标测技术观察索他洛尔对持续迷走性心房颤动(房颤)的效应并研究其对犬心房电生理特性的频率依赖性影响和终止房颤的作用机制。方法建立迷走神经性持续房颤模型,12只犬分别给予索他洛尔临床使用量(2mg·kg-1)和大剂量(6mg·kg-1,累积量为8mg·kg-1)两个剂量,观察该药对房颤的终止情况,同时在用药前后进行心房电活动实时心外膜标测及心房有效不应期的测量。结果索他洛尔2mg·kg-1负荷量可转复房颤16.7%,而6mg·kg-1可转复91.7%(P<0.001)。转复时间分别为(20.3±1.2)min和(8.2±1.4)min。转复时血药浓度分别为(6.7±1.2)mg/L和(23.4±1.6)mg/L。当S1S1为400ms时索他洛尔6mg·kg-1可使无迷走神经刺激情况下的心房有效不应期从(165±3)ms增至(235±9)ms,增加率为(43±7)%,折返波长从(18.5±1.8)cm增至(28.6±1.2)cm,增加率为(54.2±0.2)%。而S1S1为200ms时,心房有效不应期从(135±5)ms增至(155±5)ms,增加率仅为(15±1)%,心房折返波长从(15.2±0.?

Objective The atrial activations of acute atrial fibrillation(AF)using surface atrial epicardial mapping technique in canine model were studied. Method Twelve dogs were divided into two groups:control and AF induced by acetylcholine chloride The pericardial sac was opened following thoractomy,and the mapping electrodes were put on the epicardial surface of the atrium.Isochrones were displayed both during sinus rhythm and AF. Results During sinus rhythm,the atrial excitation wave front begins from sinus...

Objective The atrial activations of acute atrial fibrillation(AF)using surface atrial epicardial mapping technique in canine model were studied. Method Twelve dogs were divided into two groups:control and AF induced by acetylcholine chloride The pericardial sac was opened following thoractomy,and the mapping electrodes were put on the epicardial surface of the atrium.Isochrones were displayed both during sinus rhythm and AF. Results During sinus rhythm,the atrial excitation wave front begins from sinus node spreading to right atrium(RA)and left atrium(LA).During acute AF,there are multiple and discrete excitation wave fronts or reentry activities were found in both LA and RA,and bidirectional block regions could be shown. Conclusion Atrial activation of the acute AF evoked by acetylcholine chloride is complicated and no significant regularity is found.

目的 使用多位点心房心外膜标测技术探讨急性心房颤动 (房颤 )时心房电活动的规律。方法 将 12只实验犬分为两组 :正常对照组及用氯化乙酰胆碱建立的急性房颤模型组。在开胸状态下 ,使用自制的心外膜标测电极及信号采集系统分别对窦性心律及急性房颤心律进行心房心外膜标测 ,绘制等时图。 结果 窦性心律时窦房结发出冲动 ,激动右心房上部并向右心房下部和左心房传导 ,而房颤时双心房均可见方向不一、片段的激动波或大的折返环 ,可见多个传导阻滞区。 结论 用乙酰胆碱诱发的急性房颤的心房电活动纷乱复杂 ,未见明显的规律性。

 
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