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   结肠炎症 的翻译结果: 查询用时:0.021秒
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结肠炎症
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  colonic inflammation
     Conclusion SF can restrain the activity of activated colonic macrophages and relieve the colonic inflammation reaction in vivo in colitis rats, which may be related with the suppression of NF_κB expression.
     结论SF可减弱结肠炎大鼠结肠活化巨噬细胞的生物活性,缓解结肠炎症反应,机制可能与抑制NF_κB表达有关。
短句来源
     After 21 days of medication, the changes of UC activity index and body weight in mice were observed, and the condition of colonic inflammation and histomorphological changes in colonic tissue were observed also.
     给药治疗21天后,观察各组小鼠的疾病活动指数、体重变化,然后处死小鼠,取全段结肠,称结肠湿重,测肠重指数,比较各组小鼠的疾病活动指数、体重变化、结肠炎症反应及结肠组织病理学改变。
短句来源
     Mice in group 2 (control group) and 3 (experiment group) were treated with methylcellulose or pioglitazone(20 mg·kg -1 ·d -1 ) 3 days after enema and were administrated for consecutive 7 days. The colonic inflammation including disease activity index (DAI),macroscopic and histological changes,myeloperoxidase(MPO) activity and levels of interleukin (IL)-4,IL-5 were evaluated.
     第 2组 (对照组 )和第 3组 (实验组 )于灌肠后 3d分别给予二甲基纤维素和吡格列酮 (2 0mg·kg-1·d-1)连续 7d ,结肠炎症的评价包括炎症活动指数 (DAI)、大体形态损伤、组织学改变及肠黏膜髓过氧化物酶 (MPO)活性 ,采用ELISA法检测白细胞介素 (IL) 4和IL 5。
短句来源
     (Conclusions) The present study indicates that DG can ameliorate the colonic inflammation. Suppressing NF-κB activation and decreasing the expression of TNF-α and ICAM-1 in colon tissues may be the pos-(sible mechanism).
     结论DG可显著改善大鼠结肠炎症,其机制可能是通过影响炎症反应的信号通路NF-κB活化及ICAM-1、TNF-α的产生和表达来抑制炎症反应。
短句来源
     Pioglitazone can ameliorate the colonic inflammation by promoting the expression of PPAR-γ, and it may also inhibit the excessive apoptosis of colonic epithelial cells.
     其配体吡格列酮可促进PPAR-γ的表达,从而缓解结肠炎症,并可能具有抑制结肠上皮细胞过度凋亡的功能。
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  “结肠炎症”译为未确定词的双语例句
     Enteric microorganisms in cecum were fostered with standard methods. Results Inflammatory scores in PC group decreased compared with those in UC group(7.94±0.85 vs 10.25±1.36,P<0.05),but still higher than those in NC group(7.94±0.85 vs 4.35±0.88,P<0.01).
     结果PC组大鼠结肠炎症评分较UC组明显改善(7.94±0.85比10.25±1.36,P<0.05),但未恢复至NC组水平(7.94±0.85比4.35±0.88,P<0.01)。
短句来源
     Objective To investegate the role of B7 in DSS induced murine colitis.
     目的 研究B7分子在DSS诱导的小鼠结肠炎症中的作用。
短句来源
     C57BL/6 mice feed with 3% DSS (molecular weight is about 40 000) solution for five days may establish an ideal acute UC model.
     DSS模型结肠炎症程度的轻重与浓度无明显相关。 采用C57BL/6小鼠饮用3%DSS(分子量为40000左右)溶液5天的方法可建立理想的UC急性模型。
短句来源
     METHODS The rat colonitis was induced with the coloclysis of 2,4,6-trinitrobenzene sulfonic acid(TNBS),and then methods were established for quantifying TNF-α in rat serum and the ratio of rat colon weight/rat body weight(C/B),which were chosen as the index of colonitis degree. These indexes were used to evaluate the inhibiting action of various DSP dosage forms on TNF-α in rat serum with colonitis after their oral administration.
     方法:用2,4,6-三硝基苯磺酸钠(TNBS)诱导大鼠结肠炎,建立反应结肠炎症程度的血清TNF-α和大鼠结肠重量与大鼠重量比值(C/B)的测定方法,观察口服各种DSP制剂对TNF-α的抑制程度。
短句来源
     Conclusions: DSS-induced acute mucosal injury was dependent on DSS concentration.
     结论:DSS模型结肠炎症程度的轻重呈浓度依赖性。
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  相似匹配句对
     Its pathological characteristics are inflammationand ulcer of colon.
     结肠粘膜的炎症及溃疡是其主要病理特点。
短句来源
     Inflammation
     炎症(1)
短句来源
     Inflammation and Heart Failure
     炎症和心力衰竭
短句来源
     Domestic Sme also inhibited the colitis induced by senna.
     还能减轻由番泻叶引起的结肠炎症
短句来源
     Management of Patients with Colon Injury
     结肠损伤的处理
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  colonic inflammation
Therapeutic options for refractory colonic inflammation in patients with ulcerative colitis or Crohn's disease have recently been augmented by the introduction of biologic therapies.
      
Therapeutic options for refractory colonic inflammation in patients with ulcerative colitis or Crohn's disease have recently been expanded with the introduction of biologic therapies.
      
This finding indicates that TRPV1 receptors are required for the protection within sensory pathways that regulate the response following the initiation of colonic inflammation.
      
These unexpected results suggest that TRPV1 receptors mediate endogenous protection against experimentally induced colonic inflammation.
      
CB1 and TRPV1 receptors modulate enteric neurotransmission and colonic inflammation.
      
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Murid colon mucosal mast cell (MMC) reacted strongly to Schistosoma japonicum infection The increasing degree was 15 days post-infection (PI) and continuing until the end of the experiment (60days PI). A great deal of MMC weu present in colon of the infected mice than those in uninfected mice. MMC in 30 day PI increased gradually, the highest in 45 day PI, and decreased gradually in 60 day PI, It indicated that MMC was involved in colon local hyper-sensitivity reaction and inflammatory process.

用甲苯胺兰染色对小鼠感染日本血吸虫后结肠粘膜肥大细胞进行动态观察。结果表明:小鼠感染日本血吸虫后肥大细胞明显增多。当感染后第45天,发病高峰、炎症反应严重时,结肠粘膜肥大细胞亦达最高峰。感染后第60天又降至第15天的水平。推测肥大细胞参与结肠的炎症反应或机体的防御作用。

This study was used epidermal growth factor (EGF)and/or glutamine (GLN) to protect acetic acid-induced colitis in rats. The results suggest that combining EGF with GLN has remarkly cytoprotective effect on colonic mucosa due to increase supplies ofnutrition,mitochondria ATPase activities,energic metabolism in colon.

本文通过肌注表皮生长因子(EGF)和(或)饲饮谷氨酰胺(GLN),防治大鼠乙酸性结肠炎。结果提示联合应用EGF、GLN,能增强组织营养,提高ATP酶活性,促进肠粘膜细胞代谢和增生,防治结肠炎症,保护结肠粘膜细胞。

It was observed that the causes of lower digestive tract hemorrhage in the elderlys were,successively, colorectal carcinomas(77 cases), colorectal polyps(73 cases),colorectal inflammatory lesions(66 cases), piles and peri-anal lesions(55 cases), malformations of colorectal arteries and veins(14 cases),other colorectal diseases(45 cases). In 64 elderlys,there were no lesions found with endoscopy. Colonic carcinomas in 31 cases were found from transverse colons to cecums,which suggests that carcinomas of right...

It was observed that the causes of lower digestive tract hemorrhage in the elderlys were,successively, colorectal carcinomas(77 cases), colorectal polyps(73 cases),colorectal inflammatory lesions(66 cases), piles and peri-anal lesions(55 cases), malformations of colorectal arteries and veins(14 cases),other colorectal diseases(45 cases). In 64 elderlys,there were no lesions found with endoscopy. Colonic carcinomas in 31 cases were found from transverse colons to cecums,which suggests that carcinomas of right colons are not rare. Carcinomas in 19 cases resulted from malignant changes of adenomatous polyps. Colorectal polyps in 51 cases and multiple polyps in 23 cases were found in left colorectum,which suggest that more colorectal polyps or multiple polyps are in left colorectum. To prevent benign polyps from malignant changes,it is suggested that electronic coagulating polypectomy of colorectal polyps should be carried out in the elderlys. In the 64 cases without lesions found with endoscopy,15 cases were found later caused by lesions in jejunum or ileum.

引起老年人下消化道出血的常见病因依次为大肠癌(77例),大肠息肉(73例),结肠炎症(66例),痔核等肛周病变(55例),结肠动静脉畸形(14例),溃疡性结肠炎等病变(45例)。未发现病变者(64例)。31例大肠癌发生在横结肠至盲肠,提示老年人右半结肠癌并非少见。19例大肠癌系由腺瘤性息肉恶变所致。51例大肠息肉及23例多发性息肉发生在左半结肠,提示老年人息肉或多发性息肉仍以左半结肠多见。为防止息肉恶变,对老年人大肠息肉应尽早行电凝切除术。结肠镜未能发现病因的64例中,15例出血系空、回肠病变所致。

 
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