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心房肌     
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  atrial muscle
     Immunohistochemical method displayed that endothelins-1 staining gray values of atrial muscle and ventricular muscle in the ischemic group(167.63±4.28,154.38±4.05) were evidently lower than those in the shenqi decoction group(182.75±7.74,182.97±2.14) (P< 0.01,P< 0.05).
     免疫组化显示缺血组心肌内皮素-1染色灰度值明显低于参七汤组(心房肌:167.63±4.28,182.75±7.74,P<0.01;心室肌:154.38±4.05,182.97±2.14,P<0.05)。
短句来源
     In isolated rabbit atrial muscle the functional refractory period was found to be prolonged by DMT 3.0x 10~(-6)M.
     DMT3.0×10~(-6)使家兔离体心房肌功能不应期延长。
短句来源
     Results At the stimulation cycle length of 400 ms, MAPD_(90) of right atrial muscle prolonged gradually from young group to old group (P<0.01).
     结果在400ms刺激周长下,4组大鼠右心房肌MAPD90随年龄增长而逐渐延长〔(75±5)(、123±8)(、140±11)和(140±14)ms,均为P<0.01〕;
短句来源
     ERP of atrial muscle showed the same change trends as MAPD_(90).
     心房肌ERP的增龄性变化规律与MAPD90相同。
短句来源
     Effects of stimulation frequency(6—24/min),calciumconcentration(1.8—9 mmol/L)and temperature(2.5—18℃)on the force-velocitycurve were also observed in 12 atrial muscle preparations. The results showedthat all the curves could not fit Hill's equation,although some changes did occur.
     此外,在12例牛蛙心房肌标本中,观察了刺激频率(6—24次/min)钙离子浓度(1.8—9mmol/L)和温度(2.5—18℃)对张力速度曲线的影响,结果表明,虽然曲线发生变化,但仍不符合 Hill 公式。
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  atrial myocardium
     The amplitude of the action potential ( APA) of the atrial myocardium was decreased from 88. 00 ±9. 35 to 62. 62±20. 50 mV (n = 6, P < 0. 01) , while the APA of the ventricular myocardium did not change significantly.
     心房肌动作电位的幅度(action potential amplitude,APA)也降低,给药前后分别为88.00±9.35 mV及62.62±20.50 mV(n=6,P<0.01),而心室肌APA无明显变化。
短句来源
     The Study on the Gene Expression of AT_1- R,AT_2- R in Human Atrial Myocardium with and without Atrial Fibrillation
     心房颤动患者心房肌组织AT_1-R、AT_2-R mRNA的表达及研究
短句来源
     Study on Structural Remodeling and Ca~(2+)-CaMKⅡ-CREB Signaling Pathway in Atrial Myocardium of Pathway in Atrial Myocardium of Patients with Chronic Atrial Fibrillation
     慢性房颤病人心房肌组织重构及Ca~(2+)-CaMKⅡ-CREB信号通路的研究
短句来源
     Results (1)The apoptosis was found in 8 rabbits(8/12 rabbits) in the center and peripheral region of SAN as well as peripheral atrial myocardium were found, and their apoptosis rates were 11.3%±3.7%,15.0%±4.5% and 24.4%±5.9% respectively.
     结果 (1) IR组有8只(8/ 12 )窦房结及其周围心房肌均有细胞凋亡现象,其凋亡率(% )分别为11.3±3.7、15 .0±4 .5和2 4 .4±5 .9;
短句来源
     The expression of AT1 - R, AT2 - R in atrial myocardium of cAF group was greatly downregulated and upregulated respectively (P < 0.01 - 0.05).
     心房肌组织中AT1-RmRNA表达明显下调(P<0.01-0.05),AT2-R mRNA表达明显上调(P<0.05)。
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  atrium muscle
     The automaticity,contractility,excitability and refractoriness of the isolated atrium muscle of rats were recorded.
     用离体大鼠心房肌进行自律性、收缩性、兴奋性和不应性的研究。
短句来源
  atrial tissue
     The mRNA expression of AT_1-R,AT_2-R and CYP11B2 in human left and right atrial tissue with atrial fibrillation
     心房颤动患者左、右心房肌组织AT_1-R、AT_2-R、CYP11B2的mRNA表达
短句来源
     2. Aschoff 's body scattered in atrial tissue according to the examination of light microscope: the positive percent in control was 37.5%(3/8); experimental Ⅰgroup were 46%(5/11); experimental Ⅱgroup were 50% (6/12).
     2.光镜观察发现,多数患者心房肌组织内有散在的Aschoff小体,每组检测阳性率分别为:对照组37.5%(3/8)、实验Ⅰ组46%(5/11)、实验Ⅱ组50%(6/12)。
短句来源
     Study of atrial tissue angiotensin converting enzyme and angiotensin Ⅱ receptors mRNA expression in chronic atrial fibrillation
     慢性心房颤动患者心房肌血管紧张素转换酶和血管紧张素受体mRNA表达改变的研究
短句来源
     A Study of Chloride Channels Gene Expression in Human Atrial Tissue and Its Relationship with Atrial Fibrillation
     氯电流相关通道在人心房肌表达及与心房颤动关系的研究
短句来源
     Objective To evaluate the alteration and effect of atrial tissue angiotensin converting enzyme (ACE) and angiotensin Ⅱ receptors (AT_1, AT_2) mRNA expression on atrial structural remodeling in chronic atrial fibrillation.
     目的研究慢性心房颤动(房颤)患者心房肌中血管紧张素转换酶(ACE)和血管紧张素受体AT1R、AT2RmRNA表达改变及其对房颤心房结构重构的影响。
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  atrial muscle
The rationale behind the principle of compartimentation of the atria is the reduction of the critical atrial muscle mass neccessary to facilitate fibrillation of the atria.
      
The influence of endothelin 1 on isometrically contracting human atrial muscle strip preparations was investigated under physiological conditions (37°C, 1 Hz, Ca2+ 2.5 mM).
      
Postconditioning protects human atrial muscle through the activation of the RISK pathway
      
Unlike the adrenoceptor-mediated effect, the non-sympathomimetic inotropic effect is more pronounced in ventricular than in atrial muscle, and is not stereospecific.
      
Effect of 2-nicotinamidethyl nitrate (SG-75) on the membrane potential of left atrial muscle fibres of the dog
      
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  atrial myocardium
Amiodarone-Induced Changes in Intracellular Ca2+ Homeostasis in the Atrial Myocardium of Patients with Chronic Coronary Insuffic
      
It was found that, in 30% of the IHD patients, the atrial myocardium displayed an abnormal contractility.
      
This phenomenon may contribute to the suppression of abnormal activity of the atrial myocardium and strengthen the direct antiarrhythmic effect of Amiodarone.
      
Histochemical analysis have shown that the atrial myocardium in IHD patients is characterized by an increased density of the microvascular network, increased phosphorylase activity, and decreased succinate dehydrogenase activity.
      
Influence of vanadate on electrophysiological and contractile parameters of atrial myocardium
      
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  heart atrium
CgA has been shown to be stored in cardiomyocyte secretory granules of the rat heart atrium together with atrial natriuretic peptide (ANP).
      
The uptake of bretylium-(N-3H-methyl) iodide in the rat heart atrium in vitro was examined.
      
The apparent Kmvalue of the active bretylium uptake was 3×10-6 M, which was 10 times higher than that of the uptake of (-)-noradrenaline in the rat heart atrium (Km=3×10-7 M).
      
Neuropeptide Y inhibits acetylcholine release in human heart atrium by activation of Y2-receptors
      
We investigated whether the sympathetic cotransmitters neuropeptide Y (NPY), norepinephrine (NE), and ATP are capable of modulating acetylcholine release in human heart atrium.
      
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  atrium muscle
Electrogenic Na+ pump currents during K+-activated hyperpolarizations of bullfrog atrium muscle fibres are increased by adrenaline.
      
The ionic mechanism of the membrane outward current (ACh-current) of bullfrog atrium muscle, induced by acetylcholine in K-free solution, was analyzed by a voltage-clamp experiment.
      
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