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小鼠妊娠
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  pregnant mice
     Methods In LPS groups,pregnant mice were injected intraperitoneally with different doses of LPS(0.1~0.5 mg/kg) on gestational day(gd) 17;
     方法小鼠妊娠第17天分别注射不同剂量LPS(0.1~0.5mg/kg,ip);
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     Methods Groups of pregnant mice, 10 mice in each group, were orally administrated with TCDD in a dose of 0, 2, 50 or 100 ng·(kg·d) -1 during 1~8 d, 1~3 d, and 4~8 d of pregnancy.
     方法在NIH小鼠妊娠早期的1~8d,胚泡着床前期的1~3d和着床后期的4~8d,经口腔灌服0、2、50和100ng(kg·d)剂量的TCDD,在小鼠妊娠第9天和第18天时观察子宫内的胚胎发育形态。
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     The effects of 0 025—0 40? μg/(kg·d) dibutyltin chloride (DBTCl) on pregnant mice and fetuses were studied under 22±2℃,light:dark=12?
     雌性小鼠妊娠第 6天开始每天 1次腹腔注射 0 0 2 5— 0 40 μg/(kg·d)氯化二丁基锡 (DBTCl) ,共染毒 7d .
短句来源
     The Effect of Pu-Huang Water-Decoction on Pregnant Mice
     蒲黄水煎液对小鼠妊娠影响的实验研究
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     Meanwhile,blood was kept from pregnant mice for the determination of serum VA. In situ hybridization was used to determine Hox-3\^5 mRNA expression in the mouse embryos by digxigenin-labeled cDNA probes.
     结果 在正常情况下 ,小鼠妊娠第 12d胚胎组织Hox -3 5mRNA有表达 ,孕鼠血清VA缺乏时 ,妊娠第 12d胚胎组织Hox - 3 5mRNA的含量明显减少 ;
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  pregnant mouse
     Immunohistochemically studies showed that the quantity of CD + 57 cells in early pregnant mouse uterus increased from 16.3 at day 0 to 62.3 at day 2,decreased to 33.0 at day 4 and 9.7 at day 6 after pregnancy. It was about 4 times higher at day 6 and day 8 in abortive uterus than that at day 0 after pregnancy.
     免疫组化研究显示 ,小鼠妊娠早期子宫壁内 CD+ 57细胞数量由孕 O天时 16 .3增至孕 2天时 6 2 .3,孕 4天时已下降 ,孕 6天时降至 9.7,而发生胚胎丢失的个体孕 6天和孕 8天时细胞数量均维持较多 ,约是孕0天时的 4倍。
短句来源
     The results suggest that the quantity of CD + 57 cells, CD + 4 cells and CD + 4 cells in the early pregnant mouse uteri fluctuate respectively and early abortions have to do with the high quantity of CD + 57 cells, CD + 4 cells in uteri at day 6 and day 8 after pregnancy.
     研究结果提示小鼠妊娠早期子宫壁内 CD+ 57细胞、CD+ 4细胞和 CD+ 8细胞数量在胚胎着床前均出现先增后减的波动 ,早期胚胎丢失与孕 6天及孕 8天时子宫壁内所含大量 CD+ 57细胞、CD+ 4细胞和 CD+ 8细胞之间存在相关。
短句来源
     Methods Female murine recipients were ovariectomized,and E_2 or P_4 were orally administered until the E_2 or P4 level in peripheral blood reach that of pregnant mouse during the second trimester.
     方法雌性受体小鼠通过切除双侧卵巢去势,胃饲E2或P4,使外周血E2或P4浓度达到相当于小鼠妊娠中期水平。
短句来源
     Conclusion: Higher expression of Ang-2 in the endometrium of pregnant mouse indicates that Ang-2 may play a very important role in the cross-talk between blastocyst and maternal endometrium during the process of embryo implantation.
     结论 在小鼠妊娠过程中Ang- 2和Ang 2 mRNA在着床前小鼠子宫内膜中即开始表达,并随妊娠进程而表达增强,提示Ang 2基因在“胎 母”对话的信号传导过程中起调节作用。
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  “小鼠妊娠”译为未确定词的双语例句
     Study of uterine natural killer cells in C57Bl/6J mice at early-mid gestation stage
     C57Bl/6J小鼠妊娠早-中期子宫自然杀伤细胞研究
短句来源
     Results The pregnancy rate and number of imp lanted site was 27.3% and 5.3±0.7 respectively in the model group, significa ntl y lower than those in the normal group (90.9%,13.3±2.8), and the BAR group (72. 7%,10.7±2.2,P<0.05).
     结果(1)小鼠妊娠率和胚泡着床点数:模型组(27·3%、5·3±0·7)显著低于正常组(90·9%、13·3±2·8,P<0·01),中药组(72·7%、10·7±2·2)较模型组显著增高(P<0·05)。
短句来源
     Methods : Level of nm23 - M2 expression of the endometria from day 2 , day 5 and day 7 pregnancies were carried out by RT - PCR and immunohistochemistry.
     方法采用RT-PCR及免疫组织化学方法分别检测小鼠妊娠第2天(植入前)、第5天(植入中)、第7天(植入后)子宫内膜nm23-M2表达。
短句来源
     Results The pregnancy rate and number of implanted embryo were significantly lower in indomethacin group(27.3% and 5.3±0.7) than in normal group(90.9% and 13.3±2.8)(both P<0.01),but was higher in CHM group(72.7% and 10.7±2.2) than in indomethacin group(both P<0.05).
     结果模型组小鼠妊娠率和胚胎着床位点数(27.3%、5.3±0.7)显著低于正常组(90.9%、13.3±2.8)(均P<0.01); 中药组小鼠妊娠率和胚胎着床位点数(72.7%、10.7±2.2)较模型组显著增高(均P<0.05)。
短句来源
     mM2,as a microinjection and transfer operation mediunymproved pregnant rate(62.5%) than M2(35.9%)(p<0.05).
     用mM2培养液作为显微注射及移植工作液,小鼠妊娠率为62.5%,比用M2培养液(35.9%)显著提高(P<0.05)。
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  pregnant mice
Pregnant mice were each infected with 300 larvae 5, 7, 15 and 17 days after fertilization.
      
Excessive dexamethasone (Dex) administrated into pregnant mice during critical periods of palatal development can produce a high incidence of cleft palate.
      
This study examined the expression and distribution of angiopoietin-1/-2 (Ang-1/-2) in the endometrium of early pregnant mice.
      
With gestation time, the positive reactions of Ang-1/-2 were stronger in the endometria of the pregnant mice (P>amp;lt;0.01).
      
Exposure of pregnant mice to RE leads to rapid placental transfer of RE; 14.1% of the total amount of RE administered was detected in newborn mice.
      
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  pregnant mouse
In contrast, in the adult animal expression of the Q5k gene was limited to the thymus and uterus of the pregnant mouse.
      
The results indicated that ultrasonic exposure up to at least 100 s is not hazardous to a pregnant mouse or its offspring in utero under the conditions of our experiment.
      
Protein extracts from pregnant mouse endometria were compared with those obtained from non-pregnant and pseudopregnant mice to detect early pregnancy-specific galactose-rich glycoproteins.
      
These isoforms were also detected in the Harderian gland from 13-day pregnant mouse; however, striking quantitative changes were seen concerning the α- and ε-isoforms.
      
The 80-kDa native form of PKC-α almost doubled in the pregnant mouse in comparison with normal female mouse whereas the amount of 50-kDa catalytic domain did not change.
      
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The effects of high dose of luteinizing hormone-releasing hormone(LH-RH)analogue on early pregnancy of rats and mice were observed.On Days 5—7 ofpregnancy of rats 200 μg of LH-RH analogue were given subcutaneously daily,while the same amount of saline was given to the control animals.The animalswere sacrificed on Day 14 of pregnancy.At autopsy the number of pregnantanimals and the sites of their implantation were counted and examined.It was found that the LH-RH analogue in high dose could effectively terminatethe...

The effects of high dose of luteinizing hormone-releasing hormone(LH-RH)analogue on early pregnancy of rats and mice were observed.On Days 5—7 ofpregnancy of rats 200 μg of LH-RH analogue were given subcutaneously daily,while the same amount of saline was given to the control animals.The animalswere sacrificed on Day 14 of pregnancy.At autopsy the number of pregnantanimals and the sites of their implantation were counted and examined.It was found that the LH-RH analogue in high dose could effectively terminatethe pregnancy in rats with a rate of 72.7% as compared with zero of the controls.If progesterone(5 mg)was injected s.c.simultaneously with the LH-RH,the an-tifertility effect of the high dose of LH-RH analogue could no longer be observed.If HCG(50 IU)was injected instead of progesterone,the contraceptive effect ofthe LH-RH analogue remained.Accordingly,it seems that the antifertility effectof LH-RH analogue in high dose might be due to the blocking of the effect ofendogenous progesterone in pregnancy.However,the high dose of LH-RHanalogue showed no effect whatsoever on the pregnancy of mice when injectedon Days 4—6 or Days 7—12 of pregnancy.

本工作观察了大剂量的 LH-RH%类似物对大鼠和小鼠早期妊娠的影响。于大鼠妊娠的第5—7天,每鼠每天皮下注射200微克的 LH-RH 类似物,对照组注射等量生理盐水。于妊娠第14天处死,检查并记录妊娠动物数及正常着床胎数。结果表明,大剂量的LH-RH 类似物在大鼠具有明显的抗生育作用,终止妊娠率达72.7%。若同时皮下注射5毫克孕酮可以完全反转 LH-RH 类似物的抗生育作用;而同时注射50国际单位的HCG 并不能对抗 LH-RH 类似物的终止妊娠作用。这提示,大剂量 LH-RH 类似物的抗生育作用极可能是由于阻断了孕酮作用的结果。但在小鼠妊娠的第4—6天或第7—12天,每鼠每天注射200微克的 LH-RH 类似物,则对妊娠并无影响。

This paper reports the hormonal activity and the contraceptive action of 18-methylmestranol.In immature female mice, 18-methyl-mestranol was estrogenic as shown by the marked stimulation of uterine development. When compared with mestranol, its estrogenic potency was approximately 1/50. Given simultaneously with estradiol, 18-methylmestranol did not affect the uterine development induced by the estrogen, indicating that 18-methyl-mestranol had no anti-estrogenic action.In Clauberg test 18-methyl-mestranol did...

This paper reports the hormonal activity and the contraceptive action of 18-methylmestranol.In immature female mice, 18-methyl-mestranol was estrogenic as shown by the marked stimulation of uterine development. When compared with mestranol, its estrogenic potency was approximately 1/50. Given simultaneously with estradiol, 18-methylmestranol did not affect the uterine development induced by the estrogen, indicating that 18-methyl-mestranol had no anti-estrogenic action.In Clauberg test 18-methyl-mestranol did not stimulate the development of the rabbit endometrium but markedly inhibited the endometrium stimulating activity of norgestrel. Deciduomas in rats was provoked by injection of estrogen and norgestrel and by passing a thread through one uterine horn. Simultaneous administration of 18-methyl-mestranol with the norgestrel inhibited the development of the deciduoma. These results show that 18-methyl-mestranol possesses no progestational activity but is antiprogestogenic.Oral administration of 18-methyl-mestranol on the 1st, 2nd and 3rd day of gestation at the dosage of 1 mg/kg/day, completely prevented pregnancy in mice. When administered on the 6~8th day of gestation, 80~90% of the mice had their pregnancy interrupted. These experiments indicate that 18-methyl-mestranol is able to prevent implantation and interrupts early pregnancy.18-Methyl-mestranol was administered to mice on the 1st and 2nd day of gestation. On the 4th day of gestation, fertile eggs were seen in the oviduct in 4 out of 10 animals, while no egg was seen in the control group. Thus egg transport in the oviduct was slowed in the treated group, This slowing of egg transport might play a role inthe prevention of implantation.18-Methyl-mestranol was administered orally to mice at a daily dosage of 2 mg/kg for 14 days. No noticeable changes of body weight and morphology of the main viscera were observed. Dogs were given daily oral doses of 1 mg/kg for 14 days. At the end of this period, the blood picture, heart, SGPT and blood urea nitrogen were normal.

本文报告了18-甲基乙炔雌二醇-3-甲醚的激素活性和避孕作用。 18-甲炔雌甲醚在幼年雌性小鼠能明显刺激子宫发育,说明有雌激素活性。经与炔雌醇-3-甲醚作效价比较,结果指出其雌激素活性约为炔雌醇-3-甲醚的1/50。当与雌二醇同时应用时,18-甲炔雌甲醚并不能对抗雌二醇引起的子宫发育,说明没有抗雌激素活性。 用Claubcrg方法以及用大鼠蜕膜瘤方法进行的实验说明18-甲炔雌甲醚没有孕激素活性,但有明显的抗孕激素活性。 在小鼠妊娠第1、2、3天经口给18-甲炔雌甲醚,每日1 mg/kg小鼠不能怀孕。在妊娠第6、7、8天给18-甲炔雌甲醚,小鼠只有10~20%怀孕。说明有明显的抗着床和抗早孕作用。 在小鼠妊娠第1、2两天经口给18-甲炔雌甲醚,有4/10的动物在妊娠第四天输卵管中仍可见到受精卵,而对照组在妊娠第四天输卵管中都没有卵子,可见18-甲炔雌甲醚使受精卵在输卵管中的运行变慢,这一作用也可能是18-甲炔雌甲醚抗着床作用的一个原理。 经口给小鼠18-甲炔雌甲醚2 mg/kg,连给2周,对动物体重增长和各主要脏器形态都没有明显影响。经口给狗1 mg/kg,连给两周,狗的血象、心脏和肝、...

本文报告了18-甲基乙炔雌二醇-3-甲醚的激素活性和避孕作用。 18-甲炔雌甲醚在幼年雌性小鼠能明显刺激子宫发育,说明有雌激素活性。经与炔雌醇-3-甲醚作效价比较,结果指出其雌激素活性约为炔雌醇-3-甲醚的1/50。当与雌二醇同时应用时,18-甲炔雌甲醚并不能对抗雌二醇引起的子宫发育,说明没有抗雌激素活性。 用Claubcrg方法以及用大鼠蜕膜瘤方法进行的实验说明18-甲炔雌甲醚没有孕激素活性,但有明显的抗孕激素活性。 在小鼠妊娠第1、2、3天经口给18-甲炔雌甲醚,每日1 mg/kg小鼠不能怀孕。在妊娠第6、7、8天给18-甲炔雌甲醚,小鼠只有10~20%怀孕。说明有明显的抗着床和抗早孕作用。 在小鼠妊娠第1、2两天经口给18-甲炔雌甲醚,有4/10的动物在妊娠第四天输卵管中仍可见到受精卵,而对照组在妊娠第四天输卵管中都没有卵子,可见18-甲炔雌甲醚使受精卵在输卵管中的运行变慢,这一作用也可能是18-甲炔雌甲醚抗着床作用的一个原理。 经口给小鼠18-甲炔雌甲醚2 mg/kg,连给2周,对动物体重增长和各主要脏器形态都没有明显影响。经口给狗1 mg/kg,连给两周,狗的血象、心脏和肝、肾功能没有出现异常。

Mice were each given 200μg of LH-RH analogue subcutaneously from the 4th to the6th day of pregnancy daily,an equal amount of saline being used on the controls.On Day 4,Day 7,Day 10 or Day 14 of pregnancy,a batch of animals were sacrificed respectively.Itwas found that the high dose of LH-RH analogue showed no effect whatsoever on the preg-nancy,the level of serum progesterone and the ovary steroid-3β-ol-dehydrogenase acti-vity in mice,contrary to what was observed in rats.Following the intravenous administration...

Mice were each given 200μg of LH-RH analogue subcutaneously from the 4th to the6th day of pregnancy daily,an equal amount of saline being used on the controls.On Day 4,Day 7,Day 10 or Day 14 of pregnancy,a batch of animals were sacrificed respectively.Itwas found that the high dose of LH-RH analogue showed no effect whatsoever on the preg-nancy,the level of serum progesterone and the ovary steroid-3β-ol-dehydrogenase acti-vity in mice,contrary to what was observed in rats.Following the intravenous administration of ~(125)I-LH-RH,the radioactivity of variousorgans was determined in mice.The control animals were similarly given Na ~(125)I of thethe same radioativity.It was found that the radioactivity of ~(125)I-LH-RH was not onlyhighly concentrated in the hypophysis,but also markedly concentrated in other organssuch as the liver,the kidney,the ovary and so on.From Day 5 to Day 7 of pregnancy of rats,150 I.U.of HCG was given each subcu-taneously daily.It was found that the high dose of HCG could effectively terminate thepregnancy in 76.9 % of rats.However,the high dose of HCG showed no significant effecton the pregnancy of mice when similarly injected.Accordingly,it was inferred that theantifertility effect of the LH-RH analogue in rats might be mainly due to decrease of gona-dal responsiveness resulting from an abnormal pattern of gonadotropin secretion secondaryto the administration of the high dose of LH-RH analogue.

小鼠妊娠的第4—6天,每鼠每天皮下注射200微克LH-RH 类似物,对照组注射等量生理盐水,分别于妊娠第4天、7天、10天或第14天处死一组动物。结果表明,与大鼠不同,大剂量的LH-RH 类似物对小鼠妊娠、血清孕酮及卵巢⊿~5-3β-羟甾脱氢酶活性均无明显影响。注入大剂量~(125)I-LH-RH 在小鼠体内分布的结果表明,给小鼠颈静脉注入~(125)I-LH-RH 后,放射活性不仅高度集中于垂体,也明显地集中于肝、肾、卵巢等垂体外组织。据此我们推测,或是大剂量的LH-RH 类似物对小鼠卵巢无明显的直接抑制作用;或是小鼠卵巢对大剂量LH-RH 类似物的抑制作用耐受性较强;或可能在LH-RH 及其类似物的抗生育机理中(至少在其抗着床机理中)对性腺的直接抑制作用似乎不占主要地位。于大鼠妊娠第5—7天,皮下注射150国际单位HCG 确具有明显的抗生育作用,终止妊娠率达76.9%;相反,于小鼠妊娠第4—6天,皮下注射100国际单位HCG 对妊娠无明显影响。据此推测,LH-RH 类似物在小鼠之所以无抗生育作用,可能是由于小鼠卵巢对LH-RH 类似物引致的LH 峰形升高的失敏感作用耐...

小鼠妊娠的第4—6天,每鼠每天皮下注射200微克LH-RH 类似物,对照组注射等量生理盐水,分别于妊娠第4天、7天、10天或第14天处死一组动物。结果表明,与大鼠不同,大剂量的LH-RH 类似物对小鼠妊娠、血清孕酮及卵巢⊿~5-3β-羟甾脱氢酶活性均无明显影响。注入大剂量~(125)I-LH-RH 在小鼠体内分布的结果表明,给小鼠颈静脉注入~(125)I-LH-RH 后,放射活性不仅高度集中于垂体,也明显地集中于肝、肾、卵巢等垂体外组织。据此我们推测,或是大剂量的LH-RH 类似物对小鼠卵巢无明显的直接抑制作用;或是小鼠卵巢对大剂量LH-RH 类似物的抑制作用耐受性较强;或可能在LH-RH 及其类似物的抗生育机理中(至少在其抗着床机理中)对性腺的直接抑制作用似乎不占主要地位。于大鼠妊娠第5—7天,皮下注射150国际单位HCG 确具有明显的抗生育作用,终止妊娠率达76.9%;相反,于小鼠妊娠第4—6天,皮下注射100国际单位HCG 对妊娠无明显影响。据此推测,LH-RH 类似物在小鼠之所以无抗生育作用,可能是由于小鼠卵巢对LH-RH 类似物引致的LH 峰形升高的失敏感作用耐受性较大。因而推想,在大鼠,LH-RH 类似物抗生育作用的机理(至少在其抗着床机理中)LH 异常升高引致卵巢反应性下降可能起着较为重要的作用。

 
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