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肾慢性
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  chronic renal
     Results The rate of chronic renal allograft rejection in the recipients with TGF-β1 high producer genotype [42.9% (39/91)] was significantly higher than that in those with TGF-β1 intermediate or low producer genotypes [17.0% (9/53)] (P<0.01).
     结果TGF-β1高分泌基因型受者移植肾慢性排斥反应发生率为42.9%(39/91),中低分泌型受者为17.0%(9/53),组间差异有统计学意义(P<0.01)。
短句来源
     While in the recipients with TGF-β1 intermediate or low producer genotypes, whose donors had TGF-β1 intermediate or low producer genotypes also, the rate of chronic renal allograft rejection (12/61, 19.7%)was significantly lower than that in other recipients (23/53, 43.4%, P<0.01).
     供、受者均为中低分泌基因型的受者移植肾慢性排斥反应发生率(12/61,19.7%)明显低于所有其他基因型组合者(23/53,43.4%)(P<0.01)。 结论受者TGF-β1基因型及其与供者TGF-β1基因型的组合类型与移植肾慢性排斥反应有显著关联。
短句来源
     Objective To study the relationship between transforming growth factor-β1 (TGF-β1) genotypes of the recipieats and donors and the chronic renal allograft rejection.
     目的探讨供、受者转化生长因子β1(TGF-β1)基因型与移植肾慢性排斥反应的关系。
短句来源
     In the recipients with TGF-β1 high producer genotype, whose donors had TGF-β1 high producer genotype also, the rate of chronic renal allograft rejection (4/4) was significantly higher than that in other recipients (31/110, 28.2%).
     供、受者均为高分泌基因型的受者移植肾慢性排斥反应发生率(4/4)明显高于所有其他基因型组合者(31/110,28.2%)(P<0.01);
短句来源
     The Significance of Color Doppler Echocardiography in the Diagnosis of Chronic Renal Transplantation Rejection
     彩色多普勒对移植肾慢性排斥反应的诊断价值
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  “肾慢性”译为未确定词的双语例句
     No significant difference was detected between the donors with TGF-β1 high producer genotype [32.5% (26/80)] and those with TGF-β1 intermediate or low producer genotypes [32.4% (11/34)] (P>0.05).
     取自TGF-β1高分泌基因型和中低分泌基因型型供者的移植肾慢性排斥反应发生率分别为32.5%(26/80)和32.4%(11/34),组间比较差异无统计学意义(P>0.05)。
短句来源
     Objective: To investigate the distribution, expression and variation of MMP-2、9 and TIMP-1 in remnant renal tissues of 5/6 Nephrectomy young rats,and to evaluate the interfering effects of angiotensin converting enzyme inhibitor (ACEI) combined with angiotensin receptor agonist (ARB).
     目的:研究基质金属蛋白酶-2、9(MMP-2、9)/金属蛋白酶组织抑制物-1(TIMP-1)在5/6肾切除残肾慢性肾衰幼年大鼠的分布、表达及变化,观察联合应用血管紧张素转换酶抑制剂(ACEI)和血管紧张素II受体拮抗剂(ARB)对病变干预的影响,为儿童慢性肾功能衰竭的发病机制及临床治疗寻求新的线索和依据。
短句来源
     C4d: an effective indicator for monitoring chronic rejection of renal allograft in rat
     用补体裂解片断C4d监测大鼠移植肾慢性排斥反应
短句来源
     ③there is a close relationship between CMV infection and chronic rejection.
     ③CMV感染与移植肾慢性排异有较为密切的关系,可能是造成慢性排异反应发生的因素之一。
短句来源
     Hyperlipidemia can strengthen the increased expression of RANTES and CCR5 mRNA.
     结论:RANTES及其受体CCR5 mRNA表达可能在移植肾慢性损伤早期起作用;
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  相似匹配句对
     CT Diagnosis of Chronic Renal Infection
     慢性感染的CT诊断
短句来源
     CHANGES OF RENAL HEMODYNAMICS INE PATIENTS WITH CHRONIC COR PULMONALE
     慢性肺心病血流动力学改变
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  chronic renal
Renal transplantation (RT) is one of the renal replacement therapy options in the terminal stage of chronic renal failure.
      
New alternatives for erythropoietin therapy in chronic renal failure
      
In this review, we present some general aspects of EPO biology, with emphasis on chronic renal failure, and expose some of the alternatives to EPO used for anemia correction.
      
The objectives, however, differ from those for patients with chronic renal failure because diets that satisfy minimal requirements will not be sufficient for an acute catabolic illness.
      
In both groups progression of chronic renal failure slowed down, but the delay was more pronounced in group B.
      
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In this paper, we have observed the toxic effect to hePatocytes and renal cells of rat and human embryo by titanium in vitro.The results have showed that the toxic effect was Present by titanium in lower concentration. The morphalogical changs were observed in cells exposed to titanium, such as cytoplasm edema, disappear Cell border, nuclear edema, the granules increased in cytoplasm and cell senescence. This experiment indicated that the chronic cumulation of titanium to liver and kidney do not neglected.

用体外培养的大鼠肝细胞,人胎肝、肾细胞检测钛的毒性,测定其毒性终点和中毒后肝、肾细胞的形态改变。结果:高稀释度的钛对肝肾培养细胞均有毒性作用。中毒后的细胞出现水肿、边界模糊和消失;细胞核水肿、不规则,胞浆颗粒增多呈衰老现象,提示钛进入人体引起的肝肾慢性蓄积毒作用不容忽视。

Non-skeletal phase damage of endemic fluorosis has been noticed at present. The soft tissue and enzyme metabolism are effected by fluoride and these effects appeared before skeletal change. In this paper, in order to observe the toxic effects of fluoride and to knew the mechanism of fluoride toxication, the liver cell of rat and ceils of the liver, renal, lung of human embryo were treated by fluoride. The results indicated that the concentration of fluoride 39 ug/ml produced toxic effcts to rst liver epithelium...

Non-skeletal phase damage of endemic fluorosis has been noticed at present. The soft tissue and enzyme metabolism are effected by fluoride and these effects appeared before skeletal change. In this paper, in order to observe the toxic effects of fluoride and to knew the mechanism of fluoride toxication, the liver cell of rat and ceils of the liver, renal, lung of human embryo were treated by fluoride. The results indicated that the concentration of fluoride 39 ug/ml produced toxic effcts to rst liver epithelium cells. The concentration of fluoride I56.2 ug/ml induced toxic effects to liver, renal epithelium cells of human embryo. In this conce ntration, lung fibroblastic cells of human embryo haven't appeared any morphaiogical changes. The result suggested that non-skeletal damage of liver and kidney should be pay attention.

用体外培养的大鼠肝细胞及人胎肝、肾、肺细胞观察氟对软组织的损伤。结果表明,氟浓度在39μg/ml时对大鼠肝上皮细胞,156.2μg/ml对人胎肝、肾上皮细胞均产生明显的毒性作用。而此浓度时对人胎肺纤维母细胞未见任何形态改变。氟的非骨相损害尤其是对肝、肾的慢性损伤更应予以重视。

To assess the rule of CMV infection in kidney allograft rejection,serial survey was made in 8 cases receiving kidney transplantation. 19 biopsies of allografts were obtained from 8 cases at different post-operative period with urine specimen collected simultaneously. 7 cases had pre-operative specimen served as control. CMV-DNA in renal tissue was detected by means of in situ hybridization with a digoxigenin labelled CMV-DNA probe. While urine specimen were assessed by PCR technique. The results could be summarized...

To assess the rule of CMV infection in kidney allograft rejection,serial survey was made in 8 cases receiving kidney transplantation. 19 biopsies of allografts were obtained from 8 cases at different post-operative period with urine specimen collected simultaneously. 7 cases had pre-operative specimen served as control. CMV-DNA in renal tissue was detected by means of in situ hybridization with a digoxigenin labelled CMV-DNA probe. While urine specimen were assessed by PCR technique. The results could be summarized as following (Tab. ): The relationship between CMV infection of renal tissue and rejection was also analyzed. It was found that among 9 cases showing acute rejection only 3 had CMV-DNA in their renal tissue,while 7 of the 8 cases having chronic rejection identified CMV-DNA in their renal specimen. In conclusion:①here is a lower incidence of CMV infection in normal kidney;② after transplantation,the incidence of CMV infection in renal allograft rises gradually;③there is a close relationship between CMV infection and chronic rejection.

肾移植患者CMV感染对排异反应的影响认识不一,国内目前尚无资料可考.本文采用原位杂交及多聚酶链反应技术,动态观察了8例肾移植患者移植前供肾、移植后肾活检组织及肾活检当日尿标本中CMV-DNA的检出状况,结合肾组织病理形态学分析,发现①正常肾组织即有少数CMV感染;②肾组织CMV感染率随移植时间延长而增加;③CMV感染与移植肾慢性排异有较为密切的关系,可能是造成慢性排异反应发生的因素之一。

 
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