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myc
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  myc
     The expression intensity of c myc gene was markedly higher in GC+DENA group that in DENA group.
     cmyc基因表达强度GC+DENA组明显高于DENA组。
短句来源
     Conclusion:As2O3 can induce GLC 82 cell apoptosis mainly with regulation of c myc,p53 and p16 gene expression.
     结论:As2 O3 能显著抑制GLC82 细胞生长、诱导细胞凋亡,并主要通过调节cmyc,p16 和p53 等基因的表达来实现。
短句来源
     Detection of P53 and c myc with immunohistochemical technique were carried out for 34 surgical specimens of malignant tumor of adrenal gland,25 adrenal benign tumors and in 12 adrenal tissues free of tumor.
     为探讨肾上腺肿瘤发病机理,为鉴别肿瘤良恶性及其分级分期提供依据,收集34例肾上腺恶性肿瘤应用SP法作P53及cmyc免疫组化检测,并与25例肾上腺良性瘤及12例非瘤肾上腺组织对比。
短句来源
     Methods The pathological features of EPS were observed in 17 cases, and the expressions of HCG, HPL, EMA, PRL, PLAP, Vim, actin, c erbB 2 and c myc were analyzed immunohistochemically.
     方法:对17 例经病理检查诊断为EPS的病例进行病理学形态观察,采用免疫组化方法标记滋养细胞HCG、HPL、EMA、PRL、PLAP、Vim 、actin、cerbB2 和cmyc
短句来源
     Expression of c_fos and c_myc were inhibited and expression of p53 was enhanced in the exercise group.
     运动组cfos和cmyc明显被控制,p53表达增强。
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  “myc”译为未确定词的双语例句
     Conclusion: The expression of p21 and p62 is related to the degrees of proliferation and transformation of tumor cells in MF/SS.
     结论:p21panras和p62cmyc的表达,与MF/SS细胞增殖转化相关。
短句来源
     Each group had its expressions of bcl 2 and c m yc. According to laserdensitom etry,the expression of bcl 2 w as significantly higher in the S H R group ( P< 0.01) thanin the S H R d and W K Y groups w hich shared sim ilar densities( P> 0.05).
     各组bcl2,cm yc 均有表达,依据辉度检测表达强度,则bcl2 表达的强度为 S H R明显高于 S H Rd 及 W K Y( P< 0.01);
短句来源
     Both the expressions of p53 and cmyc in Rb were markedly correlated with the expression of PCNA (all P<005).
     Rb中p53和cmyc的表达均与PCNA的表达有相关性(P<005)。
短句来源
     The determinations of p53, cmyc and PCNA are of significance for evaluating the histologic characteristics and biological behavior of Rb.
     结论Rb的发生是多基因异常的结果,p53、cmyc及PCNA的测定对于确定Rb的组织特性和肿瘤的生物学行为具有重要意义。
短句来源
     CONCLUSION: As2O3 can induce programmed death of SGC7901 cells mainly via down regulation of cmyc gene expression.
     结论:As2O3能诱导人胃腺癌SGC7901细胞程序化死亡并可能通过降低cmyc基因的表达。
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     c myc and P53 may play a role in the transcriptive activation of hTERT gene in endometrial carcinoma.
     c myc基因、P
短句来源
     Gly.
     Gly。
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     There were a positive correlation between c myc and p53 expression in liposarcoma.
     脂肪肉瘤中,cmyc 和p53 表达呈正相关。
短句来源
     These effects were associated with decreased expression of c myc encoded protein which was shown by western blot.
     结果:①反义ODN降低cmyc蛋白的表达。
短句来源
     The clinical value of antisense c myc in preventing and treating EH is limited.
     (7)反义c-myc寡核苷酸?
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  myc
Such are the suggested effects on telomerase of Myc, p53, Waf1, protein kinases B and C, Wnt5A, TGFβ, WT1, and estrogens.
      
However, Myc, p53, WT1, estrogens, protein kinases B and C, and TGFβ can also directly influence telomerase independently of the G1-S checkpoint mechanism.
      
To study some additional factors necessary for such transformation, c-myc and N-rasAsp12 were consecutively introduced into REF52 cells by retroviral infection, and the cell cultures obtained were analyzed.
      
The proliferating-cell gene cluster included MET, VIM, MYC, TOP2A, PCNA.
      
In pCI.ori-neo, chromosomal ori was from the human c-myc locus.
      
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In apple growing region of Liaoning Province,Glomerella cingulata,the causal fungus of apple bitter rot,overwintered as dormant myc-elium in small mummified fruits, dead fruiting spurs,and rough-skinedt wigs on trees.Conidia that had remained on infected fruit mummiesfailed to germinate in the next spring.Infected fallen fruits on ground-under fruit trees played little role as source of primary infection.Und-er the climatic conditions of southern Liaoning,conidia production anddispersion started from mid-June,and...

In apple growing region of Liaoning Province,Glomerella cingulata,the causal fungus of apple bitter rot,overwintered as dormant myc-elium in small mummified fruits, dead fruiting spurs,and rough-skinedt wigs on trees.Conidia that had remained on infected fruit mummiesfailed to germinate in the next spring.Infected fallen fruits on ground-under fruit trees played little role as source of primary infection.Und-er the climatic conditions of southern Liaoning,conidia production anddispersion started from mid-June,and young fruits were infected short-tlg after petal fall, The causal fungus penetrated into the fruits thro- ugh intact epidermis. The incubation period lasted about one month du-ring the early fruit stage.Disease incidence and development covered aperiod from mid-July to mid-September. Based on the results obtainedfrom field experiments, the following spray schedule was recommerdedas practicle: spraying Bordeaux Mixture (1:2:200) or Zn-Cu Lime Mixt-ure (Zn sulfate 0.5 kg,Cu sulfate 0.5 kg, Lime 2 kg,Water 200) thricefrom mid-June at an intervals of about 20 days. The first spray shouldbe performed immediately after conidia dispersion as detected with sporetrapping method.

在辽宁地区,苹果炭疽病菌主要以菌丝在苹果树上的小僵果、死果台、粗皮、爆皮枝等部位越冬。翌年在适宜温湿度条件下产生分生孢子,进行传播侵染。越冬后的分生孢子已失去萌发力,落地病果在初侵染中不起什么作用。病菌可以直接穿透表皮侵入果实。苹果在幼果期即感病,此时不抗侵入但抗扩展,潜育期长达一个月左右。七月中旬以后病害大量发生,直至9月中旬持续为害。根据果园中病菌孢子出现期,约在6月15至25日喷施第一次药,以后每隔20天左右连续喷施160~200倍波尔多液或锌铜石灰液三次,可以有效地控制炭疽病的为害。1964年和1965年,在辽宁地区进行大面积防治试验表明效果良好,可以大面积推广。

To determine the association of fragile sites with leukemias and lymphomas,we examined 22 patients with leukemia or lymphoma (6 Hodgkin lymphomas,6 non-Hodgkin lymphomas,5 acute lymphocytic leukemia,3 acute nonlymphocytic leukemia,2 lymphosarcoleukemia) and 15 normal controls ranging in age from 4 to 56 years.The results were: (1) The average chromosome structural aberration rate of patients(12.98%) was much higher than that of the control group(0.73%).Fifteen cancer patients carried fragile sites,12 of them...

To determine the association of fragile sites with leukemias and lymphomas,we examined 22 patients with leukemia or lymphoma (6 Hodgkin lymphomas,6 non-Hodgkin lymphomas,5 acute lymphocytic leukemia,3 acute nonlymphocytic leukemia,2 lymphosarcoleukemia) and 15 normal controls ranging in age from 4 to 56 years.The results were: (1) The average chromosome structural aberration rate of patients(12.98%) was much higher than that of the control group(0.73%).Fifteen cancer patients carried fragile sites,12 of them carried multiple fragile sites,but none of the controls carried any.There was a statistically significant difference between the two groups (P<0.005).(2) These patients carried 21 autosomal fragile sites (including 14 constitutive fragile sites and 7 heritable fragile sites): 1q44,2q11,2q23,2q37,3p14,4q31,5q31,6q26,8q22,8q24,9q13,10q22,10q23,10q25,11q13,12q13,13q34,14q13,14q24,16p12 and 16q22.(3) Eight breakpoints were located at the bands where oncogenes exist: 1p31 and Blym-1,2p23 and Nmyc,6q23 and myb,8q22 and mos.8q24 and myc,11q13 and bcl-1,15q26 and fes,17q21 and erbA.(4) Four breakpoints were near the fragile sites: 1p32 near fra(1p31),2p23 near fra(2p24),7p15 near fra(7p14),7q22 near fra(7q21).One breakpoint(1p12) was near the Nras(lp13).Five of the fragile sites (8q22,11q13,12q13,16p12 and 16q22) correspond with cancer breakpoints.The role of fragile sites in carcinogenesis is not yet known,but according to our results the association of fragile sites with leukemias and lymphomas is evident which deserves further investigation.

对22例白血病、淋巴瘤病人和15例正常人进行了外周血淋巴细胞染色体脆性部位检测。结果表明,病人组的染色体畸变率、脆性部位检出率显著高于正常对照。通过G显带能准确定位的94个断点中包括了21种常染色体脆性部位,8个与癌基因在同一区带的断点。以上结果提示脆性部位同白血病、淋巴瘤之间有一定的相关。

A recently devised protocol was used to obtain macrophage (M) lines with a variety of derived DNA to see if different oncogenes would lead to generation of M lines with different phenotypes. SV 40 DNA, cellular myc genes, U 937 DNA and Insulin gene have been used and 7 cell lines obtained. All these lines show heterogeneity in the cell phenotype and in each case there are Macrophage-like cells. They all secrete collagenase and lysozyme. 30-40% of these lines have Fc receptor and Fc receptor mediated phagocytosis...

A recently devised protocol was used to obtain macrophage (M) lines with a variety of derived DNA to see if different oncogenes would lead to generation of M lines with different phenotypes. SV 40 DNA, cellular myc genes, U 937 DNA and Insulin gene have been used and 7 cell lines obtained. All these lines show heterogeneity in the cell phenotype and in each case there are Macrophage-like cells. They all secrete collagenase and lysozyme. 30-40% of these lines have Fc receptor and Fc receptor mediated phagocytosis and also complement receptor and phagocytosis. Heterogeneity within the lines is probably due to the fact that the lines are not cloned yet, It is supposed that this protocol can be used not only to derive M lines but also T and B cell lines.

本文报告应用转染技术建立巨噬细胞传代株,探讨应用不同来源的DNA获得具有不同功能特性的巨噬细胞株,应用SV40DNA、C-myc基因,U937DNA和胰岛素基因已获得7株细胞,经鉴定它们均有巨噬细胞的特性,即可分泌胶元酶、溶菌酶,其中有30—40%的细胞带有Fc受体,并有经Fc受体介导的吞噬功能。细胞株内细胞功能各异是因为细胞株尚未克隆化。从本试验结果看不仅经转染方法可获得巨噬细胞株,而且有可能经此法获得B或T淋巴细胞传代株。

 
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