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微血管并发症发生
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  micrangium complications
     Conclusions:Sex hormone disorder in female NIDDM patients in the climacterium may be one of the factors to induce micrangium complications or to foster it.
     结论 :更年期女性 NIDDM患者性激素失调可能是其微血管并发症发生或促成因素之一。
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  “微血管并发症发生”译为未确定词的双语例句
     According to the clinical discovery, leptin is associated with a series of risk factors to the development and progress of microvasculor complication of DM.
     临床观察发现,瘦素与DM微血管并发症发生发展的一系列危险因素相关,且随着DR的进展,血浆瘦素水平逐渐升高。
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     Objective:The activation of Polyol pathway has been considered as one of the causes involved in the diabetic microangiopathy.
     目的:多元醇通路的激活是糖尿病微血管并发症发生和进展的重要原因,作为该通道重要的限速酶的醛糖还原酶一直是研究的热点。
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     In this process, aldose reductase(AR) is the first and rate-limiting enzyme, has always been the focus of different studies.
     高血糖情况下醛糖还原酶的激活被认为是微血管并发症发生的始动因素,已证实在高血糖情况下组织醛糖还原酶mRNA的水平是增高的,其活性也是增强的。
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     he activities of Na+-K+-ATPase and Mg2+-ATPase and lipid peroxidation of erythrocytemembrane in 40 non-insulin-dependant diabetes mellitus(NIDDM) patients were inves-tigated, The results showed that the activities of Na+-K+-ATPase and Mg2+-ATPase oferythrocyte membrane were significantly decreased in diabetics while the level of lipidperoxide(LPO)of erythrocyte membrane was significantly increased.
     本文探讨了NIDDM患者红细胞膜Na ̄+—K ̄+—ATP酶及Mg ̄(2+)—ATP酶活性改变与脂质过氧化的关系,以及这两者在糖尿病微血管并发症发生中所起的作用。 结查显示,NIDDM患者红细胞膜Na ̄+—K ̄+—ATP酶与Mg ̄(2+)—ATP酶活性显著降低。
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     Methods 152 cases of type 2 diabetes mellitus patients were divided into three groups according to vessel lesions:no-diabetic complication group(NDC), diabetic retinopathy group(DR) and diabetic nephropathy group(DN). 32 normal persons were regarded as control group. Levels of HCY were measured by enzyme linked immunosorbent assay.
     方法152例2型糖尿病患者根据微血管并发症发生情况分为无微血管并发症(NDC)组、糖尿病视网膜病变(DR)组和糖尿病肾病(DN)组,用酶联免疫吸附试验分别检测各组患者血浆HCY水平,并同步测定可溶性血栓调节蛋白(sTM)、叶酸(FA)和维生素B12(VitB12)含量,与32名正常对照者进行比较。
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     No complications were encountered.
     无并发症发生
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     There were no other complications.
     无并发症发生
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     Early onset of severe diabetes mellitus-related microvascular complications
     早期发生的严重糖尿病相关性微血管并发症
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     Unlike classical microvascular complications, large-vessel atherosclerosis can precede the development of diabetes.
     与经典的微血管并发症不同,大血管并发症先于糖尿病的发生
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     Association of blood pressure level with complication of microvessel and great vessel in patients with type 2 diabetes mellitus
     血压水平与2型糖尿病患者微血管和大血管并发症发生的关系
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In 163 cases with IDDM over ten years,the proliferative retinopathy and clinical nephropathy were observed in 23.9% and 11.7% respectively.The onset and development of severe microvascular complications were closely related to the duration of diabetes,metabolic control,and interrupted medical care.These data showed that in IDDM patients treated continuously during the first ten-year after onset of diabetes in our clinic,constant good metabolic control effectively reduced the microvascular complications.

163例病程超过10年的胰岛素依赖型糖尿病,23.9%合并增殖性视网膜病变,11.7%合并临床肾病。严重的微血管并发症的发生与发展和糖尿病病程、病情控制程度及是否中断随访治疗密切相关。起病后第一个10年,在本院糖尿病门诊连续跟踪治疗,使血糖长期稳定控制在Ⅰ级,有效地减少了微血管并发症的发生。

The activities of superoxide dismutase (SOD) and glucose-6-phosphate dehydrogenase (G-6-PD) in erythrocyte of 17 patients with NIDDM and 14 healthy controls were evaluated, and the relationship between SOD and G-6-PD activities and renal functions and urinary protein excretion was studied. The results showed: (1) the activities of SOD and G-6-PD were obviously lower in NIDDM group than in control (P<0. 01 ,P<0. 05) ; (2) a negative correlation was found between SOD activity and GHb content (r=-0. 498, ,P<0....

The activities of superoxide dismutase (SOD) and glucose-6-phosphate dehydrogenase (G-6-PD) in erythrocyte of 17 patients with NIDDM and 14 healthy controls were evaluated, and the relationship between SOD and G-6-PD activities and renal functions and urinary protein excretion was studied. The results showed: (1) the activities of SOD and G-6-PD were obviously lower in NIDDM group than in control (P<0. 01 ,P<0. 05) ; (2) a negative correlation was found between SOD activity and GHb content (r=-0. 498, ,P<0. 05) and between G-6-PD activity and total urinary protein excretion (r=-0. 488, P<0. 05) ; (3) a positive correlation was observed between SOD activity and GFR (r = 0. 550, P<0. 05). The results suggest that: (1) there is a decrease of anti-oxidate ability in NIDDM patients; (2) the impairment of anti-oxidate ability may play an important role in the development of long-term diabetic complications. The activity of SOD in diabetic erythrocyte may be a useful indication of the risk to develop diabetic nephropathy.

测定17例非胰岛素依赖型糖尿病患者和14例正常人红细胞超氧化物歧化酶(SOD)、葡萄糖-6-磷酸脱氢酶(G-6-PD)、空腹血糖(FBS)、糖化血红蛋白(GHb)、血甘油三酯(TG)及血胆固醇(Ch)的变化,并观察糖尿病组SOD、G-6-PD活性与肾小球滤过率(GFR)、尿微白蛋白排泄率(UMA)、血肌酐(Cr)、尿β_2-微球蛋白排泄率(β_2-MG)及尿总蛋白排泄率(UPR)的相关性。结果:(1)糖尿病组FBS、GHb显著高于正常对照组,SOD、G-6-PD活性显著下降,两组间TG、Ch差异无显著性。(2)SOD活性与GHb呈显著负相关,与FBS、TG、Ch等无相关性。G-6-PD与GHb、Ch、TG、FBS均无相关性。(3)糖尿病组SOD活性与GFR呈显著正相关,G-6-PD与UPR亦呈显著负相关。提示:(1)糖尿病患者红细胞抗氧化酶活性下降;(2)糖尿病患者抗氧化酶活性异常与单次空腹血糖及单次血TG、Ch水平未明显关系,可能为慢性病变所致,与非酶促糖基化有关;(3)糖尿病患者红细胞超氧化物歧化酶活性与糖尿病慢性微血管并发症的发生有关系,是糖尿病肾病的诱发因子之一。

he activities of Na+-K+-ATPase and Mg2+-ATPase and lipid peroxidation of erythrocytemembrane in 40 non-insulin-dependant diabetes mellitus(NIDDM) patients were inves-tigated, The results showed that the activities of Na+-K+-ATPase and Mg2+-ATPase oferythrocyte membrane were significantly decreased in diabetics while the level of lipidperoxide(LPO)of erythrocyte membrane was significantly increased.Sunultaneously,the activities ofsuperoxide di smutase and glutathine peroxidase of erythrocrte were significantly...

he activities of Na+-K+-ATPase and Mg2+-ATPase and lipid peroxidation of erythrocytemembrane in 40 non-insulin-dependant diabetes mellitus(NIDDM) patients were inves-tigated, The results showed that the activities of Na+-K+-ATPase and Mg2+-ATPase oferythrocyte membrane were significantly decreased in diabetics while the level of lipidperoxide(LPO)of erythrocyte membrane was significantly increased.Sunultaneously,the activities ofsuperoxide di smutase and glutathine peroxidase of erythrocrte were significantly lowered indiabetics as compared with normal controls.The above abnormal changes were more markwt indiabetics with retinopatliy than in those without retinopathy. The changes of the activity of Na+-K+-ATPase and Mg2+-ATPase were inversely correlated with errythrocyte membraneLPO and index of errythrocyte filtrati on (IF).These suggested that the signi ficant enhancement oflipid peroxidation of erythrocyte membrane may be respousible for the decrease of the activify ofNa+-K+-ATPase and Mg2+-ATPase which may be involved in the pathogenesis of diabcticretinopathy.

本文探讨了NIDDM患者红细胞膜Na ̄+—K ̄+—ATP酶及Mg ̄(2+)—ATP酶活性改变与脂质过氧化的关系,以及这两者在糖尿病微血管并发症发生中所起的作用。结查显示,NIDDM患者红细胞膜Na ̄+—K ̄+—ATP酶与Mg ̄(2+)—ATP酶活性显著降低。而红细胞膜LPO则显著增高。红细胞SOD及GSHpx活性显著降低,伴有糖尿病视网膜病变者以上变化更为显著。红细胞膜Na ̄+—K ̄+—ATP酶和Mg ̄(2+)—ATP酶活性与红细胞膜LPO、红细胞IF呈显著负相关,红细胞膜LPO与红细胞IF呈显著正相关,表明红细胞膜脂质过化增强是导致膜Na ̄+—K ̄+—ATP酶和Mg ̄(2+)—ATP酶活性降低的重要因素,并且它们都参与糖尿病视网膜病变的发生。

 
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