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   高igg血症 的翻译结果: 查询用时:0.271秒
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高igg血症
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  hyper-igg
     Conclusion:There were two deletion sites in Fcgr2b gene promoter region in NZB mice and the deletions could induce decreasing of the expression of Fcgr2b mRNA that lead to hyper-IgG.
     结论:NZB小鼠Fcgr2b基因启动子区存在碱基缺失,且该缺失突变可引起其表达的降低而导致高IgG血症
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  “高igg血症”译为未确定词的双语例句
     Conclusion:Susceptibility allele of IgG hypergammaglobulinemia in(NZB×NZW)F 1 mice was Fcgr2b gene derived NZB strain.
     结论 :(NZB×NZW)F1 小鼠高IgG血症易感基因为NZB来源的Fcgr2b基因
短句来源
     Conclusion: Susceptibility allele of IgG hypergammaglobulinemia in (NZB×NZW)F 1 mice was Fcgr2b gene derived NZB strain.
     结论 :(NZB×NZW) F1 小鼠高 Ig G血症易感基因为 NZB来源的 Fcgr2 b基因。
短句来源
     Objective:To map the susceptibility allele of IgG hypergammaglobulinemia in SLE model (NZB×NZW)F 1 mice.
     目的 :定位自发性SLE模型——— (NZB×NZW)F1 小鼠高IgG血症的遗传易感基因。
短句来源
     Gene mapping of IgG hypergammaglobulinemia in SLE model New Zealand mice
     SLE模型新西兰小鼠高IgG血症易感基因的定位
短句来源
     Gene mutation and its effects of IgG hypergammaglobulinemia susceptibility gene-Fcgr2b gene in model of systemic lupus erythematosus
     SLE模型小鼠高IgG血症易感基因-Fcgr2b基因突变及对其表达的影响
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  相似匹配句对
     V. C.
     V.C;
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     The antibody of IgG against Sj always kept high level before and after treatment.
     IgG抗体一直维持较水平。
短句来源
     The content of IgG,IgA and IgM were higher than that of the controls;
     IgG、IgA、IgM较正常对照组 ;
短句来源
     pylori-IgG and anti-H.
     pylori-IgG、H.
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     the charges is high;
     费用
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  hyper-igg
The hyper-IgG1(A1) syndrome entails a polyclonal selective increase of the serum levels of immunoglobulin (Ig) G1 and to a lesser extent of IgA1; this is not mediated by malignancy, infectious or autoimmune diseases or environmental agents.
      
A 32: 1 chance for the linkage of this rare IgH haplotype with the hyper-IgG1(A1) syndrome in the family argues for a dominant regulator located at the human IgH locus having a selective influence on the production of IgG1 and IgA1.
      


In this study, we retrospectively

本文回顾性地分析了44例经肾活检诊断为Ⅴ型狼疮肾炎患者的临床及病理资料,并与50例Ⅳ狼疮性肾炎患者进行比较。Ⅴ型狼疮肾炎患者中,男7例,女38例,干均年龄29.6±9.2岁,肾活检光镜下表现为纯膜性改变者18例,非纯膜性改变者26例。纯膜型与非纯膜狼疮性肾炎临床表现的比较表明、纯膜型组起病年龄大于非纯膜型组,肾外表现相对较少(关节受累率38%∶73%,血液系受累33%∶50%),纯膜型组血清肌酐升高患者的比例(0%)明显低于非纯膜型组(11%),而非纯胰回组与Ⅳ型狼疮组的临床表现无显著差异:在肾病综合征比例、蛋白尿程度、镜下血尿比例上各组之间差异不显著,病理上除肾小球的基本改变外,纯膜型组小管间质改变较轻,较少伴有间质大量淋巴细胞浸润,而非纯膜型多伴系增殖及系膜插入,同时伴明显间质淋巴细胞浸润,血管周围炎细胞浸润,小管上皮细胞脱落、坏死、萎缩及慢性硬化性改变。免疫学检查表明纯膜型组患者高IgG血症程度、补体C3,C4下降幅度以及血清Anti-DsDNA阳性滴度均不及非纯膜型组。通过以上结果的比较,我们认为Ⅴ型狼疮肾炎是一组不均一的疾病,至少可分成纯膜型与非纯膜型两大亚型,治疗上应区别对待。

16 patients

16例经肾活检诊断的急性间质性肾炎除1例外均在临床用药之后出现症状。13例有不同程度的急性肾功能损害,多数病例有非选择性蛋白尿、低渗尿、溶菌酶尿和高IgG血症,仅5例同时有发热、皮疹、关节疼痛和嗜酸粒细胞血症;1例肾小球病变为局灶性节段性肾小球硬化(FSGS),其余为轻微病变和系膜增生;免疫酶标示肾小球系膜区和小管基底膜不同的免疫球蛋白沉积;13例肾功能不全患者肾间质CD_4+、CD_8+和HLA-DR+细胞显著增加,肾小管HLA-DR抗原表达与CD_4+和DR+细胞呈显著正相关。肾功能恢复与入院时血肌酐,肾小球病变,间质淋巴细胞分布和肾小管HLA-DR抗原表达有关。

Objective:To map the susceptibility allele of IgG hypergammaglobulinemia in SLE model (NZB×NZW)F 1 mice.Methods:The authors set up the (NZB×NZW)F 1×NZW backcross mice model and used polymorphic microsatellite markers and quantitative trait locus(QTL) analysis.Results:Susceptibility allele of IgG hypergammaglobulinemia was localized the telomeric region on chromosome 1 which is linked to Fcgr2b gene according to the QTL analysis.The level of serum IgG in the group of Fcgr2b gene B/W type was higher than that...

Objective:To map the susceptibility allele of IgG hypergammaglobulinemia in SLE model (NZB×NZW)F 1 mice.Methods:The authors set up the (NZB×NZW)F 1×NZW backcross mice model and used polymorphic microsatellite markers and quantitative trait locus(QTL) analysis.Results:Susceptibility allele of IgG hypergammaglobulinemia was localized the telomeric region on chromosome 1 which is linked to Fcgr2b gene according to the QTL analysis.The level of serum IgG in the group of Fcgr2b gene B/W type was higher than that of W/W type.Conclusion:Susceptibility allele of IgG hypergammaglobulinemia in(NZB×NZW)F 1 mice was Fcgr2b gene derived NZB strain.

目的 :定位自发性SLE模型——— (NZB×NZW)F1 小鼠高IgG血症的遗传易感基因。方法 :建立 (NZB×NZW )F1 ×NZW回交小鼠模型 ,采用覆盖小鼠 19条染色体的多态性微卫星遗传标记及数量性状位点 (QTL)分析进行基因定位。结果 :高IgG血症易感基因与小鼠第 1条染色体末端 92 .3cM处微卫星遗传标记D1Mit36肯定连锁 (Lods值 >3) ,该位点附近 92 .0cM处存在Fcgr2b基因 ,且回交小鼠Fcgr2b基因B W型组血清总IgG水平明显高于W W型组 (P <0 .0 0 0 1)。结论 :(NZB×NZW)F1 小鼠高IgG血症易感基因为NZB来源的Fcgr2b基因

 
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