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血小板胞浆游离钙浓度
相关语句
  platelet cytosolic free calcium concentration
     Observation of Platelet Cytosolic Free Calcium concentration and Thromboxane B_2 content in Uremic Patients
     慢性肾功能衰竭患者血小板胞浆游离钙浓度及血栓素B_2含量的观察
短句来源
     (3) The platelet cytosolic free calcium concentration and the degree of platelet aggregation were positively related to the left ventricular hypertrophic index(all P values less than 0.01).
     (3)SHR血小板胞浆游离钙浓度和血小板聚集率与左心室肥厚指数呈显著正相关 (P均 <0 .0 1 )。
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  “血小板胞浆游离钙浓度”译为未确定词的双语例句
     Objective To study the effect of Danshen injection on cytosolic free calcium concentration[Ca~(2+)]_i in normal rat platelets Methods The intraplatelet[Ca~(2+)]_i was measured with Fura-2/AM flurospectrophotometry.
     目的:研究丹参注射液对正常大鼠血小板胞浆游离钙浓度[Ca2 + ]i 的影响。 方法:以Fura - 2 /AM荧光分光光度法测定大鼠血小板内[Ca2 + ]i的变化。
短句来源
     EFFECTS OF 1-(2,6-DIMETHYLPHENOXY)-2-(3,4-DIMETHOXY PHENYLETHYLAMINO)PROPANE HYDROCHLORIDE ON CYTOSOLIC FREE CALCIUM CONCENTRATION OF RABBIT PLATELETS
     1-(2,6-二甲基苯氧基)-2-(3,4-二甲氧基苯乙氨基)丙烷盐酸盐对兔血小板胞浆游离钙浓度的影响
短句来源
     DETERMINATION OF INTRAPLATELETIC FREE CALCIUM CONCENTRATION WITH FURA 2-AM FLUORESCENCE TECHNIQUE
     用Fura 2-AM检测血小板胞浆游离钙浓度
短句来源
     And the normal WKYs were used in contrast of age pair-matching SHRs. BP-HR meter, weighing, transmission electron microscope, Fara-2/AM loading, fluorospectrophotometry and Born turbidimetry were all used to detect systolic blood pressure(SBP), heart rate(HR)、hypertrophy index, ultrastructure of myocardial cells, concentration of platelet cytoplasm free calcium(PC[Ca2+]) and platelet aggregation(PAg).
     分别采用血压心率仪、直接称左、右心室湿重,透射电子显微镜技术、Fura-2/AM负载、荧光分光光度法及Born比浊法检测SHR的收缩压(systolic blood pressure,SBP)、心率(heart rate,HR)、心肌肥大指数、心肌细胞超微结构、血小板胞浆游离钙浓度(Platelet cytoplasm free calcium concentration ,PC[Ca2+]i)和血小板聚集率(PAg)。
短句来源
     Measurement of cytoplasmic free Ca~(2+)in human platelet using Fura-2/Am
     应用Fura-2/Am检测血小板胞浆游离钙浓度
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     Determination of the level of cytoplasmic free calcium in human platelets with flow cytometry
     流式细胞仪测定血小板胞浆游离浓度
短句来源
     Thrombin-induced Cytosolic Free Calcium TransientsMonitored by Quin 2 Method in Human Platelets
     Quin2法测定人血小板胞浆游离浓度
短句来源
     Measurement of cytoplasmic free Ca~(2+)in human platelet using Fura-2/Am
     应用Fura-2/Am检测血小板胞浆游离浓度
短句来源
     DETERMINATION OF INTRAPLATELETIC FREE CALCIUM CONCENTRATION WITH FURA 2-AM FLUORESCENCE TECHNIQUE
     用Fura 2-AM检测血小板胞浆游离浓度
短句来源
     Efectsofneferineonplateletaggregationandconcentrationofcytoplasmicfrecalcium
     甲基莲心碱对血小板聚集和胞浆游离离子浓度的影响
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  platelet cytosolic free calcium concentration
Platelet cytosolic free calcium concentration in hypertension associated with early stage kidney disease
      


In this experiment, Disseminated Intravascular Coagulation(DIC) model was Prepared on New Zealand rabbits to observe the change of cytosolic calcium concentration [Ca2 +]i of thrombocytes measured with Quin-2 , in order to study the significance of [Ca2 +]i of thrombocytes in the occurance of endo-toxin-induced DIC. The effect of endotoxin on [Ca2+]i of platelets in vitro was also studied. The results indicated- the resting cytosolic calcium concentration of platelets of New Zealand rabbits is 112±24 nM (n=...

In this experiment, Disseminated Intravascular Coagulation(DIC) model was Prepared on New Zealand rabbits to observe the change of cytosolic calcium concentration [Ca2 +]i of thrombocytes measured with Quin-2 , in order to study the significance of [Ca2 +]i of thrombocytes in the occurance of endo-toxin-induced DIC. The effect of endotoxin on [Ca2+]i of platelets in vitro was also studied. The results indicated- the resting cytosolic calcium concentration of platelets of New Zealand rabbits is 112±24 nM (n= 7 ) . In vitro endotoxin can directly increase [Ca2+]i of platelet and also this effect is dose-dependent. In vivo, the experimental DIC models were prepared on New Zealand rabbits and the [Ca2+]i of platelets increase from 112 ±24 nM in normal to 343 ±48 nM, The results suggest that endotoxin can increase of [Ca2+]i of platelet in vitro and in vivo. In DIC model the platelets maintain a high level of [Ca2 +]i and result in the "hypercoagulant" of platelet, which may be important cellular mechanism of DIC.

本实验观察内毒素在体外对兔血小板胞浆游离钙浓度[Ca~(2+)]i的作用以及用二次注射内毒素法复制DIC模型时对[Ca~(2+)]i的影响。结果表明静息血小板[Ca~(2+)]i为112±24 nM,内毒素可直接作用于血小板,使[Ca~(2+)]i呈剂量依赖性升高。内毒素致DIG时(Ca~(2+)]i可升高三倍。结果提示内毒素可能是通过升高血小板[Ca~(2+)]i而激活血小板。血小板的激活可能是导致DIC的重要发病机制之一。

The effect of meglumine cyclicadenyate(MCA)on the thrombin induced cytosolicfree Ca2+transients in human platelets, comparedwith exogenous cAMP, was observed by Quin 2 meth-od. The thrombin-induced [Ca2+]i rise can be inhibitedby MCA in a dose-dependent manner(5-50 umol·L-1)but strengthened by exogenous cAMP Theresults suggest that MCA can prevent the activation ofplatelet by way of modulation of the second messengersystem, such as Ca2+ and cAMP level,therefore it is abeneficial action of MCA to the cardiovascular...

The effect of meglumine cyclicadenyate(MCA)on the thrombin induced cytosolicfree Ca2+transients in human platelets, comparedwith exogenous cAMP, was observed by Quin 2 meth-od. The thrombin-induced [Ca2+]i rise can be inhibitedby MCA in a dose-dependent manner(5-50 umol·L-1)but strengthened by exogenous cAMP Theresults suggest that MCA can prevent the activation ofplatelet by way of modulation of the second messengersystem, such as Ca2+ and cAMP level,therefore it is abeneficial action of MCA to the cardiovascular svstem

葡甲胺环腺苷酸对凝血酶诱导的人血小板胞浆游离钙浓度变化的影响陆志强,王道生,赵升皓(苏州医学院药理教研室,苏州215007)葡甲胺环腺苷酸(megluminecyclicadenylate,MCA)是环腺苷酸(cAMP)与葡甲胺结合而成的cAMP类衍...

In this study, 38 patients with acute ischemic stroke were chosen,randomly allocated to three groups and treated by niraodipine, aspirin and nimodipine plus asprin respectively, platelet baseline cytosolic free calcium concentrations and plasma TXB2 levels were determined in all patients before and after the start of treatment. The result showed nimodipine can inhibit Ca++ influx, supress membrane phosopholipid degradation decrease the release of TXA2. The biochemistry basis of anti-calcium and anti-TXA2 treatment...

In this study, 38 patients with acute ischemic stroke were chosen,randomly allocated to three groups and treated by niraodipine, aspirin and nimodipine plus asprin respectively, platelet baseline cytosolic free calcium concentrations and plasma TXB2 levels were determined in all patients before and after the start of treatment. The result showed nimodipine can inhibit Ca++ influx, supress membrane phosopholipid degradation decrease the release of TXA2. The biochemistry basis of anti-calcium and anti-TXA2 treatment were discussed briefly with relative literature. Author emphasized that drugs must be used conjunctively to guard the neurons in many aspects in the treatment of cerebral ischemia.

本研究选择急性缺血性脑卒中患者38例,随机分成三组,分别给予尼莫地平、阿斯匹林以及两药伍用治疗,于治疗前、后观察血小板胞浆游离钙浓度和血浆TXB_2含量的变化,结果提示:尼莫地平能阻滞Ca~(++)内流,抑制膜磷脂降解,减少TXA_2的合成,与阿斯匹林伍用效果更佳。作者结合文献探讨了抗钙和抗TXA_2治疗的生化学基础及临床意义,提出治疗脑缺血需联合用药,在多个环节,多种途径上对神经元进行保护。

 
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