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谷胱甘肽过氧化物酶gsh
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  gsh
     The activities of blood GSH Px in quenching,welding and sealing groups were (71.42±26.52)NU/ml,(70.77±20.12)NU/ml and (71.67 ±18.87)NU/ml respectively,significantly lower than that in the control[(97.02±21.76)NU/ml, P <0.05 or P <0.01].
     焊接组、热合组和淬火组的谷胱甘肽过氧化物酶 (GSH Px)活力分别为 (71.42± 2 6 .5 2 )、(70 .77± 2 0 .12 )、(71.6 7± 18.87)NU/ml,明显低于对照组 ,差异有显著性 (P <0 .0 5 ) ;
短句来源
     Results In normothermia group, the amount of SOD,GSH-Px, GSH and ATP were lower and MDA, lactate, water content and Ca 2+ was higher than those in the sham-operated group(P<0.05).
     结果 常温脑缺血组织中超氧化物歧化酶(SOD) ,谷胱甘肽过氧化物酶 (GSH -Px) ,还原型谷胱甘肽 (GSH)及三磷酸腺苷 (ATP)的含量明显低于假手术组 (P <0 .0 1) ,丙二醛 (MDA )、乳酸、含水量以及Ca2 + 的含量明显高于假手术组 (P <0 .0 1)。
短句来源
     Method: 23month old SD rats are observed as naturally senile rate, of brainmitochondria membrane are conjugateddienes (CD), molondialdehyde (MDA), glutathione peroxidase (GSH Px), superoxide dismutase (SOD), Na + K + ATP ase and Ca 2+ Mg 2+ ATP ase measured as indexes to observe the effect of the PNPY.
     方法 :以 2 3月龄SD大鼠为自然衰老鼠 ,以脑线粒体膜共轭双烯 (CD)、丙二醛 (MDA)、谷胱甘肽过氧化物酶 (GSH Px)、超氧化物歧化酶 (SOD)、Na+ K+ ATP酶、Ca2 + Mg2 + ATP酶为主要指标 ,观察滋补脾阴方药的作用。
短句来源
     Secondly, at 30, 90 and 180 days the activities of glutathione peroxidase (GSH Px) in arsenic group were significantly lower than that in control group (P<0.05, P<0.01).
     在染砷第 30 d、第 90 d和第 180 d大鼠肝脏谷胱甘肽过氧化物酶 (GSH- Px)活性均明显低于对照组 (P<0 .0 5 ,P<0 .0 1) ;
短句来源
     The effects of different concentrations of selenium on the growth, the activities of superoxide dismutase(SOD), catalase(CAT), ascorbate peroxidase(APX), dehydroascorbate reductase(DHAR), glutathione peroxidase(GSH PX)and the content of ascorbate(AsA), hydrogen peroxide(H 2O 2)were studied in the hydroponic spinach.
     研究了不同浓度硒对菠菜的生长、超氧化物歧化酶 (SOD)、抗坏血酸过氧化物酶 (APX)、过氧化氢酶 (CAT)、脱氢抗坏血酸还原酶 (DHAR)、谷胱甘肽过氧化物酶 (GSH PX)等酶活性以及抗坏血酸(AsA)、过氧化氢 (H2 O2 )含量的影响。
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  “谷胱甘肽过氧化物酶(gsh”译为未确定词的双语例句
     The average activity of GSH-Px in different group had no significant difference(P≥ 0.05),which was 250~350 U/mL.
     不同NO2--N浓度对鲤鱼种血液中谷胱甘肽过氧化物酶(GSH-Px)活力的影响没有显著差异(P≥0.05),其平均活性在250~350 U/mL之间。
短句来源
     Method The mice were divided into three experimental groups and given(1 382.2) mg/kg,(2 764.4) mg/kg and(4 146.6) mg/kg of 1,2,4-TCB respectively by subcutaneous injection,then measure the activities of SOD,GSH-Px and the levels of T-SH,NP-SH,P-SH and LPO in heart,liver and kidney.
     方法经皮下注射染毒,分为1 382.2、2 764.4、4 146.6 mg/kg低、中、高3个剂量,测定心、肝、肾脏中超氧化物歧化酶(SOD)、谷胱甘肽过氧化物酶(GSH-Px)、总巯基(T-SH)、非蛋白巯基(NP-SH)、蛋白巯基(P-SH)及脂质过氧化物(LPO)的含量。
短句来源
     Methods HELF were treated with Na_2AsO_2 of 0.0,0.1,0.5,1.0,5.0 and 10.0 μmol/L for 4 hours or 24 hours,respectively. The cell proliferation,the reactive oxygen species(ROS) level,the malondialdehyde(MDA) content and the activity of glutathione peroxide(GSH-Px) and the superoxide dismutase(SOD) in HELF were detected respectively.
     方法0.0、0.1、0.5、1.0、5.0和10.0μmol/L Na2AsO2处理HELF细胞4h或24 h后,检测Na2AsO2对HELF细胞增殖率、细胞中活性氧(ROS)水平、丙二醛(MDA)含量、超氧化物歧化酶(SOD)和还原性谷胱甘肽过氧化物酶(GSH-Px)活力的影响。
短句来源
     The blood sample was collected to determine the activity of superoxide dismutase(SOD),glutathione peroxidase(GSH-Px),contents of malonaldehyde(MDA),erythrocyte C3b receptor rosette rate(E-C3bRR)and immune complex rosette rate(E-ICRR)on both 0 and 20 d.
     在试验初(0d)和试验末(20d),分别测定血清超氧化物歧化酶(SOD)、谷胱甘肽过氧化物酶(GSH-Px)活性、丙二醛(MDA)含量、红细胞C3b受体花环率(E-C3bRR)和免疫复合物花环率(E-ICRR)。
短句来源
     10mg/kg lycopene could elevate the activity of serum superoxide dismutase(SOD) to 133.8U/ml,glutathione peroxidase(GSH-Px) to 158.9U/ml,decrease content of malondiadehyde(MDA) to 4.1nmol/mg.
     10mg/kg番茄红素组血清超氧化物歧化酶(SOD)活力为133.8U/m l,谷胱甘肽过氧化物酶(GSH-Px)活力为158.9 U/m l,丙二醛(MDA)含量为4.1nmol/mg,与对照组差异有显著性。
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  相似匹配句对
     STUDY ON GLUTATHIONE PEROXIDASE
     谷胱甘肽过氧化物酶的研究
短句来源
     DETERMINATION OF GLUTATHIONE PEROXIDASE IN RICE SEEDLINGS
     水稻谷胱甘肽过氧化物酶的测定法
短句来源
     GSHPx activity of EOBMB was determined.
     测定谷胱甘肽过氧化物酶GSH-Px)拟似活性。
短句来源
     Study on Glutathione Peroxidese Activity in Yeast
     酵母中谷胱甘肽过氧化物酶(GSH-Px)活性的分析
短句来源
     Biosynthesis of Glutathione
     谷胱甘肽的生物合成
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  gsh
Results revealed some definite correlation between the enzyme inhibition with GSH depletion in S.
      
The levels of malondialdehyde (MAD), total superoxide dismutase (SOD), glutathione (GSH), adenosine triphosphate (ATP) and serum amylase (SAM) were measured.
      
The levels of ATP, GSH and SOD in group A were decreased markedly and MDN increased significantly as compared with those in group B (P>amp;lt;0.01).
      
A decrease in the content of carotenoids in tissue s suppressed the activities of the key enzymes of antiradical defense-superoxide dismutase (SOD) and catalase-and is accompanied by a decrease in the reduced glutathione (GSH) pool.
      
The ferricyanide reduction rate did not correlate with GSH content in the contralateral lenses of the same mouse in the absence of MB or saponin.
      
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The results of many studies demonstrated that the chick has a specificrequirement for selenium for growth and maintenance of pancreas exocrine func-tion.The pancreas is the target organ of selenium deficiency in chicks,whileskeletal muscle,heart muscle,liver and other tissues are unaffected if vitaminE is adequate.Selenium deficiency in chicks fed adequate vitamin E results inpancreatic degeneration and fibrosis.Compared with selenium-supplementedchicks,selenium deficiency animals had lower plasma tocopherols,higher...

The results of many studies demonstrated that the chick has a specificrequirement for selenium for growth and maintenance of pancreas exocrine func-tion.The pancreas is the target organ of selenium deficiency in chicks,whileskeletal muscle,heart muscle,liver and other tissues are unaffected if vitaminE is adequate.Selenium deficiency in chicks fed adequate vitamin E results inpancreatic degeneration and fibrosis.Compared with selenium-supplementedchicks,selenium deficiency animals had lower plasma tocopherols,higher fecalneutral lipids,higher bile weigts,higher activities of plasma glutamic-oxa- loacetic transaminase,and lower activities of pancreatic lipase and trypsin.Itappeared therefore,that there was a decreased hydrolosis of fat due to pancreaticdegeneration and lack of lipase,which in turn led to the impairment of lipid-bilemicelle formation necessary for absorption of lipids and vitamin E.The additionof bile acid to the diet led to only partial correction of the impaired fat di-gestion and vitamin E absorption.Supplementation with free fatty acids andmonoolein increased vitamin E absorption and survival of chicks,but did notprevent pancreatic fibrosis.The biochemical studies showed a decrease in zymogen activity and increasein the activity of lysosomal enzymes cathepsin and acid phosphatase.Thatselenium was an integral part of the enzyme glutathione peroxidase (GSH-PX),it was demonstrated that this enzyme was related to the prevention ofexudative diathesis (ED) in the chicks.The levels of selenium in the diet wasshown to be highly correlated with the plasma GSH-PX activity,but Contorreported that GSH-PX was shown to be not correlated with pancreatic atrophy.Histochemical studies revealed that the invasion of macrophages accounted forthe observed increase in lysosomal enzymes.No evidence of lysosomal disrup-tion was obtained during the course of the pancreatic degeneration indi-cating that role of selenium in prevevention of pancreatic degeneration wasnot in the protection of lysosomal membranes of the acinar cells.The sequence of histological lesions began with vacuolization and hylinebody formation in the acinar.Subsequently,fibroblasts could be observed in in-creasing numbers in the interacinar spaces,while the acinar cytoplasm wasshrinking basally,thereby enlarging the central lumen of the acinus.Finally,theacini appeared,as rings of cells composed mainly of small,densestaning nucleiand were completely surrounded by fibrotic tissue.The islets are very conspi-cuous,but incretory function was not disordered.The β-islet cells are unaffec-ted by selenium deficiency.Athough hyaline body formation could be observed,the breathing function of the mitochondria was not impaired.Histological examination of the pancreases indicated that wheat and sele-nomethionine were the most effective-sources of selenium.Selenomethionine wasfour times as effective as either selenium or selenocystine with respect to pre-vention of pancreatic degeneration.Severe pancreatic atrophy is followed ED.The biological availability of selenium for prevention of ED in chicks has beenshown to be greater for feedstuffs of plant than for those of animal origin.Selenocystine was shown to be more available than selenomethionine,but not aseffective as sodium selenite.

大量的研究结果证实,硒对维持雏鸡生长和胰腺外分泌机能具有专一的作用。胰腺是雏鸡硒缺乏的靶器官,而骨骼肌、心肌、肝脏和其它组织在维生素E 充足时,不受缺硒的影响。硒缺乏的雏鸡,即使喂给足够的维生素 E,胰腺也发生变性和纤维化。与补硒鸡相比,硒缺乏鸡血清中生育酚较低,粪中的中性脂类和胆汁较多,血清谷草转氨酶活性升高,胰脂酶和胰蛋白酶活性较低。此结果归因于胰腺的变性和脂酶的缺乏,从而降低了脂肪的水解,破坏了脂类和维生素 E 吸收所必需的脂类——胆汁微胶粒的形成。在日粮中加入胆汁酸,仅仅部分地改善脂肪的消化和维生素 E 的吸收。补饲游离的脂肪酸和单油酸甘油脂,增加了鸡的维生素E 吸收和存活时间,但不能防止胰腺的纤维化。生物化学研究还表明,酶原活性降低,溶酶体酶中的组织蛋白酶和酸性磷酸酶活性升高。硒是谷胱甘肽过氧化物酶(GSH—Px)的重要组分,此酶与鸡的渗出性素质相关;而且日粮中的硒含量与血清中 GSH—Px 活性高度相关,但是,Contor 报道,GSH—Px 与胰腺萎缩没有相关性。组织化学的研究揭示了由于巨噬细胞的侵润,而导致溶酶体的酶增多。在变性的胰腺中未获得溶酶体破坏的证据,由此证明,硒防止胰腺纤维化的作用,...

大量的研究结果证实,硒对维持雏鸡生长和胰腺外分泌机能具有专一的作用。胰腺是雏鸡硒缺乏的靶器官,而骨骼肌、心肌、肝脏和其它组织在维生素E 充足时,不受缺硒的影响。硒缺乏的雏鸡,即使喂给足够的维生素 E,胰腺也发生变性和纤维化。与补硒鸡相比,硒缺乏鸡血清中生育酚较低,粪中的中性脂类和胆汁较多,血清谷草转氨酶活性升高,胰脂酶和胰蛋白酶活性较低。此结果归因于胰腺的变性和脂酶的缺乏,从而降低了脂肪的水解,破坏了脂类和维生素 E 吸收所必需的脂类——胆汁微胶粒的形成。在日粮中加入胆汁酸,仅仅部分地改善脂肪的消化和维生素 E 的吸收。补饲游离的脂肪酸和单油酸甘油脂,增加了鸡的维生素E 吸收和存活时间,但不能防止胰腺的纤维化。生物化学研究还表明,酶原活性降低,溶酶体酶中的组织蛋白酶和酸性磷酸酶活性升高。硒是谷胱甘肽过氧化物酶(GSH—Px)的重要组分,此酶与鸡的渗出性素质相关;而且日粮中的硒含量与血清中 GSH—Px 活性高度相关,但是,Contor 报道,GSH—Px 与胰腺萎缩没有相关性。组织化学的研究揭示了由于巨噬细胞的侵润,而导致溶酶体的酶增多。在变性的胰腺中未获得溶酶体破坏的证据,由此证明,硒防止胰腺纤维化的作用,不在于保护胰腺细胞中溶酶体的膜。胰腺变性的病理组织学损伤,开始是在胰腺细胞内形成空泡和透明体,胰腺泡成带现象消失。其次,在胰腺泡之间出现成纤维细胞,全部细胞质基本上都皱缩了,因而扩大了胰腺泡的中央腔。最后,胰腺泡呈现由小的浓染的核所组成的细胞轮,并完全被纤维组织包围。内分泌胰岛显得非常明显,但内分泌机能不紊乱,β-胰岛细胞也不受缺硒的影响。尽管组织学观察到透明体,但线粒体的呼吸机能未受影响。胰腺的组织学检查证明,大麦和硒蛋氨酸是最有效的硒源,就予防胰腺变性的效力而言,硒蛋氮酸是亚硒酸钠和硒代胱氨酸的四倍。严重的胰腺变性常常继发渗出性素质,在防止鸡的渗出性素质中,植物源性硒的生物学活性比动物源性高,硒代胱氨酸比蛋氨酸活性大,但不如亚硒酸钠有效。

Forty-four weanling rats were used and divided into 3 groups: 1. Low selenium diet group, 16 rats, were fed on a mixed diet, containing 0.017ppm Se, of cereals from the endemic area of Keshan disease and redistilled water. 2. Low selenium+Se group, 14 rats, fed on the same diet as group 1, but supplemented with selenium up to 0.30 ppm by adding Na2SeO3 in the water. 3. Control, 14 rats, fed on a routine diet containing 0.06ppm Ge. The results of the blood GSH-px activity of rats measured during 3 months were...

Forty-four weanling rats were used and divided into 3 groups: 1. Low selenium diet group, 16 rats, were fed on a mixed diet, containing 0.017ppm Se, of cereals from the endemic area of Keshan disease and redistilled water. 2. Low selenium+Se group, 14 rats, fed on the same diet as group 1, but supplemented with selenium up to 0.30 ppm by adding Na2SeO3 in the water. 3. Control, 14 rats, fed on a routine diet containing 0.06ppm Ge. The results of the blood GSH-px activity of rats measured during 3 months were 25.47±2.12-29.40±2.66U in group 1, 42.64± 2.85-59.56±2.24U in group 2 (p<0.001), and 38.05±1.50-52.47±2.19U for controls. The p value between group 1 and control was<0.025. Morphologically, in group 1 there was severe liver necrosis and hemorrhage (10/16), minute focal myocardial necrosis (3/16) and necrosis of limb muscle (5/16). In group 2, 2 rats had liver necrosis and limb muscle necrosis but no significant lesion was found in controls. In group 1, EM revealed cristolysis of myocardial and liver mitochondria, vesiculation of liver EEE and many residual bodies in limb muscle cells.

取大鼠分为:(1)低硒组,喂以含0.017 ppm Se的克山病病区混合饲料及重蒸馏水;(2)低硒加硒组,喂以低硒组饲料,从饮水中加Na_2SeO_3使之达到摄入量0.30 ppm Se;(3)对照组,饲以常备饲料,含0.06 ppm Se。在30、60、90d时测得各组大鼠血谷胱甘肽过氧化物酶(GSH-px)的活力,低硒组比低硒加硒组、对照组显著降低(p<0.001、p<0.025)。形态学所见:低硒组大鼠发生严重肝坏死肝出血(10/16),心肌灶性坏死(3/16)及骨胳肌坏死(5/16)。低硒加硒组大鼠,肝坏死及骨胳肌坏死各2只。对照组大鼠各脏器未发现明显改变。电镜显示低硒组:大鼠肝细胞、心肌细胞线粒体发生明显的嵴溶解、肝细胞粗面内浆网空泡化及骨胳肌细胞含多量残质体。

Activities of GSH peroxidase, GSH reductase and GSH S transferase in catar actous lenses of rats induced by sodium selenite have been determined and compared with normal lenses. It has been found that the activities of GSH peroxidase in nuclear opaque lenses induced by sodium selenite are twice as high as that in the normal lenses, but decreases in the advanced cataractous lenses. The activity of GSH reductase has similar changes in the cataract formation. The data indicate that GSH reductase and GSH peroxidase...

Activities of GSH peroxidase, GSH reductase and GSH S transferase in catar actous lenses of rats induced by sodium selenite have been determined and compared with normal lenses. It has been found that the activities of GSH peroxidase in nuclear opaque lenses induced by sodium selenite are twice as high as that in the normal lenses, but decreases in the advanced cataractous lenses. The activity of GSH reductase has similar changes in the cataract formation. The data indicate that GSH reductase and GSH peroxidase are metabolically related. The relationship between the activity of these enzymes and cataract formation has been discussed. Our resalts show that GSH S-transferase possibly takes no part in the formation of cataract.

测定了用亚硒酸钠诱发的大鼠白内障晶状体中谷胱甘肽过氧化物酶(GSH-Px)、谷胱甘肽还原酶(GSSG-R)和谷胱甘肽硫转移酶(GSH-S)的活性,并与正常晶休中这三种酶的活性作了比较。结果表明,核浊浑期晶状体中GSH-Px的活性比正常晶状体的高一倍,但在整个晶状体浑浊时降低,GSSG-R的活性变化与GSH-PX相似,这两种酶在代谢上是相关的。GSH-S的活性在核浑浊期不改变,但在完全浑浊后降低。

 
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